Diabetes Research Paper Introduction

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This dynamic and nuanced understanding of one T2D risk factor should be desired in all other
phases of study. High FST values and high ho- mozygosity suggest a rapid rate of divergence where
neutral, background mutation is insignificant. The molecular variant that arises from these
polymorphisms (HapB T2D ) and another molecular variant that is less understood (HapA) each
increase risk for T2D by either directly affecting ? -cell function within the pancreas or by positively
associating with BMI. For example, it has been suggested that abnormalities in the apoptosis of ?-
cells might lead to an autoimmune response. As an adaptive mechanism, HapA is still being selected
for in certain agricultural environments despite its positive association with type 2 risk because of the
advantages in energy storage and metabolism of higher BMI. This type of therapy aims to more
closely mimic insulin secretion by the pancreas and usually offers greater control of glycemia,
(Habermann, 2006), lower glycated hemoglobin (A1C), and therefore lower risk for complications
(Habermann, 2006). Patients with islet transplants can typically remain in stage 4 for considerable
periods of time if immunosuppressive therapy can sufficiently maintain viable ? cells. Insulin and
glucagons are intimately but independently involved in the regulation of the blood glucose levels.
The apparently big world is shrinking into a global village as democracy spreads, western
civilizations encroach on other civilizations. At this stage, the individual is predisposed to having
diabetes mellitus due to some factors called the risk factors. Footer Picture Dictionary English
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Calculator. PPAR?, western blot may also indicate which proteins disappeared with the combined.
Give us your email address and we’ll send this sample there. Diabetes can be both reversible as well
as chronic. The present study has demonstrated that RA and 1,25 (OH)2 D3 do not cause any. This
Metabolic disorder causes the body to lose its main source of fuel considering that the blood
contains large amounts of glucose (since diabetic people are usually obese people or people who have
large contents of glucose in their bodies). This causes the glucose to be discharged out of the human
body in the form of urine. Actually, these donuts and many sugary products like it have the ability to
sicken, and possibly kill, millions of people world-wide. HapA has been strongly correlated with high
BMI increases among certain groups of West Africans, Euro- peans, and East Asians in more energy-
plentiful environments, while the haplotypes rs12255372 and rs7903146 (HapB) have been found
unassociated, and at times negatively associated, with BMI among other sampled populations of
North and West Africa. Chronic Diabetes is caused by a lack of insulin hormone production or the
body’s resistance to the insulin produced. Limits applied to the search were journals from 2000-
2010, peer reviewed, research articles and English language. And many of these individuals are
doubly burdened: diabetes not only decreases productivity and an individual's ability to earn an
income, but it also places a strain on a family's existing income. An open and incorporative mindset
such as this can revitalize the research field. This is probably a result of increased ?-cell mass.
Gestational Diabetes occurs only during pregnancy, and once the baby is born, the condition gets
better in the mother’s body. Table 1 represents the treatment matrix for lysate generation. Since
mood swings are often a symptom, family members are often victims of mood and emotional
individuals. In Type 1 diabetes, the body’s immunity system fuelled by a genetic reason leads the
immune response to attack the beta cells and destroy them, thereby diminishing or almost inhibiting
insulin production. Helgason, A. et al. (2007). Refining the impact of TCF7L2 gene vari- ants on
type 2 diabetes and adaptive evolution. Although it has been illustrated as a risk factor for diabetes,
authors speculate that HapA’s reproductive success despite this fact is due to its advantageous
effects on BMI rela- tive to energy metabolism in agricultural environments (Hel- gason et al., 2007).
With regard s to human disease in general, we can take some additional time to ponder and
appreciate the ways in which different physiological pathways can arrive at a single disorder.
This is 100% legal. You may not submit downloaded papers as your own, that is cheating. Also you.
Those bearing the heterozygous phenotype produce a blend of normal red blood cells and “sickled”
cells—abnormal red blood cells that acquire their distinct shape from a deform- ity in the ? -globin
chains of hemoglobin. Type 2 Diabetes Type 2 Diabetes research papers examine the condition that
causes the body to no longer produce enough insulin and the cells of the body do not respond to
insulin properly. People with high cholesterol, hypertension, obesity, a sedentary lifestyle, and high
alcohol consumption are at a high risk of developing diabetes later on. 10 Lines on Diabetes Essay in
English 1. The mechanisms responsible for the loss of GSIS are unknown. Spikes in incidence rates
that are aligned with the immigration of certain groups to a new environment suggest a preexisting
set of genetic conditions that are maladaptive to that new environment. These demographic groups
were chosen because of the high incidence rates among them. Retro-analysis and comparison of the
co-evolution between P. The risk of getting affected with diabetes increases with age. We must now
track a final aspect to this story—the frequen- cies and selection for human HgbS, the allele causing
the auto- somal recessive genetic blood disorder sickle-cell anemia. This is also considered a milder
type of diabetes in comparison to type 1, but. HapA has been strongly correlated with high BMI
increases among certain groups of West Africans, Euro- peans, and East Asians in more energy-
plentiful environments, while the haplotypes rs12255372 and rs7903146 (HapB) have been found
unassociated, and at times negatively associated, with BMI among other sampled populations of
North and West Africa. They can also reveal patterns of conservation and decelerated evolution, if
changes in frequency fall below simulations that assume stable equilibrium with respect to those
alleles. Let us write or edit the research paper on your topic. The number of children and teenagers
with type 2 has skyrocketed over the past two decades due to lack of exercise, poor eating habits,
and weight gain. The exact origin of type one diabetes is not known and no known way to prevent
the disease from occurring. Upon food consumption, the pancreas automatically produces the right
amount of insulin to move and synthesize glucose from the blood into the human cells. Answer:
Diabetes can trigger coronary artery disease resulting in heart failure. Question 4. Can diabetes cause
kidney failure. As far as affected individuals are concerned the best cure is still prevention, and this
could be done by living a healthy lifestyle (eating healthy food and proper exercise), and by taking
necessary medication. Answer: Prolonged Diabetes can cause chronic kidney failure in many
patients. Type 2 diabetes is usually kept in check with medications that constitute insulin sensitizers.
Order from one of our vetted writers instead Order a paper now Free essays can be submitted by
anyone, so we do not vouch for their quality Want a quality guarantee. Original Description:
Diabetes mellitus, or simply diabetes, is a group of metabolic diseases in which a person has high
blood sugar, either because the pancreas does not produce enough insulin, or because cells do not
respond to the insulin that is produced. Proper glucose control may reduce the risks of developing
diabetes-associated complications among type 1 diabetes; adolescents usually have problems fitting
in the difficult aspects of the diabetes treatment regime. RachelPearson36 Unlocking the Power of
ChatGPT and AI in Testing - A Real-World Look, present. By continuing, you agree to our Terms
and Conditions. Type 2 diabetes is in contrast acquired during the latter stages of an individuals life
probably due to an unhealthy lifestyle. The number of overweight people is projected to increase
from 1.3 billion in 2005 to nearly 2 billion by 2030. There are also arguments that IDDM is not purely
a genetic disorder; rather, it has been suggested that certain environmental factors also play an
important role and may trigger or accelerate autoimmune diseases in genetically predisposed
individuals. Unfortunately, the answer to this question is yet to be unearthed.
One such example of this pleiotropic tradeoff is the selection of HapA in some African populations.
It appears that the incidence of T1D in youth is increasing in the U.S (American Diabetes
Association, 2008). Psychosocial factors are implicated in all aspects of the disease and its treatment.
By associating with CTNNB1 (pre- viously ? -catenin) within the cell nucleus, TCF7L2 acts on Wnt
target genes that inhibit the production of a glucagon precursor in enteroendocrine cells of th e
pancreas (Smith, 2006). This disease results when there is a high level of sugar in the blood for an
extended period of time. Here are a couple valuable information regarding diabetes. Etiology of
T1DM Type 1 diabetes is the result of the loss of. The usual symptoms to identify whether if an
individual is affected or not would be the Increased thirst and frequent urination, Extreme hunger,
Rapid weight loss, Fatigue, Blurred vision, Slow healing sores or frequent infections, and in some
cases Patches of dark velvety skin in the folds and creases of Subject’s bodies — usually in the
armpits and neck. Report this Document Download now Save Save Introduction to Diabetes For
Later 100% (1) 100% found this document useful (1 vote) 156 views 9 pages Introduction To
Diabetes Uploaded by Sameer Saurabh AI-enhanced title and description Diabetes mellitus, or
simply diabetes, is a group of metabolic diseases characterized by high blood sugar levels. Most
likely T1D is caused by interactions between genetic and environmental factors. This different form
of behavioral change has shown equally capable of influencing genetic change, and the persistence of
type 2 diabetes. Prediabetes is a borderline phase preceding Diabetes when the blood glucose levels
are higher than usual but not as high as Diabetes itself. These diagnostic criteria are defined based on
risk for diabetic retinopathy (Englegau, 1997). People are generally diagnosed with Type 1 as a child
or young adult. Concordance rates for monozygotic twins vary between 35 - 70%, while the
concordance rates for dizygotic twins is about 11 %. Type 1 diabetes is caused due to genetic
disorder, whereas Type 2 diabetes is primarily a lifestyle disorder. Soon after, the creation of the
single-use syringe for insulin, and the large portable glucose meters to monitor blood sugar were
designed. In the beginning of the 20th century, Edward Schafer concluded that the pancreas of
diabetics was unable to produce insulin and therefore excess sugar was found in their urine.
PhastCons and PhyloP software techniques have been used to analyze conservation across a variety
of ver- tebrates, revealing tracks of alignment among 44 species. Fact sheet N ? 312. URL (Last
checked 19 April 2012). Conventional insulin treatment is an older regimen only recommended for
selected patients (McCulloch, 2007). Type 1 Diabetes Mellitus (T1DM), also known as insulin-
dependent DM, childhood DM, or juvenile-onset DM, most commonly presents in children and
adolescents. This disease is a reality for urban slum citizens, in the form of type-2 diabetes mellitus.
In the last decade, a new wave of inspired research surrounding a transcription factor’s role in blood-
sugar homeostasis has emerged. The three primary types of diabetes are: Diabetes Type 1 and 2, and
during some pregnancies, Gestational diabetes. As a result, the cells fail to absorb the glucose, and
blood glucose levels increase abnormally. These rates increase with the time since proband diagnosis;
for example, concordance is 43% within 12 years of proband diagnosis, and 50% within 40 years.
There are several advancements that must be taken for more conclusive evidence. First. High blood
glucose may also cause fatigue, blurred vision, abdominal pain, and headaches. Indeed, molecular-
clock dating analyses align the emergence of HapA with the transition to agriculture within each
HapMap group (Helgason et al., 2007). In a second assay, they performed both a Fixation Index
(FST) test and long-range haplotype (LRH) test within these populations in the hopes of finding
evidence supporting positive selection.
Bradshaw for material acquisition and cell culture maintenance along with much needed. In the case
of Type 2 diabetes, the problem would either be caused by the fact that the body does not produce
enough insulin or it may be that the cells ignore the insulin. The present study has demonstrated that
RA and 1,25 (OH)2 D3 do not cause any. By continuing, you agree to our Terms and Conditions.
Metabolic syndrome is mostly associated with type 2 diabetes mellitus and caused by the
pathophysiologic changes that occur during the course of this disease. This condition is caused due
to the body’s resistance to the insulin present in the bloodstream and the pancreas’ consequent
inability to produce enough insulin to overcome this. Rangwala, S.M., et al (2003). Genetic
modulation of PPARgamma phosphorylation regulates. Sun exposure was avoided during this
process as the. The Insulin dependent diabetes strain -- accounts only for approximately 5 percent or
less of diabetes in the U. S. (Armstrong, C. 1990). People affected with Type 1 Diabetes like others
affected with other diabetes strains, have defective pancreas glands. Answer: Prolonged Diabetes
can cause chronic kidney failure in many patients. By clicking “Check Writers’ Offers”, you agree to
our terms of service and privacy policy. It appears that a combined treatment of RA and 1,25.
Diagnosis of T1DM DM is a disease of hyperglycemia and diagnosis of both T1DM and T2DM rely
on demonstrating either a fasting or postmeal hyperglycemia (Champe, 2005). Provide your email for
sample delivery Send me this sample Your email is invalid. Although the pathogenic factors are
active early on, complications usually develop later as the disease progresses and are not as common
during early stages. Although we are pursuing degrees in business administration, the REACH team
has a wealth of experience in social enterprise -- a number of us have worked for, or founded,
nonprofit organizations, and our travels have taken us from Iraq to Cambodia in the pursuit of social
good. Figures 5A-5H clearly show elongated mammalian cells with no evidence of bacteria or.
Psychosocial factors are implicated in all aspects of the disease and its treatment. Carter and Mendis
cite anthropophilic indices taken from various regions of th e world in the range of 10% - 50%. In
the same way that the practice of agriculture has created significant selective pressures that have
increased th e frequency of the HgbS allele despite the devastating effects of the recessive
homozygous genotype, agriculture has created an energy abundant environ- ment where a molecular
variant of TCF7L2 (HapA), despite its positive association with BMI and type 2 diabetes, can thrive
because of its advantages in energy metabolism. The high degree of overlap within the PhyloP
analysis, however, sugges ts that the region is highly conserved among placental mammals.
Furthermore, another thread of research has emerged out of Iceland and Germany in 2006
concerning the association be- tween rs7903146 and heightened risk of type 2 diabetes and various
forms of colorectal can cer (Grant et al., 2006). These endeavors question the regiospecificity of T2D
by sampling from various populations worldwid e. Finally, the overview concludes with a survey of
“universal” environmental and life history factors that, in all likelihood, do indeed contribute to type
2 risk among any given population. Within the liver, glucagon activates the breakdown of glycogen
into glucose and its release into the bloodstream, therefore serving to increase blood sugar and
directly antagonize insulin. It has been hypothesized that the subtropical, cultivated regions of Asia
and the Middle East were shielded from this trend by presenting mosquitos with a larger buffer of
domesticated animals, thereby dampening their dependency on human activity and distribution.
Every year, about 650,000 people learn they have the disease. Glass coverslips were seated onto
mounting media. All. Native American Indians throughout the Americas have tended to lose their
genetic isolation and recognizability post-coloni- zation. To bring a new mindset to life, we were
introduced to TCF7L2, a transcription factor coded in a highly conserved sequence of the genome
and intimately involved in glycolysis and ? -cell function. Although his long and slender build is
revealing enough, you take a crude measure of his body mass index (BMI) to confirm that he is
underweight.
Sample is in your inbox There is a way to avoid editing or writing from scratch. Some of them being
chronic kidney failure (nephropathy), neurological damage (neuropathy), coronary artery disease,
stroke, atherosclerosis, retina damage (retinopathy), Alzheimer’s disease, microbial skin infections,
foot damage, and hearing impairment. Comparing mixed treatments to individual treatments, mixed
RA and 1,25 (OH)2 D3 caused. Etiology of T1DM Type 1 diabetes is the result of the loss of. Every
year, about 650,000 people learn they have the disease. Since mood swings are often a symptom,
family members are often victims of mood and emotional individuals. Here are a couple valuable
information regarding diabetes. LIGHT Community Medicine LIGHT IS A SOURCE OF ENERGY
THERE ARE TWO TYPE OF S. The usual symptoms to identify whether if an individual is affected
or not would be the Increased thirst and frequent urination, Extreme hunger, Rapid weight loss,
Fatigue, Blurred vision, Slow healing sores or frequent infections, and in some cases Patches of dark
velvety skin in the folds and creases of Subject’s bodies — usually in the armpits and neck. Cells
were washed 3x with PBS-MgCl2 quickly at room temperature, and then fixed utilizing 3%. Trends
in Endocrinology and Metabolism 13(5): 209-14. Barbie - Brand Strategy Presentation Barbie -
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Introduction to C Programming Language Introduction to C Programming Language Brian Covello:
Diabetes Research Paper 1. Grimaldi PA (2007). Lipid Sensing and lipid sensors: peroxisome
proliferator activated. One particular letter to Nature: Genetics, “Refining the impact of TCF7L2
gene vari- ants on type 2 diabetes and adaptive evolution”, further defines the risk association
between TCF7L2 and diabetes among a selected sample of West Africans by hypothesizing its
connec- tion to a particular variant, HapB T2D —a molecular variant pro- duced by homozygosity
for rs7903146. Two major problems are caused by the disorder: the cells may become starved for
energy, as the energy is not processed in a proper manner. Diabetic foot ulcers are common but
controlled through regular foot inspection, caring of the foot, and having sufficient footwear. Let us
write or edit the research paper on your topic. The patient's blood glucose had been below the
normal, and the body broke down fatty acids into ketone bodies which were converted to glucose.
According to King, Aubert and Herman 1998, in the year 2000 there were 150 million people with
type 2 diabetes worldwide, and this number is expected to double by 2025. All the work should be
used in accordance with the appropriate policies and applicable laws. Some complications of type 2
diabetes would include: heart disease (cardiovascular disease), blindness (retinopathy), nerve
damage (neuropathy), and kidney damage (nephropathy). In any case, what is clear is that both
environmental and genetic factors are involved, as evidenced by the T1DM concordance rate of 50%
in identical twins, as well as the variable incidence of T1DM in different countries, which is most
likely due to environmental differences such as nutrition or lifestyle (Rosmalen, Leenen et al. 2002).
The role of defective immune regulation in the development of T1DM is heavily documented. Carter
and Mendis cite anthropophilic indices taken from various regions of th e world in the range of 10%
- 50%. These genetic, micro-regional, and parallel ways of thinking illuminate the necessity of a
multiple systems approach to studying type 2 diabetes and reveal a method of bringing focus to the
ambiguities and voids still remaining within type 2 diabetes research. This was followed by an
additional wash and staining with. Build Up of America Research Paper on Effective Handoff
Communication Need an original essay on Diabetes Research Paper Sample. This causes the glucose
to be discharged out of the human body in the form of urine. Insulin helps glucose get into your
body's cells and that is how your body gets the energy it needs. The cause of type 1 diabetes is not
known and it is not preventable with current knowledge. Diet, exercise, pills and insulin injections
can control it.
Protein concentrates of prepared lysates were determined using a standard curve. All the while, we
synthesize a new argument that combines this information with lessons learned from an analogous,
better understood phenomenon that has governed distributions of malaria and sickle cell anemia
across Africa for thousands of years. However, women with gestational Diabetes stand a risk of
suffering from Type 2 diabetes later on in their lives. Viruses are prime candidates as environmental
risk factors because they induce strong immune reactions and can infect the pancreas and. Figure 6A
was generated using protein standards with known concentrations and. By associating with
CTNNB1 (pre- viously ? -catenin) within the cell nucleus, TCF7L2 acts on Wnt target genes that
inhibit the production of a glucagon precursor in enteroendocrine cells of th e pancreas (Smith,
2006). High blood glucose may also cause fatigue, blurred vision, abdominal pain, and headaches.
Long and Short Essays on Diabetes for Students and Kids in English We are providing students with
essay samples on a long essay of 500 words and a short essay of 150 words on the topic of Diabetes
for reference. These realities, perhaps better than any other, truly embody th e intimate
coevolutionary rela- tionship existing between the ex tant Plasmodium agents of human malaria and
their hosts. Approximately 6% of siblings of people with T1D will also develop the disease, as
compared with a prevalence of.4% in the general population (Levin and Tomer 2003; Leoni 2003).
Epidemiologic studies depict that a sedentary lifestyle increases risk of diabetes but more exercise
reduces (image to the right for reference). Full Disclaimer. We are using Google Analytics to
enhance your experience. Furthermore, GAD, as compared with other autoantigens expressed by.
Insulin and glucagons are intimately but independently involved in the regulation of the blood
glucose levels. Table 1 represents the treatment matrix for lysate generation. Kong J, Li YC.
Molecular mechanism of 1,25-dihydroxyvitamin D3 inhibition of adipogenesis. Footer Picture
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Privacy Policy Area Volume Calculator. This condition is caused due to the body’s resistance to the
insulin present in the bloodstream and the pancreas’ consequent inability to produce enough insulin
to overcome this. Modern Science has allowed type 1 diabetes victims another lease on life when
alternative medical solutions were developed. Figures 5A-5H clearly show elongated mammalian
cells with no evidence of bacteria or. T1DM is increasing in incidence globally at a rate of about 3%
per year (Champe et al., 2005). Like all types of DM, T1DM is associated with increased risk for and
a high incidence of certain complications. This disease includes high glucose levels in the blood. As
an adaptive mechanism, HapA is still being selected for in certain agricultural environments despite
its positive association with type 2 risk because of the advantages in energy storage and metabolism
of higher BMI. Scientists nowadays are still looking into the factors which may have caused insulin
resistance. With this role in blood sugar homeostas is, it is easy to appreciate the subtle, yet powerful
physiological relation existing between TCF7L2 and type 2 diabetes. In fact, evolution and natural
selection walk a very fine line. You may not alter or remove any trademark, copyright or other notice
from copies of the content. Among them is an array of single nu- cleotide polymorphisms
(SNPs)—single base pair substitutions created along the coding sequence. Notice the increasing
overlay as the Multiz align- ment transcends from the Stickleback and other out-groups to the Rhesus
monkey. Age of proband diagnosis also seems to be a crucial factor: the concordance rate for twins
of probands diagnosed after age 24 was only 6%, whereas the co-twin of a proband diagnosed
before age 5 has a 65% risk of developing diabetes.
The World Health Organization (WHO) estimates that there are over 177 million cases worldwide
and this number will increase to at least 370 million by 2030 (Gad et al., 2003). Further, the disease
is associated with a series of secondary health complications. Insulin helps glucose get into your
body's cells and that is how your body gets the energy it needs. Short Essay on Diabetes 150 Words
in English Short Essay on Diabetes is usually given to classes 1, 2, 3, 4, 5, and 6. This swift
transition occurs in type 1 diabetes when. This different form of behavioral change has shown
equally capable of influencing genetic change, and the persistence of type 2 diabetes. Instead, the
latter variant contributes to T2D risk by af- fecting insulin secretion in the ways previously described
(Hel- gason et al., 2007). These researchers were respectful and thor- oughly sensitive to the genetic
nature and variability of their subject matter. Despite the age and usage of archaic terminology for
describing certain popula- tions in Zimmet’s article, there are many lessons that can be learned from
it concerning research fallacies therein—many of which are still employed today. Within the liver,
glucagon activates the breakdown of glycogen into glucose and its release into the bloodstream,
therefore serving to increase blood sugar and directly antagonize insulin. The causes of diabetes
depend on the type, with type 1 believed to involve genetic susceptibility triggered by viral
infections, while type 2 is caused by a combination of genetic and lifestyle factors such as obesity
and lack of exercise. Figure 6A was generated using protein standards with known concentrations
and. The number of overweight people is projected to increase from 1.3 billion in 2005 to nearly 2
billion by 2030. Apart from medications, Diabetes can be kept in check by maintaining a healthy
diet, proper exercise, optimum sleep and water consumption, and abstaining from tobacco, alcohol,
and junk food consumption. Both the methodology executed and conclusions posited in Helgason et
al. (2007) put forth creative techniques and viewpoints in studying T2D, in turn adding another
wrinkle of understanding to the disorder’s complex epidemiology. Let us write or edit the research
paper on your topic. This causal relationship is similar in principle to the ways in which agriculture
has gov- erned the frequencies and distribu tion of malaria and sickle cell trait across the African
continent. They start the ad showing an older person hurting looking very miserable. This sort of
terminology has been used as a sort of umbrella term—a catch all—that can be negligent to the
phenomena at hand and a deterrent to deeper research. One must remember that diet, along with
subsistence, infectious disease, and other sociocul- tural and environmental filters must be given the
attention they deserve in the study of any worldly distributed disorder (Jackson, 2004).
Epidemiologic studies depict that a sedentary lifestyle increases risk of diabetes but more exercise
reduces (image to the right for reference). Transcription factor 7-like 2 gene (TCF7L2 gene), also
called TCF4 gene, consti- tutes a 216 kilobase (kb) region on chromosome 10 within the human
genome. Appoximitely 3000 people have been used in this study and 85% of the original group study
continued on in further studies. Report this Document Download now Save Save Introduction to
Diabetes For Later 100% (1) 100% found this document useful (1 vote) 156 views 9 pages
Introduction To Diabetes Uploaded by Sameer Saurabh AI-enhanced title and description Diabetes
mellitus, or simply diabetes, is a group of metabolic diseases characterized by high blood sugar levels.
An open and incorporative mindset such as this can revitalize the research field. The persistent, and
in some cases, growing frequencies of these variants in studied popula- tions is often the result of
balancing selection dictated by the pleiotropic reality of TCF7L2 gene and changes in human en-
vironments and behavior. They face many complications that can lead to blindness, kidney damage,
heart disease and lower-limb amputation. Epidemiological Overview Type 2 diabetes (T2D) has
become a part of the human con- dition; it is a global phenome non with a distribution that reaches
across the entire spectrum of human populations. In 1982, P. Zimmet, an Australian-born
diabetologist, offered “An Epidemiological Overview of type 2 (non-insulin-dependent) diabetes”.
Modern Science has allowed type 1 diabetes victims another lease on life when alternative medical
solutions were developed. However, women with gestational Diabetes stand a risk of suffering from
Type 2 diabetes later on in their lives. This condition is caused due to the body’s resistance to the
insulin present in the bloodstream and the pancreas’ consequent inability to produce enough insulin
to overcome this. A decrease in glomerular filtration rate indicates the development of renal disease,
and early identification of this event is important in subjects with type 2 diabetes 1, 2.