Professional Documents
Culture Documents
Inhalation of Allergens
and Irritants (Dust and
Smoke)
Presence of infectious
microorganisms in dust
particles Output of
Immunoglobulin E (IgE)
by B Lymphocytes
Exposure to pathogen or
source of infection IgE Antibodies attach to
Mast Cells and Basophilis
in the bronchial walls
Invasion of
microorganisms into the
lungs via orifices, which
connect to upper Mast Cells degranulation
respiratory tract
Clinical Management of Bronchial Asthma in Acute
Exacerbation, to Consider Pneumonia, Emphysema
and Chronic Obstructive Pulmonary Disease
Inflammation Process
Vasolidation
Difficulty of
Hyper secretion Respiratory Respiratory Breathing/
of mucus Congestion Insufficiency Dyspnea
Increased in
Permeability
Clinical Management of Bronchial Asthma in Acute
Exacerbation, to Consider Pneumonia, Emphysema
and Chronic Obstructive Pulmonary Disease
Vasolidation
Difficulty of
Hyper secretion Respiratory Respiratory Breathing/
of mucus Congestion Insufficiency Dyspnea
Increased in
Permeability
Emigration of Leukocytes
Phagocytosis
Clinical Management of Bronchial Asthma in Acute
Exacerbation, to Consider Pneumonia, Emphysema
and Chronic Obstructive Pulmonary Disease
Impaired Expiration
Extend resistance to
airflow and decrease
flow rates (expiratory
flow)
Impaired Expiration
Air trapping
Clinical Management of Bronchial Asthma in Acute
Exacerbation, to Consider Pneumonia, Emphysema
and Chronic Obstructive Pulmonary Disease
Air trapping
Hyperinflation distal to
obstruction
CO2 retention
Absorption of trapped
air in the alveoli
Respiratory Increased lung
acidosis (if
volume
severe)
Alveolar sacs
collapsed (atelectasis)
Respiratory Response of the
failure lung receptor
triggering
hyperventilation
Perfusion without
ventilation shut
(ventilation-perfusion
mismatch)
Tachypnea
Emphysema