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LE6 TRANSCRIBERS Dee, Del Mundo, Dela Cruz, Dela EDITOR Tilbe (0925 545 2480) 2 of 4
Torre, Domalanta
6.16 Lab Plenary: Reflexes & Motor Systems PHYSIOLOGY 2020C
examiner’s finger with the tip of his or her index finger. • Inability to hop on each leg = there may be a
The examiner’s finger must be far away so that the cerebellar lesion or lesion on corticospinal tract (distal
subject must fully extend the arm with the eyes open muscles are affected)
and then with the eyes closed. • When a normal muscle with an intact nerve supply
Heel to shin test - the subject places the heel is relaxed voluntarily, it maintains a slight residual
carefully on the opposite knee and slides it slowly tension known as muscle tone
along the edge of the tibia to the ankle and back up
the knee again MUSCLE TONE
Rapid alternating test - alternately patting the front 1. Procedure:
and back of the hand on the knee as rapidly • Alternately flex and extend elbow and wrist
and regularly as possible. • Alternately flex and extend the knee joint
2. Results 2. Grading:
Test Observation + Hypotonia
Finger to nose Able to perform ++ Normal
Heel to shin Able to perform +++ Hypertonia
Rapid alternating Able to perform ++++ Hypertonia with clonus
STATION AND GAIT 3. Results:
1. Procedures: Area Tested Muscle Tone
• Station Elbow ++
o Romberg’s Test: The subject stands Wrists ++
straight with the heels together first with the
Knees ++
eyes open then with eyes closed
• Gait: Observe certain aspects of the gait of the patient
REFLEXES
wile doing the following:
o Walks normally back and forth at moderate 1. Procedure:
rate
Perform the following tests and record your results in the table:
o Walks on heels
A. Triceps jerk – Strike the patient’s elbow a few inches
o Walks on toes
above the olecranon process. Look for elbow
o Tandem walk along a straight line (touching
extension and triceps contraction
heel to toe) B. Biceps jerk – Ensure patient’s arm is relaxed and
o Hops on each leg
slightly flexed. Palpate the biceps tendon with the
*Note the following during each of the different steps
thumb and strike with tendon hammer. Look for elbow
• Length of step (vertical distance between flexion and biceps contraction
heel of one foot and the toe of the other foot) C. Knee jerk – Ensure that the patient’s leg is relaxed by
• Width of the base (horizontal distance hanging it over the edge of the bed. Tap the patellar
between both heels) tendon with the hammer and observe quadriceps
• Abnormal leg movement (example: contraction
excessively high step) D. Ankle jerk – Tap the achilles tendon and watch for
• Instability (gait ataxia) calf muscle contraction and plantar flexion of the foot
• Associated Postural Movement (example:
pelvic swaying) 2. Grading reflex:
2. Results 0 No response
+ Diminished; Low normal
STATION:
++ Normal
Eyes Open: - Romberg Sign
Eyes Closed: - Romberg Sign +++ Brisker than average; not necessarily abnormal
+ Romberg Sign with eyes open = cerebral ataxia ++++ Hyperactive
+ Romberg Sign with eyes closed = sensory ataxia
3. Results:
Gait Result Reflex Results Cranial
Normal walk at moderate rate Able to perform test Nerve/Spinal root
Walk on heels Able to perform test Triceps jerk ++ Radial N. C6-C7
Walk on toes Able to perform test Biceps jerk ++ Musculocutaneous
Tandem Walk Able to perform test C5-C6
Hop (right and left foot) Able to perform test Knee jerk ++ Femoral N. L2-L4
Ankle jerk ++ Tibial N. S1-S2
• If patient can’t walk on heels and toes, there may be a
weakness of distal muscles or corticospinal Terms to Remember:
weakness. Dysdiadochokinesia
• Incoordination may be seen during tandem walk that • Inability to perform rapid alternating movements such
couldn’t be seen during a normal walk as repeated pronation and supination of hands.
LE6 TRANSCRIBERS Dee, Del Mundo, Dela Cruz, Dela EDITOR Tilbe (0925 545 2480) 3 of 4
Torre, Domalanta
6.16 Lab Plenary: Reflexes & Motor Systems PHYSIOLOGY 2020C
•May indicate damage to the posterior lobe of the 6. Differentiate between upper and lower motor neuron
cerebellum or neocerebellum lesions by filling out this table.
Dysmetria
• Characterized by overshooting or undershooting of Upper motor Lower
range of motion needed to place the limbs correctly neuron lesion motor
during active voluntary movements neuron
• Indicated damage to the posterior lobe of the lesion
cerebellum Loss of power Pyramidal Distal,
Ataxia distribution proximal, or
• Inability to coordinate movement weakness/ general
• Indicates damage to the posterior lobe of the paralysis weakness/
cerebellum, flocculonodular lobe, or anterior lobe paralysis
o Cerebellar ataxia – swaying when eyes are Extent of paralysis Paresis Complete
open (weakness) paralysis
o Sensory ataxia – swaying when eyes are Atrophy and wasting None initially, but With muscle
closed atrophies due to atrophy
disuse
B. Guide Questions Muscle tone Spasticity Flaccidity
1. What is muscle tone? What causes muscle tone? (increased (decreased
• Muscle tone is the passive contraction of muscles muscle tone) muscle
caused by reflex arcs from muscle spindles tone)
Reflexes
2. What is coordination? a. Deep Hyperreflexia Areflexia
• Coordination is the organization of the different b. Superficial, Loss of reflex Presence of
elements of a complex body movement as to abdominal reflex
enable them to work efficiently. Muscle fasciculation With clonus No clonus
and fibrillation
3. Explain why deep reflexes can be diminished or
absent. Give some clinical conditions that result in 7. Define stance and gait.
diminished or absent DTRs. • Stance is the ability to maintain an erect posture.
• Deep reflexes can be diminished or absent due to Gait is the pattern of movement of the limbs.
lesions along the reflex arc. It is also possible that
stimulation of the afferent nerve is insufficient due 8. Describe the following forms of abnormal gait and
to human error. be able to demonstrate each example. Give some
clinical conditions that result in the following gait
4. Explain why deep reflexes can be exaggerated. Give abnormalities.
some clinical conditions that result in diminished or a. Hemiplegic gait
absent DTRs. • involves flexion of the hip due to foot drop
• Deep reflexes can be exaggerated due to upper and circumduction of the leg
motor neuron lesions. These lesions can cause • most commonly seen in stroke
loss of inhibition allowing uninhibited firing of the b. Festinating gait
reflex arc. • short steps on tiptoe with the trunk flexed
forward and legs flexed stiffly.
5. What is clonus? In which clinical condition can this • seen in Parkinson’s disease
be seen? c. Drunken gait
• Clonus is an involuntary, rhythmic muscle • ataxic gait; inability to walk heel to toe;
contraction due to lack of inhibition of the upper walking with a wide base
motor neuron lesions. Conditions such as • indicative of a cerebellar lesion
cerebral palsy, Huntington disease, multiple d. Stamping gait
sclerosis may result in clonus. • high stepping gait due to loss of sensation
• If unilateral, causes include peroneal nerve
palsy and L5 radiculopathy. If bilateral,
causes include amyotrophic lateral
sclerosis, Charcot-Marie-Tooth disease and
other peripheral neuropathies including
those associated with uncontrolled diabetes.
REFERENCES:
1. Lecture Notes of Dr. Bartolome
LE6 TRANSCRIBERS Dee, Del Mundo, Dela Cruz, Dela EDITOR Tilbe (0925 545 2480) 4 of 4
Torre, Domalanta
6.16 Lab Plenary: Reflexes & Motor Systems PHYSIOLOGY 2020C
APPENDIX
LE6 TRANSCRIBERS Dee, Del Mundo, Dela Cruz, Dela EDITOR Tilbe (0925 545 2480) 5 of 4
Torre, Domalanta