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CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS

MATERNAL AND CHILD HEALTH NURSING LECTURE


BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

GLOBAL HEALTH GOALS  Increasing health insurance coverage


 End poverty and hunger.  Dependent on countries policy on HC.
 Achieve universal primary education.  Increasing alternative settings & styles for HC
 Promote gender equality and empower women.  Satellite clinics & hospitals, health centers,
 Reduce child mortality. SPED.
 Improve maternal health.  Increasing use of technology
 Combat HIV/AIDS, malaria, and other diseases.  E-consult (Serious MD).
 Ensure environmental sustainability.  Meeting work needs of pregnant &
 Develop a global partnership for development. breastfeeding women
2020 NATIONAL HEALTH GOALS  Milking room, extended maternity leave
Intended to help citizens more easily understand  Regionalizing intensive care
the importance of health promotion and disease  Increasing use of alternative treatment
prevention. modalities
The two main overarching national health goals  Increasing reliance on home care
are:  Increasing concern for QoL
 Increase quality and years of healthy life.  Increasing awareness of the individuality and
 Eliminate health disparities. diversity of clients
MEASURING CHILD HEALTH  Empowerment of health care consumers
CHILD MORTALITY RATE PROBLEMS RELATED TO MATURITY
 Probability a newborn would die before 5 years PRETERM INFANT
of age (per 1,000 lives).  Defined as a live-born infant before the end of
 PH data – 17.544 deaths/1000 live births. week 37 of gestation.
CHILDHOOD MORBIDITY RATE  Early preterm (born between 24 and 34 weeks),
 Rate at w/c disease or illness occurs in a and late preterm (born between 34 and 37
population. weeks).
COMMON CHILDHOOD MORBIDITY  Neonatal assessments such as inspection of
(ILLNESS) sole creases, skull firmness, ear cartilage, and
 Congenital anomalies neurologic development plus the mother’s
 Chronic Respiratory Disease report of LMP along with a sonographic
 Non communicable disease estimation of age all can be helpful to
 Acquired heart disease determine gestational age.
 Childhood Cancer  Preterm birth occurs in approximately 10% of
 Diabetes live births of non-Hispanic White infants. In
 Obesity African American infants, the rate is
COMMON CAUSE OF CHILDHOOD approximately 17.47%.
MORBIDITY (ILLNESS)  Prone to hypoglycemia.
 Infectious disease  Lack of lung surfactant – Respiratory Distress
 Preterm birth Syndrome (RDS).
 Asphyxia & trauma COMMON FACTORS ASSOCIATED W/
 Congenital anomalies PRETERM BIRTH
TRENDS IN HEALTHCARE ENVIRONMENT  Low socioeconomic level
 Initiating cost containment  Poor nutritional status
 Cost effectiveness.  Lack of prenatal care
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

 Multiple pregnancy  Bowel develops necrotic patches, interfering


 Previous early birth with digestion and possibly leading to a
 Race (nonwhites have a higher incidence) paralytic ileus, perforation, and peritonitis.
 Cigarette smoking OTHER PROBLEMS RELATED TO
 Age of the mother (younger than age 20 PREMATURITY
years)  RETINOPATHY OF PREMATURE INFANT
 Order of birth (1st & 4th) (ROP)
 Closely spaced pregnancies  Eye disease that can happen in babies who are
 Abnormalities of the mother’s reproductive premature, or who weigh less than 3 lbs.
system  ROP happens when abnormal blood vessels
 Infections grow in the retina.
 Pregnancy complications (PRoM, PSP)  Caused by vasoconstriction of immature retinal
 Early induction of labor blood vessels.
 Elective cesarean birth  S & SX: wandering eye, eye doesn’t follow
POTENTIAL COMPLICATIONS OF objects, pupils look white, trouble recognizing
PREMATURE INFANTS faces.
1. ANEMIA OF PREMATURITY  Mgt: regular check-up, surgery for severe
 All babies have some anemia when they are cases.
born. Normalizes around 3-6 months.  TEMPERATURE INSTABILITY
 There is a low level of hemoglobin w/ the  Preterm infants are prone to rapid heat loss &
infant. consequent hypothermia because of large
 Due to excessive blood drawing for electrolyte surface area to body mass ratio, decreased
or blood gas analysis (iatrogenic blood loss). subcutaneous fat, immature skin, high body
 S & SX: lethargy, difficult to feed, not very water content, poorly developed metabolic
active. mechanism & altered skin blood flow.
 Nrsg mgt: monitoring.  Heat loss via: evaporation, convection,
 Mgt: BT, iron (Fe), erythropoietin (EPO) as last conduction, radiation.
resort.  Premature brain, heart, lungs & kidneys.
2. ACUTE BILIRUBIN ENCEPHALOPATHY POTENTIAL COMPLICATIONS OF
(KERNICTERUS) PREMATURE INFANTS
 High level of bilirubin may cause seizures 1. PERSISTENT PDA (AO-PA)
leading to brain cell damage.  PDA (Patent Ductus Arteriosus) is important
 May cause problems w/ vision, teeth, & during fetal circulation.
intellectual disabilities.  It should close after the baby is delivered (2-3
 S & SX: lethargy, drowsiness, poor feeding days).
habits, fever, shrill high-pitched cry, absence of  Mgt: indomethacin or ibuprofen may be used.
moro reflex.  Surgical or interventional catheterization to
 Mgt: phototherapy, or exchange transfusion. close PDA.
3. NECTROTIZING ENTEROCILITIS (NEC) 2. PERIVENTRICULAR / INTRAVENTRICULAR
 Premature GI tract. HEMORRHAGE
 Intestinal dysfunction that develops in  Bleeding in the brain of infant.
approximately 5% of all infants in intensive  Rapid change in cerebral blood pressure.
care nurseries. MEDICAL INTERVENTION
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

1. CARDIAC CATHETERIZATION PROBLEMS RELATED TO GESTATIONAL


 Procedure in which a small radiopaque catheter WEIGHT
is passed through a major vein in the arm, leg, SMALL FOR GESTATIONAL AGE (SGA)
or neck into the heart to secure blood samples  Infant w/ BW below 10th percentile.
or inject dye, is a major method used to  Intrauterine growth restriction.
evaluate cardiac function.  Infant could be born preterm, term, or
 Diagnostic/interventional. postterm.
 PDA closure.  Thermal regulation problem.
 Pacemaker insertion.  Hypoglycemia.
2. HEART SURGERY IN CHILDREN LARGE FOR GESTATIONAL AGE (LGA)
Open-heart or intracardiac catheterization surgery  Infant w/ BW above 90th percentile.
remains the chief cure for congenital heart disease.  Infant could be born term, or postterm.
 Extracorporeal Membrane Oxygenation (ECMO).  They tend to be infants of diabetic women, and
 Vascular Assistive Devices (VAD). they are particularly prone to birth trauma.
 PDA closure. LUBCHENCO CHART
 Heart transplant.  To determine maturity.
PROBLEMS RELATED TO MATURITY
POSTTERM INFANT
 More than 42 weeks of gestation.
 Prone to hypoglycemia and intracranial
hemorrhage.
 Prone to meconium stain/meconium
aspiration.
POSTTERM SYNDROME
 Dry, cracked, leather like skin, & absence of
vernix caseosa.
 LGA, less amniotic fluid at birth & may be
meconium stained.
 Grown fingernails beyond the end of the
fingertips.
 May demonstrate an alertness much more like a
2-week-old baby than a newborn.
PROBLEMS RELATED TO POSTTERM
INFANTS
A. Meconium stain
B. Meconium aspiration
C. Difficult extraction
D. Temperature regulation
E. Polycythemia ALTERATION IN OXYGENATION
Illness that occur in New Born:
F. Utero placental insufficiency
 Respiratory Distress Syndrome (RDS)
G. Oligohydramnios
 Meconium Aspiration
H. Increased risk for intra uterine infection
 Transient Tachypnea of NB (TTN)
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

 Apnea  Low APGAR score, limp, tachypneic, shallow


RESPIRATORY DISTRESS SYNDROME retractions, tachycardic.
(RDS)  Chest X-ray: course, irregular patchy infiltrates,
 Often occurs in preterm infants, infants of snow storm pattern.
diabetic mothers, infants born by CS, or those  Labs: abnormal ABGs, SGPT, Crea.
who for any reason have decreased blood  Mgt: close monitoring, high O2, ET intubation.
perfusion of the lungs.  Prognosis: poor.
 Cause of RDS is a low level or absence of
surfactant, the phospholipid that normally lines
the alveoli and reduces surface tension to keep
the alveoli from collapsing on expiration.
 S & SX:
 Low body temperature, nasal flaring, sternal
and subcostal retractions.
 Tachypnea (more than 60 breaths/min),
cyanotic mucous membranes.
 Mgt:
 Surfactant replacement therapy
 Oxygenation TRANSIENT TACHYPNEA OF NB (TTN)
MECONIUM ASPIRATION  Rapid RR due to late absorption of placental
 Meconium staining occurs in approximately fluids in the lungs.
10% to 20% of all births.  S & SX:
 In 2% to 4% of these births, infants will  Tachypnea, retractions, cyanosis,
aspirate enough meconium to cause MAS. desaturation.
 S & SX:  May be same presentation w/ pneumonia.
 Tachypnea an coarse bronchial sounds  Usually resolves in 2-3 days.
 Chest retractions  Mgt:
 Chest X-ray may reveal pneumonia  Close observation & monitoring.
 PE – barrel chest (anteposterior)  O2 support as needed.
 Poor gas exchange evidenced by a decreased APNEA
O2 and an increased Pco2.  As a result of fatigue or the immaturity of their
 Mgt: respiratory mechanisms.
 O2 support (intubation, CPAP)  High incidence of apnea is common on babies
 Antibiotics with secondary stresses ie. infection,
 MECONIUM ASPIRATION PNEUMONIA hyperbilirubinemia, hypoglycemia, hypothermia.
 Good APGAR score, active, tachypneic, shallow  S & SX:
retractions, tachycardic.  Pause in respirations longer than 20 seconds
 Chest X-ray: course, irregular patchy infiltrates, with accompanying bradycardia.
snow storm pattern.  Mgt:
 Labs: normal ABGs, SGPT, Crea.  Gently shaking an infant or flicking the sole of
 Mgt: monitoring, none-low O2 therapy. the foot often stimulates the baby to breathe
 Prognosis: good. again.
 MECONIUM ASPIRATION SYNDROME
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

 Maintain a neutral thermal environment and


use gentle handling to avoid excessive
fatigue.
 Always suction gently to minimize
nasopharyngeal irritation, which can cause
bradycardia because of vagal stimulation.
FETAL CIRCULATION
CONGENITAL HEART DISEASE  Umbilical vein delivers oxygenated blood
 CHDs are present at birth & can affect the from the placenta to the fetus.
structure of a baby’s heart & the way it works.  Umbilical arteries transport deoxygenated
 8% of term infants and 9% - 10% of preterm blood away from the fetal tissue & back
infants are born with a congenital towards the placenta for re-oxygenation.
cardiovascular abnormality.  Ductus venosus allows blood from the
 CHDs can affect the blood flows through the placenta to bypass the highly demanding, but
heart & out of the rest of the body. inactive liver.
 CHDs happen due to failure of the heart  Ductus arteriosus is the fusion of the
structure to progress beyond an early stage of primitive PA (Pulmonary Artery) to the AO
embryonic development. (Aorta), allowing blood to pass straight from
 CHDs can vary from mild to severe. the RV (Right Ventricular) in to the AO and
 Small hole in the heart to missing or poorly bypass the inactive lungs.
formed parts of the heart.  Foramen ovale creates a shunt between the
 ASD: Atrial Septal Defect RA (Right Atrium) and LA (Left Atrium) so
 VSD: Ventricular Septal Defect oxygenated blood from the placenta can move
 PDA: Patent Ductus Arteriosus to the left atrium. This allows for the
 AVCD: Atrioventricular Canal Defect oxygenated blood to pass through the LV (Left
 COA: Coarctation of Aorta Ventricular) and into the ascending AO,
 PVS: Pulmonary Valve Stenosis oxygenating the brain.
 AVSD: Atrioventricular Septal Defect
 TOF: Tetralogy od Fallot
 TGA: Transposition of the Great Arteries
 TA: Tricuspid Atresia
 TrA: Truncus Arteriosus
 HLHS: Hypoplastic Left Heart Syndrome

1. ATRIAL SEPTAL DEFECT


CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

 Birth defect of the heart in w/c there is a hole S & SX:


in the wall that divides the upper chambers of  SOB, fast/heavy breathing, sweating, tiredness
the heart. while feeding, poor weight gain.
 Hole can vary in size & may close on its own.  PE: whooshing sound (heart murmur).
 Echocardiography will generally reveal the  85% of VSDs are so small they close
enlarged right side of the heart and the spontaneously.
increased pulmonary circulation.  Many children are managed only by close
 Volume problem- oxygen remains normal in observation.
early stages. MX MGT:
 Acyanotic because the pressure of LA is higher  Diuretics or digoxin to help prevent fluid from
than the pressure of RA, so blue discoloration is accumulating in the lungs.
not highly evident.  Surgery or cardiac to help prevent fluid from
THERAPEUTIC MANAGEMENT: accumulating in the lungs.
 Interventional catheterization to close the  Complications include: pulmonary HPN,
disorder is usually done electively by cardiac arrhythmias, or stroke.
catheterization between 1 and 3 years of age.
 The edges of the opening in the septum are
approximated and filled in with a septal occlude
device (amplatzer).

3. PATENT DUCTUS ARTERIOSUS (PDA)


 Persistent opening between 2 major blood
vessels leading from the heart (PA-AO).
 DA – ductus arteriosus is part of a baby’s blood
2. VENTRICULAR SEPTAL DEFECT (VSD) flow system in the womb. It usually closes after
 Most common type of congenital cardiac birth.
disorder, occurs in about 4 to 5 of every 1,000  Small PDA often doesn’t cause problems and
live births. might never need treatment.
 Birth defect of the heart in w/c there is a hole S & SX:
in the wall that separates the 2 lower chambers  Poor eating/poor growth, fast breathing, easy
of the heart. tiring, rapid heart rate.
 The extra blood being pumped into the lungs
forces the heart & lungs to work harder.
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

 Complications includes: pulmonary HPN


(hypertension), CHF (Congenital Heart Failure),
heart infection (endocarditis).
MGT:
 Ibuprofen or indomethacin.
 Surgery & cardiac catheterization.

5. COARCTATION OF AORTA (COA)


 Is a birth defect in w/c a part of the AO is
narrower than usual.
 If the narrowing is severe enough &
undiagnosed, the baby may have serious
problems & may need surgery or other
procedures soon after birth.
 The narrowing blocks normal blow flow to the
body. This can back up flow into the LV.
 Narrowing of AO is usually located after arteries
branch to the upper body, coarctation can lead
to normal or high blood pressure & pulsing of
blood in the head & arms, weak pulses in the
4. ATRIOVENTRICULAR CANAL DEFECT legs & lower body.
(AVCD)  Is often considered a critical congenital heart
 Comprise a spectrum of defects including defect.
ventricular or atrial defects, septal defects, AV  Extra work on the heart can cause walls of the
anomalies, & endocardial cushion defects. heart to become thicker in order to pump
 Happens when valves & septum do not form harder (this weakens the heart muscle & may
completely while baby is still in the womb. lead to heart failure).
 Maybe complete or partial. S & SX:
 COMPLETE: have a combination of 3  Pale ski, irritability, profuse sweating, DOB.
separate defects (ASD, VSD, 1 large valve  PE: noticeable weak pulse in the legs or groin,
between atrium & ventricle). heart murmur.
 PARTIAL-HOLE: hole in the atrial septum  Older children often have high BP in the arms.
(ASD), & partial abnormality or “cleft” in the  Dx test: echocardiogram (primary), ECG/EKG,
left valve (mitral valve). cardiac catheterization.
 Common in children with Down syndrome  Mx/Tx: surgery or cardiac catheterization
(Trisomy 21). (balloon angioplasty).
S & SX: MGT:
 Slow/poor growth, undernourishment, hard &  Regular check-up.
fast breathing pattern, sweating while eating,
and weight loss.
 Dx tests: ECG/EKG, chest X-ray,
echocardiogram.
 Close monitoring of VS & O2 sat.
 Treatment: surgery.
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

7. ATRIOVENTRICULAR SEPTAL DEFECT


(AVSD)
 Is a congenital heart condition in w/c there is a
hole at the center of the heart.
6. PULMONARY VALVE STENOSIS (PVS)  Incidence rate is about 2/10,000 babies.
 Narrowing of the valve between the lower right  There is no clear reason why it occurs, but
heart chamber & lung arteries. most are related to genetic condition such as
 In a narrowed heart valve, the valve flaps may down syndrome.
become think or stiff w/c reduces blood flow. S & SX:
S & SX:  Fast breathing, SOB when feeding, arrhythmia,
 Depends on how much blood flow is blocked. fatigue, and palpitations.
 Mild PS doesn’t have symptoms.  Untreated, AVSD can cause heart failure &
 Severe PS: whooshing sound (heart murmur), pulmonary HPN.
fatigue, SOB during activity, chest pain, MGT:
fainting.  Surgery (6 y/o and above).
 Echocardiography will reveal right ventricular
hypertrophy.
 Dx: ECG/EKG, echocardiogram, cardiac
catheterization.
MX & TX:
 Balloon angioplasty by way of cardiac
catheterization is the procedure of choice for
severe PS.
 Pulmonary valve replacement (surgery or
cardiac catheterization).
 Following the procedure, although children may
always have residual heart murmur, they can
expect to have a normal life span.
 Children who have had surgery need to take
antibiotics before dental procedure or other
surgeries to prevent endocarditis.

8. TETRALOGY OF FALLOT
 Is a CHD that affects the normal blood flow
through the heart.
 Made up of 4 defects of the heart & blood
vessels.
 VSD, PSD, enlarged aortic valve, & RV
hypertrophy.
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

 TOF can cause decreased O2 in the blood that S & SX:


flow to the rest of the body.  Pounding heart, weak pulse, ashen or
S & SX: bluish skin color, breathing problems, poor
 Infants w/ TOF may develop sudden episodes feeding.
of bluish discoloration during crying or feeding
MGT:
called “tet spells.”
 Surgery (arterial switch operation or atrial
 Infants w/ TOF are prone to endocarditis,
switch operation).
arrhythmia, seizure, & delayed growth.
 Dx: TOF is often diagnosed after the baby is
born.
 NB screening (low pulse ox),
echocardiography.
MX & TX:
 Surgery can be done right after the baby is
born.
 PV replacement, PA replacement, patch
replacement on VSD.

10. TRICUSPID ATRESIA


 A birth defect of the heart where the valve that
controls the blood flow from the R upper
chamber of the heart to the right lower
chamber of the heart does not form at all.
 Blood cannot flow correctly through the heart &
the rest of the body.
 No blood can go from the atrium through the
9. TRANSPOSITION OF THE GREAT RV to the lungs for oxygenation.
ARTERIES  Since blood cannot directly flow from RA to RV,
 TGA is a birth defect of the heart in w/c the 1 blood must use other routes to bypass the
main arteries (PA & AO) carrying blood out of unformed tricuspid valve.
the heart are switched in positions.  TA is common on in infants w/ Down syndrome.
 In babies w/ TGA, unoxygenated blood from  Familial tendency and viral illness (rubella).
the body enters the R side of the heart. But,  Is a critical congenital heart defect and needs
instead of going to the lungs, blood is pumped procedures & surgery soon after birth.
directly back out to the body through the AO. S & SX:
 Oxygenated blood from the lungs entering the  Problem breathing, ashen or bluish skin color,
heart is pumped straight back to the lungs via poor feeding, extreme sleepiness.
the main PA.  Evident murmur (whooshing sound).
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

MX MGT:  Dx test: echocardiography, pulxe ox (NB


 Surgery. screening).
 SEPTOSTOMY: done within babies few days of TX:
life. Purposely creates or enlarges an ASD.  Surgery to repair the heart & blood vessels.
 BANDING: temporary placement of band 12. HYPOPLASTIC LEFT HEART
around the artery w/ in the baby’s few days of SYNDROME (HLHS)
life to control blood flow to the lungs.  A CHD that affects the normal blood flow
 SHUNT PROCEDURE: surgeons put a bypass through the heart.
from AO to PA.  As the baby develops, the L-side of the heart
 BI-DIRECTIONAL GLENN PROCEDURE: does not form correctly.
performed within 4-6 months of age. Procedure  LV, mitral valve, aortic valve, ascending portion
that creates a direct connection of the main PA of the AO.
to SVC.  Babies w/ HLHS also have ASD.
 FONTAN PROCEDURE: done within 2 yrs. of S & SX:
age. Connection of the main PA with the IVC.  Breathing problem, pounding heart, weak
pulse, ashen/bluish skin color, poor feeding.
MGT:
 3 stages of surgery.
1. NORWOOD SURGERY: done w/ in first 2
weeks of life, doctors create new AO.
2. BI-DIRECTIONAL GLENN SHUNT
PROCEDURE: done w/ in 4-6 months of age.
Doctors create a direct connection PA & SVC.
3. FONTAN PROCEDURE: done w/ in 18months
to 3 years of age. Doctors connect the PA to
11. TRUNCUS ARTERIOSUS (TrA) IVC.
 A rare CHD in w/c a single blood vessel comes
out of the heart, instead of 2 vessels (main PA
and AO).
 It occurs when blood vessel coming out of the
heart in the developing baby fails to separate
completely during development, leaving a
connection between the AO & PA.
 Usually, there’s a hole between the ventricles
(VSD).
 Unoxygenated & oxygenated blood are mixed
together as blood blows to the lungs and the
rest of the body.
S & SX: EINSENMENGER SYNDROME
 Breathing problem, pounding heart, weak  Develops when too much blood flows through
pulse, ashen/bluish skin color, poor feeding, the arteries that carry blood to your lungs from
extreme sleepiness. your heart. The result is pulmonary HPN.
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

 It also leads to blood shunting between the left  Associated w/ Hirsch sprung disease & cystic
and right sides of your heart. This means fibrosis.
there’s an abnormal connection that causes  Predominantly affects neonates w/ low BW,
blood flow from the left side of your heart to infants of mothers treated w/ MgSo4
the right side of your heart. (eclampsia, preeclampsia, preterm labor).
 Eisenmenger syndrome is usually the result of S & SX:
one or more untreated congenital (present at  Absence of stool, abdominal distention, &
birth) heart defects. Over time, it can lead to vomiting.
permanent lung damage and other TX:
complications.  Radiographic contrast enema (therapeutic
NURSING CARE OF HIGH RISK NEWBORN TO effect).
MATURITY C. MECONIUM ILEUS
 Meconium aspiration syndrome  Bowel obstruction that occurs when the
 Meconium plug syndrome meconium is blocking the last part of the baby’s
 Meconium ileus small intestine.
 Sepsis  This can happen when the meconium is thicker
 Hyperbilirubinemia & stickier than normal.
 Sudden infant death syndrome S & SX:
A. MECONIUM ASPIRATION SYNDROME  Abdominal distention, greenish vomit, no
(MAS) passage of meconium.
 Meconium is the first feces or stool of the baby. TX:
 MAS occurs when NB breathes a mixture of  Radiographic enema or laparotomy (ileostomy
meconium and amniotic fluid into the lungs w/ lavage).
around the time of delivery. D. NEONATAL SEPSIS
 A leading cause of severe illness & death in NB  Neonatal sepsis is a blood infection that occurs
(5-10% of births). in infant younger than 90 days old.
 Occurs when the baby is stressed during labor,  Early onset – 1st week, late onset – 2 weeks
especially when the infant is past its due date. to 3 months of age.
S & SX:  Occurs when the body has an extreme
 Bluish skin color, breathing problem, dark response to an infection (inflammation
greenish stain, limpness upon birth. throughout the body) w/c leads to reduced
MGT: blood flow to limbs & vital organs.
 Antibiotics, O2 therapy, warmer/drop light, PREDISPOSING FACTORS:
chest clapping.  Bacterial infection (E-coli, listeria, strains of
B. MECONIUM PLUG SYNDROME (MPS) streptococcus).
 Colonic obstruction caused by thick meconium.  Preterm delivery.
 Transient disorder of the NB colon  ROM longer than 18hrs before birth.
characterized by delayed passage of meconium  Infection of placenta & amniotic fluid
& intestinal dilatation. (chorioamnionitis).
 Also termed functional immaturity of the colon, OTHER RISK FACTORS:
colonic immaturity, small left colon syndrome, &  Having catheter in blood vessel for a long
functional colonic obstruction. time.
 1 in 500 live births.
CHAPTER 1: CARE OF CHILD AT RISK OF WITH PROBLEMS
MATERNAL AND CHILD HEALTH NURSING LECTURE
BS Nursing 2-1 | PROF. Edwin Rommel Grantoza | SEM 2 2023

Staying in the hospital for an extended period


  Use frim mattress w/ fitted sheets.
of time.  No fluffy blankets & stuffed animals.
S & SX:  Smoke free environment.
 Body temperature changes, breathing  Ambient room temperature while the baby
problems, diarrhea or decreased bowel sleeps.
movements, low blood sugar, reduced  Proper prenatal care & feeding the baby.
movements, reduced sucking, seizures, slow or
fast heart rate, swollen belly area, vomiting,
yellow skin & whites of the eyes (jaundice).
 Lab tests: blood culture, C-reactive protein, CBC
MX MGT:
 IV antibiotics.
E. HYPERBILIRUBINEMIA
 Happens when there is too much bilirubin in the
baby’s blood.
 60% of term NBs & 80% of preterm NBs get
jaundice (jaune-yellow).
S & SX:
 Yellowing of the skin & whites of eyes.
TX:
 Phototherapy.
F. SUDDEN INFANT DEATH SYNDROME
(SIDS)
 Sudden & unexplained death of a baby younger
than 1 y/o.
 Sometimes called crib death because death is
usually associated w/ sleep.
 Leading cause of post neonatal death of babies
in the USA.
RISK FACTORS:
 Placing baby on his side or stomach to sleep.
 Premature or low birth weight babies.
 Overheating the baby during sleep.
 Sleeping on too soft surface w/ loose
blankets.
 Having a sibling who died of SIDS or family
hx.
OTHER FACTORS:
 Mothers under 20 yrs. of age.
 Smoking during pregnancy.
 Mother w/ little or no prenatal care.
MGT:
 Placing baby on his back to sleep.

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