Professional Documents
Culture Documents
Department of Emergency
Dr. Zong
1
2
Case 1
• Male, 37y, motorcycle accident
• Transferred to the closest (40km) level 1 trauma center
• Injuries at ED: GCS 14, rib fractures (V-VIII, left), lung contusions,
left femur and bilateral tibial fracture, soft tissue contusions,
distorted splenic shape on FAST, suspected subcapsular
hematoma on CT
• Hemodinamicaly stabile, temporary immobilized
• Transferred to the specialized orthopedic hospital (80km) for
definitive repair 4 hours later
• Day 2: During preparation for surgery, patient becomes
tachycardic, tachypneic, anxious, hypoxic, complains on chest
and abdominal pain.....
Case 1
• Anesthesiologist refuses to anesthetize him and requests
CTPA as pulmonary embolism was suspected
• CT at the nearby hospital (1h later): Diffuse ground- glass
opacities, abdominal free fluid, signs of splenic rupture
How many survival
• Transfer to the Emergency center (30 mins)
chances
• Emergency center were missed?
ED: somnolent, pale, Fr 135/min, TA 75/40,
tachypneic, hypoxic, intubated, immediately transferred to
OR
• OR: Splenectomy, massive transfusion
• ICU: Severe ARDS
• Died on the 3rd day due to MOF
Case 2
• A 28-year-old woman was involved in a motor vehicle
accident (MVA) and suffered major trauma. The trauma
mechanism was a high speed MVA with head-on collision on a
motorway.
• Initial Glasgow Coma Scale (GCS) of 8.
• Complained about severe pain in her head, chest and right
leg. She was intubated, immobilized and transferred.
• After initial trauma management following the Advanced
Trauma Life Support (ATLS)-scheme, a whole body CT scan
was performed.
• Damage control concept-- stabilize open
tibial fracture (external fixator)--open
mandibular fracture surgery.
• Transferred to ICU--reassessment
showed an anisocoria (right > left).
• Reassessment--Additional diagnostics
were initiated. A CT angiography
revealed the suspicion of a bilateral
internal carotid artery (ICA) dissection.
Second CT scan with coronal
reconstructions of the bilateral
ICA dissection. “String sign”
indicated with an arrow.
• 48 h after the accident, the patient was transferred via
helicopter non-intubated to the emergency department of
our level I trauma center.
• GCS of 8 under sufentanil analgesia
• Transferred to the ICU. High-dose intravenous
heparinization was started immediately (PTT 60–80 s).
• MRI on the next day (3 days after trauma) revealed
bihemispheric, mainly left-sided, ischemic lesions near the
border zone.
• The high-dose heparinization was continued.
8
Axial T-2 sequences of a brain MRI scan of the 28-year-old patient. Left
posttraumatic bihemispheric, mainly left-sided, ischemic lesions near the
border zone, 3 days after trauma (indicated with arrows). Right 6 months
follow-up MRI scan with good recovery. 9
• 8 days after the accident and ICU treatment the patient was
referred to the stroke unit with a score of 8 on the National
Institutes of Health Stroke Scale(NIHSS, min. 0 points = no
neurological deficit, max.42 points = worst outcome).
• proximal tibial fracture was treated with osteosynthesis and the
intravenous heparinization was converted to oral
anticoagulation
• The patient was discharged to rehabilitation 22 days after the
trauma with a NIHSS of four points.
• 6 months later, besides slight concentration difficulties and fine
motor dysfunctions, no neurological deficiencies remained
(NIHSS = 0). The MRI scan showed good recovery.
10
Axial T-2 sequences of a brain MRI Angio scan of the 28-year-old
patient. Left posttraumatic intracranial dissection with intramural
hematomas of both internal carotid arteries 3 days after trauma (indicated
with arrows). Right 6 months follow-up MRI scan with good perfusion
of both internal carotid arteries.
11
Epidemiology of Trauma
• Leading cause of death in ages 1-44
• 3rd leading cause of death for all ages
• 100,000 deaths/year
• 60 million injuries/year
Epidemiology
Road Traffic Accidents are major cause of long term morbidity and
mortality in developing nations
– In the first quarter of 2009, 372 deaths in Ghana from Road Traffic
Accidents
– 25% increase from previous year
WHO predicts that by 2020, Road Traffic Accidents will be second
leading cause of loss of life for world’s population
High Morbidity = Loss of income to society
Challenges in Developing Countries
– Technological Advances in Trauma Care
– Lack of Infrastructure for Trauma Management
EMS
Pre-hospital notification
MD/RN Training in trauma care
13
Epidemiology
Trimodal Distribution of Trauma Deaths
Immediate death
aortic rupture, lacerations of the brain stem, decapitating injuries
Early death ( golden hour )
tension pneumothorax, hemorrhagic shock ( intraabdominal or
intrathoracic bleeding , pelvic ring disruptions with massive
retroperitoneal hemorrhage, or due to severe traumatic brain injury with
acute cerebral edema or intracranial hematoma )
Late death—days to wks
septic complications, multiple organ failure and due to untreatable increased
ICP associated with cerebral edema.
14
aortic rupture
lacerations of the brain stem
Decapitation/Internal
Decapitation
tension pneumothorax
hemorrhagic shock
pelvic ring disruptions
Traumatic brain injury(TBI)
Epidemiology
Trimodal Distribution of Trauma Deaths
30%
20%
22
Trauma topics
Definition
1665 reports ( 1995-2008 ) A consensus yet to be
47 definitions established
25
Pathophysiological change
• Cardiovascular : hypodynamic flow, the
hyperdynamic flow, and the recovery
• Immune system : systemic inflammatory response
syndrome (SIRS) and the counter anti-inflammatory
response syndrome (CARS)
Anti-inflammatory Cytokines
29
Trauma-induced
systemic inflammatory
response syndrome
(SIRS) and
complications
Host response to polytrauma
Ischemia/Reperfusion Injury
Haber-Weiss
Fe (Ⅲ)+ O2-→fe(Ⅱ) + O2
Fe(Ⅱ)H2O2 →Fe(Ⅲ)+OH-+OH·
What really matters
34
Acidosis
• Poor tissue perfusion is the main contributor in trauma patients
• Decreased cardiac output, hypoxia and anemia lead toward
cellular anaerobic metabolism and cause lactic acid accumulation
• Resuscitation with normal saline induces hyperchloremic acidosis
• Acidosis diminishes cardiac output leading to worse tissue
perfusion
• Aggravates coagulopathy
PH drop from 7.4 to 7.0 reduces the effectiveness of coagulation cascade by 50-
75%
Procoagulant drugs (rFVII) cannot work in acidotic environment
35
Hypothermia
• The greatest contributor to hypothermia are environmental
temperature, cold crystalloids and PRBCs
• Tissue hypoperfusion and anaerobic metabolism exhaust ATP
which is required for maintenance of normothermia.
• Hypothermia causes coagulopathies:
Coagulation cascade is temperature dependent
Relative thrombocyopenia by plateled sequestration and dysfunction
• Induces shivering with further depletion of ATP and
progression of acidosis
36
Coagulopathy
• Present at admission in 25% of trauma patients
• 4 fold increase in mortality
Endotoxin
neutrophils ECs TF
cytokines macrophages
( TF ) activate FX to FXa
FVIIa
converts prothrombin to thrombin
(FIIa)
FVII
activates FV, FVIII, and FXI thrombin formation
Fibrin clot
Surgical control of bleeding is unlikely to be successful!
History of Trauma System Development
Standardized Trauma Assessment
– Nebraska Cornfield, 1976
– Orthopedic Surgeon
– Lead to development of ATLS
Trauma Systems Development
– First developed my military in wartime
i.e. MASH Units Otisarchives1 (flickr)
38
Mechanisms of Injury
Blunt Trauma
– Compression Forces
Cells in tissues are compressed and crushed
E.g. Spleen
– Shear Forces
Acceleration/Deceleration Injury
E.g. Aorta
– Shearing force = Spectrum from Full thickness tear
(Exsanguination) to Partial tear (Pseudoaneurysm)
– Overpressure
Body cavity compressed at a rate faster than the tissue
around it, resulting in rupture of the closed space
E.g. Plastic bag
E.g. in trauma = diaphragmatic rupture, bladder injury
39
Mechanisms of Injury
Frontal Impact Collisions
Lateral Impact Collisions (T bone)
Rear Impact Collisions
Rollover Mechanism Nico.se (flickr)
41
Preparation for Patient Arrival
Organize Trauma
Response Team
Mild : 13-15
Moderate : 9-12
Severe : 3-8
CRAMS
Abbreviated Injury Scale(AIS)
An example of the ISS calculation is shown below:
Region Injury AIS Square
Description Top Three
Face No Injury 0
Gets to
53
Primary Survey
Key Principles
– When you find a problem during the
primary survey, FIX IT.
– If the patient gets worse, restart from the
beginning of the primary survey
– Some critical patients in the Emergency
Department may not progress beyond
the primary survey
54
Airway and Protection of Spinal Cord
Why first in the algorithm?
– Loss of airway can result in death in < 3 minutes
– Prolonged hypoxia = Inadequate perfusion, End-organ damage
Airway Assessment
– Vital Signs = RR, O2 sat
– Mental Status = Agitation, Somnolent, Coma
– Airway Patency = Secretions, Stridor, Obstruction
– Traumatic Injury above the clavicles
– Ventilation Status = Accessory muscle use, Retractions, Wheezing
Clinical Pearls
– Patients who are speaking normally generally do not have a need
for immediate airway management
– Hoarse or weak voice may indicate a subtle tracheal or laryngeal
injury
– Noisy respirations frequently indicates an obstructed respiratory
pattern
55
Airway Interventions
Maintenance of Airway Patency
– Suction of Secretions
– Chin Lift/Jaw thrust Dept. of the Army, Wikimedia Commons
– Nasopharyngeal Airway
– Definitive Airway
Airway Support
– Oxygen
– NRBM (100%) Ignis, Wikimedia Commons
– Bag Valve Mask
– Definitive Airway
Definitive Airway
– Endotracheal Intubation
In-line cervical stabilization
– Surgical Crichothyroidotomy
U.S. Navy photo by Photographer's
Mate 2nd Class Timothy Smith,
Wikimedia Commons 56
Protection of Spinal Cord
General Principle: Protect the entire spinal cord until injury has been
excluded by radiography or clinical physical exam in patients with
potential spinal cord injury.
Spinal Protection
– Rigid Cervical Spinal Collar = Cervical Spine
– Long rigid spinal board or immobilization on flat surface such as
stretcher = T/L Spine
Etiology of Spinal Cord Injury (U.S.)
– Road Traffic Accidents (47%)
– High energy falls (23%)
Clinical Pearls
– Treatment (Immobilization) before diagnosis
– Return head to neutral position
– Do not apply traction
– Diagnosis of spinal cord injury should not precede resuscitation
– Motor vehicle crashes and falls are most commonly associated with
spinal cord injuries
– Main focus = Prevention of further injury
57
C-spine Immobilization
Return head to neutral position
Maintain in-line stabilization
Correct size collar application
Blocks/tape
Sandbags
Paladinsf (flickr)
59
Breathing and Ventilation
Identify Life Threatening Injuries
– Tension Pneumothorax
Air trapping in the pleural space
between the lung and chest wall
Sufficient pressure builds up and
pressure to compress the lungs and
shift the mediastinum
Delldot (wikimedia)
Physical exam
– Absent breath sounds
– Air hunger
– Distended neck veins
– Tracheal shift
Treatment
– Needle Decompression
2nd Intercostal space, Midclavicular line
– Tube Thoracostomy
5th Intercostal space, Anterior axillary line
Author unknown,
www.meddean.luc.edu/lumenMedEd/medicine/pulmonar/cxr/pneumo1.ht
m 60
Breathing and Ventilation
Hemothorax
– Blood collecting in the pleural space and is
common after penetrating and blunt chest
trauma
– Source of bleeding = Lung, Chest wall
(intercostal arteries), heart, great vessels
(Aorta), Diaphragm
– Physical Exam
Absent or diminished breath sounds
Dullness to percussion over chest
Author unknown, Hemodynamic instability
http://www.trauma.org/index.php/mai
n/images/C11/
– Treatment = Large Caliber Tube Thoracostomy
10-20% of cases will require Thoracostomy for control of bleeding
61
Breathing and Ventilation
Flail Chest
– Direct injury to the chest resulting in an
unstable segment of the chest wall that moves
separately from remainder of thoracic cage
– Typically results from two or more fractures on
2 or more ribs
– Typically accompanied by a pulmonary
contusion
– Physical exam = paradoxical movement of chest
segment
http://images1.clinicaltools.com/images/trauma/
flail_chest_wounded.gif – Treatment = improve abnormalities in gas
exchange
Early intubation for patients with respiratory
distress
Avoidance of overaggressive fluid resuscitation
Author unknown,
http://www.surgical-tutor.org.uk/default-home.htm 62
?specialities/cardiothoracic/chest_trauma.htm~right
Breathing and Ventilation
Open Pneumothorax
– Sucking Chest Wound
– Large defect of chest wall
Leads to rapid equilibration of
Author unknown, atmospheric and intrathoracic
http://www.trauma.org/index.php/main/image/
902/
pressure
Impairs oxygenation and ventilation
– Initial Treatment
Three sided occlusive dressing
Provides a flutter valve effect
Chest tube placement remote to site
of wound
Avoid complete dressing, will create a
tension pneumothorax
Middle and bottom images:
Author unknown,
http://www.brooksidepress.org/Products/Ope
rationalMedicine/DATA/operationalmed/Pro 63
cedures/TreataSuckingChestWound.htm
Needle Thoracostomy
Needle Thoracostomy
– Midclavicular line
– 14 gauge angiocath
– Over the 2nd rib
– Rush of air is heard
Author unknown,
www.trauma.org/index.php/main/article
/199/index.php?main/image/95/
64
Tube Thoracostomy
Insertion site
– 5th intercostal space,
– Anterior axillary line
Sterile prep, anesthesia with lidocaine
2-3 cm incision along rib margin with #10 blade
Dissect through subcutaneous tissues to rib margin
Puncture the pleura over the rib
Advance chest tube with clamp and direct posteriorly and
apically
Observe for fogging of chest tube, blood output
Suture the tube in place
Complications of Chest Tube Placement
– Injury to intercostal nerve, artery, vein
Author unknown, – Injury to lung
http://www.trauma.org/images/image_lib – Injury to mediastinum
rary/chest0051a.jpg – Infection
– Allergic reaction to lidocaine
– Inappropriate placement of chest tube
65
Shock
Circulation
– Impaired tissue perfusion
– Tissue oxygenation is inadequate to meet metabolic demand
– Prolonged shock state leads to multi-organ system failure and cell
death
Clinical Signs of Shock
– Altered mental status
– Tachycardia (HR > 100) = Most common sign
– Arterial Hypotension (SBP < 120)
Femoral Pulse – SBP > 80
Radial Pulse – SBP > 90
Carotid Pulse – SBP > 60
– Inadequate Tissue Perfusion
Pale skin color
Cool clammy skin
Delayed cap refill (> 3 seconds)
Altered LOC
Decreased Urine Output (UOP < 0.5 mL/kg/hr)
66
Circulation
Types of Shock in Trauma
– Hemorrhagic
Assume hemorrhagic shock in all trauma patients until proven
otherwise
Results from Internal or External Bleeding
– Obstructive
Cardiac Tamponade
Tension Pneumothorax
– Neurogenic
Spinal Cord injury
Sources of Bleeding
– Chest
– Abdomen
– Pelvis
– Bilateral Femur Fractures
67
Circulation
Emergency Nursing Treatment
– Two Large IV Lines
– Cardiac Monitor
– Blood Pressure Monitoring
General Treatment Principles
– Stop the bleeding
Apply direct pressure
Temporarily close scalp lacerations
– Close open-book pelvic fractures
Abdominal pelvic binder/bed sheet
– Restore circulating volume
Crystalloid Resuscitation (2L)
Administer Blood Products
– Immobilize fractures
Responders vs. Nonresponders
– Transient response to volume resuscitation = sign of ongoing blood loss
– Non-responders = consider other source for shock state or operating room
for control of massive hemorrhage
68
Circulation
Pericardial Tamponade
– Pericardium or sac around heart fills with
blood due to penetrating or blunt injury to
Pericardium
chest
Blood – Beck’s Triad
Distended jugular veins
Hypotension
He
– Treatment
Epicardium Rapid evacuation of pericardial space
Performed through a pericardiocentesis
(temporizing measure)
Aceofhearts1968(Wikimedia) Open thoracotomy
69
Pericardiocentesis
Puncture the skin 1-2 cm inferior to xiphoid process
45/45/45 degree angle
Advance needle to tip of left scapula
Withdraw on needle during advance of needle
Preferable under ultrasound guidance or EKG lead V
attachment
Complications
– Aspiration of ventricular blood
– Laceration of coronary arteries, veins,
epicardium/myocardium
Author unknown,
http://www.trauma.org/images/image_library/ch – Cardiac arrhythmia
est0054_thumb.jpg – Pneumothorax
– Puncture of esophagus
– Puncture of peritoneum
Author unknown,
70
www.brooksidepress.org/ProductsTrauma_Surgery?M=A
Circulation
A word about cardiac arrest . . .
– Care of the trauma patient in
cardiac arrest
CPR
Bilateral Tube Thoracostomy
Pericardiocentesis
Volume Resuscitation
– Traumatic cardiac arrest due to
blunt injury has very low survival
rate (< 1%)
No point for emergency thoracotomy
– Selected cases of cardiac arrest due
Author unknown,
to penetrating traumatic injury may
http://www.trauma.org/images/image_library/chest0 benefit from emergent
046.jpg
thoracotomy
Pericardial tamponade
Cross clamp aorta
71
Disability
Baseline Neurologic Exam
– Pupillary Exam
Dilated pupil – suggests transtentorial herniation on ipsilateral side
– AVPU Scale
Alert
Responds to verbal stimulation
Responds to pain
Unresponsive
– Gross Neurological Exam – Extremity Movement
Equal and symmetric
Normal gross sensation
– Glasgow Coma Scale: 3-15
– Rectal Exam
Normal Rectal Tone
Note: If intubation prior to neuro assessment, consider quick
neuro assessment to determine degree of injury
72
Glasgow Coma Scale
Disability GCS ≤ 8
Intubate
– Eye
Spontaneously opens 4
To verbal command 3
To pain 2
No response 1
– Best Motor Response
Obeys verbal commands 6
Localizes to pain 5
Withdraws from pain 4
Flexion to pain (Decorticate Posturing) 3
Extension to pain (Decerebrate Posturing) 2
No response 1
– Verbal Response
Oriented/Conversant 5
Disoriented/Confused 4
Inappropriate words 3
Incomprehensible words 2
No response 1
73
Disability
Key Principles
– Precise diagnosis is not necessary at this point in
evaluation
– Prevention of further injury and identification of
neurologic injury is the goal
– Decreased level of consciousness = Head injury until
proven otherwise
– Maintenance of adequate cerebral perfusion is key
to prevention of further brain injury
Adequate oxygenation
Avoid hypotension
– Involve neurosurgeon early for clear intracranial
lesions
74
Disability
Cervical Spinal Clearance
– Patients must be alert and oriented to person,
place and time
– No neurological deficits
– Not clinically intoxicated with alcohol or drugs
– Non-tender at all spinous processes
– No distracting injuries
– Painless range of motion of neck
75
Exposure
Remove all clothing
– Examine for other signs of injury
– Injuries cannot be diagnosed until seen by provider
Logroll the patient to examine patient’s back
– Maintain cervical spinal immobilization
– Palpate along thoracic and lumbar spine
– Minimum of 3 people, often more providers required
Avoid hypothermia
– Apply warm blankets after removing clothes
– Hypothermia = Coagulopathy
Increases risk of hemorrhage
76
Exposure
Author unknown,
http://www.trauma.org/index.php/main/image/98/C11
77
Exposure
Author unknown,
http://www.trauma.org/images/image_library/chest0044b.jpg
78
Secondary Survey
Secondary Survey is completed after primary
survey is completed and patient has been
adequately resuscitated.
No patient with abnormal vital signs should
proceed through a secondary survey
Secondary Survey includes a brief history
and complete physical exam
79
History
AMPLE History
–Allergies
–Medications
–Past Medical History, Pregnancy
–Last Meal
–Events surrounding injury, Environment
History may need to be gathered from family
members or ambulance service
80
Physical Exam
Head/HEENT
Neck
Chest
Abdomen
Pelvis
Genitourinary
Extremities
Neurologic
81
Adjuncts to Secondary Survey
Radiology
– Standard emergent films
C-spine, CXR, Pelvis
– Focused Abdominal Sonography in Trauma
(FAST)
– Additional films
Cat scan imaging
Angiography
Foley Catheter
– Blood at urethral meatus = No Foley catheter
Pain Control
Tetanus Status
Antibiotics for open fractures
82
Definitive Care
Secondary Survey followed by radiographic
evaluation
– CatScan
– Consultation
Neurosurgery
Orthopedic Surgery
Vascular Surgery
Transfer to Definitive Care
– Operating Room
– ICU
– Higher level facility
83
Damage control
COU CONTRECO
P UP
Acceleration injury Deceleration injury
( Coup injury ) ( Countercoup injury )
Skull Fractures
Terminology
• Direct head injury
• Open & closed head injuries
• Coup & Contrecoup injury
• Missile injury
Signs & Symptoms
Anterior
cranial fossa
injury
posterior
cranial fossa middle
injury cranial fossa
injury
Primary brain injury
• Occurs at time of insult as a result of the conta
ct between the brain and the interior of the sk
ull or a foreigh body.
• Direct traumatic forces that injure & kill brain
cells
• Four categories: contusion, diffuse axonal inju
ry, disruptive brain injuries, skull fractures
Secondary brain injury
• Results from cerebral hypoxia, increased intra-
pressure, and decreased cerebral blood flow,
culminating in further hypoxic neuronal dama
ge
• Cause:hematoma
– Epidural
– Subdural
– Intracerebral
Subdural Hematoma with SAH ( su
Epidural Hematoma barachnoid hemorrhage )
Management
• C-Spine Immobilization Be prepared for seizures.
• Airway management If patient is on a backboa
– have suction available
rd with C-Spine immobiliz
• High concentration oxygen
ation you can raise the h
or ventilate 12/min
ead of the board slightly
• Manage bleeding from
scalp lacerations (4-6 inches) to possibly lo
– do not attempt to stop wer ICP.
bleeding from Evacuation:craniotomy
ears / nose
Thoracic trauma
Chest Cavity
Two Lungs (right and left)
Heart
Diaphragm
Pleural Space
Terminology
Traumatic asphyxia
• Rib fractures Myocardial contusion
• Flail chest Pericardial tamponade
(Becks triad:distended neck
• Open pneumothorax veins,quiet heart tones,and
• Closed pneumothorax hypotension:pericardiocente
• Tension pneumothorax sis)
Pulmonary contusion
• Hemothorax
Violence related penetrating
• Hemo-pneumothorax trauma
Open Versus Closed
• Closed Open
– Rib fractures Sucking chest wound
– Flail chest
– Pneumothorax
– Hemothorax
– Myocardial contusion
– Pulmonary contusion
Open Pneumothorax
• An opening in the chest wall causes air to ente
r the pleural space
• Caused by trauma
• Causes the lung to collapse
• Can be a severe life-threatening condition, tha
t can deteriorate rapidly
Open Pneumothorax S & S
• Dyspnea
• Diminished breath sounds on the affected side
• Presence of an open wound in chest wall (suck
ing chest wound)
• Poor skin color
• Accessory muscle use, shock, and possibly sub
cutaneous emphysema
Closed Pneumothorax S & S
• Blunt trauma to chest or back
• Dyspnea
• Possible hypotension
• Shock
• Diminished lung sounds on affected side
• Accessory muscle use
• History of previous pneumothorax
Open Pneumothorax
Inhale Exhale
Pneumothorax
Each time we inhale,the lung
collapses further. There is no
place for the air to escape..
mediastinum
Flail Chest
• Segment of the chest that becomes free to move wit
h the pressure changes of respiration
• Three or more adjacent rib fracture in two or more pl
aces
• Serious chest wall injury with underlying pulmonary
injury
• Reduces volume of respiration
• Adds to increased mortality
• Paradoxical flail segment movement
• Positive pressure ventilation can restore tidal volume
Flail chest
Tension Pneumothorax
• Buildup of air under
pressure in the thorax.
• Excessive pressure
reduces effectiveness of
respiration
• Air is unable to escape
from inside the pleural
space
• Progression of Simple or
Open Pneumothorax
Hemothorax
Accumulation of blood in the pleural space
Serious hemorrhage may accumulate 1,500
mL of blood
Mortality rate of 75%
Each side of thorax may hold up to 3,000
mL
Blood loss in thorax causes a decrease
in tidal volume
Ventilation/Perfusion Mismatch & Shock
Typically accompanies pneumothorax
Hemopneumothorax
Management of the Chest Injury Patient
• Flail Chest
– Place patient on side of injury
• ONLY if spinal injury is NOT suspected
– Expose injury site
– Dress with bulky bandage against
flail segment
• Stabilizes fracture site
– High flow O2
• Consider PPV or ET if decreasing respiratory st
atus
– DO NOT USE SANDBAGS TO STABILIZE FX
Management of the Chest Injury Patient
• Open Pneumothorax
– High flow O2
– Cover site with sterile occlusive
dressing taped on three sides
– Progressive airway
management if indicated
(closed tube thoracostomy)
Management of the Chest Injury Patient
• Tension Pneumothorax
– Confirmation
• Auscultation & Percussion
– Pleural Decompression
• 2nd intercostal space in mid-cla
vicular line
– TOP OF RIB
• Consider multiple decompressio
n sites if patient remains sympt
omatic
• Large over the needle catheter:
14ga
• Create a one-way-valve: Glove
tip or Heimlich valve
Management of the Chest Injury Patient
• Hemothorax
– High flow O2
– 2 large bore IV’s
• Maintain SBP of 90-100
• EVALUATE BREATH SOUNDS
for fluid overload
• Pericardial Tamponade
– High flow O2
– IV therapy
– Consider pericardiocentesis; rapidly d
eteriorating patient
Abdominal Trauma
Liver -Spleen -
Pancreas
Good
Bad
Liver contusions
Hollow Viscous Injury
• Injury can involve stomach, bowel, or mesentery
• Symptoms are a result from a combination of blood l
oss and peritoneal contamination
• Small bowel and colon injuries result most often fro
m penetrating trauma
• Deceleration injuries can result in bucket-handle tear
s of mesentery
• Free fluid without solid organ injury is a hollow viscus
injury until proven otherwise
bowel
mesentery
See normal
Liver and kidney
Free fluid in Morrison's
Pouch between liver and
kidney
Spinal Injury
Mechanisms of Spinal Injury
151