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Spinal shock was first defined by Whytt in 1750
as a loss of sensation accompanied by motor
paralysis with initial loss but gradual recovery
of reflexes, following a spinal cord injury (SCI) --
most often a complete transection.
Reflexes in the spinal cord caudal to the SCI are
depressed (hyporeflexia) or absent (areflexia).
Note that the 'shock' in spinal shock does not refer
to circulatory collapse
Sumber Ditunno JF, Little JW, Tessler A, Burns AS. Spinal shock revisited: a
four-phase model. Spinal Cord. 2004 Jul;42(7):383-95.
Physiological loss of spinal cord function distal
to level of injury
• Flaccid paralysis
• Anesthesia
• Incontinence
• Loss of reflex activity
• Priapism
In spinal cord injuries above T6, autonomic
dysreflexia may occur, from the loss of autonomic
innervation from the brain. Sacral
parasympathetics (S2-S4) are lost, as are many
sympathetic levels, depending on the level of the
SCI. Cervical lesions cause total loss of
sympathetic innervation and lead to vasovagal
hypotension and
bradyarrythmias -- which resolve in 3-6 weeks.
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