Scribd Logo
Upload

Welcome to Scribd!

  • Xi. Pathophysiology: Further Physically Rectal Exam Tenesmu S

    Uploaded by

    Sheana Tmpl
    1 views2 pages
    This document outlines the pathophysiology of fecal contamination of food or water ingestion. It begins with direct invasion and destruction of the bowel wall by pathogens, releasing endotoxins. This causes stimulation and obstruction of the mucosal lining, leading to ulceration. Symptoms include abdominal pain, bleeding, diarrhea, nausea and vomiting. The lining destruction and secretion of fluids overwhelms the bowel's ability to reabsorb, causing watery diarrhea and fluid/electrolyte deficits. This puts the patient at risk for dehydration, metabolic acidosis, and other imbalances if not properly managed with antibiotics, antispasmodics, rehydration and other treatments.

    Original Description:

    Gastroenteritis Patho

    Original Title

    XI. Pathophysiology

    Copyright

    © © All Rights Reserved

    Available Formats

    DOCX, PDF, TXT or read online from Scribd

    Did you find this document useful?

    Is this content inappropriate?

    Report this Document
    This document outlines the pathophysiology of fecal contamination of food or water ingestion. It begins with direct invasion and destruction of the bowel wall by pathogens, releasing endotoxins. This causes stimulation and obstruction of the mucosal lining, leading to ulceration. Symptoms include abdominal pain, bleeding, diarrhea, nausea and vomiting. The lining destruction and secretion of fluids overwhelms the bowel's ability to reabsorb, causing watery diarrhea and fluid/electrolyte deficits. This puts the patient at risk for dehydration, metabolic acidosis, and other imbalances if not properly managed with antibiotics, antispasmodics, rehydration and other treatments.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    1 views2 pages

    Xi. Pathophysiology: Further Physically Rectal Exam Tenesmu S

    Uploaded by

    Sheana Tmpl
    This document outlines the pathophysiology of fecal contamination of food or water ingestion. It begins with direct invasion and destruction of the bowel wall by pathogens, releasing endotoxins. This causes stimulation and obstruction of the mucosal lining, leading to ulceration. Symptoms include abdominal pain, bleeding, diarrhea, nausea and vomiting. The lining destruction and secretion of fluids overwhelms the bowel's ability to reabsorb, causing watery diarrhea and fluid/electrolyte deficits. This puts the patient at risk for dehydration, metabolic acidosis, and other imbalances if not properly managed with antibiotics, antispasmodics, rehydration and other treatments.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    of 2

    Ingestion of

    fecally
    contaminated
    food/water

    XI. PATHOPHYSIOLOGY

    Endotoxin are
    released

    Direct invasion of
    the bowel wall
    Stimulation and
    obstruction of
    mucosal lining of
    the bowel wall

    Destruction of
    lining of the
    bowel wall

    (Abdominal
    pain/crampin
    g)
    Bleeding
    Hematochezi
    a
    Hematemesis

    tenesmu
    s

    Management

    Further
    physically
    rectal exam

    Digestive and
    absorptive
    malfunction

    Antispasmodics
    Antibiotics ( to
    directly kill pathogen)
    H2- receptor blocker

    Ulceration in the
    bowel wall

    Excessive gas
    formation

    Borborygmi
    Mild diarhea
    (2-3 stool)
    Nausea
    Vomiting
    Feeling of
    fullness

    Flatus
    GI distention

    Greater secretion
    of water and
    electrolyte

    antispasmodics

    Increase
    peristaltic
    movement
    LI Is overwhelmed
    Diarrhea
    fluid
    volume
    and
    unable
    to
    (
    watery
    stool)
    deficit
    reabsorb
    the
    lost

    Active secretion
    of chloride and
    bicarbonate ions
    in the small bowel

    Antimotility
    Antiemetics
    Antidiarrhea
    rehydration

    Inhibition of
    sodium
    reabsorption
    Fluid and
    electrolyte
    imbalance

    hypermatrem
    ia

    Increased
    protein-rich
    fluids in the
    lumen

    Continuous
    rehydration
    especially
    through IV line

    Severe diarrhea
    (>10 episodes)

    Large intestine is
    overwhelmed and
    unable to
    reabsorb the lost
    fluid

    Metabolic
    acidosis

    Kussmauls
    breathing

    Hyperventilation
    through keeping
    the PCO22 ion

    You might also like