SPINAL ANAESTHESIA
Dr. Apexa Chhatrola
Guided by:
DEFINATION OF SPINAL ANAESTHESIA
A form of neuraxial regional anesthesia
producing reversible sympathetic block
involving the injection of a local anesthetic into
the subarachnoid space.
Commonly at L3-L4
Largest Interspace
INDICATIONS OF SA
All procedures carried out on the lower half of the
body.
Indications include surgery on the lower limb, pelvis,
genitals, perineum, lower abdominal surgeries and
most urological procedures.
CONTRAINDICATIONS OF SPINAL
ANAESTHESIA
ABSOLUTE -
Patient refusal
Infection at the site of injection
Coagulopathy and other bleeding disorders
Severe hypovolemia
Increased intracranial pressure
Severe MS & AS
Contd…
Relative –
Sepsis
Uncooperative patient
Preexisting neurological deficits
Severe spinal deformity
Prolonged operation
Major blood loss
THE ADVANTAGES OF SPINAL
ANAESTHESIA
Prolonged block: Pain free postoperative period
Alternative to GA in certain patients esp.
Difficult airway
Respiratory disease
Blunt the stress response to surgery
Cost
Patent airway
Contd…
VERTEBRAE ANATOMY
CURVES OF SPINE:
The cervical curve: convexity anterior
The dorsal curve: convexity posterior
Lumbar curve: convexity anterior
Sacrococcygeal: convexity posterior
When pt lies in supine position,
•The highest point : Third lumbar vertebra
•The lowest point : Fifth thoracic vertebra.
•Solution heavier than spinal fluid deposited at L3 will flow away
both cranial and caudal directions from the third lumbar vertebra
and tend to pool in region of the fifth thoracic vertebra.
SPINAL CORD
Extends from foramen magnum to
Adult : lower border of L1 / upper border
of L2
Infants/children : Lower border of L2.
It is about 42-45 cm long.
Duramater, Subarachnoid space & subdural
space: S2 in adults( S3 in children)
S. C gives 31 pairs of spinal
nerves.(8+12+5+5+1)
An extension of piamater, the FILUM
TERMINALE penetrates the dura and attaches
with the terminal end of spinal cord [conus
SPINAL ANAESTHESIA/ANALGESIA
SURFACE LANDMARKS
SURFACE ANATOMY
Anatomic Landmarks to Identify
Vertebral Levels
Anatomic Features
Landmark
C7 Vertebral prominence, the
most prominent process in the
neck
T7 Inferior angle of the scapula
L4 Line connecting highest
points of iliac crests(Tuffier’s
line)
S2 Line connecting the posterior
superior iliac spines
Sacral Groove or depression just
hiatus above or between the gluteal
clefts above the coccyx
SITE
Adult : L3-L4 or L4-L5 ( or even L2-
L3)
Infant : L4-L5
A line drawn b/w the highest pt. of
iliac crests (Tuffier’s line) usually crosses
either body of L4 or the L4-L5
interspace
POSITION
• Sitting
• lateral
• Prone(anorectal procedure, hypobaric
solution, jackknife position)
POSITIONING OF PATIENT
Sitting
With Legs hanging over side of bed
Put Feet up on a Stool (no wheels)
Assistant MUST keep the patient from Swaying
Curve his/her back like a “C”,
Lateral Decubitus (Left or Right)
Needs to be Parallel to the Edge of the Bed
Legs Flexed up to Abdomen
Forehead Flexed down towards Knees
Jack-knife Position
Chosen for ano-rectal surgery
CSF will not drip from hub of needle
Use hypobaric solution
Anesthetic dose is injected at a rate of approximately 0.2 ml/sec
.
The patient and operating table should then be placed in the
position appropriate for the surgical procedure and drugs chosen.
Lateral decubitus positioning for a neuraxial block. The
assistant can help the patient assume the ideal position of
“forehead to knees.”
SPINAL : APPROACHES
1) MIDLINE APPROACH Midline Approach Paramedian
2) PARAMEDIAN APPROACH approach
Skin Skin
Subcutaneous fat Subcutaneous fat
Supraspinous ligament
Structure Pierced Interspinous ligament
Ligamentum flavum Ligmentum flavum
Dura mater Dura mater
Subdural space Subdural space
Arachnoid mater Arachnoid mater
Subarachnoid space Subarachnoid
space
STRUCTURES PIERCED
MIDLINE APPROACH
•With advancement of needle, two “resistance” are felt. The
first is penetration of the L. flavum & second is the
penetration of dura-arachnoid membrane.
• The stylet is then removed, and CSF should appear at the
needle hub.
•For spinal needles of small gauge, this usually takes 5-10 sec
PARAMEDIAN APPROACH
Used in case of Calcified interspinous ligament or difficulty in
flexing the spine
The needle : 1 cm lateral of the superior spinous process of
desired level.
Angle : 10-25 toward midline.
TECHNIQUE OF LUMBAR PUNCTURE
Monitors should be placed.
Airway, oxygen and resuscitation equipment should be
readily available.
All equipments for the spinal blockade should be ready
for use.
All necessary medications should be drawn up prior to
positioning the patient for spinal anesthesia.
Preload, Secure large bore IV Cannula in iv line; attach
pint with the help of IV set to vigo, start @10-20ml/kg drip
set.
Proper positioning of the patient is must.
The skin should be painted and drapped in sterile fashion.
The midline should be palpated. The iliac crests are palpated,
and a line is drawn between them in order to find the body of L4
or the L4-5 interspace.
Local anesthetic is then administered.
Bupivacaine 0.5%, Ropivacaine 0.75%, are most common local
anesthetic drugs used in spinal anesthesia.
DERMATOMES
Dermatome level Surface Landmark
(sensory block)
C8 Little finger
T1,T2 Inner aspect of the arm
T4 Nipple line, root of
scapula
T6 Inferior border of
scapula ,Tip of xiphoid
T10 Umbilicus
L2 to L3 Anterior thigh
S1 Heel of foot
CHECK EFFECT OF ANAESTHESIA
MODIFIED BROMAGE SCALE FOR ONSET OF MOTOR BLOCK
Scale Criteria Degree of motor
block
0 Free movement of legs and feet with None
ability to raise extended leg
1 Inability to raise extended leg, knee Partial (33%)
flexion is decreased, but full flexion of
feet and ankles
2 Inability to raise leg or flex knee, Partial(66%)
flexion of ankle and feet present
3 Inability to raise leg, flex knee or move Complete paralysis
toes
STOUTS PRINCIPLE FOR SPREAD OF
SOLUTION
• Height of anesthesia directly proportional to
1.Speed of injection
2.Volume of the fluid
3.Specific gravity of fluid
4.Concentration of drug
• Height of anesthesia inversely proportional to
5. Rapidity of fixation
6.Spinal fluid Pressure
7. Height of anesthesia varies with position of patient
ADDITIONAL FACTORS TO CONSIDER WITH
SAB…
Patient Age
Elderly patients
Patient Height
Intra-abdominal Pressure(Pregnancy &
Obesity )
Addition of opioids
Barbotage Technique
Site of injection
DIFFERENTIAL BLOCK WITH SAB
Sympathetic Block- 2 dermatomes higher
than the sensory block
Motor Block- 2 dermatomes lower than
sensory block
SPINAL NEEDLE
Spinal needles fall into two main
categories:
(i) those that cut the dura :
Quincke- Babcock needle,
the traditional disposable
spinal needle
(ii) those with a conical tip(Pencil
tip) : Whitacre and Sprotte
needles
If a continuous spinal technique
is chosen, use of a Tuohy or
Hustead needle can facilitate
passage of the catheter QUINCKE WHITACRE SPROTEE
Blunt tip (pencil-
point) needle decreased
the incidence of PDPH.
Sprotte is a side-
injection needle with a
long opening.
It has the advantage of
more vigorous CSF flow
compared with similar
gauge needles.
Hustead Tuohy
Examples of continuous spinal needles, including a disposable, 18-gauge
Hustead (A) and a 17-gauge Tuohy (B) needle. Both have distal tips designed to
direct the catheters inserted through the needles along the course of the bevel
opening; 20-gauge epidural catheters are used with these particular needle
sizes.
BARICITY OF LOCAL ANESTHETICS
Isobaric – Stays where you put it
LA has the same density or specific gravity as CSF (1.003-
1.008) – Normal Saline
Hypobaric – “Floats” up – Lighter than CSF
LA has a density or specific gravity that is less than CSF
(<1.003) – Sterile Water
Hyperbaric – Settles to Dependent aspect of the
subarachnoid space – Heavier than CSF
LA has a density or specific gravity that is greater than
CSF (>1.008) - Dextrose
HYPOBARIC & ISOBARIC SPINAL ANAESTHESIA
To make a drug hypobaric to CSF, it must be less dense than
CSF, with a baricity appreciably less than 1.000 or a specific
gravity appreciably less than 1.0069 (the mean value of the
specific gravity of CSF).
A common method of formulating a hypobaric solution is to
mix solution with sterile water & for hyperbaric mix with
dextrose
SPINAL BLOCK ADDITIVES
Fentanyl(<25µg)
Clonidine(25-50µg)
Dexmedetomidine (3-5 µg)
Neostigmine
Tramadol
Epinephrine (0.2 mg) or phenylephrine (5 mg)
CARDIOVASCULAR EFFECTS OF SA
Hypotension and bradycardia are the most common side
effects.(30%of baseline fall.)
Arterial and venodilation both occur.
Total peripheral vascular resistance (TPVR) decreases only by
15% to 18%. So,MAP decreases by 15% to 18%,
Because preload determines cardiac output.
patient positioning is a major factor in determining preload.
Most patients do not experience a significant change in heart
rate after spinal anesthesia.
Sympathetic cardiac accelerator fibers emerge from the T1 to
T4 spinal segments, and blockade of these fibers is proposed as
the cause of bradycardia.
Decreased venous return may also cause bradycardia, due to a
fall in filling pressures.
Even though bradycardia is usually well tolerated after spinal
anesthesia, asystole and second- and third-degree heart block
can occur, so it is wise to be vigilant when monitoring.
RESPIRATORY EFFECTS OF SA
Very little effect on pulmonary function.
Lung volumes, resting minute ventilation, dead space, arterial blood gas
tensions, and shunt fraction show minimal change after spinal anesthesia.
Effect occurs during high spinal blockade when active exhalation is
affected due to paralysis of abdominal and intercostal muscles.
During high spinal blockade, expiratory reserve volume, peak expiratory
flow, and maximum minute ventilation are reduced.
Patients of COPD that rely on accessory muscle use for adequate
ventilation should be monitored carefully after spinal blockade.
AFFECTIVE DYSPNEA
• Can be seen in patients with normal pulmonary
function with high spinal block.
• Complain: pt can’t breath.
•Cause:loss of proprioception in thoracic cage
structures will provoke a sensation of not
breathing.
•It can be combated simply by asking patient to
take deep breaths or by having the patient smell
aromatic spirits.
ABGA:
Does not change during high spinal anesthesia in patients
who are spontaneously breathing room air; . The main effect
of high spinal anesthesia is on expiration, as the muscles of
exhalation are impaired.
Since a high spinal usually does not affect the cervical area,
sparing of the phrenic nerve and normal
diaphragmatic function occurs, and inspiration is minimally
affected
GASTROINTESTINAL EFFECTS OF SA
Sympathetic blockade(from T6 to L2) and unopposed
parasympathetic activity.
Secretions increase, sphincters relax, and the bowel becomes
constricted.
Increased vagal activity after sympathetic block causes
increased peristalsis of the gastrointestinal tract, which leads
to nausea and vomiting.
THE USE OF SA IN OBSTETRICS
Resuscitative equipment and emergency medication must be
readily available.
Require less local anesthetic to achieve the same level of
anesthesia as nonpregnant women.
A T4 level block is usually required for a cesarean section due to
traction on the peritoneum and uterine exteriorization.
Because of the sympathetic blockade, hypotension may result,
It is must to monitor the blood pressure very carefully and
frequently.
It should be treated immediately with medications or fluid
administration, or both.
WHY LESS VOLUME OF DRUG IS NEEDED
IN LSCS?
Explained by two mechanisms.
1) Mechanical factor:
Obstruction of inferior vena cava by gravid uterus.
Distension of epidural venous plexuses.
Effective volume of epidural space and CSF are reduced.
Enhances cephalad spread of LA agents during both spinal and epidural
anesthesia.
2) Hormonal factor:
Mediated by progesterone which makes the nervous tissue more
sensitive to LA agents.
SA IN GERIATRIC PATIENTS
Slightly higher spinal levels of sensory and motor blockade.
At somewhat greater risk of hypotension.
Thus, bolus doses of local anaesthetic should be reduced.
The duration of blockade last longer in the elderly after peripheral nerve
blocks also.
The clearance of LA decreases with age.
Consequently, infusion rates or top-up bolus doses may need to be adjusted.
Regional anaesthesia has several beneficial effects for elderly patients,
ex.,reduced blood loss,
better peripheral vascular circulation,
suppression of the surgical stress response and
better postoperative pain control.
COMPLICATIONS
BRADYCARDIA -
Defined as HR < 50 beats/ min.
T1-4 involvement, unopposed vagal tone, decreased
venous return which leads to bradycardia and asystole
NAUSEA AND VOMITING -
Causes(Hypotension, Increased peristalsis, Opioid
analgesia)
Associated with neuraxial block in up to 20% of patients,
Ondensetron is almost universally effective in treating the
nausea associated with neuraxial anesthesia.
HYPOTENSION
Fall of >30% from baseline systolic BP of the patient.
Prevented by - Volume loading with 10-20 mL/kg of
intravenous fluid.
Treatment of hypotension:
100% O2
FLUIDS -
Crystalloid
Colloid [500-1000ml] preferred due to
increased intravascular time, maintaining
Cardiac output, uteroplacental circulation.
SYMPATHOMIMETICS:
Mephentermine:
causes vasocnstriction, increases CO,SBP,DBP.
Variable effect on HR(depends on vagal tone)
Epinephrine:
increases HR, CO, SBP, decrease DBP.
Phenylephrine:
Increase in SVR, SBP, DBP. Causes reflex
bradycardia, coronary blood flow increased.
Ephedrine:
increase myocardial contractility and rate.
SHIVERING
Cover the patient with sterile cloth.
Slow down the speed of pint.
Give Tramadol preceded by emset to control
shivering.
HIGH REGIONAL BLOCK
Due to inappropriately high spread of LA affecting spinal
nerves.
• High spinal block: spread of LA above T4.
Effects are of variable severity depending upon the
maximum level that is involved.
Includes cardiovascular and/or respiratory
compromise.
•Total spinal block: Intracranial spread of LA resulting in
loss of consciousness.
Spinal level Areas affected Symptoms/signs
T1-T4 Cardiac sympathetic Hypotension
fibers Bradycardia
C6-C8 Hands and arms Parasthesia or numbness
Accessory respiratory in hands and arms
muscles Weakness of hands/arms
Shortness of breath
C3-C5 Diaphragm and Shoulder weakness
shoulders Hypoventilation and
desaturation
Respiratory arrest
Intracranial Cranial nerves/ Sedation
spread(Total spinal) brainstem Slurred speech
Loss of consciousness
Management of total spinal –
Airway - Secure airway and administer 100%
oxygen
Breathing - ventilate by facemask or intubate.
Circulation - treat with i/v fluids and vasopressor
e.g. ephedrine 3-6mg or 0.5-1ml adrenaline 1:10
000 as required
Continue to ventilate until the block wears off (2
- 4 hours)
As the block recedes the patient will begin
recovering consciousness followed by breathing and
then movement of the arms and finally legs.
POST DURAL PUNCTURE HEADACHE
Leak of CSF leads to traction in supporting structure especially in dura and
tentorium.
vasodialatation of cerebral blood vessels.
Usually bifrontal or occipital.
Usually worse in upright , coughing , straining
Causes nausea, photophobia, tinnitus, diplopia[6th nerve], cranial nerve
palsy
Treatment plan
• Keep patient supine.
• Adequate hydration.
• NSAIDS.
• Epidural blood patch (if not relieved within 12-24 hrs)
Factors that May Increase the Incidence of Post–spinal Puncture
Headache
Age Younger more frequent
Gender Females > males
Needle size Larger > smaller
Less when the needle bevel is
Needle bevel placed in the long axis of the
neuraxis
Pregnancy More when pregnant
Dural punctures (no.) More with multiple punctures
Onset of headache :Usually 12-72 h following the procedure
Time to shift patient :
Patients should be allowed to leave the recovery room
once it can be demonstrated that…
• Their block is receding appropriately (at least two
dermatomes regression or a spinal level of less than
T10).
•Hemodynamically stable.
THANK YOU…