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  • Pathopyhsiology DM Type 2

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    Czarina Cullen
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    Insufficient insulin leads to reduced glucose uptake in tissues, causing intracellular hypoglycemia and triggering gluconeogenesis and glucogenesis to produce more glucose. This breakdown of fats and proteins leads to high levels of ketones and the dangerous condition of diabetic ketoacidosis. Extracellular hyperglycemia causes a hyperosmotic plasma, dehydrating cells and potentially causing a hyperglycemic coma. High blood glucose above the renal threshold also causes glucosuria, osmotic diuresis, polyuria, polydipsia, and electrolyte imbalances.

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    Pathophysiology of DM type 2

    Original Title

    Pathopyhsiology DM type 2

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    Insufficient insulin leads to reduced glucose uptake in tissues, causing intracellular hypoglycemia and triggering gluconeogenesis and glucogenesis to produce more glucose. This breakdown of fats and proteins leads to high levels of ketones and the dangerous condition of diabetic ketoacidosis. Extracellular hyperglycemia causes a hyperosmotic plasma, dehydrating cells and potentially causing a hyperglycemic coma. High blood glucose above the renal threshold also causes glucosuria, osmotic diuresis, polyuria, polydipsia, and electrolyte imbalances.

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    © All Rights Reserved
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    50 views1 page

    Pathopyhsiology DM Type 2

    Uploaded by

    Czarina Cullen
    Insufficient insulin leads to reduced glucose uptake in tissues, causing intracellular hypoglycemia and triggering gluconeogenesis and glucogenesis to produce more glucose. This breakdown of fats and proteins leads to high levels of ketones and the dangerous condition of diabetic ketoacidosis. Extracellular hyperglycemia causes a hyperosmotic plasma, dehydrating cells and potentially causing a hyperglycemic coma. High blood glucose above the renal threshold also causes glucosuria, osmotic diuresis, polyuria, polydipsia, and electrolyte imbalances.

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    © All Rights Reserved
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    of 1

    Book Based

    Pathophysiology

    Insufficient insulin
    Reduced tissue
    uptake of glucose
    Intracellular
    hypoglycaemia
    Glucogenesis
    and
    gluconeogenesi
    s
    Breakdown
    of fats
    High levels
    of ketones
    Diabetic
    ketoacidosi
    s
    Decrease protein
    synthesis;
    cachexia, lethargy
    and polyphagia,.
    Decrease gamma
    globulins
    susceptibility to
    infections
    impaired wound
    healing
    Extracellular
    hyperglycemia
    Hyperosmoti
    c plasma
    Dehydration
    of cells
    Hyperglycaemi
    c coma
    Blood glucose
    > renal
    threshold
    Glocosuria
    urine has a
    high sg
    Osmotic diuretics
    - Polyuria
    - Polydipsia
    - Hypokalemia
    - hyponatremia

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