Professional Documents
Culture Documents
Female
MALE External genitalia
External genitalia ❖ mons pubis
❖ Penis: reproductive and urinary elimination. ❖ labia majora and minora
❖ Scrotum: External sac that houses testes. ❖ Clitoris
Protects the testes from trauma & testicular ❖ Vestibule
temperature regulation. ❖ perineum
Internal reproductive organs
Internal reproductive organs ❖ Vagina: muscular tube that leads from the vulva to the
❖ Testes: produce male sex hormone and from uterus
spermatozoa ❖ Cervix: dips into the vagina and forms fornices, which are
❖ Ductal system: “ vas deferens” the tube in which arch-like structures or pockets.
sperm begin the journey out of the body. ❖ Ovaries :two sex glands homologous to the male testes,
❖ Accessory glands: The seminal vesicles are are located on either side of the uterus. (Hatfield 55)
paired glands that empty an alkaline, fructose-rich Fallopian tubes: The paired fallopian tubes (also known as oviducts)
fluid into the ejaculatory ducts during ejaculation. are tiny, muscular corridors that arise from the superior surface of the
Prostate: muscular gland that surrounds the first part of the uterus near the fundus and extend laterally on either side toward the
urethra as it exits the urinary bladder. The alkaline fluids ovaries. The fallopian tubes have three sections
secreted by these glands are nutrient plasmas with several ❖ Isthmus
key functions, including the following: ❖ Ampulla
❖ Enhancement of sperm motility (i.e., ability to ❖ infundibulum
move) Uterus: uterus, or womb, is a hollow, pear-shaped, muscular
❖ Nourishment of sperm (i.e., provides a ready structure located within the pelvic cavity between the bladder and
source of energy with the simple sugar fructose) the rectum.
❖ Protection of sperm (i.e., sperm are maintained in The uterus is divided into four sections.
an alkaline environment to protect them from the ❖ cervix
acidic environment of the vagina) (Hatfield 51) ❖ uterine isthmus
❖ corpus
❖ fundus (Hatfield 53)
Cellular development
Menstrual cycle Soma cells:
❖ Makeup organs and bodily tissue of the human body.
Two main components : Ovarian cycle and Uterine cycle ❖ Gametes: germ cells/ sex cells found only in the reproductive
Ovarian cycle : Cyclical changes in the ovaries occur in glands
response to two anterior pituitary hormones: ❖ Nucleus: contains 23 pairs of chromosomes
follicle-stimulating hormone (FSH) and luteinizing hormone ❖ Each parent donates 1 par of chromosomes ( 46 Chromosomes
(LH). There are two phases of the ovarian cycle, each equals little Mikey)
named for the hormone that has the most control over that ❖ Each parent donates 22 pairs of autosomes: genetic traits such
particular phase. The follicular phase, controlled by FSH, as eye color, hair color, ear wax consistency.
encompasses days 1 to 14 of a 28-day cycle. LH controls ❖ One pair of sex chromosomes
the luteal phase, which includes days 15 to 28
❖ Follicular phase
❖
Fetal development
Luteal phase
Signs of pregnancy
❖ Presumptive: subjective data the
woman reports to the HCP for
example, “ My breasts hurt”
❖ Probable : objective data such as
cervical changes
❖ Positive : diagnostic confirmation
such as, fetal heartbeat & ultrasound
FETAL HEART TONES
CONDITION CAUSE GRADE
Fetal decelerations
The nurse should administer 02 and the baby needs to be
delivered as quickly as possible.
Memory trick
Early decelerations : A decrease in FHR during uterine contraction mirrors
uterine contractions . caused by uterine squeeze
❖ FHR slows as the contraction begins
V: variable deceleration C: cord compression ❖ Lowest point coincides with the highest point ACME of the
E:early deceleration H: head compression contraction
A: acceleration O: ok ❖ Deceleration ends with the contraction
Late deceleration Placental insufficiency Late deceleration: occurs after the peak of contraction due to uteroplacental
insufficiency, pitocin, HTN, diabetes, placental abruption.
❖ Too many decelerations will indicate a need for C-section
❖ Prepare for fetal resuscitation
Variable decelerations: may indicate cord compression. Occur at different
times during a contraction, resulting in fetal HTN that causes the aortic arch
to slow the FHR. usually abrupt and sudden.
Measures to clarify NONreassuring FHR patterns
❖ Fetal stimulation
❖ Fetal scalp sampling
❖ Fetal scalp oximetry
Hematologic Changes
❖ Blood volume increases by
45-50%
PREGNANCY ❖
Weight gain
A woman should increase her
❖ Red blood cell count caloric intake by 300 kcal/day
during 2nd & 3rd trimesters.
Signs of pregnancy
increases up to 30%
❖ Recommended weight gain
❖ Plasma increases up tp 50%
depends on pre pregnancy BMI.
❖ Hemoglobin decreases ❖ Presumptive: subjective data ❖ FIRST TRIMESTER : 3-4 lb total
❖ Hematocrit decreases the woman reports to the HCP ❖ REMAINDER OF PREGNANCY: 1
❖ for example, “ My breasts hurt” lb per week.
❖ Total weight gain: 25-35 lb for a
Cardiac changes
❖ Probable : objective data such
as cervical changes woman with a normal BMI
❖ Positive : diagnostic
❖ Blood pressure slightly
confirmation such as, fetal
decreases
heartbeat & ultrasound
Nutrition
❖ Heart rate increases by
10-15 BPM
❖ Cardiac output increases ❖ When a woman isn't getting the proper nutrients this can cause
Amenorrhea which can inhibit the ability to become pregnant.
❖ Lack of folic acid can cause neural tube defects( spina bifida) and cause
damage to the growing fetus.
❖ Deficits in Vit C have been shown to also cause birth defects and
Integumentary changes
cancer.
❖ Pica:
❖ Chloasma : “ pregnancy mask” ❖ persistent ingestion of nonfood substances such as clay, laundry
brown blotchy areas on the skin of starch, freezer frost, or dirt.It results from a craving for these
substances that some women develop during pregnancy.
the face, cheeks, nose and
❖ These cravings disappear when the woman is no longer pregnant.
forehead.
❖ Pica is associated with iron-deficiency anemia, but it is unknown
❖ Linea nigra: a dark line down the whether iron deficiency is the cause or the result
middle of the skin on the abdomen
Nutritional requirements
❖ Striae: develop in response to
increased glucocorticoid levels.
Also known as stretch marks ❖ Proteins: Growth and repair of fetal tissue, placenta, uterus,
breasts, and maternal blood volume
❖ Minerals: Prevent deficiencies in the growing fetus and maternal
stores
Musculoskeletal changes
❖ Iron : Formation of hemoglobin; essential to the oxygen-carrying
capacity of the blood
❖ Calcium: Nerve cell transmission, muscle contraction, bone
❖ Lordosis: Excessive inward building, and blood clotting
curvature of the spine ❖ Phosphorus: Promotes strong bone growth
❖ Diastasis rectus abdominis: ❖ Zinc: Fetal growth and maternal milk production
tearing of the rectus abdominis ❖ Iodine : Promotes normal thyroid activity, preventing specific birth
muscles defects
Vitamin requirements
Respiratory changes Folic acid (Vitamin B9)
❖ Nasal mucosa edematous due to ❖ Necessary for formation of the nervous system
vasocongestion ❖ Prevents up to 70% neural tube defects
❖ Nasal congestion and voice ❖ Diet should include at least 400 mcg of folic acid per day
changes possible
❖ Accommodations to maintain lung Vitamin A
capacity ❖ Recommended intake via beta-carotene
❖ May feel short of breath when ❖ Too much can be toxic to the fetus
❖ Too little can stunt fetal growth and cause impaired dark adaptation
eupneic
and night blindness
❖ Third trimester diaphragm pressure
Vitamin C
❖ Essential in the formation of collagen, a necessary ingredient to
GI changes
wound healing
Vitamin B6
❖ Necessary for the healthy development of the
❖ Intestines are displaced fetus’s nervous system
upwards & to the side. Vitamin B12
❖ Pressure changes in the ❖ Needed to maintain healthy nerve cells, RBCs, form DNA
esophagus & stomach
which leads to heartburn.
❖ constipation
Assessment
Admission
❖ Birth imminence Components of assessment
❖ Fetal status Obstetric History
❖ Maternal status ❖ Number and outcomes of previous pregnancies in
❖ Risk assessment GTPAL (gravida, term, preterm, abortions, living)
format (see Chapter 7 for a detailed explanation of
these terms)
❖ Estimated delivery date
❖ History of prenatal care for current pregnancy
Prenatal visits
❖ Amount of smoking during pregnancy
❖ Drug and alcohol use during pregnancy
Ist visit : Desires/Plans for Labor and Birth
❖ Presence of a partner, coach, and/or doula (see
❖ Family History, Medical Surgical History,
Chapter 7 for discussion of doulas)
Social History, Teaching, Avoiding
❖ Pain management preferences
teratogenic, substance ingestion, Alcohol, ❖ Other personal preferences affecting intrapartum
tobacco, illegal drugs, etc., Diet, nutrition, and nursing care
exercise, Infection control ❖ Presence of a birth plan
❖ Medication use ❖ Desires/Plans for Newborn
❖ Determining due dates ❖ Plans for feeding—breast or formula
❖ Naegele's rule ❖ Choice of pediatrician
❖ Add seven days to the date of the first day of ❖ Circumcision preference, if the infant is male
the LMP, then subtract three months (and ❖ Rooming-in preference (Hatfield 208)
add a year)
❖ Pelvic examination
❖ Practitioner sizes the uterus to estimate term
❖ Obstetric sonogram: High frequency sound
waves reflect off fetal and maternal pelvic
Tips
structures, allowing structure measurement If a woman presents with ℅
bleeding ask her how man
sanitary napkins she has
saturated in an hour.
Labor positions
Anticipatory signs of labor
The Four P’s of Labor
❖ Lightening or sense that the baby has
❖ “dropped”
❖ Increased frequency, intensity of
❖ Passageway: Pelvic shape
Braxton Hicks contractions
❖ Passenger: fetus
❖ Gastrointestinal disturbances
❖ Powers: contractions
❖ Expelling the mucus plug
❖ Psyche
❖ Feeling a burst of energy
Clinical signs
❖ Ripening (softening)
❖ effacement (thinning) of the cervix
Fetal lie
❖ Longitudinal lie: Long axis of the fetus is parallel to
maternal long axis
❖ Oblique lie: Between longitudinal and transverse lie
❖ Transverse lie: Long axis of fetus is perpendicular to
Maternal adaptation to maternal long axis
labor
❖ Maternal physiologic adaptation
Fetal presentation
❖ Increased demand for oxygen Foremost part of the fetus that enters the pelvic inlet
during the first stage of labor Three main presentations
❖ Increased heart rate ❖ Head: Cephalic presentation
❖ Increased cardiac output ❖ Feet or buttocks: Breech presentation
❖ Increased respiratory rate ❖ Shoulder: Shoulder presentation
❖ Gastrointestinal and urinary
systems are affected Fetal attitude
❖ Laboratory values impact Relationship of fetal parts to one another
❖ Flexion (ovoid shape):Most favorable for
Pushing
❖ Assess woman’s ability to cope family
❖ Assess maternal status
❖ Assess fetal status
❖ She will often express irritability, ❖ Vigorous pushing: take a deep breath, hold the breath, and push while
restlessness, and will feel out of control. counting to 10. She is encouraged to complete three “good” pushes in
She may tremble, vomit, or cry. It is this manner with each contraction.
important to assess for hyperventilation ❖ open-glottis pushing: method of expelling the fetus that is
during this phase. characterized by pushing with contractions using an open glottis so that
air is released during the pushing effort.
❖ urge-to-push method, in which the woman bearsdown only when she
feels the urge to do so using any technique that feels right for her
Pain management during labor
Pain General concepts of pain
❖ Individual &
❖ Pain threshold: Level of pain
interventions
Principles of pain relief ❖ Continuous labor support
Opioids Anesthesia
❖ Local: Used to numb the perineum just before birth,
Medications with opium-like properties (also
allowing for episiotomy and repair
known as narcotic analgesics); the most ❖ Regional: Blocks a group of sensory nerves, supplying
frequently administered medications to provide a particular organ or body area
analgesia during labor. (ex.:Demerol IV, IM) ❖ General :Not frequently used in OB due to risks
Advantages involved
❖ Increased ability for a woman to cope Complications of anesthesia
with labor ❖ Hypotension
❖ Total spinal blockade (rare)
❖ Medications may be
❖ Inadvertent injection into the bloodstream
nurse-administered ❖ Spinal headache
Disadvantages ❖ Pruritus
❖ Frequent occurrence of uncomfortable ❖ Respiratory distress
side effects ❖
Fatal complications of anesthesia
❖ Nausea and vomiting; pruritus;
❖ Failed intubation
drowsiness; neonatal depression ❖ Aspiration
❖ Pain not completely eliminated ❖ Malignant hyperthermia: is a disease that causes a
❖ Possible overdose fast rise in body temperature and severe muscle
contractions when someone with the disease gets
general anesthesia. It is passed down through families(
google)
DELIVERY
Getting ready for the newborn Birthing the placenta
❖ If the urinary bladder is full, the birth
attendant may request that you perform a ❖ nursing care focuses on monitoring for
straight cath placental separation and providing
❖ bed is “broken”—the lower part of the bed
physical and psychological care to the
is removed to allow room for the birth
attendant to control the delivery. woman.
❖ place the woman’s feet on foot pedals or ❖ the fundus rises in the abdomen, the
stirrups uterus takes on a globular shape,
❖ clean the woman’s perineum with an blood begins to trickle steadily from
antiseptic solution
the vagina, and the umbilical cord
❖ Position the instrument table close to the
birthing bed and uncover it. lengthens as the placenta separates
❖ Eye shields, gowns, and gloves may be from the uterine wall.
necessary for protection from contact with
bodily fluids.
Recovery
❖ The new mother is at highest risk for hemorrhage during the
first two to four hours of the postpartum period.
❖ Monitor the woman’s vital signs, and palpate the fundus for
position and firmness.
❖ The fundus should be well contracted, at the midline, and
approximately one fingerbreadth below the umbilicus
immediately after delivery.
❖ Assess the lochia (vaginal discharge after birth) for color and
quantity. The lochia should be dark red and of a small to
moderate amount. If she saturates more than one perineal pad
in an hour, palpate and massage the fundus,
❖ Monitor for signs of infection. The temperature may be
elevated slightly, as high as 100.4°F, because of mild
dehydration and the stress of delivery.
❖ The woman should void within six hours after delivery.
❖ Assess cramping from uterine contractions (referred to as
“afterbirth pains”) and perineal pain from edema or episiotomy
repair
❖ ibuprofen to be given every six to eight hours
❖ ice pack to the perineum.
The newborn
Complications
Neonatal resuscitation
❖ If the newborn doesn't cry immediately: Transport him or
❖ Neonatal Resuscitation Program (NRP) her to a preheated radiant warmer for prompt resuscitation
❖ Must be able to initiate resuscitation and ❖ Dry him or her quickly to prevent heat loss
assist throughout process ❖ Bag and mask connected to 100% oxygen are used to
❖ First 6 to 12 hours after birth are a critical provide respiratory support
transition period for the newborn ❖ Most newborns do not require resuscitation, and the ones
❖ Must be alert to early signs of distress: who do generally respond well to a short period of positive
Bluish color of the skin and mucous pressure ventilation with a bag and mask.
membranes (cyanosis),Brief stop in ❖ However, a very small number of infants require chest
breathing (apnea), Decreased urine output. compressions, intubation, and medications
Nasal flaring. Rapid breathing.,Shallow ❖ Give constant attention to the airway
breathing.Shortness of breath and grunting ❖ Position newborn on side; bulb syringe is used to suction
sounds while breathing. mouth first, then nose
❖ Must be ready to intervene quickly to
Thermoregulation
Assessment
prevent complications and poor outcomes
❖ Critical to protecting the newborn from chilling
❖ Cold stress increases amount of oxygen and glucose
needed
❖ Heart and respiratory rates at least every 30 ❖ Can quickly deplete body’s glucose and develop
minutes during the first two hours of transition. hypoglycemia( < 40 mg/ dl )
❖ Monitor the axillary temperature every 30 ❖ Easily develop respiratory distress and metabolic
minutes until it stabilizes in the expected range acidosis if exposed to prolonged chilling ( PH < 7.20 )
❖ Dry the newborn while on the mother’s abdomen
between 97.7°F and 99.5°F ❖ Swaddle him snugly, and apply a cap to prevent heat
❖ Be alert for signs of hypoglycemia. loss
❖ A full physical assessment including gestational ❖ Kangaroo care
age assessment is completed within the first few
Hypoglycemia
hours of life.
❖
first and then the nose.
Keep the bulb syringe with the newborn, and
INFECTION CONTROL OF THE NEONATE
teach the parents how and when to suction
❖ UMBILICAL CORD STUMP: Use strict aseptic technique
the baby. when caring for the cord
❖ Triple dye, bacitracin ointment, or povidone-iodine used
initially to paint the cord to help prevent the development
of infection.
❖ PREVENT OPHTHALMIA NEONATORUM: a severe eye
infection contracted in the birth canal of a woman with
gonorrhea or chlamydia.
❖ 0.5% erythromycin
Principles
Newborn nutrition
Feeding types
❖ At birth, the passive intake of nutrition
ends and the newborn must actively Two main types of nourishment suitable for the healthy
consume and digest food term newborn:
❖ Newborn has unique nutritional needs
❖ Breast milk
❖ Healthy term newborn requires ❖ Commercial formula
❖ 80 to 100 mL/kg/day of water to maintain
fluid balance and growth Two delivery methods:
❖ 100 to 115 kcal/kg/day to meet energy
needs for growth and development ❖ Breast
❖ Bottle
Breastfeeding
Feeding method choices:
❖ Breastfeed exclusively
● Recommended method for feeding newborns
❖ Breastfeed and supplement with expressed breast
● Breast milk is nutritionally superior to commercial formulas
milk in a bottle
❖ Breastfeed and supplement with formula
● The American Academy of Pediatrics (AAP) recommends
❖ Formula-feed exclusively
● Exclusive breast-feeding until 6 months of age
● Continuation of breast-feeding until at least 12 months of
age
● Healthy People 2020 goals
● Increase proportion of women who breastfeed their babies
Nose breathers
Education
Relieving common maternal breast-feeding problems
WHEN THE BABY ISN'T FEEDING WELL
❖ Sore nipples
❖ Engorgement ❖ Dry mouth
❖ Plugged milk ducts
❖ Mastitis ❖ Not enough wet diapers per day
❖ Breastfeeding amenorrhea ❖ Difficulty rousing the newborn for feeding
❖ Return of woman’s menstrual cycle occurs between ❖ Not enough feedings per day
six and 10 weeks post delivery
❖ Ovulation can occur in absence of a menstrual ❖ Difficulty with latching on or sucking
period, and she can become pregnant
❖ By end of third day of life at least six wet diapers and
about three bowel movements per day
❖ Monitor the newborn’s weight daily during the
hospital stay
Cesarean section delivery
Indication
❖ History of previous cesarean Cesarean Birth
birth or other uterine incision Maternal risks
❖ Labor dystocia (failure to ❖ Major surgery risks and risks of birth itself
progress in labor) ❖ Increases maternal risk of death
❖ Nonreassuring fetal status ❖ Thrombophlebitis, laceration of uterine artery,
❖ Fetal malpresentation bladder, ureter, bowel
❖ Active herpes, prolapsed cord( ❖ Hemorrhage, infection, pneumonia, etc.
emergency)
❖ ruptured uterus(emergency)
❖ placenta previa Fetal risks
❖ abruptio placenta. ❖ Inadvertent delivery of premature fetus
(miscalculation of dates)
❖ Increases incidence of neonatal respiratory
Induction of Labor
unfavorable
Never schedule an induction without
asking the bishop score.
Artificial rupture of membranes (AROM) – amniotomy
❖ Causes release of prostaglandins, which enhance
Causes
❖ Pressure of baby moving
through the birth canal against
a previous uterine scar.
❖
❖
abnormal presentation
prolonged labor Nursing interventions
❖ multiple gestation
❖ improper use of oxytocin
❖ traumatic effects of forceps use ❖ The nurse should prepare IV fluid
or traction.
replacement.
❖ IV oxytocin administration .
❖ Laparotomy to control the
bleeding and repair the rupture.
❖ Cesarean hysterectomy or tubal
ligation
Postpartum care
Maternal Adaptation During
Postpartum Period Weight loss
Physiologic adaptation
Reproductive system ❖ Immediate 12 to 14 lb = baby, placenta, and
❖ Uterine contraction leads to amniotic fluid
involution(shrinking of the uterus ❖ 5 to 15 lb (early postpartum) = fluid loss from
❖ Measured by assessing fundal height diaphoresis, urinary excretion
❖ Factors promote, inhibit involution ❖ Return to prepregnancy weight six months after
❖ Afterpains childbirth (if within recommended weight gain
range)
❖
exercises
Lactation can lead to vaginal dryness, Nursing management & Discharge
Breasts
dyspareunia (painful intercourse)
planning
❖ Colostrum; prolactin ❖ Preventing injury from Rh-negative blood type or non immunity
Cardiovascular system to rubella
❖ High plasma fibrinogen levels and other ❖ Is the woman a candidate for Rho(D) immune globulin
coagulation factors mark postpartum period (RhoGAM) I'M within 24 hours of delivery. Given and checked
Vital signs as if you were administering a blood product.
❖ Temperature may be slightly elevated first 24 ❖ Providing patient teaching
❖ Breast care; fundal massage
hours
❖ Perineum and vaginal care
❖ Slow pulse a first, then WNL first week post
❖ Pain management
delivery ❖ Nutrition; constipation
❖ Blood pressure should not be elevated ❖ Proper rest
For every 250 mL of blood loss, the hemoglobin and ❖ Stress importance of prioritizing self-care
hematocrit (H&H) fall by one and two points, ❖ MMR (subq) right before DC if needed
respectively. So, if the woman’s H&H were 12 and 34, ❖ Evaluation: Goals and expected outcomes
then fall to 10 and 30, the approximate blood loss is
500 mL.
❖ Musculoskeletal system: Abdomen is soft,
sagging immediately postpartum
❖ Gastrointestinal system: Very hungry;
constipation
❖ Urinary system: Transient glycosuria,
proteinuria, and ketonuria are normal
immediately postpartum
❖ Voiding issues
❖ Integumentary system: Copious diaphoresis
common
❖ Striae (stretch marks) on abdomen and breasts
Newborn adaptation
Respiratory system
The Birth process:
❖ Helps expel fetal lung fluid
❖
Thermoregulatory adaptation
Stimulates lung inflation
❖ Stimulates surfactant
production
❖ Surfactant keeps alveoli from ❖ Thermoregulation is the physiologic process
collapsing after expansion of balancing heat production with heat loss
to maintain adequate body temperature
Circulation through the heart ❖ Newborn thermoregulation difficulties
❖ Fetal circulation ❖ Prone to heat loss due to lower proportion of
❖ High pressure in the lungs heat-producing tissue
causes pressure in right atrium ❖ Not readily able to produce heat
> left atrium ❖ Vulnerable to cold stress
Pressure differences help route
blood:
❖ Through the foramen ovale, Newborn loses heat in four ways:
❖ Conduction—body heat transfers to cold
ductus arteriosus object, infant placed in cold scale
❖ Away from non functioning ❖ Convection—air currents blow over infant’s
lungs body, infant susceptible to draft
❖ Back into general circulation ❖ Evaporation—wet skin dries and
❖ Ductus venosus shunts fetal evaporates
❖ Radiation—cold object close but not
blood away from the liver touching, infant close to cold windowpane
causing body heat to radiate to window
Causes
miscarriage.
❖ Chronic conditions. Women who have a chronic
Chromosomal abnormalities might lead to: condition, such as uncontrolled diabetes, have a
higher risk of miscarriage.
❖ Blighted ovum. Blighted ovum occurs when no ❖ Uterine or cervical problems. Certain uterine
embryo forms. abnormalities or weak cervical tissues (incompetent
❖ Intrauterine fetal demise. In this situation, an cervix) might increase the risk of miscarriage.
embryo forms but stops developing and dies ❖ Smoking, alcohol and illicit drugs. Women who
before any symptoms of pregnancy loss occur. smoke during pregnancy have a greater risk of
❖ Molar pregnancy and partial molar pregnancy. miscarriage than do nonsmokers. Heavy alcohol use
With a molar pregnancy, both sets of and illicit drug use also increase the risk of
chromosomes come from the father. A molar miscarriage.
pregnancy is associated with abnormal growth of ❖ Weight. Being underweight or being overweight has
the placenta; there is usually no fetal been linked with an increased risk of miscarriage.
development. ❖ Invasive prenatal tests. Some invasive prenatal
❖ Uncontrolled diabetes genetic tests, such as chorionic villus sampling and
❖ Infections amniocentesis, carry a slight risk of miscarriage.
❖ Hormonal problems ❖
❖ Uterus or cervix problems
Treatment
❖ Thyroid disease
Prevention
❖ Bed rest
❖ If it is sure that a miscarriage is
❖ Seek regular prenatal care. happening : prepare to start an IV,
administer blood & D&C
❖ Avoid known miscarriage risk factors
— such as smoking, drinking alcohol
and illicit drug use.
❖ Take a daily multivitamin.
❖ Limit your caffeine intake. A recent
study found that drinking more than
two caffeinated beverages a day
appeared to be associated with a
higher risk of miscarriage.
Complications: Ectopic pregnancy
What Am I ? ASSESSMENT Diagnostics
❖ Pain ❖ H&H: Low if rupture occurs.
Fetal growth somewhere
❖ Referred shoulder pain ❖ Diagnosable with ultrasound.
outside of the uterus, usually
❖ Spotting ❖ Human chorionic gonadotropin
within the fallopian tubes.
❖ Bleeding into the peritoneum level (serum) (hCG) is abnormally
❖ Bleeding from vagina if rupture low; when the test is repeated in
occurs 48 hours, the level remains lower
Causes ❖ Normal signs/ symptoms of
pregnancy
than usual for a normal
(intrauterine) pregnancy.
❖ Congenital defects in reproductive tract ❖ Progesterone level (plasma) is
❖ Diverticula lower than expected for an
❖ Ectopic endometrial implants in the tubal
intrauterine pregnancy.
mucosa
Risk Factors
❖ Endosalpingitis
❖ History of multiple elective abortions
❖ Intrauterine device
❖ Previous surgery, such as tubal ligation ❖ History of tubal surgery
or resection ❖ Previous ectopic pregnancy
❖ Sexually transmitted tubal infection ❖ History of pelvic inflammatory disease
❖ Transmigration of the ovum ❖ Pelvic adhesions
❖ Tumor pressing against the tube ❖ Use of intrauterine device
❖ Hormonal imbalance ❖ History of endometritis
❖ Progesterone-only contraceptive use
❖ Use of assisted reproductive technologies
❖
Complications
Diethylstilbestrol exposure in utero
❖ Cigarette smoking
❖ Age between 35 and 44
❖ Rupture of fallopian tube ❖ Multiple sexual partners
❖ Hemorrhage ❖ Vaginal douching
❖ Shock ❖ Young age at first sexual intercourse
❖ Peritonitis
❖ Infertility
Interventions
❖ Disseminated intravascular
coagulation
❖ Death
❖ Vital signs
Treatments
❖ Vaginal bleeding
❖ Pain level and effectiveness of
❖ Transfusion with whole blood or packed red blood interventions
cells to treat hypovolemic shock if the tube has ❖ Fluid balance status
ruptured. ❖ Intake and output
❖ IV fluid replacement ❖ Signs and symptoms of hypovolemia and
❖ Supplemental iron if anemia occurs from blood loss impending shock
❖ Methotrexate sodium (Trexall) as primary treatment ❖ Surgical site (postoperatively)
for unruptured ectopic pregnancy (single I.M. dose
or multidose treatment via I.M. or IV route)
❖ Leucovorin I.N. between doses of multi dose
methotrexate therapy
❖ Rho(D) immune globulin, human, if the patient is
Rh-negative
Complications : Placenta Previa
Assessment complications
What am i? ❖ Cord being the presenting part, possible
❖ Three types: Marginal, partial,
❖ Painless bleeding cord prolapse
and total
❖ Soft, nontender uterus ❖ Fetal hypoxia or blood loss
❖ Common cause of bleeding during
❖ Fetal malpresentation ❖ Preterm delivery
the second half of pregnancy
❖ Minimal descent of fetal ❖ Dystocia
❖ Good maternal prognosis if
presenting part ❖ Anemia
hemorrhage can be controlled
❖ Good fetal heart tones ❖ Hemorrhage
❖ Usually necessitates pregnancy
❖ Possible contractions ❖ Abruptio placentae
termination if bleeding is heavy
❖ Disseminated intravascular coagulation
❖ Fetal prognosis dependent on
❖ Shock
gestational age and amount of
❖ Placenta accreta, increta, percreta
blood lost; risk of death greatly
❖ Intrauterine growth restriction
reduced by frequent monitoring
❖ Abnormal fetal presentation
and prompt management
❖ Kidney damage
❖ Cerebral ischemia
❖ Maternal or fetal death
Patho
Improper implantation of the placenta
in the lower uterine segment has Medications
caused partial or total coverage of the
cervical os.With development of the ❖ I.V. fluids, such as lactated Ringer solution
lower uterine segment and gradual or normal saline solution, using a large-bore
changes in the cervix during the third catheter
trimester, shearing forces at the ❖ Oxygen
attachment site lead to partial
❖ Fresh frozen plasma and platelets, as
detachment and bleeding.
necessary, for coagulation problems
❖ Tocolytics, such as terbutaline sulfate,
Risk Factors
calcium channel blockers, or magnesium
sulfate short-term to halt preterm labor and
to allow time for doses of betamethasone
❖ Advanced maternal age (over age 35) dipropionate (Diprolene)
❖ Defective vascularization of the decidua ❖ Betamethasone dipropionate to enhance
❖ Endometriosis fetal lung maturity if less than 34 weeks'
❖ Multiparity Interventions
gestation
❖ Infertility treatments ❖ Pad counts, the patient should not saturate
❖ Multiple pregnancy more than one pad an hour.
❖ Previous uterine surgery or cesarean birth ❖ Monitor blood counts
❖ Monitor fetal heart tones
❖ Smoking
❖ Monitor contractions
❖ Male fetus ❖ Prepare for c-section
❖ Cocaine use ❖ Do not perform cervical exams
❖ History of placenta previa
❖ High altitudes
❖ Uterine abnormalities inhibiting normal
embryonic implantation (such as prior
curettage or the presence of uterine
fibroids)
Complications: Abruptio Placenta
What Am I? Assessment
❖ Premature separation of the ❖
❖
Rigid board-like abdomen
Abdominal pain
Priorities
placenta from the uterine wall ❖ Keep baby safe,
❖ Difficulty palpating baby.
❖ Usually occurs after 20 weeks' ❖ Signs of fetal distress: prolonged continuous monitoring
gestation, most commonly during fetal bradycardia, repetitive late ❖ Manage maternal shock
the third trimester, and peaks at decelerations, and decreased
24 to 26 weeks' gestation short-term variability; absent fetal
heart tones
Complications
❖ Common cause of bleeding
❖ Uterine hypertonicity
during the second half of ❖ Abdominal tenderness
pregnancy
❖ Cesarean delivery
PATHo ❖ Hemorrhage/coagulopathy
Improperly implanted placenta ❖ Retroperitoneal
separates before the pregnancy bleed/bleeding into the
reches term. If the abruption is abdomen
classified as concealed it is bleeding ❖ Shock
into the uterus.
Can be classified on scale of 0-3, 3
❖ Acute kidney injury
Causes
being the worst prognosis. ❖ Disseminated intravascular
coagulation (DIC)
❖ Car accidents ❖ Adult respiratory distress
❖ Domestic or IPV
syndrome
❖ Previous C-section
❖ Rupture of membranes ❖ Multisystem organ failure
❖ Cocaine use ❖ Maternal death
❖ Smoking ❖ Fetal hypoxia or asphyxia
❖ Pregnancy induced ❖ Precipitous labor and
hypertension
delivery
❖ Prematurity
❖ Fetal death
❖ Sepsis
Interventions
❖ Insert an indwelling urinary : monitor urine Labs
output.
❖ Obtain blood specimens for Hb level and ❖ Serum hemoglobin level test and
hematocrit, coagulation studies, and typing and platelet count are decreased.
crossmatching. ❖ Fibrin degradation products test
❖ Evaluate the extent and amount of bleeding; shows progression of abruptio
perform a pad count, placentae and indicates the
❖ Provide continuous external electronic fetal presence of DIC.
monitoring if the fetus is viable. ❖ Hypofibrinogenemia suggests
severe abruption (fibrinogen
❖ Give I.V. fluids and blood products. Maintain
levels less than or equal to 200
one to two large-bore I.V. lines; inspect I.V.
mg/dL).
insertion sites frequently for signs and
❖ Kleihauer-Betke test is positive if
symptoms of inflammation or infiltration.
fetal-maternal transfusion has
Provide I.V. site care according to your facility's occurred.
policy. ❖ Rho(D) antibody screening is
❖ Position the patient on her left side to enhance positive if isoimmunization has
uteroplacental perfusion. occurred.
❖ Administer oxygen, as ordered, on the basis of
pulse oximetry levels and respiratory status.
❖ Prepare the patient for emergency delivery, as
appropriate.
What is it?
Hyperemesis gravidarum
Excessive vomiting that
leads to dehydration,
starvation, and even Assessment Interventions
death among pregnant ❖ Hypotension ❖ 48 hours of NPO status
populations ❖ Elevated H&H ❖ IV fluids
Related to increased ❖ Decreased urine ❖ Antiemetics as ordered
estrogen levels output ❖ Vitamins
❖ Hypokalemia ❖ Decrease environmental
❖ Weight loss stimuli
❖ Ketonuria ❖ Clear liquids and small dry
feedings as tolerated.
❖ Give either cold or hot food,
nothing room temp.
Critical labs
❖ Potassium
❖ Have the patient on tele
❖ Monitor for symptoms of
shock and fluid volume
deficit.
Complications
❖ Dehydration
❖ Wernicke's encephalopathy from
vitamin B1 deficiency
❖ Mallory-Weiss tears (esophageal
tears and bleeding)
❖ Esophageal bleeding
❖ Pneumothorax
❖ Acute tubular necrosis
❖ Electrolyte and acid‑base
imbalances
Diagnostic studies
❖ Potassium, sodium, chloride, and protein levels
are decreased due to losses from vomiting.
❖ Blood urea nitrogen, non protein nitrogen, and
uric acid levels are increased due to renal
compromise and hemoconcentration.
Hemoglobin (Hb) level and hematocrit (HCT) are
increased due to hemoconcentration.
❖ Urinalysis reveals ketones and, possibly, protein;
urine specific gravity increases.
❖ Vitamin B1 and B6 levels are decreased due to
impaired intake.
❖ Thyroid-stimulating hormone, thyroxine, and
triiodothyronine levels may be mildly increased.
What is it?
Extreme elevation in
blood pressure during
preeclampsia
pregnancy with the
presence of protein in the Assessment Types
urine after 20 weeks of ❖ Sudden weight gain ❖ Mild : 30/15 mmhg off of baseline
❖ Swelling of the face and six hours apart. Increase the
gestation.
hands amount of protein in the diet
❖ Headache
because they are spilling it into the
❖ Blurry vision
❖ Hyperreflexia urine. Glomerular damage is
❖ Clonus ( seizures) present.
❖ Severe: 160/110 mmHg 6 hours
apart. May have an episode of
seizure activity. Have mag sulfate
ready.
Interventions
Magnesium sulfate ❖ Magnesium sulfate : have
calcium gluconate at
❖ Vasodilator & sedative bedside.
❖ Monitor for pulmonary ❖ Monitor for sedation and
edema hyporeflexia.
❖ Seizure precautions
❖ Monitor for signs of mag
❖ Safety checks
toxicity: decreased
DTRS, BP, respiration,
Risk factors
decreased LOC.
❖ Labor will halt: have
oxytocin ready if ❖ History of preeclampsia.
indicated. ❖ Chronic hypertension.
❖ First pregnancy.
❖ New paternity
Care
❖ Age. The risk of preeclampsia is higher for very
young pregnant women as well as pregnant
❖ Decrease environmental
women older than 40.
stimuli. This decreases the
risk of seizures. ❖ Obesity.
❖ Initiate seizure precautions ❖ Multiple pregnancy. Preeclampsia is more
❖ Monitor mom and baby common in women who are carrying twins,
triplets or other multiples.
❖ Interval between pregnancies. Having babies
less than two years or more than 10 years apart
leads to a higher risk of preeclampsia.
❖ In vitro fertilization. Your risk of preeclampsia is
increased if your baby was conceived with in
vitro fertilization.
Nclex tip!
Blood pressure that exceeds
140/90 millimeters of mercury
(mm Hg) or
greater,documented on two
occasions, at least four hours
apart , is abnormal.
PRETERM LABOR Assessment
What is it? ❖ Regular or frequent sensations of
abdominal tightening (contractions)
Onset of labor anywhere
between 20-37 weeks ❖ Constant low, dull backache
gestation. ❖ A sensation of pelvic or lower abdominal
pressure
❖ Mild abdominal cramps
❖ Vaginal spotting or light bleeding
❖ Preterm rupture of membranes — in a
gush or a continuous trickle of fluid after
the membrane around the baby breaks
Risk factors or tears
❖ Previous preterm labor or premature birth, ❖ A change in type of vaginal discharge —
particularly in the most recent pregnancy or watery, mucus-like or bloody
in more than one previous pregnancy
❖ Pregnancy with twins, triplets or other
multiples
❖ Problems with the uterus, cervix or placenta
❖ Smoking cigarettes or using illicit drugs Prevention
❖ Certain infections, particularly of the ❖ Seek regular prenatal care.
amniotic fluid and lower genital tract ❖ Eat a healthy diet
❖ Some chronic conditions, such as high ❖ Avoid risky substances.
blood pressure and diabetes ❖ Consider pregnancy spacing.
❖ Stressful life events, such as the death of a ❖ Be cautious when using assisted reproductive
loved one technology (ART).
❖ Too much amniotic fluid (polyhydramnios)
❖ Vaginal bleeding during pregnancy
❖ Presence of a fetal birth defect
❖ An interval of less than six months between
pregnancies
❖ Infection of tissues that surround and
support your teeth (periodontal disease)
Treatment
❖ Terbutaline: Tocolytic
❖ Magnesium sulfate
❖ Betamethasone : to stimulate maturation of
babies lungs.
❖ Hydration
❖ Treatment of UTI
Prolapsed cord
Diagnosis
What is it Umbilical cord prolapse should always be
Umbilical cord prolapse considered a possibility when there is a
occurs when the umbilical sudden decrease in fetal heart rate or
cord comes out of the uterus variable decelerations, particularly after the
with or before the presenting rupture of membranes. With overt
part of the fetus. It is a prolapses, the diagnosis can be confirmed
relatively rare condition and if the cord can be palpated on vaginal
occurs in fewer than 1% of examination. Without overt prolapse, the
pregnancies. Cord prolapse is diagnosis can only be confirmed after a
more common in women who cesarean section, though even then it will
have had rupture of their not always be evident at time of procedure
amniotic sac
Treatments
❖ Lift the baby's head off the cord.
❖ Trendelenburg or knee chest
position
❖
Causes
Hyperoxygenate mom
❖ Never push the cord back in
❖ Premature delivery of the baby
❖ Delivering more than one baby
per pregnancy (twins, triplets,
etc.)
❖ Excessive amniotic fluid
❖ Breech delivery (the baby comes
❖
through the birth canal feet first)
An umbilical cord that is longer
Management
than usual ❖ Monitor fetal heart tones
❖ manual elevation of the presenting fetal part
❖ repositioning of the mother to be head down
with feet elevated
❖ filling of the bladder with a foley catheter, or
tube through the urethra to elevate the
presenting fetal part
NCLEX Tip
❖ use of tocolytics (medications to suppress labor)
have been proposed, usually in addition to
If the cord stops pulsating fetal bladder filling rather than a standalone
death has occured.
intervention
Assessment of Growth and Development of the Infant
Head Should measure 13.75 cm at birth
-Posterior fontanelle should close by 2nd month Well checkup schedule
-Anterior fontanelle should close in 12-18 months
❖ Second week of life
Height and weight ❖ 2, 4, 6, 9 months of age.
- In the first 6 months birth weight doubles and baby should grow 6 inches
- By 12 months birth weight should triple and baby should grow 10-12 in. Vitals
Skelton ❖ HR: 70 resting - 180 awake and crying
- Is made up of cartilage at 3 month gestation and continues to ossify and grow ( accurate HR is taken apically)
throughout life.
❖ RR: 30 but can range from
- Bone age, injury, abuse or nutritional deficits can be determined by X-Ray.
Circulation
20-50 with increase or
decrease of activity.
- Hemoglobin and RBCs decrease when respiratory system takes over until
3 months of age
❖ BP: 85/60 mmHg
❖
Neuro
Temp: 98.6
- Nerve cells grow and coordination begins in an orderly pattern.
Language Nutrition
❖ 0-3 mo: Cries, grunts and Rapid growth causes a need for the greatest amount of nutrients
Coo ❖ 4-6 mo- 12 mo: breast milk or commercial formula, introduction of solid foods.
❖ 0-6 mo: babbling, vowels, One food at a time starting with veggies.
❖ You may need to supplement Vit C/D iron, fluoride.
half consonants
❖ 6mo: iron rich foods are needed to supplement
❖ 12 mo: 1-2 words, imitation, ❖ 7-8 mos: self feeding begins by grasping and bringing food to mouth. Ends with
responding to simple use of utensils
commands ❖ WIC program helps children and women get proper nutrition when they qualify
❖
Height and weight ❖ Put away poisons and ❖ Tell child the behavior is bad, not them
medications with locks
- Gain 5 to 10 LBs per year
- Grow 3 inches per year
❖ Burns from hot Well visits
appliances and water are
- Normal to go on food jags ❖ 15 mo for shots
common
❖ Annually after that
Learns to stand alone and walk, ❖ Assess growth/ development, caregiver skill,and
1 year Need 12-14 hours a day of sleep relationship between toddler and parent
3 years need 10-12 hours
Language Nutrition
❖ Vocabulary begins to increase names objects, ❖ Require about 1000-1400 calories a day
body parts, animals, and familiar locations ❖ Toddlers should be active 60 min a day
❖ Primary method of communication ❖ Fruites: 1-1.5 cups
❖ Continuous questioning “why” ❖ Veggies: 1-1.5 cups
❖ Toys that talk are preferred ❖ Grains : 3-5 oz
❖ Brief sentences ❖ Protein : 2-4 oz
❖ Dairy : 2-2.5 cups
❖ Allow children to eat when hungry instead of forcing
meals.
Language Nutrition
❖ 3-4: non communicative w/ ❖ Do not need large quantities of food, keep portions small.
language ❖ Requires high amount of protein
❖ 4: communicate with language ❖ Erratic appetite, frequent small meals are better
❖ 4-5: use naughty words ❖ Guide them when choosing food
❖ Converse in a way they can ❖ Provide healthy snacks
understand ❖ Rituals are important.
❖ Delays can be caused by:
hearing impairment, lack of
stimulation,
Erik Erikson Initiative vs guilt Child develops a Monitor and protect from
3-5 yrs conscious and sense injury and poisoning.
of right and wrong Encourage them to ask
questions
Jean Piaget Pre operational The child sees the Plan drawing and writing
2-7 yrs world egocentrically activities.
phase
Growth and Development of the school aged CHILD
Head Should measure 13.75 cm at birth Well checkup schedule
❖ School starts
❖ Thinking skills develop ❖ Annual physicals
Social Milestones
❖ Develop confidence in family and explore Emotional growth
relationships outside of family ❖ Greater understanding of complex emotions
❖ Peers become important ❖ Understands they can have more than one
❖ Motivated by accomplishment emotion
❖ success/ failure have a strong impact ❖ Greater ability to control emotion
❖ Uses strategies to redirect feelings
Language Nutrition
❖ Language is refined vis ❖ Requires more food for increased energy demands
grammar education ❖ Choose foods from all food groups
❖ Ability to use words to express ❖ Food jags and increased appetite are normal
knowledge ❖ Limit fat intake, supervise snack habits
❖ Narrative skills improve ❖ Offer choices.
❖ Able to make inferences
❖ Able to evaluate speech and
messages
Jean Piaget Pre operational The child sees the Plan drawing and writing
2-7 yrs world egocentrically activities.
phase
Assessment of Growth and Development of the Adolescent
11-18yrs
Early adolescents
Well checkup schedule
❖ -puberty : reproductive maturity
❖ - starts at age 10 in girls and ends with menstruation
❖ - starts age 12 in boys and ends sperm production ❖ 2x during teens
❖ Immunizations, hearing, vision,
Growth scoliosis, thyroid and pelvic for
sexually active girls..
❖ Girls achieve 98% of height of 16 ❖ BP, height and weight
❖ Skeletal growth outpaces muscle growth
❖ Nonvoluntary with early menses, 13-15 ovulation begins
❖ Boys : grow rapidly from 13-20
❖ Muscle strength and coordination develop rapidly
❖ Larynx becomes enlarged
❖ Both sexs : body takes on contours
❖ Primary sex organs develop, hormonal activity increases
❖ Bone growth continues until 20s
❖ Girls : 9-11: growth spurts last 18 months ❖ They wonder who they are
❖ Grows 3 in annually until menarche ❖ What will they become
❖ Begin to develop figure ❖ More mobile
❖ Boys: 11-13: slower and steadier than girls ❖ Seek out intimate relationships
❖ Changes in penis testes and scrotum ❖ Most are heterosexual, homosexuality can be
❖ Nocturnal emissions “ wet dreams” difficult emotionally
❖ Body image is closely related to self esteem
❖ Underdevelopment causes anxiety
Language Nutrition
● Able to communicate Rapid growth causes a need for the greatest amount of nutrients
complex thoughts ● Appetite increases and teens eat frequently
● Food choices not always wise
● May skip meals
● Can have nutritional deficits of vit A, D and B. folic acid, iron and zinc. Due to
menses girls need additional iron.
❖ Adequate Pulses
❖ adventitious breath sounds ❖ strength and regularity
❖ central vs. peripheral
Respiratory Distress
❖ Tachypnea Perfusion
Mechanics of breathing ❖ capillary refill
❖ skin color (e.g. pale, mottled)
❖ Retractions
❖ Tracheal Tug Cardiovascular Collapse
❖ Nasal flaring ❖ Tachycardia
❖ Head bobbing ❖ Altered perfusion
❖ Grunting on exhalation
❖ Prolonged expiratory phase Skin
❖ Diminished air entry ❖ Prolonged capillary refill > 2 sec
❖ Change in breath sounds ❖ Increased core to skin temperature gradient
❖ Stridor
❖ Wheezing
Brain
Neurological
❖ Level of consciousness Late signs
❖ Decreased response to pain
❖ Mental status,
❖ Flaccid tone
interaction
❖ Hypotension
❖ Activity, movement,
❖ Bradycardia
muscle tone
❖ Age appropriate
responses
Vital signs
GI/GU
❖ Bowel sounds
Newborn ❖ Appetite
❖ Resp: 30 - 50
❖ Bowel movement
❖ Heart: 120 - 160
/Emesis
Infant (1-12 months)
❖ Resp: 20 - 30 Hydration status
❖ heart: 80 - 140
Toddler (1-3 yrs.) ❖ Urine output
❖ Resp: 20 - 30 ❖ Moist oral mucosa
❖ Heart: 80 - 130 ❖ Skin Turgor
Preschooler (3-5 yrs.) ❖ Fontanelle
❖ Resp: 20 - 30
❖ Heart: 80 - 120
LARYNGOTRACHEOBRONCHITIS
WHAT IS IT? Assessment Treatment
Also known as croup, a type of ❖ Steam
❖ "barking" cough,
respiratory infection that is usually ❖ Car Rides with
stridor
caused by a virus.The infection leads windows down at night
to swelling inside the trachea, which ❖ hoarseness,
❖ difficulty breathing ❖ Cool down the
interferes with normal breathing and environment to open
produces the classic symptoms of which usually
up the airway and
"barking" cough, stridor, and a worsens at night
blood vessels.
hoarse voice. Fever and runny nose
may also be present.[1] These
symptoms may be mild, moderate,
or severe. Often it starts or is worse
at night. It normally lasts one to two
days
Medical management
❖ Nebulized epinephrine
❖ Corticosteroids
Respiratory syncytial virus: Bronchiolitis
What am I ? Assessment Diagnostics
Respiratory syncytial virus (RSV) ❖ Sneezing ❖ Secretions from the
is a very common virus that leads ❖ Rhinorrhea nose and throat are
to mild, cold-like symptoms in ❖ Low grade fever cultured.
adults and older healthy children. ❖ Coughing ❖ ELISA test
It can be more serious in young ❖ Wheezing ❖ IFA test
babies, especially those in certain ❖ Retractions
high-risk groups ❖ Nasal flaring
❖ Dyspnea
❖ Prolonged expiratory phase
❖ Intermittent cyanosis
Patho
Infection that inflames the air
sacs in one or both lungs. The air Treatments
sacs may fill with fluid or pus
❖ Antibiotics for bacterial
CLASSIFICATIONS
(purulent material), causing cough
pneumonia after you
with phlegm or pus, fever, chills,
❖ Lobular: Large portion of collect the sputum
and difficulty breathing. A variety
lung involvement sample.
of organisms, including bacteria,
❖ Interstitial : Involves the ❖ Hydration
viruses and fungi, can cause
alveolar walls, ❖ Nebulizers
pneumonia.
peribronchial, and ❖ 02 therapy
interlobular tissue. ❖ Chest physiotherapy
❖ Bronchial: involves the
bronchi and lung fields
Risk factors
❖ Cigarette smoking
❖ Recent viral respiratory infection—a cold,
laryngitis, influenza, etc.
❖ Difficulty swallowing (due to stroke, dementia,
Parkinson's disease, or other neurological
conditions), which can lead to aspiration
(breathing in a foreign object)
❖ Chronic lung disease such as COPD,
bronchiectasis, or cystic fibrosis
❖ Cerebral palsy Interventions
❖ Other serious illnesses, such as heart ❖ Airway is the priority
disease, liver cirrhosis, or diabetes ❖ Oxygen therapy
❖ Living in a nursing facility ❖ Increase fluids to
❖ Impaired consciousness (loss of brain increase sputum
function due to dementia, stroke, or other production and thin
neurologic conditions) secretions
❖ Recent surgery or trauma ❖ Antibiotics for bacterial
❖ Having a weakened immune system due to pneumonia
illness, certain medications, and autoimmune ❖ Supportive care
disorders
asthma
What am I ? Assessment
A respiratory condition marked
by spasms in the bronchi of the ❖ Expiratory wheezing
lungs, causing difficulty in ❖ Chronic cough Diagnostics
breathing. It usually results from ❖ Dyspnea ❖ Decreased peak
an allergic reaction or other forms ❖ Nonproductive cough expiratory flow rates
of hypersensitivity. ❖ Tachypnea ❖ Pulmonary function
❖ Irritability test
❖ Restlessness
❖ Use of accessory muscles to
breathe
❖ Orthopnea
Treatments
Patho W: Wheezing ❖ Corticosteroids
Characterized by chronic ❖ Cromolyn
E: Extra cranky
inflammation , ❖ Metered dose inhaler
E: EXtra fast breathing
bronchoconstriction, and ❖ Salmeterol
Z: Zones Of muscles ( accessory muscle
bronchial ❖ Methylxanthines
use)
hyperresponsiveness. Most ❖ Anticholinergics
Y: Yes they cough
common respiratory disorder ❖ Leukotriene modifiers
in childhood. Do not give beta blockers to an
asthmatic
Triggers
❖ air pollutants
❖
❖
strong odors
seasonal and perennial allergens
Interventions
❖ stress and emotional distress ❖ High-Fowler's
❖ medications (aspirin, NSAIDS, beta ❖ O2 therapy
blockers, cholinergics) ❖ Monitor cardiac rate and rhythm
❖ enzymes (laundry detergents) for changes during acute attack
❖ chemicals (cleaners) ❖ Initiate and maintain IV
❖ sinusitis with post nasal drip
❖ viral respiratory tract infection
STATUS Asthmaticus
life threatening episode of airway
obstruction that is often unresponsive
to common treatment
-extreme wheezing, labored
breathing, use of accessory muscles,
distended neck veins
-creates risk for cardiac and /or
respiratory arrest
Acute epiglottitis
What am I ?
Acute infection of
Assessment
Treatments
supraglottic structures
resulting in edema and
Remember the 4 D’s ❖ Antipyretics
swelling cause the upper
❖ Drooling ❖ Antibiotics ( swelling of
airway to be obstructed
❖ Dysphasia epiglottis usually subsides,
A flap of cartilage at the
❖ Dysphonia 24 hrs after) c
root of the tongue, which is
❖ Distress ❖ Constant monitoring of
depressed during
swallowing to cover the respiratory status
Respiratory distress ❖ o2 humidification
opening of the windpipe.
❖ nasal flaring ❖ Intubation
❖ sternal/intercostal retraction ❖ let child sit in position they
❖ pale/blue find most comfortable
❖ tachycardia
Patho ❖
❖
increased restlessness
Tripod position with tongue
Epiglottitis is a potentially
protruding
life-threatening condition that
occurs when the epiglottis a
small cartilage "lid" that
covers your windpipe and
swells, blocking the flow of air
into your lungs.
.
Interventions
NEVER USE A TONGUE DEPRESSOR
Triggers TO ASSESS AIRWAY! THIS COULD
❖ Swallow a chemical that CAUSE SPASMS AND FURTHER
burns your throat BLOCK THE AIRWAY!
❖ Swallow a foreign object ❖ protect airway
❖ Smoke drugs, such as crack ❖ avoid throat culture or using
cocaine tongue blade
❖ Streptococcus infection ❖ prepare for intubation
❖ provide humidified oxygen
❖ monitor pulse ox
❖ Administer corticosteroids and IV
fluids
❖ Administer abx therapy starting
with IV then transition to oral to
Emergency management complete 10 day course
Respiratory arrest ❖ Droplet isolation precaution first
❖ Bag ventilation 24 hr after IV initiation
❖ Attempt to intubate ❖
❖ Preform needle or surgical
cricothyroidotomy
❖ Start IV- ceftriaxone or cefotaxime
❖ Racemic epi & steroids are
ineffective