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You are in emergency department when an unconscious patient with BP : 90/50 mmHg Pulse : 92 bpm How will you approach ?
APPROACH
ABC Immediate management Examination History Investigations
Immediate management
Assessment of the patient's : Airway Breathing Circulation inspect the tongue for signs of biting supplemental O2 Take a set of vitals at least every 15 minutes
Two large bore IV accessone IV for fluids and another for medications.
Examination
Examination
Pulse
Tachycardia Hypovolemia/haemorrhage Intoxication hyperthermia Bradycardia Raised intracranial pressure Heart blocks
Temperature
Increased Sepsis Meningitis ,encephalitis Malaria ,Pontine haemorrhage Decreased Alcohol,barbiturate or phenothiazine intoxication Hypoglycemia Myxedema
Blood pressure
Increased Hypertensive encephalopathy Cerebral haemorrhage Raised intracranial pressure Decreased Hypovolemia Myocardial infarction Intoxication/poisoning Profound hypothyroidism/hyperthyroidism, Addisonian crisis
Respiratory rate
Increased Pneumonia Acidosis (DKA, renal failure) Pulmonary embolism Respiratory failure Decreased Intoxication/poisoning Brain tumours/ increase ICP
Endocarditis
Neurological assessment
General posture
Level of conciousness
Posture
Lack of movements on one side Intermittent twitching
Multifocal myoclonus
DECORTICATION DECEREBRATION
A system for assessing the depth of unconsciousness AVPU A alert V respond to voice stimulus P respond to pain U - unresponsive
Level of conciousness
Brainstem reflexes
Spontaneous and elicited eye movements Pupillary responses to light Corneal responses Respiratory movements
Ocular movements
Downward conjugate deviation of eyes Eyes turn down and inward in Dysconjugate ocular deviation Ocular bobbing Ocular dipping Oculovestibulo responses
Pupillary changes
Sr no 1 pupils B/L Pin-point pupils ( less than 1mm) but responsive B/L small pupils but responsive causes Opiates poisoning ,extensive pontine hemorrhage B/L diencephalon involvement or destructive pontine leison Metabolic encephalopathies ,deep B/L hemisphere lesion or thalamic hgr.
Sr. no.
Pupil
cause
Horner syndrome
Ipsilateral dilated pupil with no Compression of 3rd cranial direct or consensual reflexes nerve e.g, uncal herniation
Respiratory movements
Cheyne-stokes respiration Kussmaul breathing Irregular respiration with random deep & shallow breaths Prolonged inspiratory gasp with a pause at full inspiration
Neck rigidity
Meningitis
Subarachnoid haemorrhage
Fundoscopy
Raised intracranial pressure Hypertensive changes Subarachnoid haemorrhage
Diabetic retinopathy
History
Onset of the symptoms Antecedent symptoms Use of medications Chronic liver ,kidney ,lung or heart disease
Possible Causes
Alcohol Epilepsy Trauma Infection Psychiatric Stroke, syncope
Insulin
Overdose Uremia (and other metabolic causes)
Causes of unconsciousness
Focal signs
Trauma Intracranial bleed Cerebral contusion
Vascular SAH Stroke Space occupying lesion Tumour Abscess
Diffuse signs
Infection Metabolic/Endocrine
Hyper/hypoglycaemia Hypothyroidism Acid-base disturbance Hyper/hyponatraemia Hepatic encephalopathy addisonion crisis
Toxic
Alcohol Recreational drugs Overdose
Hypothermia /Hyperthermia
Immediate investigations
RBS ESR LFTs Urea and Creatnine Blood and urine cultures
Other investigations
CRP ABGs Urine Ketone Toxic screen , drug levels