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    Hypoglycemic drugs used in the treatment of Type 2 diabetes work through various mechanisms such as binding to pancreatic beta cell channels to stimulate insulin secretion, inhibiting the absorption of carbohydrates from the small intestine, or preventing the inactivation of GLP-1 to stimulate insulin secretion while inhibiting glucagon. These drugs help lower blood sugar levels by different pathways that either stimulate insulin, increase insulin sensitivity, inhibit gluconeogenesis or block glucose absorption and reabsorption.

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    Hypoglycemic drugs used in the treatment of Type 2 diabetes work through various mechanisms such as binding to pancreatic beta cell channels to stimulate insulin secretion, inhibiting the absorption of carbohydrates from the small intestine, or preventing the inactivation of GLP-1 to stimulate insulin secretion while inhibiting glucagon. These drugs help lower blood sugar levels by different pathways that either stimulate insulin, increase insulin sensitivity, inhibit gluconeogenesis or block glucose absorption and reabsorption.

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    Uploaded by

    Lisa L
    Hypoglycemic drugs used in the treatment of Type 2 diabetes work through various mechanisms such as binding to pancreatic beta cell channels to stimulate insulin secretion, inhibiting the absorption of carbohydrates from the small intestine, or preventing the inactivation of GLP-1 to stimulate insulin secretion while inhibiting glucagon. These drugs help lower blood sugar levels by different pathways that either stimulate insulin, increase insulin sensitivity, inhibit gluconeogenesis or block glucose absorption and reabsorption.

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    of 1

    Hypoglycemic Drugs used in the Treatment of Type 2 Diabetes

    Across Down
    2 Bind to pancreatic beta cell channels, similar to 1 Inhibits the absorption of
    sulfonylureas but with faster dissociation. carbohydrates from the small
    3 Lower blood sugar levels by preventing renal intestine
    reabsorption of glucose. Options:
    4 Prevents inactivation of GLP-1, stimulating insulin
    secretion and inhibiting glucagon. Biguanides
    5 Stimulate insulin secretion from pancreatic beta-cells Sulfonylureas
    by closing ATP-sensitive K+ channels.
    Meglitinides
    6 Decreases hepatic gluconeogenesis and increases
    insulin sensitivity SGLT2Inhibitors
    7 Increase insulin sensitivity by binding to nuclear αglucosidaseinhibitor
    transcription regulator PPAR gamma to increase
    adiponectin release and GLUT 4 expression Thiazolidinediones
    DPP4inhibitors

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