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A Review Paper
U
.S. surgeons annually perform more than 150,000 hip arthroplasty. CT provides 3-dimensional information
total hip arthroplasties (THAs), 90% of which about anterior and posterior column deficiencies, socket
are primary procedures.1 Improved surgical size, and thickness of the anterior and posterior walls and
technique and instrumentation have expanded allows visualization of the external iliac vessels to ensure
the clinical indications for THA to include patients who
previously would not have been considered eligible for this
procedure. Such complex cases, which often require mod-
ular implants2 and/or bone grafting similar to that used in
revision arthroplasty, fall into the categories of dysplastic
hip, ankylosed hip, fractures about the hip, protrusio
acetabuli, neuromuscular conditions, skeletal dysplasias,
and previous bony procedures about the hip.
Indications for complex THA include pain not relieved
with conservative treatment and functional impair-
ment with radiographic evidence of hip degeneration.
Contraindications include active sepsis and major medi-
cal comorbidities. Preoperative range of motion (ROM)
A
that they can be avoided during placement of anchorage Given the complexity in operating on Crowe group
screws for autograft.5 IV/high-dislocation hips and the increased likelihood
Challenges in clinical evaluation and surgical interven- of failure, Charnley and Feagin15 recommended against
tion vary according to disease entity. We describe these reconstruction in these cases. Modern implants, we believe,
challenges in the following sections. make this recommendation no longer applicable. We prefer
a surgical technique that uses a transverse osteotomy for
Hip Dysplasia subtrochanteric femoral shortening and derotation for high-
Hip dysplasia is the most common cause of secondary osteo- riding developmental dislocation as it reduces the complex-
arthritic degeneration of the hip.6 Although developmental ity of the acetabular and femoral reconstruction.16
dysplasia of the hip (DDH) is the predominant type, other Other surgeons prefer the transtrochanteric approach,
conditions are associated with similar findings, such as which not only reduces the complexity of the acetabular
Perthes disease and neuromuscular diseases.7 In the initial and femoral reconstruction but facilitates tensioning the
phases of hip dysplasia, pelvic osteotomies7 may obviate or abductors to improve stability at the end of the procedure
delay the need for hip arthroplasty, though later degeneration by advancing it distally and laterally.9,11 Trochanteric
of the hip warrants consideration for THA. On the basis of migration has not been associated with an increased dislo-
disease severity, anatomical aberrations determine the com- cation rate.11
plexity of the arthroplasty procedure.8 The dysplastic hip is The longevity of a THA performed for dysplastic
characterized by these features9-11: hips is determined by how closely a near-restoration of
•There may be significant femoral head subluxation, normal anatomy with stable fixation of the components
with the head articulating with a false acetabulum instead is achieved.17 By approximating the site of the native
of the true acetabulum. acetabulum (inferior, medial, and anterior), the joint reac-
•The true acetabulum is often porotic, triangular, and tion forces decrease, and improved survivorship of ace-
shallow, with a thick posterior ischial wall and thin anterior tabular components results.18-24 During medialization, we
acetabular walls. direct the reamer posteromedially to prevent destruction
•The true acetabulum can be distinguished from the false of the deficient anterior wall.9,17 The thin anterior wall
acetabulum by presence of a fibrofatty pulvinar and pres- increases the tendency to position the cup in excessive
ence of a capsular attachment at its rim. anteversion, which can increase the anterior dislocation
•The neurovascular structural anatomy is often altered rate, especially if the femoral component is also placed in
but not usually shortened. excessive anteversion.9 Some investigators have suggested
•There is accentuated femoral neck anteversion. that medialization can be achieved by performing a type
•The proximal femur is hypoplastic, with a stenotic of acetabuloplasty called cotyloplasty.11,13 Cotyloplasty
medullary canal. involves creating a controlled fracture of the medial wall,
•The greater trochanter is posteriorly oriented, which augmenting with autogenous bone graft, and then cement-
compromises the abductor lever arm. ing a small acetabular component onto the deepened
acetabular cavity.
Crowe and colleagues12 classified hip dysplasias by In some cases, acetabular deficiency leads to exposure
quantifying the acetabular deficiency into 4 subtypes. The of the superior portion of the acetabular component and
hip is categorized as subluxed if the medial head–neck a resulting need for bone graft augmentation.5,15,25 Rim
junction distance referenced off the interteardrop line is at augmentation can be performed with femoral head auto-
least 10% of the measured height of the pelvis on the AP graft or allograft, thereby eliminating the segmental defect
x-ray of the pelvis and is quantified as follows: and restoring bone stock for future revision surgery.10,17,26
Group Subluxation Any peripheral segmental bone defects can be converted
I <50% to contained defects using metal meshes with bone grafts
II 50% to 75% with cemented cups.17 After anchorage of the bone graft,
III 75% to 100% any gap between the ilium and the bone graft can be packed
IV >100% with morselized bone graft to augment bony union.5,25
Histologic studies after impaction bone grafting of the
An alternative classification scheme is based on the acetabulum in revision arthroplasty reveal neovasculariza-
position of the femoral head in relation to the acetabu- tion of the bone graft with eventual bone incorporation.26,27
lum.11,13,14 The 3 types of hip dysplasia are: Failure of bony incorporation with resorption, however,
Dysplasia. The femoral head has relative articulation leads to cup failure.28-30 Moreover, some authors have
with the native (true) acetabulum, despite subluxation. reported that, even with bony union, these cemented cups
Low dislocation. The femoral head articulates with a false tend to loosen and fail.31 Failure of the acetabular sockets
acetabulum, which partially overlaps the true acetabulum. is correlated with younger age and increased cup cover-
High dislocation. The femoral head migrates superiorly age by the graft. As patients with DDH are more likely to
and posteriorly and articulates only with the false acetabu- require revision arthroplasty, we prefer using uncemented
lum. acetabular implants.
Figure 3. Anteroposterior x-
ray of a 42-year-old woman
who presented with severe
developmental dysplasia
of the hip and a signifi-
cantly anteverted femoral
neck. A modular S-ROM
total hip system (DePuy
Orthopaedics, Warsaw,
Indiana) was used to restore
hip mechanics.
Hip Ankylosis
Ankylosis of the hip can occur spontaneously or be
acquired surgically. Spontaneous and surgical fusions
present different operative challenges during THA, as
both retained hardware and anatomical deformity of the
proximal femur increase the complexity of the procedure.
Historical and current indications for surgical hip fusion
include Legg-Calve-Perthes disease, congenital dyspla-
sia of the hip, slipped capital femoral epiphysis, juvenile
rheumatoid arthritis, significant degenerative changes in a
young patient, recurrent hip infections, and tumor resec-
tion.52,53 Spontaneous hip fusion has been associated with
tuberculous and pyogenic arthritis, osteoarthritis, trauma,
A
inflammatory arthritis, and ankylosing spondylitis.52-55
In general, a fused hip is painless and, if in a functional
position, provides a stable platform for ambulation.56,57
Fusion takedown and conversion to a THA may be indicat-
ed to facilitate sitting or for unrelenting back and knee pain,
inability to perform functional activities (driving, work), or
nonunion or malunion of fusion.52,53,58
Preoperative evaluation should include assessment of the
degree of limb shortening, deformity, presence of a joint
line, need to remove fusion hardware, presence of a greater
trochanter, and hip abductor function as indicated by
muscular contraction.52,54,56,59,60 Electromyography57,59 or
magnetic resonance imaging (Figure 4) may be necessary
to confirm the adequacy of hip abductor innervation.
Surgical exposure is difficult because of loss of hip B C
ROM. Although some authors have described using a pos-
Figure 4. (A) Anteroposterior x-ray of a 34-year-old woman with
terolateral approach61,62 and an anterolateral approach,57 at a surgical fusion of the hip performed 15 years previously for
our institution we prefer a direct lateral approach.52,54,56,63 recurrent tuberculous arthritis of the right hip. (B) Given the
The crucial step in the procedure is identification of the patient’s symptoms of back pain and functional activity restric-
surgical landmark—that is, the vastus tubercle. The latter tion attributable to the fused right hip, total hip arthroplasty
was indicated. This magnetic resonance image was obtained
ridge marks the distal limit for performing a standard tro-
to evaluate the hip abductors, which were noted to be attached
chanteric osteotomy, which increases hip exposure. to the greater trochanter. (C) Anteroposterior x-ray obtained
An in situ femoral neck osteotomy is performed by after total hip arthroplasty with transtrochanteric approach and
angling the saw blade in line with the native acetabulum modular implants shows satisfactory outcome.
and leaving sufficient quantity of bone in the ilium.8
Culture specimens should be taken to rule out infection in Some authors have suggested using antibiotic-impregnated
cases of previous infection.52,56 The true acetabular cav- cement for both components to decrease risk for infec-
ity can be identified by triangulating the 3 vital surgical tion,53,54 but others have reported no significant difference
landmarks: the obturator foramen inferiorly, the sciatic with use of cemented and cementless techniques in this
notch posteriorly, and the pubic bone or anterior inferior category of patients.61 After femoral and acetabular prepa-
iliac spine anteriorly.61 Intraoperative x-rays taken with ration, assessment with trial components should be done. If
an inferior acetabular retractor in the estimated position there is difficulty in reduction, or if the trochanteric frag-
of the inferior landmark can confirm the position before ment cannot be reduced at an abduction angle of <25°, the
reaming. During reaming, identification of pulvinar-like hip abductors must be adequately released from the iliac
tissue in the inferior acetabulum is a good indication of the wing, and consideration may have to be given to revising
correct position. Nevertheless, the true position of antever- the femoral osteotomy.56 Other authors have recommended
sion and abduction of the cup may be skewed because of avoiding elevation of the abductor mass from the iliac wing
overall abnormal pelvic fixed abnormalities. If this aberrant to prevent vascular injury and eventual scarring.60
anatomy is not appreciated, excessive cup anteversion with After completion of the THA, we typically perform a
anterior dislocation of the cup may occur. percutaneous adductor tenotomy if the abduction angle is
Femoral canal preparation can be challenging because of limited (<30°).54 The postoperative regimen is dictated by
abnormal proximal femoral anatomy with a sclerotic femo- the quality of the osteotomized trochanteric fragment and
ral canal. One must be prepared to use modular implants the hip abductor muscle. For cases involving a small oste-
to accommodate anatomical deficiencies or abnormalities. otomized fragment or thin and fibrotic abductor musculature,
538
536 The
The American
American Journal
Journal of
of Orthopedics
Orthopedics®®
S. S. Sathappan et al
Figure 6. Anteroposterior x-ray of a 50-year-old woman shows Figure 7. Anteroposterior x-ray of a 27-year-old woman who
bilateral acetabular protrusio with rheumatoid arthritis. The had realignment osteotomy for developmental dysplasia of the
breach in Kohler’s line is distinct. hip at age 11 and was now presenting with degenerative arthri-
tis of the right hip.
Secondary. Central fracture dislocation of acetabulum, Patients with Down syndrome represent a significant
Paget disease, Marfan syndrome, rheumatoid arthritis, percentage of cases of hip dysplasia, and their management
ankylosing spondylitis, osteomalacia, sickle cell disease is similar to that of patients with DDH. In general, relative
(Figure 6).77-79 lack of muscle tone rules out hip arthroplasty in patients
with myelomeningocele84 and leads to poor outcomes
Although the cause of protrusio may be obvious in in patients with a history of poliomyelitis, though use of
trauma, in other conditions certain mechanisms have been constrained liners may allow successful hip replacement
proposed. In sickle cell disease, protrusio acetabuli has in selected cases. Patients with cerebral palsy develop
been attributed to pelvic marrow hyperplasia that weakens progressive femoral head subluxation caused by muscular
the bone, leading to a discontinuity in the Kohler line.77,78 imbalance, spasticity, soft-tissue contractures, coxa valga,
The technical principles to consider when performing accentuated femoral anteversion, and increased acetabular
THA in these patients include: index. One often needs to perform adductor tenotomies and
•The hip dislocation may be difficult and thus necessitate use hip spicas after surgery.83,85
an in situ femoral neck osteotomy. Pain relief and functional improvement can be expected
•The hip center should be restored to within 10 mm of its in the majority of patients with neuromuscular condi-
anatomical location for improved cup survivorship.80 tions in general and cerebral palsy specifically.83,86 As
•Medialization should be avoided and reaming limited the functional demands of patients with cerebral palsy are
primarily to the periphery of the acetabulum. low, prosthesis survival can be as high as 95% at 10 years;
•Peripheral rim support should be maintained when a however, because of spasticity and muscular imbalances
cementless acetabular cup is used. there is a high rate of dislocation.86
•The cavitary defect behind the cup should be augmented
with particulate cancellous bone graft and impaction graft- Skeletal Dysplasias
ing techniques.81 Being of short stature and having significant joint
•Excessive limb-lengthening may be avoided by doing deformities predispose patients with skeletal dysplasia
a low femoral neck resection and using components with to accelerated degenerative changes about the hip.87
increased medial offset.8 Patients often have degenerative changes in multiple
joints, including the spine, and therefore a diagnostic
Outcomes of cemented primary THAs in patients with fluoroscopy-guided local anesthetic injection into the
protrusio acetabuli have been shown to be similar to those of hip joint may be needed to differentiate hip pain from
cementless primary THAs at a mean follow-up 60 months.82 spine pain.87,88 The small osseous anatomy with periar-
ticular deformity necessitates careful preoperative plan-
Neuromuscular Conditions ning with appropriate implant selection and possibly a
Patients with neuromuscular conditions can be divided into modular or custom prosthesis. These patients have a
those with decreased muscle tone (eg, poliomyelitis, Down higher predisposition to cervical spinal instability and
syndrome, myelomeningocele) and those with increased may require fiber-optic intubation if general anesthesia
muscle tone (eg, cerebral palsy, Parkinson disease, stroke).83 is used.87,89 The revision rate in these patients is particu-
Patients with these conditions may require THA because of larly high (29%) because of aseptic loosening of the cup
subsequent hip dysplasia attributable to abnormal neuromus- and/or stem, periprosthetic fracture, infection, and/or
cular tone or progressive degenerative changes. extensive osteolysis.87,90
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Surgical Challenges in Complex Primary Total Hip Arthroplasty
540
536 The
The American
American Journal
Journal of
of Orthopedics
Orthopedics®®
S. S. Sathappan et al
28. Mulroy RD Jr, Harris WH. Failure of acetabular autogenous grafts in total hip 59. Amstutz HC, Sakai DN. Total joint replacement for ankylosed hips. Indications,
arthroplasty. Increasing incidence: a follow-up note. J Bone Joint Surg Am. technique, and preliminary results. J Bone Joint Surg Am. 1975;57(5):619-
1990;72(10):1536-1540. 625.
29. Enneking WF, Mindell ER. Observations on massive retrieved human 60. Kilgus DJ, Amstutz HC, Wolgin MA, Dorey FJ. Joint replacement for anky-
allografts. J Bone Joint Surg Am. 1991;73(8):1123-1142. losed hips. J Bone Joint Surg Am. 1990;72(1):45-54.
30. Hooten JP Jr, Engh CA, Heekin RD, Vinh TN. Structural bulk allografts in 61. Kim YH, Oh SH, Kim JS, Lee SH. Total hip arthroplasty for the treatment of
acetabular reconstruction. Analysis of two grafts retrieved at post-mortem. osseous ankylosed hips. Clin Orthop. 2003;(414):136-148.
J Bone Joint Surg Br. 1996;78(2):270-275. 62. Reikeras O, Bjerkreim I, Gundersson R. Total hip arthroplasty for arthrodesed
31. Shinar AA, Harris WH. Bulk structural autogenous grafts and allografts for hips. 5- to 13-year results. J Arthroplasty. 1995;10(4):529-531.
reconstruction of the acetabulum in total hip arthroplasty. Sixteen-year-aver- 63. Hardinge K, Murphy JC, Frenyo S. Conversion of hip fusion to Charnley low-
age follow-up. J Bone Joint Surg Am. 1997;79(2):159-168. friction arthroplasty. Clin Orthop. 1986;(211):173-179.
32. Perka C, Fischer U, Taylor WR, Matziolis G. Developmental hip dysplasia 64. Brewster RC, Coventry MB, Johnson EW Jr. Conversion of the arthrodesed
treated with total hip arthroplasty with a straight stem and a threaded cup. hip to a total hip arthroplasty. J Bone Joint Surg Am. 1975;57(1):27-30.
J Bone Joint Surg Am. 2004;86-A(2):312-319. 65. Di Cesare PE, Nimni ME, Peng L, Yazdi M, Cheung DT. Effects of indo-
33. Edwards BN, Tullos HS, Noble PC. Contributory factors and etiology of sci- methacin on demineralized bone–induced heterotopic ossification in the rat.
atic nerve palsy in total hip arthroplasty. Clin Orthop. 1987;(218):136-141. J Orthop Res. 1991;9(6):855-861.
34. Nercessian OA, Piccoluga F, Eftekhar NS. Postoperative sciatic and femoral 66. Harder AT, An YH. The mechanisms of the inhibitory effects of nonsteroidal
nerve palsy with reference to leg lengthening and medialization/lateralization anti-inflammatory drugs on bone healing: a concise review. J Clin Pharmacol.
of the hip joint following total hip arthroplasty. Clin Orthop. 1994;(304):165- 2003;43(8):807-815.
171. 67. Panagiotopoulos KP, Robbins GM, Masri BA, Duncan CP. Conversion of
35. Symeonides PP, Pournaras J, Petsatodes G, Christoforides J, Hatzokos I, hip arthrodesis to total hip arthroplasty. Instr Course Lect. 2001;50:297-
Pantazis E. Total hip arthroplasty in neglected congenital dislocation of the 305.
hip. Clin Orthop. 1997;(341):55-61. 68. Matta JM. Fractures of the acetabulum: accuracy of reduction and clinical
36. Russotti GM, Harris WH. Proximal placement of the acetabular component results in patients managed operatively within three weeks after the injury.
in total hip arthroplasty. A long-term follow-up study. J Bone Joint Surg Am. J Bone Joint Surg Am. 1996;78(11):1632-1645.
1991;73(4):587-592. 69. Mears DC, Velyvis JH, Chang CP. Displaced acetabular fractures managed
37. Lengsfeld M, Bassaly A, Boudriot U, Pressel T, Griss P. Size and direction operatively: indicators of outcome. Clin Orthop. 2003;(407):173-186.
of hip joint forces associated with various positions of the acetabulum. 70. Mears DC. Surgical treatment of acetabular fractures in elderly patients with
J Arthroplasty. 2000;15(3):314-320. osteoporotic bone. J Am Acad Orthop Surg. 1999;7(2):128-141.
38. Callaghan JJ, Salvati EA, Pellicci PM, Wilson PD Jr, Ranawat CS. Results of 71. Bellabarba C, Berger RA, Bentley CD, et al. Cementless acetabular recon-
revision for mechanical failure after cemented total hip replacement, 1979 to struction after acetabular fracture. J Bone Joint Surg Am. 2001;83-A(6):868-
1982. A two to five-year follow-up. J Bone Joint Surg Am. 1985;67(7):1074- 876.
1085. 72. Weber M, Berry DJ, Harmsen WS. Total hip arthroplasty after operative
39. Yoder SA, Brand RA, Pedersen DR, O’Gorman TW. Total hip acetabular treatment of an acetabular fracture. J Bone Joint Surg Am. 1998;80(9):1295-
component position affects component loosening rates. Clin Orthop. 1305.
1988;(228):79-87. 73. Jimenez ML, Tile M, Schenk RS. Total hip replacement after acetabular frac-
40. Kelley SS. High hip center in revision arthroplasty. J Arthroplasty. ture. Orthop Clin North Am. 1997;28(3):435-446.
1994;9(5):503-510. 74. Mears DC, Velyvis JH. Acute total hip arthroplasty for selected displaced
41. Pagnano W, Hanssen AD, Lewallen DG, Shaughnessy WJ. The effect of acetabular fractures: two to twelve-year results. J Bone Joint Surg Am.
superior placement of the acetabular component on the rate of loosening 2002;84-A(1):1-9.
after total hip arthroplasty. J Bone Joint Surg Am. 1996;78(7):1004-1014. 75. McKinley JC, Robinson CM. Treatment of displaced intracapsular hip frac-
42. Linde F, Jensen J. Socket loosening in arthroplasty for congenital dislocation tures with total hip arthroplasty: comparison of primary arthroplasty with
of the hip. Acta Orthop Scand. 1988;59(3):254-257. early salvage arthroplasty after failed internal fixation. J Bone Joint Surg Am.
43. Stans AA, Pagnano MW, Shaughnessy WJ, Hanssen AD. Results of total 2002;84-A(11):2010-2015.
hip arthroplasty for Crowe type III developmental hip dysplasia. Clin Orthop. 76. Mears DC, Shirahama M. Stabilization of an acetabular fracture with cables
1998;(348):149-157. for acute total hip arthroplasty. J Arthroplasty. 1998;13(1):104-107.
44. Hedlundh U, Carlsson AS. Increased risk of dislocation with collar reinforced 77. Al-Mousawi F, Malki A, Al-Aradi A, Al-Bagali M, Al-Sadadi A, Booz MM. Total
modular heads of the Lubinus SP-2 hip prosthesis. Acta Orthop Scand. hip replacement in sickle cell disease. Int Orthop. 2002;26(3):157-161.
1996;67(2):204-205. 78. Martinez S, Apple JS, Baber C, Putman CE, Rosse WF. Protrusio acetabuli
45. Scifert CF, Noble PC, Brown TD, et al. Experimental and computational in sickle-cell anemia. Radiology. 1984;151(1):43-44.
simulation of total hip arthroplasty dislocation. Orthop Clin North Am. 79. McBride MT, Muldoon MP, Santore RF, Trousdale RT, Wenger DR. Protrusio
2001;32(4):553-567. acetabuli: diagnosis and treatment. J Am Acad Orthop Surg. 2001;9(2):79-
46. Barrack RL, Thornberry RL, Ries MD, Lavernia C, Tozakoglou E. The effect 88.
of component design on range of motion to impingement in total hip arthro- 80. Bayley JC, Christie MJ, Ewald FC, Kelley K. Long-term results of total hip
plasty. Instr Course Lect. 2001;50:275-280. arthroplasty in protrusio acetabuli. J Arthroplasty. 1987;2(4):275-279.
47. Hartofilakidis G, Stamos K, Karachalios T. Treatment of high dislocation of the 81. Slooff TJ, Huiskes R, van Horn J, Lemmens AJ. Bone grafting in total hip
hip in adults with total hip arthroplasty. Operative technique and long-term replacement for acetabular protrusion. Acta Orthop Scand. 1984;55(6):593-
clinical results. J Bone Joint Surg Am. 1998;80(4):510-517. 596.
48. Huo MH, Salvati EA, Lieberman JR, Burstein AH, Wilson PD Jr. Custom- 82. Sotelo-Garza A, Charnley J. The results of Charnley arthroplasty of hip per-
designed femoral prostheses in total hip arthroplasty done with cement for formed for protrusio acetabuli. Clin Orthop. 1978;(132):12-18.
severe dysplasia of the hip. J Bone Joint Surg Am. 1993;75(10):1497-1504. 83. Cabanela ME, Weber M. Total hip arthroplasty in patients with neuromuscular
49. Cameron HU, Botsford DJ, Park YS. Influence of the Crowe rating on the disease. Instr Course Lect. 2000;49:163-168.
outcome of total hip arthroplasty in congenital hip dysplasia. J Arthroplasty. 84. Weber M, Cabanela ME. Total hip arthroplasty in patients with low-lumbar-
1996;11(5):582-587. level myelomeningocele. Orthopedics. 1998;21(6):709-712.
50. Marti RK, Schuller HM, van Steijn MJ. Superolateral bone grafting for ace- 85. Weber M, Cabanela ME. Total hip arthroplasty in patients with cerebral palsy.
tabular deficiency in primary total hip replacement and revision. J Bone Joint Orthopedics. 1999;22(4):425-427.
Surg Br. 1994;76(5):728-734. 86. Buly RL, Huo M, Root L, Binzer T, Wilson PD Jr. Total hip arthroplasty in cere-
51. Inao S, Matsuno T. Cemented total hip arthroplasty with autogenous acetab- bral palsy. Long-term follow-up results. Clin Orthop. 1993;(296):148-153.
ular bone grafting for hips with developmental dysplasia in adults: the results 87. Chiavetta JB, Parvizi J, Shaughnessy WJ, Cabanela ME. Total hip arthroplas-
at a minimum of ten years. J Bone Joint Surg Br. 2000;82(3):375-377. ty in patients with dwarfism. J Bone Joint Surg Am. 2004;86-A(2):298-304.
52. Hardinge K, Williams D, Etienne A, MacKenzie D, Charnley J. Conversion 88. Crawford RW, Ellis AM, Gie GA, Ling RS. Intra-articular local anaesthesia for
of fused hips to low friction arthroplasty. J Bone Joint Surg Br. 1977;59- pain after hip arthroplasty. J Bone Joint Surg Br. 1997;79(5):796-800.
B(4):385-392. 89. Papagelopoulos PJ, Morrey BF. Hip and knee replacement in osteogenesis
53. Strathy GM, Fitzgerald RH Jr. Total hip arthroplasty in the ankylosed hip. A imperfecta. J Bone Joint Surg Am. 1993;75(4):572-580.
ten-year follow-up. J Bone Joint Surg Am. 1988;70(7):963-966. 90. Peltonen JI, Hoikka V, Poussa M, Paavilainen T, Kaitila I. Cementless hip
54. Joshi AB, Markovic L, Hardinge K, Murphy JC. Conversion of a fused hip to arthroplasty in diastrophic dysplasia. J Arthroplasty. 1992;7(suppl):369-376.
total hip arthroplasty. J Bone Joint Surg Am. 2002;84-A(8):1335-1341. 91. Papagelopoulos PJ, Trousdale RT, Lewallen DG. Total hip arthroplas-
55. Welch RB, Charnley J. Low-friction arthroplasty of the hip in rheumatoid ty with femoral osteotomy for proximal femoral deformity. Clin Orthop.
arthritis and ankylosing spondylitis. Clin Orthop. 1970;72:22-32. 1996;(332):151-162.
56. Hamadouche M, Kerboull L, Meunier A, Courpied JP, Kerboull M. Total hip 92. Ferguson GM, Cabanela ME, Ilstrup DM. Total hip arthroplasty after failed
arthroplasty for the treatment of ankylosed hips: a five to twenty-one-year intertrochanteric osteotomy. J Bone Joint Surg Br. 1994;76(2):252-257.
follow-up study. J Bone Joint Surg Am. 2001;83-A(7):992-998. 93. Peters CL, Beck M, Dunn HK. Total hip arthroplasty in young adults after
57. Rittmeister M, Starker M, Zichner L. Hip and knee replacement after long- failed triple innominate osteotomy. J Arthroplasty. 2001;16(2):188-195.
standing hip arthrodesis. Clin Orthop. 2000;(371):136-145. 94. Soballe K, Boll KL, Kofod S, Severinsen B, Kristensen SS. Total hip replace-
58. Lubahn JD, Evarts CM, Feltner JB. Conversion of ankylosed hips to total hip ment after medial-displacement osteotomy of the proximal part of the
arthroplasty. Clin Orthop. 1980;(153):146-152. femur. J Bone Joint Surg Am. 1989;71(5):692-697.
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