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ADIPOSITY 101.

ADIPOSITY 101.

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Published by Dr. Heath Motley
Huge amount of research on studies dealing with weight loss. Se the end for numerous links.
Huge amount of research on studies dealing with weight loss. Se the end for numerous links.

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Published by: Dr. Heath Motley on Feb 05, 2013
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07/16/2013

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1 per cent Successful Long Term Weight Loss
-Robert Jeffrey and colleagues
"dieting may be the major cause of obesity"
-Jean-Paul Deslypere, University of Ghent Professor of human nutrition
EXECUTIVE SUMMARY
Adiposity is the result of an excessive number or size of white adipose cells.Adiposity is caused by genetics
and 
the environment. New chromosome variations causingobesity are constantly added to the list. Identical twins reared apart have the same weight,(unless one has been exposed to a fattening virus!).Maternal diabetes, smoking, and malnutrition predispose the unborn to grow up fat. Earlywithdrawl of breast feeding, early introduction of a high carbohydrate diet predispose thechild to grow up fat. Exposure to at least one virus causes permament weight gain.Normal adults do not retain weight brought on by a period of overeating.Conversely, individuals whose weight gain was not caused by overeating are rarelysuccessful at long term weight loss. The weight they lose usually comes back withconsiderable "interest" (rebound). This rebound may be mediated by diet induced increasein fat cell numbers.
High carbohydrate low fat dietsfor weight loss have been recommended for 
three millennia
. Low fat diets have been extensively studied for the last 5 decades. In the last decade Americans have reduced their fat intake only to getfatter than ever. For the first time in history, a majority of males are overweight.Previously reported assocations between higher fat consumption and obesity have not held up to careful study.Previously reported associations between higher fat consumption and breast cancer have been refuted. A 14-year study of nearly 89,000 women found no evidence that a high-fat diet promotes breast cancer or that a low-fat diet protects against it. Women who ate the least fat appeared to have a 15 percent higher rate of breast cancer. (Journalof the American Medical Association 3/10/99)The low fat/low cholesterol diet is ineffective.Some researchers now think low-fat high carbohydrate diets aremaking us fat. Meanwhile, traditional nutritionists have ignored the lowering of female puberty from 17 to 13 years in the last century, revealing a tremendous increase in bioavailable dietary carbohydrate.In the future, drugs, antibodies to fat cells, and/or cellular removal will control adiposity. In the meantime, people atrisk of adiposity would be wise to check with a competent endocrinologist to prevent the early rise in insulin levels that triggers adiposity and related diseases.Adiposity 101 surveys the rapidly evolving field of adiposity research.
 
F L A S H
Compound C75, developed for cancer treatment, inhibits feeding, but does not allow themetabolism to shut down. It tricks the animal into thinking it's well fed. It drops weightlike a stone. C75 treated mice lost weight 45 per cent faster than
untreated fasting mice.
C75also reversed a fat-related form of diabetes.C75 blocks Fatty Acid Synthase, a powerful fat making enzyme. "This is the enzyme thatturns your pasta into fat," Dr. Frank Kuhajda told United Press International. FAS is thelast enzyme on an assembly line of about 25 enzymes that builds fat molecules to storeenergy. Kuhajda says that in a test tube, purified FAS will "make fat before your eyes" if given the right building blocks. This may have been very useful when primitive humanshad to sprint across the savanna and kill an animal for supper. It has become a curse in theage of carbohydrate. "It makes us fat," Kuhajda says.
FLASH
NEW YORK, Feb 18 2000 (Reuters Health) -- The extremely carbohydrate-restrictedAtkins diet is a safe, effective way to lose weight, according to studies presented at theSouthern Society of General Internal Medicine in New Orleans.
In a press release, the researchers also say that their study did not find any of the safety concernsvoiced by the American Dietetic Association, such as potentially dangerous effects on liver andkidney function."In four short months on the Atkins Diet, we were able to confirm scientifically what Dr. Atkinsstates he has seen in his practice over the past decades. The diet lowers cholesterol andtriglycerides and raises HDL... which may represent an entirely new approach to the control and prevention of heart disease," said lead researcher Dr. Eric C. Westman, assistant professor of medicine at North Carolina's Duke University.
ADIPOSITY 101
Chuck Forsberg caf@omen.com
 
FOREWORD
Obesity ruins the quality of life for more than 100 million Americans. Medical advances have circumvented thenatural eugenic selection that previously limited the diabetic/obese to less than 10% of the population. Increases inobesity prone minorities and high carbohydrate gestation and suckling environments add to the weight.Traditional weight control technology has changed little since Greek antiquity. 30 years of applied research intotraditional weight control technology and the resulting recommendations have only made Americans fatter. No study has ever shown dieting to extend the life of fat people, but more than twenty have reported ill effects fromdietary weight cycling. For years dieters have complained that weight loss regimes made them fatter, but theseobservations fell on deaf ears. Recent research has shown that dieting is a major cause of obesity. While the longterm success rate from dieting is less than one per cent, about 30 per cent of dieters regain more than they lose as adirect result of their dieting.
"In the last 25 years there has been no progress in treatment for obesity and the long-term results aremiserable."
(Marian Apfelbaum, University of Paris Professor of Nutrition)
"dieting may be the major cause of obesity"
(Jean-Paul Deslypere, University of Ghent Professor of human nutrition)Recent obesity research has disproven public stereotypes and the conventional wisdom of most health professionals.Identical twins grow up with virtually the same body fatness, even when raised by different families, (those thatdon't may have been exposed to a virus that causes obesity)while adopted children raised by fat parents are no fatter than those raised by thin parents. The opposite would be true if adiposity were environmental instead of inborn. Thisevidence has yet to register on diet promoters and exercise gurus who continue to claim obesity is mostlyenvironmental. When all you have is a hammer every problem looks like a nail.For the first time in history, research has placed true cures for human obesity within sight. But before this canhappen, the public must first be weaned from its belief that the obese eat much more than other people, that this isthe cause of their obesity, and that they could become lean and remain slender simply by eating normal amounts of food. This belief is particularly resistant to change since it was the accepted scientific position until recently.Misleading weight loss advertising perpetuates this belief, and the sheer volume of this commerce discourages themedia from educating the public.Less than one research dollar is spent for each overweight American compared to a thousand dollars for each HIV positive American. It is high time overweight Americans got their fair share of the billions and billions of tax dollarsthey pay for medical research. In addition, we should add a checkoff to income tax forms allowing taxpayers toearmark money for the research and deployment of new weight control technology.In the meantime, the protections of the Americans with Disabilities Act should be extended to those Americanswhose diligence in dieting has only made them fatter.The purpose of this paper is to set out the case for new weight loss technology and thereby give hope to the millionsof fat Americans for whom conventional weight loss technology has been ineffective or worse.This paper is a summary of recent progress in obesity research. It identifies topics and issues concerning obesity.The reader should study the references given below if questions or doubts remain.Many of the topics related to adiposity are interrelated. Since this document was only recently converted tohypertext, few links are available. The reader must carefully study the entirety of this document to understandadiposity.
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