HIDROSEFALUS NONKOMUNIKAN PADA PERDARAHAN INTRASEREBRAL/INTRAVENTRIKULAR

Patofisiologi Perdarahan intraserebral/intaventrikular perdarahan masuk ke dalam ventrikel lateral kiri dan mendesak ventrikel lateral kanan obstruksi sistem ventrikel dilatasi venterikel lateral kanan Hidrosefalus nonkomunikan

If there is an obstacle to the flow of CSF within the ventricular system, then a portion of the ventricles enlarges while the remainder does not. This pattern is referred to as noncommunicating hydrocephalus and is most commonly seen with masses at the formamen of Monro or aqueduct of Sylvius. In communicating hydrocephalus all of the ventricular system is enlarged; here the cause is most often reduced resorption of CSF (Robbins Basic Pathologi 8 ed)

In 1914, Dandy and Blackfan introduced the also somewhatinaccurate but now well-established terms communicating and noncommunicating (obstructive) hydrocephalus. The concept of a communicating hydrocephalus was based on the observations that dye injected into a lateral ventricle would diffuse readily downwarinto the lumbar subarachnoid space and that air injected into the lumbar subarachnoid space would pass into the ventricular system; in other words, the ventricles were in communication with the spinal subarachnoid space. If the lumbar spinal fluid remained colorless after the injection of dye, the hydrocephalus was assumed to be obstructive or noncommunicating. In actuality, the distinction between these two types is not fundamental, because all forms of tension hydrocephalus are obstructive at some level, and the obstruction is virtually never complete. Acute and complete aqueductal occlusion is said to be incompatible with survival for more than a few days. The authors suggest that a more appropriate terminology is one in which the site of the presumed obstruction is indicated, e.g., meningeal, aqueductal, or third ventricular tension hydrocephalus.