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Cardiotocography (CTG) is used during pregnancy to monitor both the foetal heart and the
contractions of the uterus. It is usually only used in the 3rd trimester. Its purpose is to
monitor foetal well-being and allow early detection of foetal distress. An abnormal CTG
indicates the need for more invasive investigations and may lead to the need for emergency
caesarian section.
How it works
women.
One transducer records the fetal heart rate using ultrasound.
DR Define Risk
C Contractions
BRa Baseline Rate
V Variability
A Accelerations
D Decelerations
O Overall impression
Define risk
You first need to assess if this pregnancy is high or low risk.
This is important as it gives more context to the CTG reading e.g. If the pregnancy is
high risk, your threshold for intervening may be lowered.
Obstetric complications
Multiple gestation
Post-date gestation
Previous cesarean section
Intrauterine growth restriction
Premature rupture of membranes
Congenital malformations
Oxytocin induction/augmentation of labor
Pre-eclampsia
Contractions
Record the number of contractions present in a 10 minute period e.g. 3 in 10
Each big square is equal to 1 minute, so look at how many contractions occurred
within 10 squares.
Individual contractions are seen as peaks on the part of the CTG monitoring uterine
activity.
You should assess contractions for the following:
o
The baseline rate is the average heart rate of the foetus within a 10 minute window.
Look at the CTG and assess what the average heart rate has been over the last 10
minutes.
Ignore any accelerations or decelerations.
A normal foetal heart rate is between 110-150 bpm.
Foetal tachycardia
Foetal tachycardia is defined as a baseline heart rate greater than 160 bpm.
It can be caused by:
o
Foetal hypoxia
Hyperthyroidism
Foetal tachyarrhythmia
Foetal bradycardia
Foetal bradycardia is defined as a baseline heart rate less than 120 bpm.
Mild bradycardia of between 100-120bpm is common in the following situations:
o
Post-date gestation
Severe prolonged bradycardia (< 80 bpm for > 3 minutes) indicates severe hypoxia.
Causes of prolonged severe bradycardia are:
o
Cord prolapse
Maternal seizures
Variability
Baseline variability refers to the variation of foetal heart rate from one beat to the next.
Variability occurs as a result of the interaction between the nervous system,
chemoreceptors, baroreceptors and cardiac responsiveness.
Therefore it is a good indicator of how healthy the foetus is at that particular moment
in time.
This is because a healthy foetus will constantly be adapting its heart rate to respond
to changes in its environment.
Normal variability is between 10-25 bpm
To calculate variability you look at how much the peaks and troughs of the heart rate
deviate from the baseline rate (in bpm)
Variability can be categorised as: 4
o
Reassuring 5 bpm
Foetal sleeping this should last no longer than 40 minutes most common cause
Foetal acidosis (due to hypoxia) more likely if late decelerations are also present
Foetal tachycardia
Accelerations
Accelerations are an abrupt increase in baseline heart rate of >15 bpm for >15
seconds.
Accelerations
Early deceleration
Early decelerations start when uterine contraction begins and recover when uterine
contraction stops.
This is due to increased foetal intracranial pressure causing increased vagal tone.
It therefore quickly resolves once the uterine contraction ends and intracranial pressure
reduces.
This type of deceleration is therefore considered to be physiological and not pathological.
Variable deceleration
Variable decelerations are observed as a rapid fall in baseline rate with a variable recovery
phase.
They are variable in their duration and may not have any relationship to uterine contractions.
They are most often seen during labour and in patients with reduced amniotic fluid volume.
Variable decelerations are usually caused by umbilical cord compression:
o
When pressure on the cord is reduced another acceleration occurs and then the baseline rate
returns.
Accelerations before and after a variable deceleration are known as the shoulders of
deceleration.
Their presence indicates the foetus is not yet hypoxic and is adapting to the reduced blood
o
flow.
Variable deceleration
Late deceleration
Late decelerations begin at the peak of uterine contraction and recover after the contraction
ends.
This type of deceleration indicates there is insufficient blood flow through the uterus and
placenta.
As a result blood flow to the foetus is significantly reduced causing foetal hypoxia and
acidosis.
Reduced utero-placental blood flow can be caused by:
o
Maternal hypotension
Pre-eclampsia
Uterine hyper-stimulation
The presence of late decelerations is taken seriously and foetal blood sampling for pH is
indicated.
If foetal blood pH is acidotic it indicates significant foetal hypoxia and the need for
emergency C-section.
Late deceleration
Prolonged deceleration
A deceleration that last more than 2 minutes.
If it lasts between 2-3 minutes it is classed as non-reassuring.
If it lasts longer than 3 minutes it is immediately classed as abnormal.
Action must be taken quickly e.g. foetal blood sampling / emergency C-section
Prolonged deceleration
Sinusoidal pattern
This type of pattern is rare, however if present it is very serious.
It is associated with high rates of foetal morbidity & mortality.
.
It is described as:
o
Foetal/maternal haemorrhage
.
Immediate C-section is indicated for this kind of pattern.
Outcome is usually poor.
Overall impression
Once you have assessed all aspects of the CTG you need to give your overall
impression.
The overall impression can be described as either:
o
Reassuring
Suspicious
Pathological
The overall impression is determined by how many of the CTG features were either
reassuring, non-reassuring or abnormal. The NICE guideline below demonstrates how
to decide which category a CTG falls into.4