PARTURITION is defined as the process of bringing forth of

young which comprises of multiple transformations in

both uterine and cervical functions
There are four phases :
Quiescence
Activation phase
Stimulation phase
Involution phase.
 QUIESCENCE ACTIVATION STIMULATION INVOLUTION
FROM CONCEPTION BEGINNING OF UP TO TILL THE
TO INITIATION PARTURITION DELIVERY OF TIME
OF TO ONSET OF CONCEPTUS FERTILITY IS

PHASES OF PARTURITION
PARTURITION LABOUR RESTORED

PREDOMIN INHIBITORS UTEROTROPI UTEROTONICS OXYTOCIN

-ANTLY PROGESTRONE , C OXYTOCIN THROMBINS
INFLUENC PROSTACYCLIN, ESTROGEN, PROSTAGLANDI
-ING NITROUSOXIDE OXYTOCIN , NS
FACTOR , RELAXIN PROSTAGLAND
INS->
INCREASED
GAP JUNC.
UTERINE CONTRACTILE PREPARATION CONTRAC INVOLUTION
ACTIVITY UNRESPONSIVE FOR LABOUR TIONS ALONG
NESS. WITH FETAL &
PLACENTAL
EXPULSION
CERVIX SOFTENING RIPENING DILATATION & REPAIR
EFFACEMENT
LABOUR
It is the third phase of parturition, comprising three
stages:
First stage: from onset of labour pains till cervix is fully
dilated.

Second stage of labour: from complete dilatation of

cervix till the delivery.

Third stage of labour: placental separation &expulsion

FIRST STAGE OF LABOUR

Following are the major events during labour:

 Gradually increasing uterine contractions
Retraction
Dilatation of cervix
Effacement of cervix
Lower uterine segment formation
UTERINE CONTRACTIONS IN
LABOUR
Characteristics of normal uterine contractions:
Pace maker: situated in the region of tubal ostia from where
wave of contraction spread downwards.
Sometimes there is emergence of multiple pace maker foci
leading to less efficient contractions and hence causing
primary dysfunction labour
Fundal dominance with gradual diminishing contractions
towards the lower segment.
Polarity of uterus : when upper segment contracts, retracts
and pushes the fetus down the lower uterine segment and
cervix dilates in response.
Lack of fundal dominance and the reverse polarity leads to
spastic lower uterine segment. Here pacemaker does not
work in rhythm.
Good synchronization of contraction waves from
both sides of uterus.
Regular pattern of contractions
Good relaxation in between the contractions
Intra amniotic pressure during relaxation is 8mm
rising beyond 20mm during contraction
INTENSITY: describes degree of uterine systole.
increases with progress of labour.Maximum during 2nd
stage of labour
DURATION: initially last for 10-15 seconds gradually
increases up to 40-45 sec.
FREQUENCY: in the early stage of labour, contractions
come at the interval of 10-15min and increases to
maximum in 2nd stage of labour.
Clinically contractions are said to be good when they come
after interval of 3-5minutes and at the height of
contractions uterine wall can not be indented by fingers.
TONUS : intra uterine pressure in between the
contractions.
During Quiscent stage- 2-3mm Hg
During first stage of labour 8-10mmHg.
Factors governing tonus are:
Contractility of uterine muscles
Intra abdominal pressure
Over distension of uterus as in twins and
hydramnios.
If the intensity diminishes, duration is shortened and
period between the increases it leads to hypotonic
uterine dysfunction. Here intrauterine pressure
during the contractions remains below 25mm of Hg.
if there is increased frequency and duration without
adequate relaxation in between it leads to inco-
ordinate uterine action.
It comprises a rise in the base line tone which and
hence diminishing the circulation in the intervillous
space of placenta
LABOUR PAINS
Pain during contractions is along the cutaneous nerve
distribution of T10 to L1
Pain of cervical dilatation is radiated to back through sacral
plexus
Causes of pain:
Myometrial hypoxia
Streching of peritonium over the fundus
Streching of cervix during dilatation
Compression of nerve ganglia
Retraction
Permanent shortening of uterine muscle.
net effects are :
Formation of lower uterine segment.
Maintain advancement of presenting part made during
contractions
Reduce the surface area of uterus and hence favouring
placental separation.
Effective haemostasis after separation of placenta.
FRIEDMAN graph of cervical
dilation
Latent phase : during which there is little dilatation
occurs with considerable changes taking place in the
connective tissue component of cervix which include:
Breaking down of collagen by collagease and elastases.
Accumulation of fluid between collagen fibres.
Fibro- muscular glandular hypertrophy.
Increased vascularity
Acceleration phase with cervical dilatation 2.5-4 cm.
Phase of maximum slope: between 4-9cm
Phase of decelaration: 9-10cm
Caused by:
(a)Uterine contraction and retraction: bucket
handle manner of attachment of longitudinal muscle
fibres of upper uterine segment with circular muscle
fibres of lower uterine segment and cervix. Thus
during contraction of upper segment the canal-
shortens, retracts and opens.
(b)Bag of membranes : during labour the membranes
attached to the lower uterine segment are detached

herniation of membranes through the cervical canal

due to ball valve action of well flexed head, during uterine

contraction hydrostaic pressure in forewaters increases

cervical dilatation
Fetal axis pressure: contractions of circular muscles of
body of uterus

Straightening of vertebral column of fetus

Fundal contractions transmit through podalic pole in to

fetal axis

Mechanical streching of lower uterine segment and

opening of cervical canal
Effacement of cervix
Muscular Fibres of cervix are pulled upwards and merge
with
lower uterine segment.
Effacement precedes the dilatation in primegravidae
While it occurs simultaneously with dilatation in multiparae
Lower uterine segment formation
During labour lower uterine segment is demarcated by
physiological retraction ring above and fibromuscular
junction of cervix and uterus below.
 formed maximally during labour.
7.5-10 cm when fully formed and cylindrical during 2nd
stage of labour
Poor retractile property as compared to upper uterine
segment.
 gradual thinning of lower uterine segment due to
relaxation of its muscle fibres to allow elongation and
descent of presenting part
1)implantation of placenta of in lower uterine
segment leads to placenta praevia.

2)poor decidual reaction in this segment facilitates

morbid adherent placenta.

3)lower segment is entirely the passive segment of

uterus. Because of poor retractile property,there is
chance of post partum haemorrhage if placenta is
implanted over the area.
Uterine tetany: when there is no physiological
differentiation between upper active and lower
passive segment of uterus whole of the uterus goes in
to a tonic muscular spasm holding the fetus inside.
Poor decidual reaction in this segments facilitates
morbid adherent placenta if implanted here
Poor retractile property leads to post partum
haemorrhage.
SECOND STAGE OF LABOUR
It two phases:
(a)propulsive: from full dilatation until head
touches the pelvic floor.
 (b)expulsive: since the time there is irresistible
maternal desire to bear down until the baby is
delivered .
Factors leading to expulsion of fetus from uterine cavity are :
Reduced volume due to escape of large amount of amniotic
fluid.
Elongation of uterus due to contraction of circular muscle
fibers keeping the fetal axis straight.
 Reduced transverse or anterioposterior diameter.
Downward thurst offered by uterine contractions
supplimented by voluntary contractions of abdominal
muscle.
Retraction of uterus which counterbalance the resistance
offered by pelvic floor.
Third stage of labour

It comprises expulsion of placenta with membranes

SEPERATION OF PLACENTA: due to shearing force
instituted between the placenta and placental site due to
marked reduction in the surface area in the placental site
and inelasticity of placenta.

PLANE OF SEPERATION: runs through spongy layer of

decidua basalis.
METHODS OF SEPERATION :
Marginal separation Of Placenta(Mathew Duncan):
more frequent . Separation starts at the margins as it
is mostly unsupported.
Central separation (Schultze): detachment starts at
centre with opening of few uterine sinuses and
collection of retroplacental haematoma. Gradually
due to weight of placenta and retroplacental blood
collection more and more placenta separates.
SEPARATION OF MEMBRANES: The membranes in
the upper part are thrown in to folds while those in
the lower part are already detached due to stretching.
Expulsion of placenta : After complete separation the
placenta is forced in to the lower uterine segment and
then in the vagina.
Complete expulsion occures due bearing down efforts
of by manual procedure.
HAEMOSTASIS
Living ligature : as the arterioles pass tortuously through
interlacing intermediate layers of myometrium they are
actually clamped during uterine contractions.

Thrombosis: occlude torn sinuses as pregnancy is

hypercoagulation state.

Myotamponade: apposition of walls of uterus after

expulsion of placenta.