The herpes virus enters the body through open wounds or the mouth from an infected person. It attaches to host cells and injects its RNA, causing the host cell to produce more virus cells until it bursts, spreading the virus. This causes small blisters on the skin that contain fluid and eventually pop, as the body tries to remove the virus. There are two types that typically cause sores on the mouth or genitals and can reoccur due to the virus lying dormant in nerve cells until reactivated by stress or illness. Once a person is infected, the virus remains in their body for life, though outbreaks can be avoided.
The herpes virus enters the body through open wounds or the mouth from an infected person. It attaches to host cells and injects its RNA, causing the host cell to produce more virus cells until it bursts, spreading the virus. This causes small blisters on the skin that contain fluid and eventually pop, as the body tries to remove the virus. There are two types that typically cause sores on the mouth or genitals and can reoccur due to the virus lying dormant in nerve cells until reactivated by stress or illness. Once a person is infected, the virus remains in their body for life, though outbreaks can be avoided.
The herpes virus enters the body through open wounds or the mouth from an infected person. It attaches to host cells and injects its RNA, causing the host cell to produce more virus cells until it bursts, spreading the virus. This causes small blisters on the skin that contain fluid and eventually pop, as the body tries to remove the virus. There are two types that typically cause sores on the mouth or genitals and can reoccur due to the virus lying dormant in nerve cells until reactivated by stress or illness. Once a person is infected, the virus remains in their body for life, though outbreaks can be avoided.
The virus enters your body, from someone who is already infected with the virus, through an open wound or through a persons mouth. Once it's in your system the virus works by sticking its glycoproteins, which surround the cell, onto any one of your good cells, and injects its RNA, (the cells DNA), into your good cell. Your cell then reads the viruses RNA and copies many herpes cells inside your good cell. Eventually the cell containing all the copied herpes will burst and the virus cell will stick on to other cells and continue spreading. Before you know it, you are sick with the herpes virus. As a result, small blister type bumps will appear on your skin. They usually sit on the surface of your skin for 5- 10 days. Clear liquid fills the bump and eventually pops. That
is your body trying to get rid of the
virus. You can get HSV1- oral herpes, or HSV2- genital herpes. They often occur, go away, then come back again. The reasoning behind that is the herpes lie dormant, or stagnant in the nerve cell and the virus will reactivate from illness, stress or temperature. That is the physical trait of the herpes virus. But of course everything starts from the way the virus reacts with your bodys cells. Inside the herpes cell, there is a double stranded RNA, (viruses genetic information). That, is surrounded by an icosahedral protein cage called capsid. The capsid is wrapped in a lipid bilayer (fat layer). The capsid and lipid layers are called the envelope. Herpes can be fairly harmless, but once you are infected with herpes, it is in your system forever. You can avoid break outs, but the virus cells will always remain in your body.
Right: Actual microsc ope picture of herpes virus. Left: Diagra m of herpes cell structur e virus