Professional Documents
Culture Documents
Lesson
Shock & Fluid
6 Resuscitation
Developed by the
National Association of
Emergency Medical Technicians
In cooperation with
The Committee on Trauma,
American College of Surgeons
6-2
Shock:
A rude unhinging of the
machinery of life.
6-3
Shock Redefined:
6-4
Scenario
You are caring for the sole victim of a
motorcycle crash. He is lying on the
ground next to his motorcycle. The
scene appears safe.
6-5
Findings
A - Open, clear.
B - RR fast. BS clear. Bruising noted
over sternum; no other chest
deformity.
C - Radial pulse weak & rapid.
D - PEARL. Anxious & confused.
Normal movement & sensation.
E - No deformity noted. Skin cool &
diaphoretic.
6-6
Discussion
Is this patient in shock?
Why?
6-7
Cell Perfusion
6-8
Anaerobic Metabolism
Inadequate oxygenation
for metabolism.
By-products:
Less energy.
More acid.
Potassium.
6-9
Leads to Acidosis
• Inadequate oxygen delivery, cellular
extraction, and consumption result in
cellular conversion to anaerobic
metabolism for energy substrate
production (ATP production)
Tissue injury
Death
Cellular Death
SHOCK
Cellular Hypoxia - Anaerobic
Metabolism
Hypotension
Hypoperfusion
Cellular Hypoxia
Anaerobic Metabolism
If cellular death is not
Cell Death prevented, organism
death will follow.
6-10
Staged Death
Organism death:
May be quick - prior to EMS.
May be prolonged - 2 to 3 weeks later.
Occurs in stages:
Stages occur as the body tries to
compensate.
We see the stages through signs &
symptoms.
6-11
What Organs Fail?
Combination of respiratory
failure (ARDS) with renal
failure, metabolic failure,
or cardiac failure
– Poor prognosis
Mortality rate
– One organ/systems 40%
– Two organ/systems 60%
– Four or more 100%
Prevent Cellular Death
Recognize shock early.
Restore cellular perfusion.
Restore aerobic metabolism.
6-12
3 Phases of Shock
Compensatory
Progressive
Irreversible
Early Recognition
Signs & Symptoms of Uncompensated Shock
6-13
Early vs. Late
What are the earliest signs of
shock?
6-14
Restore Cellular Perfusion
6-15
Chemical Balance continued...
Decrease in oxygen.
Detected by chemical receptors in the carotid
arteries and in the arch of the aorta.
Respirations increase in rate & depth.
Rise in acidity.
Buffer system converts acid to CO2 & water.
Medulla senses increased CO2.
Respirations increase; lungs “blow off” CO2.
6-16
Common Shock Pathways
Mechanisms Compensation
Oxygen deficiency ↑ Catecholamines
Cellular hypoxia ↑ HR, contractility
Ischemia Vasoconstriction of
Anoxia both arterial and
venous beds
– ↑ oxygen delivery
↑ Cellular extraction
of oxygen
Chemical Balance cont’d.
6-18
Fluid Decrease continued...
Hormonal response:
Various hormones are released,
causing vasoconstriction and fluid
retention.
Compensation:
Blood vessels constrict.
Heart increases strength & rate.
Fluid is retained.
6-19
Fluid Decrease cont’d.
6-22
Scenario
You are called to the scene of a house
fire, where firefighters have just removed
one patient. He is unresponsive. Pulse
102, RR 6, and BP 104/76.
6-23
Scenario
You respond to a scene where a skier has
struck a tree at high speed. Exam reveals pain
on palpation of the right lateral chest, absent
breath sounds on the right and labored
breathing. Pulse 142, RR 40, and BP 88/70.
6-24
Scenario
You are dispatched to an MVC. On arrival, you
find a 47-year-old male driver still in the
vehicle. There are 10-15 inches of intrusion to
the front of the vehicle. The patient is
unresponsive as you approach.
6-25
Findings
A - Blood & vomitus in airway.
B - Slow & labored. BS absent on
left.
C - No palpable radial pulse. Cool,
wet skin. No external bleeding.
D - Pupils equal but reacting slowly.
E - Obvious fracture of left femur.
Shock is staged
death...
…catch it in the first act!
6-27