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NCM 106 Acute Biologic Crisis
NCM 106 Acute Biologic Crisis
Virac, Catanduanes
College of Health Sciences
Course Description:
Objectives:
At the end of the course, and given actual
clients with problems in acute biologic crisis,
the student should be able:
1.Academic Excellence
2. Virtues
A.Observe bioethical principles and the core
values (love of God, caring, love security and of
people
Utilize the bioethical principle and core
values and nursing standards in the care
of client
c. Integrate the various principles, concept and
application of bioethics in the care of the client.
A.Determine the different principles and tech
niques of nursing care management inpromot
ing the health of the community
D e s c r i p t i o n - I s t h e i n a b i l i t y o f
the heart to pump sufficient b
l o o d t o m e e t t h e needs of the tissues fo
r oxygenation and nutrients
-CHF is most commonly used when referring
to left-sided and right-sided failure
- Formerly called Congestive Heart Failure
Etiologic Factors:
-Hypoxia
-Anemia Pathophysiology:
Cardiac failure most commonly occurs with disord
ers of cardiacmuscles that result in decreased
contractile properties of the heart. Common
underlying conditions that lead to decreased
myocardial contractility include myocardial
dysfunction, arterial hypertension, and alular
dysfunction. Myocardial dysfunction may be
due to coronary artery disease, dilated
cardiomyopathy, or inflammatory and
degenerative diseases of the myocardium.
Atherosclerosis of the coronary arteries is the
primary cause of heart failure. Ischemia
causes myocardial dysfunction because of
resulting hypoxia and acidosis (from
accumulation of lactic acid). Myocardial
infarction causes focal my cellular necrosis, the
death of myocardial cells, and a loss of
contractility; the extent of the infarction is
prognostic of the severity of CHF. Dilated
cardiomyopathy causes diffuse cellular
necrosis, leading to decreased contractility.
Inflammatory and degenerative diseases of the
myocardium, such as myocarditis, may also
damage myocardial fibers, with a resultant
decrease in contractility. Systemic or
pulmonary HPN increases afterload which
increases the workload of the heart and in turn
leads to hypertrophy of myocardial muscle
fibers; this can be considered a compensatory
mechanism because it increases contractility.
Alular heart disease is also a cause of cardiac
failure. The valves ensure that blood flows in
one direction. With alular dysfunction, valve
has increasing difficulty moving forward. This
decreases the amount of blood being ejected,
increases pressure within the heart, and
eventually leads to pulmonary and venous
congestion.
Clinical Manifestations
- Dyspnea on exertion
- Cough
- Adventitious breath sounds
- Restless and anxious
- Skin appears pale and ashen and feels
cool and clammy
- Tachycardia and palpitations
- Weak, thread pulse
Easy fatigability and decreased activity
tolerance
Right-Sided
Cardiac Failure
- When the right ventricle fails,
congestion of the viscera and the peripheral
tissues predominates. This occurs because the
right side of the heart cannot eject blood and
thus cannot accommodate all the blood that
normally returns to it from the venous
circulation.
Clinical Manifestations
Edema of the lower extremities (dependent e
dema)
- Weight gain
- Hepatomegaly (enlargement of the liver)
- Distended neck veins
- Ascites (accumulation of fluid in the peritone
al cavity) - Anorexia and nausea
- Nocturnal (need to urinate at night)
- Weakness
Diagnostics
-
Chest X-ray (may show cardiomegaly or
vascular congestion)
-
Echocardiogram (shows decreased ventricular
function and decreased ejection fraction
CVP (elevated in right-sided failure)
*pulmonary artery pressure monitoring may
be used as guide treatment in serious case of
pulmonary edema
Nursing Diagnoses-
- Excess fluid volume r/t excess fluid/sodium
intake or retention secondary to CHF and its
medical therapy
Anxiety r/t breathlessness and restlessness s
econdary to inadequate oxygenation
- Non-compliance r/t to lack of knowledge-
Powerlessness r/t inability to perform role res
ponsibilitiessecondary to chronic illness and
hospitalizationNursingManagementa. Acute
phase
- Monitor and record BP, pulse, respirations,
ECG and CVP to detect changes in cardiac
output
- maintain client in sitting position to decrease
pulmonarycongestion and facilitate improved
gas exchange
- auscultate heart and lung sounds frequently:
increasing crackles, increasing dyspnea,
decreasing lung sounds indicate worsening
failure
- administer O2 as ordered to improve gas ex
change and increaseoxygenation of blood;
monitor arterial blood gases (ABG) as ordered
to assess oxygenation
- administer prescribed medications on accura
te schedule
- Monitor serum electrolytes to detect hypokal
emia secondary to diuretic therapy
- Monitor accurate input and output (may requ
ire Foley catheter tallow accurate
measurement of urine output) to evaluate fluid
status
- If fluid restriction is prescribed, spread the flu
id throughout the dayto reduce thirst
- encourage physical rest and organized activi
ties with frequentrest periods to reduce the
work of the
heart- provide a calm reassuring environment
to decrease anxiety; thisdecreases oxygen
consumption and demands on the heart.
Chronic heart
failure- educate client and family about the rati
onale for the regimen- establish baseline asse
ssment for fluid status and functionalabilities-
monitor daily weights to evaluate changes in fl
uid status- assess at regular intervals for chan
ges in fluid status or functionalactivity
level PharmacologicTherapy- ACE Inhibitors (
promotes vasodilation and diuresis by decrea
singafterload and preload eventually
decreasing the workload of the
heart.)- Diuretic Therapy. A diuretic is one of
the first medications prescribed to a patient
with CHF. Diuretics promote the excretion of
sodium and water through the
kidneys- Digitalis
(increases the force of myocardial
contraction and slows conduction through the
AV node. It improves contractility thus,
increasing left ventricular output.)-
.
(Dobutrex) is an intravenous medication given
topatients with significant left ventricular
dysfunction. Acatecholamine, it stimulates the
beta1-adrenergic receptors. Itsmajor action is
to increase cardiac
contractility.- Milrinone (Primacor). A phospho
diesterase inhibitor that prolongsthe release
and prevents the uptake of calcium. This in
, causing a decrease in preload and afterload
The workload of the heart.
- Nitroglycerine (a vasodilator reduces preload
) - Morphine to sedate and vasodilator,
decreasing the work of theheart- Anticoagulan
ts may be prescribed. Beta-adrenergic
blockersmaybe indicated in patients with mild
or moderate failure Client Education
-
Include family member or others in teaching as
appropriate
-
Weight monitoring: teach client the importance
of measuring and recording daily weights and
report unexplained increase of 3-5pounds
-
Diet: sodium restriction to decrease fluid
overload and potassium rich foods to replenish
loss from medications; do not restrict
water intake unless directed
-
Medication regime: explain the importance of
following all medication instruction
-
Activity: help client plan paced activity to
maximize available cardiac output
-
Symptoms: report to MD promptly any of the
following: chest pain, new onset of dyspnea on
exertion, paroxysmal and nocturnal dyspnea
-
Report even minor changes to MD as they
may be an early signoff
Etiologic factors
- Physical exertion
- Emotional stress- Weather extremes
- Digestion after a heavy meal
- Valsalva maneuver
- Hot baths or showers
- Sexual excitation
Pathophysiologic characteristic (Coronary arte
ry disease)
Pathophysiology
Coronary artery blood flow is blo
c k e d b y a t h e r o s c l e r o t i c narrowing,
thrombus formation or persistent vasospasm;
myocardium supplied by the arteries is
deprived of oxygen; persistent ischemia may
rapidly lead to tissue death
ClinicalManifestations
- Chest pain or discomfort (described as
aching or squeezing pain, most common
location is sub sternal, radiating to neck, jaw,
back, shoulders, left arm or occasionally the
right arm)
-Complain of heartburn or indigestion
-pallor, diaphoresis, cold skin, shortness of
breath, weakness, dizziness, anxiety, and
feelings of impending doom.
DiagnosticsLaboratory Tests
- Electrocardiogram (12-lead)
– capable of diagnosing MI in 80% of patients
, making it an indispensable, noninvasive, and
cost-effective tool. Reading shows ST
elevation, accompanied by T-wave inversion;
and later new pathologic Q wave
Definedasafall in arterial
oxygentension and a rise
i n a r t e r i a l carbon dioxide tension.
- The ventilation and/or
perfusion mechanisms in the lung are
impaired.
Etiologic factors
-Alveolar hypoventilation
-Diffusion abnormalities
-Ventilation-perfusion mismatching
-Shunting Pathophysiology
-Progression of pulmonary edema
occurs when capillary hydrostatic
pressure is increased, promoting
movement of fluid into the interstitial
space of the alveolar-capillary
membrane. Initially, increased
lymphatic flow removes the excess
fluids, but continued leakage eventually
overwhelms this mechanism. Gas
exchange becomes impaired by the
thick membrane. Increasing interstitial
fluid pressure ultimately causes leaks
into the alveolar sacs, impairing
ventilation and gas exchange.
ClinicalManifestations
-Tachypnea
-Tachycardia
-Cold, clammy skin and frank
diaphoresis are apparent especially
around the forehead and face
Etiologic factor.
Prerenal - caused by decrea
se blood flow to kidneys like
s e v e r e dehydration,diuretic therapy,
circulatory collapse,hypovolemia
or shock; readily reversible when
recognized and treatedb.Intrarenal –
caused by disease process,
ischemia, or toxic conditions such as
acute glomerulonephritis, vascular
disorders,toxicagents, or severe
infection
Post renal – caused by any condition
that obstructs urine flow such as benign
prostatic hyperplasia, renal or urinary
tract calculi, or tumors.
Pathophysiology
-Acute renal failure is classified as
perennial, intracranial or post renal. All
conditions that lead to perennial failure
impair blood flow to the kidneys (renal
perfusion), resulting in a decreased
glomerular filtration rate and increased
tubular desorption of sodium and water.
Intracranial failure results from damage
to the Kidneys. Post renal failure results
from obstructed urine flow.
ClinicalManifestations
*A change in blood pressure and
volume signals pre renal failure,
the patient may have the following:
-Oliguria
-Tachycardia
-Hypotension
-Dry mucous membranes
-Flat jugular veins
-Lethargy progressing to coma
-Decreased cardiac output and cool,
clammy skin in patient with heart
failure
*As renal failure progresses, the patient
may manifest the following signs and
symptom:
- uremia
- confusion
- GI complaints
- fluid in the lungs
- infection
Diagnostics
-Blood studies reveal elevated BUN,
serum keratinize, and potassium levels
and decreased blood pH, bicarbonate,
HCT, and Hob levels
-Urine studies show cats, cellular
debris, decreased specific gravity and,
in glomerular diseases, proteinuria and
urine osmolality close to serum
osmolality.
-Keratinize clearance testing is used to
measure the GFR and estimate the
number of remaining functioning
nephrons
-Electrocardiogram (ECG) shows tall,
peaked T waves, widening QRS
complex, and disappearing P waves if
increased potassium is present
*Other studies used to determine the
cause of renal failure
:- kidney ultrasonography
- plain films of the abdomen
- KUB radiography
- excretory urography
- renal scan
- retrograde pyelography
computed tomography scan and nephr
otomography
Nursing Diagnoses
-Excess Fluid Volume
-Imbalanced Nutrition: Less than Body
Requirements
-Deficient Knowledge
-Riskfor Infection
NursingManagement
-Monitor intake and output
-Observe for oliguria followed
by polyuria
-Weigh daily and observe for edema
-Monitoring of complications of
electrolyte imbalances, such as
acidosis and hyperkalemia
-Allow client to verbalize concerns
regarding disorder
-Encourage prescribed diet: moderate
protein restriction, high in
carbohydrates, restricted potassium
-Once diuresis phase begins, evaluate
slow return of BUN, keratinize,
phosphorus, and potassium to
normalPharmacologicTherapy
-Use volume expanders are prescribed
to restore renal perfusion in
hypotensive clients and Dopamine IV to
increase renal blood flow
-Loop diuretics to reduce toxic
concentration in nephrons and establish
urine flow
-ACE inhibitors to control hypertension
-Antacids or H2 receptor antagonists to
prevent gastric ulcers
-Kayexelate to reduce serum potassium
levels and sodium bicarbonate to treat
acidosis
*Avoid nephrotoxic drugs
Client Education
-Dietary and fluid restrictions, including
those that may be continued after
discharge
-Signs of complications such as fluid
volume excess, CHF, and
hyperkalemia
-Monitor weight, blood pressure, pulse,
and urine output
-Avoid nephrotoxic drugs and
substances: NSAIDs, some antibiotics,
radiologic contrast media, and heavy
metals; consult care provider prior to
taking any OTC drugs
-Recovery of renal function requires up
to 1 year; during this period, nephrons
are vulnerable to damage from
nephrotoxinsStroke/Cerebrovascular
accident Description
-Is a condition where neurological
deficits occur as a result of decreased
blood flow to a localized area of the
brain?
Description- prolonged pressure grea
ter than 15mmHg or 180mmH2O measu
r e d i n the lateral ventricles
Etiology
-Cerebral Edema is an increase in volume of brain tissue due to
alterations in capillary permeability, changes in functional or the
structural integrity of the cell membrane or an increase in the
interstitial fluids
-Hydrocephalus is an increase in the volume of CSF within the
ventricular system; it may be no communicating hydrocephalus
where the drainage from the ventricular system is impaired
Pathophysiology
-Blood flow exerts pressure against a weak arterial wall,
stretching it like an overblown balloon and making it to rupture;
rupture is followed by a subarachnoid hemorrhage, in which
blood spills into space normally occupied by CSF. Sometimes,
blood spills into brain tissue, where a clot can cause potentially
fatal increased ICP and brain tissue.
Clinicalmanifestations
-Blurring of vision, decreased visual acuity and diplopia are the
earliest signs of increased ICP
-Headache, papilledema or the swelling of optic disk
and vomiting
-Change of LOCDiagnostics
-Skull radiography
-CT scan
-MRI
* Lumbar puncture is not performed because of brain herniation
caused by sudden release of pressure*Laboratory tests are
performed to augment and monitor treatment approaches;
serum osmolality monitors hydration status and ABGsmeasure
pH, oxygen and carbon dioxide
Nursing Diagnoses
-Ineffective Cerebral Tissue Perfusion related to Increased ICP
-Risk for Infection
-Impaired Physical Mobility
-Risk for Ineffective Airway Clearance
NursingManagement
- Assess neurological status every 1 to 2 hours and report any
deterioration; include LOC, behavior, motor/sensory function,
pupil size and response, vital signs with temperature
-Maintain airway; elevate head of 30 degree or keep flat as
prescribed; maintain head and neck in neutral position to
promote venous drainage
-Assess for bladder distention and bowel constipation; assist
client when necessary to prevent Val Sava maneuver
-Plan nursing care so it is not clustered because prolonged
activity may increase ICP; provide quiet environment and limit
noxious stimuli; limit stimulants such as radio, TV and
newspaper; avoid ingesting stimulants such as coffee, tea, cola
drinks and cigarette smoke
-Maintain fluid restriction as prescribed
-Keep dressings over catheter dry and change dressings as
prescribed; monitor insertion site for CSF leakage or infection;
monitor clients for signs and symptoms of infection; use aseptic
technique when in contact with ICP monitor
Pharmacologic therapy
-Osmotic diuretics such as Manito and loop diuretics such as
Furosemide (Lasix) are mainstays used to decrease ICP
-Corticosteroids are effective in decreasing ICP especially
withtumorsSurgical Intervention
-A drainage catheter, inserted via ventriculostomy into lateral
ventricle, can be done to monitor ICP and to drain CSF to
maintain normal pressure; if used the system is calibrated with
transducer is leveled 1 inch above the ear; sterile is of utmost
Etiology
-Decreased or missed dose of insulin
-Illness or infection
-Undiagnosed and untreated diabetes Pathophysiology
-In the absence of endogenous insulin, the body breaks down
fats for energy. In the process, fatty acids develop too rapidly
and are converted to ketones, resulting to severe metabolic
acidosis. As acidosis worsens, blood glucose levels increase
and hyperkalemia worsens. The cycle continues until coma and
death occur.
Clinical manifestations
- Acetone breath
- Poor appetite or anorexia
- Nausea and vomiting
- Abdominal pain
- Blurred vision
- Weakness
- Headache
- Dehydration
- Thirst or polydipsia
- Orthostatic hypotension
- Hyperventilation (Kussmaul respirations)
- Mental status changes in DKA vary from patient to patient
- weight loss
- Muscle wasting- leg cramps- recurrent infections
Diagnostics
-Serum glucose is elevated (200 to 800 mg/dl)
-Serum Ketone Level is increased
-Urine acetone test is positive
-Arterial Blood Gas analysis reveals metabolic acidosis
-ECG findings shows tall tented T waves and widened
QRScomplex changes related to hyperkalemia; later with
hypokalemia, shows flattened T wave and the presence of
Wave
-Serum osmolality is elevated
Nursing Diagnoses
-Deficient Fluid Volume
-Risk for Injury
-Risk for Skin Impaired Integrity
-Ineffective Breathing Pattern
-Disturbed Sensory Perception
-Knowledge Deficit
-Anxiety
Nursing Management
-Restore fluid, electrolyte and glucose balance with IV infusions
and medications, analyze intake and out, blood glucose, urine
ketones, vital signs, oxygenation and breathing pattern
-Maintain skin integrity; promote healing of impaired skin;
prevent infection by turning and positioning client every 2 hours;
provide pressure relief as indicated; manage incontinence and
perspiration with skin protective barriers and cleansing; provide
appropriate nutrition and oxygen support
-Promote safety by analyzing vital signs, client communication,
LOC and emotional response, and activity tolerance; implement
falls prevention measures
-Assist client to verbalize concerns and cope effectively with
illness and fears
-Assist client to update Medic-Alert bracelet information
asappropriatePharmacotherapy
-Administer IV Insulin and fluid and electrolyte replacements
based on laboratory test results Client
Education
-Instruct client about the nature and causes of DKA (such as
excess glucose intake, insufficient medications or physiological
and/or psychological stressors) any new medications.
Etiology
-Medications
-Infections
-Acute illness
-Invasive procedure
-Chronic illness
Pathophysiology
-glucose production and release into the blood is increased
or glucose uptake by the cells is decreased; when the cells don’t
receive glucose, the liver responds by converting glycogen to
glucose for release into the bloodstream; when all excess
glucose molecules remain in the serum, osmosis cause fluid
shifts.; the cycle continues until fluid shifts in the brain cause
coma and death.
Clinical Manifestations
-Severe dehydration
-Hypotension and tachycardia
-Diaphoresis
-Tachypnea
-Polyuria, polydipsia and polyphagia
-Lethargy and fatigue
-Vision changes
-Rapid onset of lethargy
-Stupor and coma
-Neurologic changes
Diagnostics
-Serum glucose is elevated, sometimes 800 to 2,000 mg/dl
-Ketones are absent, urine and serum ketones are absent
-Urine glucose levels are positive
-Serum osmolality is increased
-Serum Sodium levels are elevated and the serum potassium
level is usually normal
-ABG results are usually normal, without evidence of acidosis
Nursing Diagnoses
-Decreased Cardiac Output
-Deficient Fluid Volume
-Hyperthermia
-Disturbed Sensory Perception
-Risk for Impaired Skin Integrity
-Risk for Aspiration
-Deficient Knowledge
Nursing Management
-Assess the patient’s LOC, respiratory status and oxygenation
-Monitor the patient’s VS; changes may reflect the patient’s
hydration status
-Monitor patient’s blood glucose and serum electrolytes
-Administer regular insulin IV as ordered, by continuous infusion
and titrate dosage based on the patient’s blood glucose levels
-Maintain intact skin integrity by turning every 2 hours, use
of pressure relief aids, nutritional support, use of skin
moisturizers and barriers, and management of incontinence
-Prevent aspiration by using appropriate feeding precautions,
elevate head of bed 15 to 30 degrees during and after feeding
for 1 hour; if BP is too unstable to elevate head of bed with
feeding, then withhold oral feedings.
Pharmacotherapy
-IV infusion of NS to replace fluids and sodium, regular insulin
Ivo manage the hyperglycemia, and potassium to replace losses
and shifts
Client Education
-Instruct client and family about HHNK, symptoms to report, and
administration of new medications
-Provide patient and family education to foster prevention
of future episodes.
Massive Bleeding
Description
-Uncontrolled bleeding
Etiology
-Result of blunt or penetrating trauma
-Gastrointestinal or genitourinary bleeding
-Hemoptysis
Pathophysiology
-Due to the lack of adequate circulating blood volume causing
creased tissue perfusion and metabolism resulting in hypoxia,
vasoconstriction and shunting of the available circulating blood
volume to the vital organs(heart and brain);
Symphatheticnervous system stimulation, hormonal release of
antidiuretic hormone and the angiotensin-renin mechanisms and
neural responses attempt to compensate for the loss of
circulating volume but eventually metabolic acidosis, multi organ
system failure occurs.
Clinical lManifestations
-Cool, clammy, pale skin (esp. distal extremities)
-Delayed capillary refill (>3 seconds)
-Weak, rapid pulses
-Decreased blood pressure (systolic pressure <90mmHg)
-Rapid shallow respirations (>28/ min)
-Restless, anxious, decreased LOC
-Cardiac dysrhythmias (abnormalities of cardiac rhythm)
-Decreased urinary output
Diagnostics
-Evidence of bleeding from thoracotomy that indicates bleeding
from chest area
-Abdominal or pelvic CT scan, abdominal ultrasound or
peritoneal lavage indicate intra-abdominal bleeding
-Endoscopy indicates upper or lower GI bleeding
-Angiography procedures diagnose severe vascular damage
-Extremity radiographs show long bone fractures
-Hemoglobin and hematocrit from the CBC are decreased due
to blood loss
-Elevated serum lactate if bleeding continues and client
becomes acidotic
-ABGs show metabolic acidosis as blood loss continues
-Baseline coagulation studies should be reviewed; initial
PT/Stand platelet counts will be within normal limits but as
coagulation factors become depleted, clotting times will increase
and platelet counts will decrease
-Serum electrolytes to assess renal function
Nursing Diagnoses
-Impaired Tissue Perfusion
-Deficient Fluid volume
-Decreased cardiac Output
Nursing Management
-Establish an adequate airway, breathing pattern, and applying
supplemental oxygen
-Give priority interventions to control bleeding such as direct
pressure to wound site, or assisting with surgical interventions
-Establish IV access and begin with fluid replacement
-Draw blood specimens as ordered to assist in evaluation
of hemoglobin, hematocrit, electrolyte, and oxygenation
andhydrationstatus
-Insert an indwelling catheter and NG tube to assist in accurate
recording of fluid balance status
-Perform and document continuous serial assessments
of hemodynamic parameters such as VS, capillary refill, CVP,
cardiac rhythm, LOC, urinary output and laboratory findings
Pharmacotherapy
-Crystalloids and blood products to maintain adequate
circulating volume status
-Sodium Bicarbonate to correct acidosis state
-Vasopressor such as Dopamine
Client Education
-Explain procedures to the client
-Support the family by explaining emergency measures
BURNS
Description
-An alteration in skin integrity resulting in tissue loss or injury
caused by heat, chemicals, electricity or radiation
E t i o l o g y
T y p e s o f b u r n
i n j u r y
a.Thermal: results from dry heat (flames) or moist
heat (steam or hot liquids); it is the most common type; it
causes cellular destruction that results in vascular, bony,
muscle, or nerve complications; thermal burns can also lead to
inhalation injury if the head and neck area is affected
Pathophysiology
-It depends on the cause and classification of the burn; the
injuring agents denatures cellular proteins; some cells die
because of traumatic or ischemic necrosis; loss of collagen
cross-linking also occurs with denaturation, creating abnormal
osmotic and hydrostatic pressure gradients that cause
intravascular fluid to move into interstitial spaces; Cellular
injury triggers the release of mediators of inflammation,
contributing to local and in the case of major burns , systemic
increases in capillary permeability.
ClinicalManifestations
-Localized pain and erythema, usually without blisters in the
first24 hours (first degree burn)
-Chills, headache, localized edema, nausea and vomiting (most
severe first degree burn)
-Thin-walled, fluid filled blisters appearing within minutes of the
injury, with mild to moderate edema and pain (second degree
superficial partial thickness burn)
-White, waxy appearance to damaged area (second degree
partial-thickness burn)
-White, brown or black leathery tissue and visible
thrombosedvessels due to destruction of skin elasticity (dorsum
of hand, most common site of thrombosis veins), without blisters
(third-degree burn)
-Silver-colored, raised or charred area, usually at the site
of electrical contact
D i a g n o s t i c s *
Rule of Nines chart determines the percentage of body surface
area (BSA) covered by the burn
-ABG levels may be normal in the early stages but may reveal
hypoxemia and metabolic acidosis
-Carboxyhemoglobin level may reveal the extent of smoke
inhalation due to the presence of carbon monoxide
-Complete blood count may reveal decrease hemoglobin due
O hemolysis, increased hematocrit and leukocytosis
-Electrolyte levels show hypernatremia and hyperkalemia,
other laboratory tests reveals elevated BUN, decreased total
protein and albumin
-Keratinize kinase (CK) and myoglobin levels may be elevated
-Presence of myoglobin in urine may lead to acute
tubular necrosis
Nursing Diagnoses
Nursing Management
-Assess patient’s ABCs; monitor arterial oxygen saturation and
serial ABG values and anticipate the need for ET intubation and
mechanical ventilation
-Auscultate breath sounds
-Administered supplemental humidified oxygen as ordered
-Perform or pharyngeal or tracheal suctioning as indicated by
the patient’s inability to clear his airway
-Monitor the patient’s cardiac and respiratory status
-Assess LOC for changes such as confusion, restlessness
or decreased responsiveness
-solution for chemical burns
-Place the patient in semi-Fowler’s position to maximize chest
expansion; keep patient as quiet and comfortable to minimize
oxygen demand
-Prepare the patient for an emergency escharotomy of the chest
and neck for deep burns
-Administer rapid fluid replacement therapy as ordered
Client Education
-Environmental safety: use low temperature setting for hot
water heater, ensure access to and adequate number of
electrical cords/outlets, isolate household chemicals, and avoid
smoking imbed
-Use of household smoke detectors with emphasis on
maintenance
-Proper storage and use of flammable substances-
Evacuation plan for family
-Care of burn at home
-Signs and symptoms of infection
-How to identify risk of skin changes
-Use of sunscreen to protect healing tissue and other protective
skin care
Poisoning
Description
-Substances that are harmful to humans that are inhaled,
ingested (food, drug overdose) or acquired by contact
Etiology
-Carbon monoxide inhalation
-Food poisoning
-Drug overdose
-Insecticide surface absorption
Pathophysiology
-The pathophysiology of poisons depends on the substance
that’s inhaled or ingested. The extent of damage depends on the
of the substance, the amount ingested, its form and the length of
exposure to it. Substances with an alkaline pH cause tissue
damage by liquefaction necrosis, which softens the tissue. Acids
produce coagulation necrosis. Coagulation necrosis denatures
proteins when substance contacts tissue. This limits the extent
of the injury by preventing penetration of the acid into the tissue.
-*The mechanism of action for inhalants is unknown, but they’re
believed to act on the CNS similarly to a very potent anesthetic.
Hydrocarbons sensitize the myocardial tissue and allow it to be
sensitize to catecholamine’s, resulting in arrhythmias.
ClinicalManifestations
a.Carbon monoxide inhalation: mild exposure –
nausea, vomiting, mild throbbing headache, flu-like symptoms;
moderate exposure – dyspnea, dizziness, confusion, increased
severity of mild symptoms; severe/prolonged exposure –
seizures, coma,respiraotory arrest, hypotension and
dysrhythmias
B.Food poisonings: nausea, vomiting, diarrhea, abdominal
cramps, fever , chills, dehydration, headache
c.Drug overdose: depends upon the substance
ingested; symptoms may include nausea, vomiting, CNS
depression or agitation, altered pupil response, respiratory
changes such as tachypnea or bradypnea, alterations in
temperature control, seizures or cardiac arrest
D i a g n o s t i c s *
The diagnosis of many poisonings is based on a thorough
client history and clinical manifestations
laboratory toxicology screens (serum,vomitus, stool and urine)d
etermine the extent of the absorption
baseline blood work such as CBC, electrolytes, renal and
hepatic studies enable future determination of organ and tissue
damage
Chest Xray may show aspiration pneumonia in inhalation
poisoning
-Abdominal X-rays may reveal iron pills or other radiopaque
substances
-ABG analysis used to evaluate oxygenation Nursing Diagnoses
-Risk for Ineffective Airway Clearance
-Risk for Decreased Cardiac Output
-Deficient Fluid Volume
-Ineffective Breathing Pattern
-Impaired Tissue Perfusion
-Risk for Injury
-Anxiety
-Risk for Self-directed Violence
-Hopelessness
Nursing Management
-Assist with the management of an effective airway, breathing
pattern and circulatory status
-Give treatment of life-threatening dysrhythmias and conditions
as ordered; continual monitoring of vital signs, cardiac rhythm
and neurological status and supportive care is essential
-Assist in the hastening in the elimination of the medication
or poison, decrease the amount of absorption and
administer antidotes as ordered
-for specific treatment contact the poison center
Pharmacotherapy
Client Education
-Assist the client and family in seeking the appropriate referrals
and provide client education to further complications
or incidence of overdose
-Ensure that the client and family understand discharge
instruction for follow up care or reason for admission.
Multiple Injuries
Description
-Is a physical injury or wound that’s inflicted by an external
or violent act; it may be intentional or unintentional; involve
injuries to more than one body area or organ
Etiology
-Weapons
-Automobile collision
-Physical confrontation
-Falls
-Unnatural occurrence to the body
*Type of trauma which determines the extent of injury
-Blunt trauma – leaves the body intact
-Penetrating trauma – disrupts the body surface
-Perforating trauma – leaves entrance and exit Pathophysiology
-A physical injury can create tissue damage caused by stress
and strain on surrounding tissue which results to infection, pain,
swelling and potential compartment syndrome or it can be life-
threatening if it affects a highly vascular or vital organ
Diagnostics
-Chest X-ray – detect rib and sterna fractures, pneumothorax,
flail chest, pulmonary contusion and lacerated or ruptured aorta
-Angiography studies – performed with suspected aortic
laceration or rupture
-Ct scan, cervical spine X-rays, skull X-rays, Angiogram – test
for a patient with head trauma
-ABG analysis to evaluate respiratory status and determine
acidotic and alkaloid states
-CBC to indicate the amount of blood loss
-Coagulation studies to evaluate clotting ability
-Serum electrolyte levels to indicate the presence of
electrolyteimbalances
Nursing Diagnoses
-Ineffective Airway Clearance
-Ineffective Breathing Pattern
-Impaired Gas Exchange
-Deficient Fluid Volume
-Decreased Cardiac Output
-Impaired Tissue Perfusion
-Impaired Skin Integrity
-Risk for infection
-Anxiety
-Pain
-Disturbed Body Image
Nursing Management
Pharmacotherapy
-Tetanus immunization
-Antibiotics for infection control
-Analgesics for pain Client Education
-Provide explanations of all procedures done
-Families usually require emotional support and honest
discussions about therapeutic interventions and plans
- Pharmacotherapy
Tetanus immunization
-Antibiotics for infection control
-Analgesics for pain Client Education
-Provide explanations of all procedures done
-Families usually require emotional support
and honest discussions about therapeutic
interventions and plans
2. Assessment
: A systematic procedure for collecting
qualitative and quantitative data to describe
progress and ascertain deviations from
expected outcomes and achievements.
3. Attributes: Characteristics that underpin
competent performance.
4. Benchmark: Essential standard
5. Client: An individual, family, group or
community that is a consumer of nursing
service.
6. Competence: The combination of skills,
knowledge, attitudes, values and abilities that
underpin effective performance as a nurse.
7. Competent: The person has competence
across all domains of competencies applicable
tithe registered nurse, at a standard that is
judged to appropriate for the level of nurse
beingassessed.
8. Competency: A defined area of skilled
performance.
9. Context: The setting/environment where
competence can be demonstrated or applied.
10. Domain: An organized cluster of
competencies in nursing practice.
11. Effective: Having the intended outcome.
12. Enrolled nurse: A nurse registered under
the enrolled nurse scope of practice.
13. Indicator: Key generic examples of
competent performance. They are
neither comprehensive nor exhaustive.
They assist the assessor when using their
professional judgment in assessing nursing
practice. They further assist curriculum
development.
14. Performance criteria: Descriptive
statements that can be assessed and that
reflect the intent of a competency in terms of
performance, behavior and circumstance.
15. Registered nurse: A nurse registered under
the registered nurse scope of practice
16. Reliability: The extent to which a tool will
function consistently in the same way with
repeated use.
17. Validity: The extent to which a
measurement tool measures what it purports
to measure.
CORE COMPETENCY
3 Gets involved in professional organizations
and civic activitiesIndicators:○ Participates
actively in professional, social, civic and
religious activities○ Maintain membership to
professional organizations○ Support activities
related to nursing and health issues
CORE COMPETENCY
4 Projects a professional image of
nurseIndicators:○ Demonstrate good manners
and right conduct at all times.○ Dresses
appropriately.○ Demonstrates congruence of
words and actions.○ Behaves appropriately at
all times.
CORE COMPETENCY
5 Possesses positive attitude towards change
and criticismIndicators:○ Listens to suggestions
and recommendations.○ Tries new strategies
or approaches.○ Adapts to changes willingly.
CORE COMPETENCY
6 Performs function according to professional
standardsIndicators:○ Assesses own
performance against standards of practice.○
Sets attainable objectives to enhance nursing
knowledge and skills.○ Explains current
nursing practices, when situations call for it.
VII. RESEARCH
CORE COMPETENCY 1:
Gathers data using different
methodologiesIndicators:Identifies
researchable problems regarding patient care
and community health Identifies appropriate
methods of research for a particular
patient/community problem Combines
quantitative and qualitative nursing design thru
simple explanation on the phenomena
observed Analyzes data gathered
CORE COMPETENCY 2:
Recommends actions for
implementationIndicator:Based on the analysis
of data gathered, recommends practical
solutions appropriate for theproblemCORE
COMPETENCY 3:Disseminates results of
research findingsIndicators:Communicates
results of findings to colleagues/patients/family
and to others
CHARACTERISTICS OF AN ASSESSMENT
THAT PROMOTES
COMPETENCY1 . P U R P O S E F U L
To promote Critical thinking, your approach to
assessment must change, depending onyour
purpose and the circumstances(c0ntext) of
your patient situation.For example:Are you
aiming to assess all aspects of care, or are
you monitoring one specificproblem?Are your
assessing a hospitalized patient or someone in
the home?Is the person an adult or a child?
NOTE: Your aim is to gain all the information
needed to ensure that your patients
haveindividualized plans that are designed to
help them achieve outcomes in the best
waypossible, in context of their particular
situation (eg, their age, culture, and level
of independence)
2.FOCUSED AND RELEVANT
Your assessment must be focused to gain
relevant information, depending onpurpose
and context as above.For example:Physician’s
Data: (Disease focus)
“ Mrs. Garcia has pain and swelling in all
joints. Diagnostic studies indicates that she has
rheumatoid arthritis. We will start her on a
course of anti inflammatory drugsto treat the
rheumatoid arthritis.” (focus on the treatment
modalities)
Nurse’s Data: (holistic focus, considering both
problems and their effect on theperson’s ability
to function independently)“Mrs. Garcia has
pain and swelling in all joints, making it difficult
to feed and dressherself. She has voiced that
it’s difficult to feel worthwhile when she can’t
feedherself. She states that she is depressed
because she misses seeing her two
smallgrandchildren. We need to to develop a
plan to help her with her pain, to assist her with
feeding and dressing, to work through feelings
of self-esteem, and for specialvisitations with
the grandchildren.” ( Focus is on Mrs. Garcia)
3.SYSTEMATIC
Developing a systematic approach to
assessment helps you pay attention to what
isimportant, learn how to prioritize, be
comprehensive, and avoid omission errors.For
example:
•
What are your symptoms?
•
Can you point out with one finger to the areas
that are bothering you?
•
When did they start?
•
What makes them better?
•
What makes them worse?
•
Are you taking any medications- prescribed,
over-the-counter, or herbalremedies- that may
be causing some of these symptoms?
•
Can you think of anything else that might be
contributing to your symptoms?
4.COMPREHENSIVE AND
ACCURATE
The most common error that happens in critical
thinking is identifying problems or making
judgments based on sufficient or incorrect
information. Your information mustbe factual,
and as complete as is warranted by your
purpose.For example:An assessment aims to
get information about one specific problem is
shorter thanone that aims to get
comprehensive data about all aspects of care.
DISPLAY B
.
1.1
:How to ensure Comprehensive Data
CollectionComprehensive data collection often
occurs in three phases:1.Before you see the
person: You find what you can. This
information may belimited( only name and
age) or extensive ( medical records may be
available for you to read)2.When you see the
person: You interview the person and
do PhysicalExamination (PE).3.After you see
the person: You review
the resources(consumer like patient,
familyand community, significant others,
nursing and medical records, verbal andwritten
consultations, diagnostic and laboratory
results) you used and determineswhat other
resources may offer additional information (e.g.
You may consult apharmacist to gain more
information about a medication
regimen)Comprehensive Data Collection have
several factors:1.The purpose of the
assessment- example is when you do data
base(start of care) assessment or a focus
assessmentData base assessment-
Comprehensive information gathered on
initialcontact with the person to assess all
aspect of health statusFocus Assessment-
Data gathered to determine the status of a
specificcondition like someone’s bowel
habits2.The needs and problems commonly
encountered in a particular
clinicalsetting.For example: An adult
assessment tool is different from a
newbornassessment tool.3.Standards of care
for the assessment as defined by
regulatory agencies andprofessional
associationsFor example: Maternal and Child
Nursing Association of the
Philippines/MCNAP, Operating Room Nurses
association of the
Philippines/ORNAP,Philippine Nurses
Association/PNA etc.4.The nursing model
or theory adopted by the school or
facilitiesFor example: Gordon’s Functional
Health Patterns or Orem’s Self Caretheory.
5.RECORDED IN A STANDARDIZED
WAY
Like pilots who follow computerized or pre-
printed checklists (instead of relying
onmemory), you must value the importance of
completing a standardized tool that isdesigned
to promote an assessment that is purposeful,
relevant, systematic, andcomplete.
NOTE: You cannot rely your brain to do it all,
even if you have years of experience
DISPLAY B.1.2:
Major Intellectual Skills & Critical
Thinking Skills R/T Assessment
(Behavior Evidence Suggesting
Competence in Nursing
Practice)
The competent nurse:
•
Applies standard and principles
•
Assesses systematically and comprehensively;
uses a nursing framework toidentify nursing
concerns; uses a body systems framework to
identify medicalconcerns
•
Detects bias; determines credibility of
information sources
•
Distinguishes normal from abnormal; identifies
risks for abnormal
•
Determines significance of data; distinguishes
relevant from irrelevantclusters relevant data
together
•
Identifies assumptions and inconsistencies;
checks accuracy and reliability ;recognizes
missing information; focuses assessment as
indicated
•
Communicates effectively orally and in writing
•
Establishes empowered partnerships with
patients, families, peers, and coworkers
•
Sets priorities and make decisions in a timely
way; includes key stakeholdersin making
decisions
•
Weigh risks and benefits
•
Identifies ethical issues and take appropriate
action
•
Identifies and uses technologic, information,
and human resources
•
Address conflicts fairly, fosters positive
interpersonal relationships
•
Facilitates and navigates change
•
Organize and manages time and environment
•
Facilitates teamwork ( focuses on common
goals; helps and encouragesothers to
contribute in their own way)
•
Demonstrates systems thinking (shows
awareness of the interrelationshipsexisting
within and across health care systems)
CLINICAL SCENARIO
Listening Empathetically Promotes
Understanding of the Real Issues,Fostering
Caring Human Responses
Today Patricia/Pat is caring for Sharon, who’s
just given birth to her fifth child,a healthy baby
girl. Pat never has been able to conceive, has
always wanted children, and feels a little
envious of Sharon’s family of two boys and
(now) of three girls.Pat notes that Sharon
seems very quiet. Recognizing the importance
of beingempathetic listener, Pat has the
following conversation with Sharon.Pat:
“You’ve been pretty quiet since I came
on.”Sharon: “I can’t help it. I’m supposed to be
happy, but I’m really disappointed-I was so
sure I’d had a baby boy.”Pat: (making a
conscious effort to eliminate thoughts about the
fact that she’dbe happy with any child, and
rephrasing what Sharon seems to be feeling):
“you feel like you’re supposed to be happy, but
you really feel sort of sad?”Sharon: “yes”,Pat
pauses to reflect on the feeling of sadness and
encourages Sharon tocontinue.Sharon: “I was
going to name this baby after my father. He
died 2 monthsago.”Pat (connecting to what
Sharon must be feeling): “I’m sorry. That would
be adisappointment. Being able to name the
baby after him would have been alovely thing
to do.”Sharon (crying): “Yes, I had it all pictured
in my mind.”Pat conveying acceptance and
understanding, sits quietly, allowing Sharon
tocry.Pat (detaching and coming back to her
own frame of reference):“Sharon, I think you
needed to cry and you may need to cry again.
But right now you’ve got a very beautiful baby
girl; with the longest hair I’ve ever seen, waiting
to meet her mother. How would you feel if I