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ON
INTESTINAL
OBSTRU CTION
(Y.SOUJANYA)
LESSON PLAN
By the of the class students will gain knowledge regarding intestinal obstruction
The structured teaching program is amid at bringing a complete awareness about intentional obstruction
Specific objectives:
INCIDENCE:
2min
Know the Intestinal obstruction has a high mortality rate. If it is not Taking
diagnosed and treated within 24 hours. The mortality rate for notes
incidence
acute obstruction in the small bowel is 10% are in the large
bowel 30%
DEFINITION:
5min
Impairment of the forward flow of intestinal content is
Define intestinal known as intestinal obstruction. Two types of processes
Over head
obstruction can impete this flow.
projector
- Brunner and Siddhartha
Discuss 4min
th Black
ETIOLOGY:
eetiological
Obstruction of the large intestine may be caused by
narrowing of
factors and risk the intestinal lumen as a result of inflammation, neoplasm, Lecture cum Taking board What are the
factors adhesion, hernia, food blockages. A cancer account for about Discussion notes etiological
80% of obstruction of the large intestine with most occurring in factors
colitis, and previous abdominal surgery.
RISK FACTORS:
1. Factors that cause intestinal obstruction
2min
may be mechanical, Neurogenic, Vascular. Lecture Active
a. Mechanical factors: cum listening What are risk
● Adhesion: loops of intestine become adherent to Discussion factors
Flash cards
areas that heal slowly or scar after abdominal surgery.
Adhesion is probably the most common cause of
obstruction in boththe small and large intestine.
● Hernia: protrusion of intestine through a weakened area
in the abdominal muscle or wall, may or may not cause
obstruction depending on the size of the hernial ring
Neurogenic factors:
Neurogenic factors are responsible for a dynamic
obstruction, the most common type of intestinal
obstruction. A dynamic obstruction called paralytic ileus is
caused by lack of peristaltic movement.
Listening
Vascular factors: actively
Body is interrupted, the part ceases to function and pain
occurs.
4min
Understand the PATHOPHYSIOLOGY:
pathophysiology Intestinal contents, fluid and gas accumulate above the
of intestinal
intestinal obstruction. The abdominal distention and retention
obstruction
of fluid reduce the absorption of fluids and stimulate Model
Lecture
more gastric secretion. With increasing distention, pressure
cum
within
Discussion
the intestinal lumen increases, causing a decrease in
venous andarteriolar capillary pressure. This causes edema,
congestion,
necrosis, and eventual rupture of perforation of the
intestinal wall, with resultant peritonitis.
Reflux vomiting may be caused by abdominal distention.
Vomiting results in loss of hydrogen ions and potassium from Listening
IF OBSTRUCTION IS COMPLETE
● The peristaltic waves initially become extremely vigorous
and eventually assume a reverse direction, with the
intestinal contents propelled toward the mouth instead
of toward the rectum. If the obstruction is in the ileum,
fecal vomiting takes place. First, the patient vomits the
stomach contents, then the bile-stained contents of the Listening
duodenum and the jejunum, and finally, with each actively
paroxysm of pain, the darker, fecal-like contents of the
ileum. The signs of dehydration become evident: intense
thirst, drowsiness, generalized malaise, aching, and a Lecture
parched tongue and mucous membranes. The abdomen cum
becomes distended. The lower the obstruction is the Discussion
GI tract, the more marked the abdominal distension. If
the obstruction continues uncorrected, hypovolemic
shock occurs from dehydration and loss of plasma
volume.
Discuss
nursing NURSING DIAGNOSIS:
diagnosis Based on the assessment data, the major nursing diagnosis may
include the following List out the
nursing
● Imbalanced nutrition (less than body requirements
diagnosis
related to nausea and anorexia
● Risk for deficient fluid volume related to vomiting and
dehydration
1min
● Anxiety related to impending surgery and the diagnosis
ofcancer
● Risk for ineffective therapeutic regimen management
related to knowledge deficit concerning the diagnosis, Pamphlet
the surgical procedure, and self care after discharge
Listening
REMOVING AND APPLYING THE COLOSTOMY APPLIANCE actively
The colostomy begins to function 3 to 6 days after surgery.
The nurse manages the colostomy and teaches the patient
about this care until the patient can take over its management.
The nurse teaches skin care and how to apply and remove the
drainage pouch. Care of the peristomal skin is an ongoing
concern because excoriation or ulceration can develop quickly.
The presence of such irritation makes adhering the ostomy
appliance to irritated skin can worsen the skin condition. The
effluent discharge and the degree to which it is irritating vary
with the type of ostomy. With a transverse colostomy, the Handout
stool is soft, mushy and irritating to the skin. With a descending
or sigmoid colostomy, the stool is fairly solid and less irritating
to the skin. Other skin problems include yeast infections and
allergic dermatitis.
If the patient wants to bath or shower before or shower
before putting on the clean appliance, microphone tape to the Lecture
sides of the pouch will keep is secure during bathing. To remove cum
the appliance, the patient assumes a comfortable sitting or Discussion
standing position and gently pushes the skin down from the
faceplate while pulling the pouch up and away from the stoma.
Gentle pressure prevents the skin prevents the skin from being
traumatized and any liquid fecal contents from spilling out. The
nurse advises the patient to protect the peristomal skin by then
washing the area gently with a moist soft cloth and a mild soap.
Soap acts as a mild abrasive agent to remove enzyme residue
from fecal spillage. The patient should remove any excess skin
barrier. While the skin, is being cleansed, a guaze dressing can
cover the stoma, or a vaginal tampon can be inserted gently to
absorb excess drainage. Completely dry with a guaze pad,
taking care not to rub the area. The patient can lightly dust
nystatin (Mycostatin) powder on the peristomal skin if irritation Listening
of yeast growth is present. actively
SUMMARY
We have discussed regarding the intestinal obstruction,
definition, etiology, Pathophysiology, clinical manifestations,
assessment, diagnostic findings and management.
CONCLUSION:
Students have gained knowledge regarding the intestinal
obstruction and the medical, surgicaland Handout
nursing management
of the client with the intestinal obstruction.
ASSIGNMENT:
Write nursing care plan for the client suffering from
intestinalobstruction
BIBLIOGRAPHY:
✓ Lippincot Williams and Wilkins Text book of manual of
nursing practice, , ninth edition page no- 687-690