List of human hormones

From Wikipedia, the free encyclopedia

The following is a list of hormones found in Homo sapiens. Spelling is not uniform for many
hormones. For example, current North American and international usage is estrogen,
gonadotropin, while British usage retains the Greek digraph in oestrogen and favors the earlier
spelling gonadotrophin (from trophē ‘nourishment, sustenance’ rather than tropē ‘turning,
change’).

Contents
 1 Amino acid
 2 Eicosanoid
 3 Peptide
 4 Steroid
 5 References

Amino acid

Target
Name Abbreviation Tissue Cells Receptor Effect
Tissue
blood pressure,
adrenal adrenergic nearly all
Epinephrine EPI glycogenolysis,
gland receptor tissues
lipolysis, etc.
CNS and
melatonin
Melatonin MT pineal gland peripheral circadian rhythm
receptor
tissue
peripheral
thyroid nearly every
tissue of increased
Triiodothyronine T3 hormone cell in the
thyroid metabolism
receptor body
gland
thyroid
thyroid similar effect as T3
Thyroxine T4 hormone
gland but much weaker
receptor

Eicosanoid

Target
Name Abbreviation Tissue Cells Receptor Effect
Tissue
seminal prostaglandin
Prostaglandins PG vasodilation
vesicle receptor
 white G protein-
increase vascular
Leukotrienes LT blood coupled
permeability
cells receptors
prostacyclin
Prostacyclin PGI2 endothelium
receptor
thromboxane
Thromboxane TXA2 platelets vasoconstriction
receptor

Peptide

Abbrevi Target
Name Tissue Cells Receptor Effect
ation Tissue
slowing down gastric
Amylin (or
pancreatic β- amylin emptying, inhibition of
Islet Amyloid IAPP pancreas
cells receptor digestive secretion, and
Polypeptide)
reducing food intake
Anti-
Müllerian
hormone (or Inhibit release of prolactin
Müllerian AMH testes Sertoli cell AMHR2 and TRH from anterior
inhibiting pituitary
factor or
hormone)
adiponect
adipose
Adiponectin Acrp30 in
tissue
receptors
synthesis of
Adrenocortic
ACTH corticosteroids
otropic anterior
ACTH corticotrope receptor (glucocorticoids and
hormone (or pituitary
→ cAMP androgens) in
corticotropin)
adrenocortical cells
vasoconstriction
Angiotensino angiotensi
release of aldosterone
gen and AGT liver n receptor
from adrenal cortex
angiotensin → IP3
dipsogen.
Antidiuretic ADH posterior Parvocellular AVPRs, retention of water in
hormone (or pituitary neurosecreto VACM-1 kidneys
vasopressin, ry neurons in moderate vasoconstriction
arginine hypothalamu Release ACTH in anterior
vasopressin) s pituitary
Magnocellul
ar
neurosecreto
ry cells in
posterior
 Abbrevi Target
Name Tissue Cells Receptor Effect
ation Tissue
pituitary
Atrial-
ANP
natriuretic
ANP heart receptor
peptide (or
→ cGMP
atriopeptin)
(To a minor degree than
ANP) reduce blood
Brain pressure by:
heart[dubious Cardiac
natriuretic BNP – discuss] NPR reducing systemic
myocytes
peptide vascular resistance,
reducing blood water,
sodium and fats
CT
thyroid parafollicula Construct bone, reduce
Calcitonin CT receptor
gland r cell blood Ca2+
→ cAMP
Release of digestive
enzymes from pancreas
Cholecystoki CCK
CCK duodenum Release of bile from
nin receptor
gallbladder
Hunger suppressant
Corticotropin
hypothala CRF1 → Release ACTH from
-releasing CRH
mus cAMP anterior pituitary
hormone
depression of neuronal
activity; induction of
slow-wave sleep;
Somatost
cerebral inhibitory reduction of locomotor
Cortistatin CORT atin
cortex neurons activity; activation of
receptor
cation selective currents
not responsive to
somatostatin
Chromaffin Opioid
Enkephalin Kidney Regulate pain
cells receptor
Vascular Smooth muscle
Endothelial ET
Endothelin endotheliu contraction of medium-
cells receptor
m sized vessels
Extraglomer
Erythropoieti ular Stimulate erythrocyte
EPO kidney EpoR
n mesangial production
cells
Follicle- FSH anterior gonadotrope FSH In female: stimulates
stimulating pituitary receptor maturation of Graafian
hormone → cAMP follicles in ovary.
 Abbrevi Target
Name Tissue Cells Receptor Effect
ation Tissue
In male: spermatogenesis,
enhances production of
androgen-binding protein
by the Sertoli cells of the
testes
central
nervous GALR1,
modulation and inhibition
system GALR2,
Galanin GAL of action potentials in
and and
neurons
gastrointes GALR3
tinal tract
mucosa of
Gastric the
inhibitory GIP duodenum K cell GIPR Induces insulin secretion
polypeptide and the
jejunum
stomach, Secretion of gastric acid
Gastrin GAS G cell CCK2
duodenum by parietal cells
Stimulate appetite,
ghrelin secretion of growth
Ghrelin stomach P/D1 cell
receptor hormone from anterior
pituitary gland
glycogenolysis and
Glucagon
gluconeogenesis in liver
Glucagon GCG pancreas alpha cells receptor
increases blood glucose
→ cAMP
level
Stimulates the adenylyl
Glucagon- GLP1R, pancreatic cyclase pathway, resulting
GLP1 ileum L cells
like peptide-1 GLP2R beta cells in increased synthesis and
release of insulin
Gonadotropi GnRH
hypothala Release of FSH and LH
n-releasing GnRH receptor
mus from anterior pituitary.
hormone → IP3
Growth
GHRH
hormone- hypothala Release GH from anterior
GHRH receptor
releasing mus pituitary
→ IP3
hormone
ferroporti inhibits iron export from
Hepcidin HAMP liver
n cells
Human hCG placenta syncytiotrop LH promote maintenance of
chorionic hoblast cells receptor corpus luteum during
gonadotropin → cAMP beginning of pregnancy
Inhibit immune response,
 Abbrevi Target
Name Tissue Cells Receptor Effect
ation Tissue
towards the human
embryo.
increase production of
Human insulin and IGF-1
placental HPL placenta increase insulin resistance
lactogen and carbohydrate
intolerance
stimulates growth and cell
Growth GH or anterior GH reproduction
somatotropes
hormone hGH pituitary receptor Release Insulin-like
growth factor 1 from liver
Sertoli cells
of testes
testes, granulosa
anterior
Inhibin ovary, cells of Inhibit production of FSH
pituitary
fetus ovary
trophoblasts
in fetus
Intake of glucose,
glycogenesis and
insulin glycolysis in liver and
receptor, muscle from blood
Insulin INS pancreas beta cells
IGF-1, intake of lipids and
IGF-2 synthesis of triglycerides
in adipocytes Other
anabolic effects
Insulin-like
growth factor insulin insulin-like effects
(or IGF liver Hepatocytes receptor, regulate cell growth and
somatomedin IGF-1 development
)
adipose decrease of appetite and
Leptin LEP LEP-R
tissue increase of metabolism.
lipolysis and
anterior Corticotrope steroidogenesis,
Lipotropin LPH
pituitary s stimulates melanocytes to
produce melanin
In female: ovulation
Luteinizing anterior gonadotrope LHR → In male: stimulates Leydig
LH
hormone pituitary s cAMP cell production of
testosterone
Melanocyte MSH or anterior Melanotroph melanoco melanogenesis by
stimulating α-MSH pituitary/p rtin melanocytes in skin and
 Abbrevi Target
Name Tissue Cells Receptor Effect
ation Tissue
ars receptor
hormone hair
intermedia → cAMP
Small Motilin
Motilin MLN stimulates gastric activity
intestine receptor
wakefulness and increased
hypothala
Orexin OX1, OX2 energy expenditure,
mus
increased appetite
Favors muscle function,
Muscle
memory formation,
Brain
Osteocalcin OCN Skeleton Osteoblastss Gprc6a testosterone synthesis and
Pancreas
energy expenditure[1]
Testes
release breast milk
Contraction of cervix and
Magnocellul vagina Involved in
OXT
posterior ar orgasm, trust between
Oxytocin OXT receptor
pituitary neurosecreto people.[2] and circadian
→ IP3
ry cells homeostasis (body
temperature, activity level,
wakefulness).[3]
Self-regulation of
pancreatic pancreatic secretions
Pancreatic polypepti (endocrine and exocrine).
Pancreas PP cells
polypeptide de It also affects hepatic
receptor 1 glycogen levels and
gastrointestinal secretions.
increase blood Ca2+:
 indirectly stimulate
osteoclasts
 Ca2+ reabsorption
in kidney
 activate vitamin D
PTH
Parathyroid parathyroi parathyroid (Slightly) decrease blood
PTH receptor
hormone d gland chief cell phosphate:
→ cAMP

 (decreased
reuptake in kidney
but increased
uptake from bones

 activate vitamin D)
Pituitary PACAP multiple ADCYAP Stimulates
 Abbrevi Target
Name Tissue Cells Receptor Effect
ation Tissue
adenylate
1R1,
cyclase- enterochromaffin-like
VIPR1,
activating cells
VIPR2
peptide
lactotrophs
of anterior milk production in
anterior
pituitary PRL mammary glands
Prolactin PRL pituitary,
Decidual receptor sexual gratification after
uterus
cells of sexual acts
uterus
Prolactin
hypothala Release prolactin from
releasing PRH
mus anterior pituitary
hormone
Corpus
luteum,
Uterus,
Decidual RLN
Relaxin RLN placenta, Unclear in humans
cells receptor
and
Mammary
gland
Activates the renin-
Juxtaglomer angiotensin system by
Renin Kidney
ular cells producing angiotensin I of
angiotensinogen
Secretion of bicarbonate
from liver, pancreas and
SCT duodenal Brunner's glands
Secretin SCT duodenum S cell
receptor Enhances effects of
cholecystokinin Stops
production of gastric juice
Somatostatin SRIF hypothala delta cells in Somatost Inhibit release of GH and
mus, islets islets atin TRH from anterior
of Neuroendocr receptor pituitary
Langerhan ince cells of Suppress release of
s, the gastrin, cholecystokinin
gastrointes Periventricul (CCK), secretin, motilin,
tinal ar nucleus in vasoactive intestinal
system hypothalamu peptide (VIP), gastric
s inhibitory polypeptide
(GIP), enteroglucagon in
gastrointestinal system
Lowers rate of gastric
emptying
Reduces smooth muscle
 Abbrevi Target
Name Tissue Cells Receptor Effect
ation Tissue
contractions and blood
flow within the intestine[4]
Inhibit release of insulin
from beta cells[5]
Inhibit release of glucagon
from alpha cells[5]
Suppress the exocrine
secretory action of
pancreas.
liver,
Thrombopoie kidney, TPO megakary
TPO Myocytes produce platelets[6]
tin striated receptor ocytes
muscle
Thyroid- Thyrotrop
stimulating anterior in thyroid secrete thyroxine (T4) and
TSH thyrotropes
hormone (or pituitary receptor gland triiodothyronine (T3)
thyrotropin) → cAMP
Release thyroid-
Thyrotropin- Parvocellular
hypothala TRHR → anterior stimulating hormone
releasing TRH neurosecreto
mus IP3 pituitary (primarily)
hormone ry neurons
Stimulate prolactin release
gut,
stimulates contractility in
pancreas,
the heart, causes
and Vasoactiv
vasodilation, increases
Vasoactive suprachias e
glycogenolysis, lowers
intestinal VIP matic intestinal
arterial blood pressure and
peptide nuclei of peptide
relaxes the smooth muscle
the receptor
of trachea, stomach and
hypothala
gall bladder
mus
guanylate
cyclase
2C (heat regulates electrolyte and w
Guanylin GN gut stable ater transport
enterotoxi in intestinal epithelia.
n
receptor)
guanylate
cyclase
2C (heat regulates electrolyte and w
renal
Uroguanylin UGN stable ater transport in renal
tissues
enterotoxi epithelia.
n
receptor)
Steroid

Targ
Chemical Abbrevi Recep et
Name Tissue Cells Effect
class ation tor Tiss
ue
libido, Anabolic:
growth of muscle
mass and strength,
increased bone
density, growth and
strength,
androgen Testosterone testes Leydig cells AR Virilizing:
maturation of sex
organs, formation of
scrotum, deepening
of voice, growth of
beard and axillary
hair.
Zona
fasciculata
and Zona
reticularis
testes,
Dehydroepiandr cells of Virilization,
androgen DHEA ovary, AR
osterone kidney anabolic
kidney
theca cells of
ovary
Leydig cells
of testes
adrenal
Substrate for
androgen Androstenedione glands, AR
estrogen
gonads
androgen Dihydrotestoster DHT multiple AR 5-DHT or DHT is a
one male reproductive
hormone that targets
the prostate gland,
bulbourethral gland,
seminal vesicles,
penis and scrotum
and promotes
growth/mitosis/cell
maturation and
differentiation.
Testosterone is
converted to 5-DHT
by 5alpha-reductase,
 Targ
Chemical Abbrevi Recep et
Name Tissue Cells Effect
class ation tor Tiss
ue
usually with in the
target tissues of 5-
DHT because of the
need for high
concentrations of 5-
dht to produce the
physiological
effects.
Increase blood
adrenal volume by
cortex reabsorption of
mineralocor
Aldosterone (zona MR sodium in kidneys
ticoid
glomerul (primarily)
osa) Potassium and H+
secretion in kidney.
estrogen Estradiol E2 females: females: ER Females:
ovary, granulosa
males cells, males: Structural:
testes Sertoli cell
 promote
formation of
female
secondary
sex
characteristi
cs
 stimulate
endometrial
growth
 increase
uterine
growth
 maintenance
of blood
vessels and
skin
 reduce bone
resorption
 increase
 Targ
Chemical Abbrevi Recep et
Name Tissue Cells Effect
class ation tor Tiss
ue
hepatic
production
of binding
proteins

Coagulation:

 increase
circulating
level of
factors 2, 7,
9, 10,
antithrombin
III,
plasminogen
 increase
platelet
adhesiveness

Fluid balance:

 salt (sodium)
and water
retention
 increase
growth
hormone
 increase
cortisol,
SHBG

Gastrointestinal
tract:

 reduce
bowel
motility
 increase
cholesterol
in bile
 Targ
Chemical Abbrevi Recep et
Name Tissue Cells Effect
class ation tor Tiss
ue

Lung function:

 promote
lung
function by
supporting
alveoli.[7]

Males: Prevent
apoptosis of germ
cells[8]
granulosa
estrogen Estrone ovary cells, ER
Adipocytes
syncytiotrop
estrogen Estriol E3 placenta ER
hoblast
Stimulation of
gluconeogenesis
Inhibition of
adrenal
glucose uptake in
cortex
muscle and adipose
(zona
tissue Mobilization
glucocortico fasciculat
Cortisol GR of amino acids from
id a and
extrahepatic tissues
zona
Stimulation of fat
reticulari
breakdown in
s cells)
adipose tissue anti-
inflammatory and
immunosuppressive
progestogen Progesterone ovary, Granulosa PR Support pregnancy:
[9]
adrenal cells theca
glands, cells of ovary  Convert
placenta endometriu
(when m to
pregnant) secretory
stage
 Make
cervical
mucus
permeable to
 Targ
Chemical Abbrevi Recep et
Name Tissue Cells Effect
class ation tor Tiss
ue
sperm
 Inhibit
immune
response,
e.g. towards
the human
embryo.
 Decrease
uterine
smooth
muscle
contractility[
9]

 Inhibit
lactation
 Inhibit onset
of labor
 Support fetal
production
of adrenal
mineralo-
and
glucosteroid
s

Other:

 Raise
epidermal
growth
factor-1
levels
 Increase core
temperature
during
ovulation[10]
 Reduce
spasm and
 Targ
Chemical Abbrevi Recep et
Name Tissue Cells Effect
class ation tor Tiss
ue
relax smooth
muscle
(widen
bronchi and
regulate
mucus)
 Antiinflamm
atory.
Regulate
immune
response
 Reduce gall-
bladder
activity[11]
 Normalize
blood
clotting and
vascular
tone, zinc
and copper
levels, cell
oxygen
levels, and
use of fat
stores for
energy
 Assist in
thyroid
function and
bone growth
by
osteoblasts
 Resilience in
bone, teeth,
gums, joint,
tendon,
ligament and
skin healing
by regulating
 Targ
Chemical Abbrevi Recep et
Name Tissue Cells Effect
class ation tor Tiss
ue
collagen
 Nerve
function and
healing by
regulating
myelin

 Prevent
endometrial
cancer by
regulating
effects of
estrogen
Active form of
vitamin D3
skin/prox Increase absorption
Calcitriol (1,25-
imal of calcium and
secosteroid dihydroxyvitami VDR
tubule of phosphate from
n D3)
kidneys gastrointestinal tract
and kidneys inhibit
release of PTH
skin/prox
Calcidiol (25-
imal Inactive form of
secosteroid hydroxyvitamin VDR
tubule of vitamin D3
D3)
kidneys

References
1.

 http://www.cell.com/cell/abstract/S0092-8674(16)30193-3
  Kosfeld M, Heinrichs M, Zak PJ, Fischbacher U, Fehr E (June 2005). "Oxytocin increases
trust in humans". Nature. 435 (7042): 673–6. doi:10.1038/nature03701. PMID 15931222.
  Scientific American Mind, "Rhythm and Blues"; June/July 2007; Scientific American
Mind; by Ulrich Kraft
  http://www.vivo.colostate.edu/hbooks/pathphys/endocrine/otherendo/somatostatin.html
Colorado State University - Biomedical Hypertextbooks - Somatostatin
  Physiology: 5/5ch4/s5ch4_17 - Essentials of Human Physiology
  Kaushansky K (May 2006). "Lineage-specific hematopoietic growth factors". N. Engl. J.
Med. 354 (19): 2034–45. doi:10.1056/NEJMra052706. PMID 16687716.
  Massaro D, Massaro GD (2004). "Estrogen regulates pulmonary alveolar formation, loss,
and regeneration in mice". American Journal of Physiology. Lung Cellular and Molecular
Physiology. 287 (6): L1154–9. doi:10.1152/ajplung.00228.2004. PMID 15298854.
  Pentikäinen V, Erkkilä K, Suomalainen L, Parvinen M, Dunkel L (2000). "Estradiol acts
as a germ cell survival factor in the human testis in vitro". J. Clin. Endocrinol. Metab. 85 (5):
2057–67. doi:10.1210/jcem.85.5.6600. PMID 10843196.
  Placental Hormones
  Physiology: 5/5ch9/s5ch9_13 - Essentials of Human Physiology
 Hould F, Fried G, Fazekas A, Tremblay S, Mersereau W (1988). "Progesterone receptors
regulate gallbladder motility". J Surg Res. 45 (6): 505–12. doi:10.1016/0022-4804(88)90137-0.
PMID 3184927.

Bio-Identical Hormone Regeneration

Balance bio-identical hormone regeneration is a special program designed by Dr. Lander
who has 25 years of experience in replacing men and women’s hormones. We focus on
replacement when needed and ideally regeneration when possible to help the body make its
own natural hormones. When replacement is required, we emphasize bio-identical
hormones that are exactly what the body makes genetically and naturally, rather than
hormones that are modified as a pharmaceutical.

Bio-Identical Hormone Regeneration for Women

Menopause can creep up silently or may be very sudden such as after hysterectomy. Hormonal
imbalance can significantly impact your ability to enjoy youthful aging and vigorous health.

Are you feeling fatigue or experiencing difficulty losing weight?

Have you felt depressed or had mood swings?
Are you having trouble dealing with menopausal changes?
What about difficulty concentrating and remembering?
Does your skin have less elasticity than it used to?

When your hormones change, you might notice your skin feels different or you may experience
changes in your muscles and body composition. Women may start wondering where their libido
went. Many symptoms that are commonly ignored or treated with pharmaceuticals are actually
related to the changes we begin to experience due to hormonal imbalances associated with
normal aging. That’s where age management medicine steps in and helps patients achieve their
health goals in a natural way.

The hormone system is a complex network: Changing one hormone affects all the others so the
regeneration program must be methodical and comprehensive. We have special programs for
patients that cannot take estrogen replacement due to a history of breast cancer.
The Balance program is a customized system developed to replace hormones with biologically
identical agents or restore your body’s ability to produce natural hormones where possible.
Beneficial effects can be dramatic and immediate.

Bio-Identical Hormone Regeneration for Men

Men naturally go through a decline in hormone production in midlife. This is called Andropause.
Sometimes it is quite dramatic and can result in a sexual decline and significant detrimental
changes to your health and body. Perhaps you’ve seen ads on television for “low T” and you
recognize many of the symptoms?

Are you feeling fatigue or experiencing difficulty losing weight?

Have you felt depressed or had mood swings?
Is your sex drive decreased and erection quality diminished?
What about difficulty with concentration, focus and multi-tasking?

When your Testosterone declines, you may experience changes in your sports performance,
muscle mass and body composition (weight gain around the middle and over the chest area).
Many symptoms that are commonly ignored or treated with pharmaceuticals are actually related
to the changes we begin to experience due to hormonal imbalances associated with normal aging.
That’s where age management medicine steps in and helps patients achieve their health goals in a
natural way.

“Low T” is also common in patients with obesity, diabetes and other chronic illnesses, chronic
pain and chronic stress.

The hormone system is a complex network: Changing one hormone affects all the others so the
regeneration program must be methodical and comprehensive. Often several hormones must be
restored.

Methods of male hormone replacement:

Creams: We have specially compounded bio-identical testosterone creams using the latest
technology (Sindi) to provide steady absorption and distribution for optimal effects. These
creams can be compounded specially with other ingredients based on individual needs such as
preventing conversion into other hormones or preserving testicular volume.

Pellets: Pellets are compressed bio-identical hormones into small cylinders that can be concealed
under the skin. The pellets are placed under the skin in a brief office procedure associated with
minimal discomfort. The pellets are produced under sterile manufacturing conditions by a top
quality compounding pharmacy and are custom ordered for strength and quantity for each
individual patient. Not everyone absorbs and metabolizes the pellets at the same rate and
individual variations must be accounted for in dose planning. Success with pellet therapy
requires a fundamental knowledge of dosing and the various insertion methods. Pellets are
placed in different positions in men vs. women utilizing basic differences in men’s and women’s
bodies, making sure that they do not interfere with any daily activities. Patients report that pellets
inserted under the skin maintain the highest steady state levels with maximum convenience,
requiring re-insertion a few times per year.

Shots: Injections can be either given at our office or we can train our patients to do their own
convenient self injections.

Oral Pills: Testosterone pills can be safe if they are manufactured a special way to avoid
absorption into the liver. Our patients have access to special oral testosterone pills that avoid
liver toxicity and can be very convenient for daily use.

Click Here to Watch Dr. Lander Discuss Male Menopause

FAQS
Are Bio-identical hormones safe?
Bio-identical hormone replacement is natural and very safe if supervised carefully. Normal levels
of hormones in both women and men are associated with mental, sexual, and physiologic effects
that are vital to good health and quality of life. These are the exact same compounds your body
would be making naturally under optimal functioning conditions.

What kind of hormones do women and men need?

Most women need a healthy balance of estrogen, progesterone, and even testosterone. Symptoms
in both men and women who lack adequate hormones may include weight gain, mood swings,
lack of energy, osteoporosis, loss of muscle and skin elasticity, and changes in mental acuity.
Women may complain of decreased libido, vaginal dryness, bladder irritability, and hot flashes,
Men need adequate levels of testosterone for energy, to maintain muscle and bone, and to
function sexually.

Do women also need testosterone?

Testosterone is not just a male hormone. It is naturally produced in small amounts by women and
is necessary for bone density, healthy skin elasticity, cardiac health, energy and the development
of lean muscle mass. Testosterone can help stabilize mood and can improve memory.
Testosterone is an important hormone for libido in both women and men. Women’s testosterone
levels should be approximately one tenth of the levels found in men.

What about other hormones?

It is important to identify other hormonal imbalances such as pituitary, thyroid or adrenal stress
related hormones to optimize a person’s well-being. Hormones can influence each other and the
same hormone can have different effects on different parts of the body. A simple blood test can
help sort out the situation and lays the foundation for a replacement plan that makes good
medical sense and gets results.

How are hormones replaced?

Hormones can be replaced with biologically identical agents that can be given either as a
compounded daily cream, a slow release system consisting of injected pellets placed under the
skin every few months, or oral pills, and also injections in some cases.
Is “pellet” therapy the most convenient method?
Patients will report that pellets inserted under the skin in both men and women maintain the
highest steady state levels with maximum convenience. The pellets are placed under the skin in a
brief office procedure associated with minimal discomfort. Dr. Lander is one of the few
physicians in this region of California that has extensive experience with pellet therapy.

How long does it take to get results?

Treatment often makes a dramatic difference very early on in a person’s sense of wellbeing. The
effects of the hormonal regeneration program are optimized when combined with good nutrition,
exercise, and a healthy lifestyle.

How much does it cost and does insurance cover it?

Unfortunately insurance companies do not recognize the value of this level of customized care.
Patient fees include medications, evaluation, monitoring of progress and ong