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K - 11 Cedera Kepala (Bedah Neurologi)
K - 11 Cedera Kepala (Bedah Neurologi)
KULIT
TULANG TENGKORAK
DURAMATER
ARACHNOID
PIAMATER
PEMERIKSAAN CEDERA KEPALA DI UGD
PRIMARY SURVEY
Airway
Breathing
Circulation
Disability
Exposure
SECONDARY SURVEY
HIPOKSIA
HIPERKARBIA
HIPOTENSI
HIPOVOLEMIA
GELISAH
HYPOXIA
RETENTION URINE
NYERI
PROSES INTRACRANIAL
Intracranial contents,
Brain (including the neurological elements [70%] and
interstitial fluid [10%] ) ;
volume 1400 ml or 80%
Blood (arterial and venous) ;
volume 150 ml or 10%
Cerebrospinal fluid (CSF) ;
volume 150 ml or 10%
PEMERIKSAAN NEUROLOGIS
TINGKAT KESADARAN
PUPIL DAN GERAKAN BOLA MATA
REAKSI TERHADAP RANGSANG DARI LUAR
REAKSI MOTORIK
POLA PERNAFASAN
SINDROMA HERNIASI
BRAIN DEATH
GLASGOW COMA SCALE
• Eye Response. (4) Motor Response. (6)
1. No eye opening 1. No motor response
2. Eye opening to pain 2. Extension to pain
3. Eye opening to verbal command 3. Flexion to pain
4. Eye open spontaneously 4. Withdrawal from pain
5. Localizing pain
• Verbal Response. (5) 6. Obey Commands
1. No verbal response
2. Incomprehensible sounds Klasifikasi
3. Inappropriate words Mild 14 - 15
4. Confused Moderate 9 - 13
5. Orientated Severe 3-8
SYMPTOMS AND SIGNS
Headache, worse at night or recumbent position,
because of the increase in CO2 tension and increased
venous pressure
Nausea and vomiting
Ataxia, papilledema, and cranial nerve paralysis
Irregular breathing patterns
Decorticate or decerebrate
Pupillary inequality
PEMERIKSAAN RADIOLOGIS
FRAKTUR LINEAR
FRAKTUR DIASTASE
FRAKTUR COMMUNITED
FRAKTUR DEPRESSED
FRAKTUR KONVEKSITAS / KUBAH
FRAKTUR BASIS CRANII
ANTERIOR - ANOSMIA, RHINORRHOE
MEDIA - OTORRHEA, HEMATYMPANI, BATTLE’S SIGN
POSTERIOR - INFRA TENTORIAL
TANDA FRAKTUR BASIS KRANII
HAEMOTYMPANUM
OTORRHEA
RHINORRHEA
RACOON EYES
BATTLE’S SIGN
CEREBRAL EDEMA
VASOGENIC EDEMA
Increased permiability of capillaries ; the tight junctions
between the endothelial cell become incompetent,
allowing plasma filtrate to escape into the intercellular
space
Contrast enhancement because of the breakdown of the
BBB
Edema is more marked in white matter than in gray
matter
Edema is seen with trauma, tumor, and abscess
CYTOTOXIC EDEMA
Hypoxia of the neural tissue and water intoxication
Hypoxia affects the ATP-dependent sodium pump
mechanism in the cell membrane, promoting an
accumulation of intracellular sodium and subsequent
flow of water into cell to maintain osmotic equilibrium
Edema is intracellular and affects all cells : endothelial
cells, astrocytes, and neurons (interstitial space is
narrowed)
Subtle or no changes in CT scan, indicative in early
phases of ischemic stroke
INTERSTITIAL EDEMA
Transudation of CSF in obstructive hydrocephalus
Best observed on CT or MRI as periventricular low
density areas because of the retrograde transependymal
flow of CSF into the interstitial space of the white
matter (mostly in frontal region) , indicates active
hydrocephalus requiring surgical therapy
TYPES OF BRAIN HERNIATION
CINGULATE HERNIATION
Focal mass lesion in the supratentorial compartement
pressure locally on the ipsilateral hemisphere
The mass lesion may displace the cingulate gyrus, which
is next to the free edge of the falx cerebri, and cause it to
herniate under the falx to the opposite side
Usually displacement of the ventricular system
Arterior cerebral artery, tight, sharp edge of the falx
No clinical signs and symptoms specific
UNCAL HERNIATION
When lesions of the middle cranial fossa, such as acute
epidural hematoma, subdural hematoma, temporal lobe
contussions, or temporal lobe neoplasms
An expansile mass of the middle fossa cause the uncus,
the inferomedial structure of the temporal lobe, to
herniate between the rostral brainstem and tentorial
edge into posterior fossa
The medial displacement of the brainstem may cause
compression of the brainstem againts the opposite
tentorial edge, producing a notch called Kernohan’s
notch (ipsilateral hemiplegia)
CENTRAL TRANSTENTORIAL HERNIATION
Mass lesions located to the tentorial hiatus
Bilateral mass lesions, such as bilateral subdural
hematomas, can also cause herniation
Downward displacement of the diencephalon and
midbrain centrally through the tentorial incisura
Clinical symptom,
Bilaterally small, reactive pupils
Cheyne-Stokes respirations
Loss of vertical gaze
TONSILLAR HERNIATION
The tonsil of the cerebellum herniates through the
foramen magnum into the upper spinal canal,
compressing the medulla
Manifestations of acute medullary compression are,
Cardiorespiratory impairment
Hypertension
High pulse pressure
Cheyne-Stokes respirations
Neurogenic hyperventilation
Impaired consciousness
Stiff neck or opisthotonic position
Decorticate or decerebrate posturing
INDIKASI OPERASI
Epidural Hematoma (EDH)
- EDH >30 ml
- EDH,Koma,GCS <9, pupil anisokor
- Bila EDH <30 ml dan ketebalan <15 mm
serta midline shift <5 mm dan GCS >8 tanpa
fokal defisit ------ tidak operasi
Akut Subdural Hematoma (SDH)
- ketebalan >10 mm atau midline shift >5 mm
pada CT Scan
- koma (GCS<9), ketebalan <10 mm dan
midline shift <5 mm,operasi bila GCS
menurun 2 atau lebih (waktu antara kejadian
dan masuk RS) atau ICP >20 mmHg
- koma (GCS<9) ----- monitoring ICP
Traumatic parenchymal lesions
- GCS 6-8 dengan kontusi frontal atau
temporal <20 ml, midline shift >5 mm
dan kompresi sisterna pada CT Scan
- lesi >50 ml