Professional Documents
Culture Documents
• Retained placenta
• Amniotic Fluid Embolism
Dr Sushma Sharma
Professor
Department of Obstetrics & Gynaecology
MIMER Medical College, Talegaon(D), Pune
Uterine Inversion
Dr
Introduction
• Uterine inversion occurs when the uterine fundus collapses into the
endometrial cavity, turning the uterus inside out.
• A rare complication of vaginal delivery /cesarean
• Life threatening obstetric emergency
• Needs prompt recognition and management
• Less than 5% maybe spontaneous
Classification
By the extent of inversion:
• First degree (Incomplete) – The fundus
is within the endometrial cavity
• Second Degree (Complete) – The
fundus protrudes through the os.
• Third Degree (Prolapsed) – The fundus
protrudes into or beyond the introitus
• Fourth Degree (Total) – Both the uterus
and vagina inverted
Classification-contd.
By time of occurrence:
• Subacute – more than 24 hours but less than four weeks post partum
Pathogenesis -
Incompletely understood.
Maybe attributed to:
• Use of excessive cord traction
• Fundal pressure
• Crede’ manoeuvre
• Fundal implantation of placenta
• Likely that other factors play a role – eg connective tissue disorders like Marfan’s
syndrome
Risk factors –
• Short cord
• Prolonged labour
• Precipitate labour
• Macrosomia
• Use of uterine relaxants
• Uterine anomalies
• Retained placenta
• Placenta accreta spectrum
Clinical Features
Depends on extent and time of occurrence. May include:
• Mild to severe vaginal bleeding
• Mild to severe lower abdominal pain
• Mass protruding from cervix/ vagina
• Urinary retention
Management
Resuscitate, IV
access, fluids/ bolus
replacement
Immediate
replacement
UTERUS
YES NO
REPLACED
Principle :
“ The part of the uterus which has come down last , should go back first. “
Procedure
Manual ⦿ If the diagnosis is made immediately after the
reposition- inversion has occurred, then that same degree of
relaxation of myometrium and cervix (which is
Johnson’s required for the inversion to occur) will allow
technique uterine replacement easily…
7. Once the fundus has been replaced keep the hand in the uterus while
rapid infusion of oxytocin is given to contract the uterus.
Initially, bimanual compression aids in control of further hemorrhage
until uterine tone is recovered.
8. When the uterus is felt contracting, the hand is slowly withdrawn.
If placenta is attached, it is to be removed only after the uterus
becomes contracted.
If the placenta is partially attached , it should be peeled out
before replacement of uterus.
O’Sullivan’s hydrostatic method
Abdominal Vaginal
Huntington ; Haultain Kustner ; Spinelli
Modifications
of the classical
abdominal techniques
Uterine Malformation
Septic Endometritis
Risk Factors :
High Parity
Advanced Maternal Age
Down Syndrome
High level of Maternal Serum AFP.
High level of Maternal free Beta HCG.
ETIOLOGY
:
Defective decidual formation :
from,
- Severe Hemorrhage
- Infection
- Inversion of Uterus
DIAGNOSI
S
Clinical suspicion
Ultrasound
Color Doppler
MRI
Biochemical Marker
Histopathology
USG
First-line investigation for suspected
placental invasion of the myometrium.
[ Chou MM, Ho ES, Lee YH. Prenatal diagnosis of placenta previa accreta by transabdominal color
Doppler ultrasound. Ultrasound Obstet Gynecol 2000;15:28–35. ]
3 D USG
Diagnostic Criteria :
Irregular intraplacental vascularization with
tortuous confluent vessels crossing placental
width.
Hypervascularity of uterine serosa– bladder
wall interface.
Colour Doppler
Diffuse or focal
intraparenchymal lacunar
flow.
Vascular lakes with
turbulent flow.
Hypervascularity of
serosa-bladder interface.
Prominent subplacental
venous complex.
M.R.I.
No more sensitive than USG , But used as an
adjunct to USG , when there is strong clinical suspicion
of accreta.
MRI achieves better images than Ultrasonography in
- Posteriorly sited MAP and
- With prior myomectomy,
preserved )
Localized Resection with uterine repair
Over sewing of the ut. Defect
Blunt dissection followed by curetting the uterine cavity
• Uterus fails to contract (Multipara) :
Hysterectomy
Non Surgical
Involve UROLOGIST.
Preoperative Ureteric
stenting aids in identifying
the ureters, which will
help reduce ureteric
injuries.
Care must be taken during
surgery not to attempt to
dissect the bladder off the
lower uterine segment
which results in torrential
bleeding.
Anterior bladder wall
incision is particularly
helpful in defining
dissection planes and the
location of the ureters.
Reality :
Even today, the ground reality is that
a majority of morbidly adherent placenta
are diagnosed during the third stage of
labour or during caesarean section and
which results in adverse consequences
including exanguinating haemorrhage.
To Conclude…
Amniotic Fluid
• Protects the unborn fetus from its surrounding environment.
• Isolated from the maternal intravascular compartment.
• When this isolation is disrupted in one way or another, a
systemic maternal reaction can ensue, affecting the heart,
lungs, and brain.
• This reaction can be lethal to both mother and fetus.
• The syndrome is know as amniotic fluid embolism (AFE) and
is one of the most serious complications of
pregnancy and delivery.
• Given the unpredictable and unexpected nature of AFE, every
obstetrician should be prepared to include this condition in
differential diagnosis to optimize the chances for survival of
both mother and infant.
Background
• The presence of fetal cellular debris in the maternal circulation,
associated with maternal complications, was reported for the first
time in the 1920s by Ricardo Meyer from Brazil.
• In an effort to better understand this condition, several animal
models have been developed.
• Studies to human beings cannot be easily made, since not all the
findings in animals are consistent with the clinical manifestations.
• Recently, the term Anaphylactoid syndrome of pregnancy has
been proposed instead of AFE.
Incidence
• The true incidence of AFE may not be known.
• The disparity between reported figures is large, with rates ranging
from 1 in 8,000 to 1 in 83,000 deliveries.
• A recent study from California reports an incidence of 1
per 20,646 deliveries.
Mortality
• AFE constitutes the leading cause of mortality during labor
and the first few postpartum hours.
• The disorder occurs during the last stages of labor when amniotic fluid
enters the circulatory system of the mother.
• Suspect AFE when confronted with any pregnant patient who has
sudden onset of - respiratory distress,
- cardiac collapse,
- seizures,
- unexplained fetal distress, and
- abnormal bleeding
• AFE is suspected when a woman giving birth experiences very
sudden insufficient oxygen to body tissues, low blood
pressure, and profuse bleeding due to defects in blood
coagulation.
• They also can be entirely absent.
Clinical Conditions Associated With A F
E
• Pharmacologic treatment
• The goals of therapy are:
(1) maintenance of systolic blood pressure above 90 mm Hg
(2) maintenance of arterial PO2 over 60 mm Hg