DERMATOLOGY-NEET IMAGE QUESTIONS

PREPARED BY

• DR MANJUNATH MBBS MD (MAMC NEW DELHI)

• AUTHOR OF NEET BOOK & E BOOK
• DIRECTOR DOCTORS ACADEMY
• SHIVAMOGGA/DAVANAGERE
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA(www.doctorsacademydvg.com)

DERMATOLOGY VISUALS
1. IDENTIFY THE LESION?
A.LICHEN NITIDUS
B.LICHEN PLANUS
C.ATOPIC DERMATITIS
D.POMPHOLYX

ANS: A. LICHEN NITIDUS

FLAT TOPPED SHINY
HYPOPIGMENTED LEISONS
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA

DERMATOLOGY VISUALS

2. IDENTIFY THE LESION?

(A)ERYTHEMA MULTIFORME
(B) GIANOTTI-CROSTI
(C) PITYRIASIS
(D) ACNE ROSACEA

ANS: (A)ERYTHEMA MULTIFORME.

CLUE : BLANCHING CENTRE AND TARGET
LEISONS
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA(www.doctorsacademydvg.com)

DERMATOLOGY VISUALS
2.EXPLANATION: ERYTHEMA MULTIFORME
• NOTICE THE TARGET SHAPED LESIONS RESEMBLING BULLS’ EYE
DIAGNOSTIC OF REACTIVE RASH SECONDARY TO INFECTIONS LIKE
MYCOPLASMA AND HERPES SIMPLEX. THE TARGET LESION
AFFECTS THE DISTAL EXTREMITIES AND CAN BECOME
WIDESPREAD.
• GIANOTTI-CROSTI SYNDROME IS A REACTIVE POPULAR RASH
SEEN IN CHILDREN SECONDARY TO VIRAL INFECTIONS INVOLVING
THE BUTTOCKS, EXTREMITIES, FACE AND IS ASYMPTOMATIC. THE
PAPULES ARE FIRM ON POPULATION. CHILDREN ARE
SYSTEMATICALLY WELL AND LESIONS SETTLE OVER 6-8 WEEKS.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
3. IDENTIFY THE LESION?
(A)PITYRIASIS VERSICOLOR
(B) CAFÉ AU LAIT PATCH
(C) PITYRIASIS ALBA
(D) PITYRIASIS ROSEA

ANS: (A) PITYRIASIS VERSICOLOR

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
3. ANS.A. PITYRIASIS VERSICOLOR.
• PITYRIASIS VERSICOLOR IS A FUNGAL INFECTION CAUSED BY A YEAST MALASSEZIA
FURFUR PRESENTING AS HYPER OR HYPOPIGMENTED LESIONS ON THE TRUNK AND
PROXIMAL EXTREMITIES AND FACE. LESIONS ARE SMALL SCALY AND ASYMPTOMATIC.
MICROSCOPYSHOWS PSEUDOHYPHAE AND SPORES, AN APPEARANCE TERMED AS
SPAGHETTI AND MEATBALLS.
• CAFÉ AU LAIT MACULES (CALM) ARE LIGHT BROWN MACULES HAVING AN IRREGULAR
BORDER AND PRESENCE OF>5CALM>5MM IN DIAMETER AND OTHER SIGNS LIKE
AXILLARY FRECKLES, NEUROFIBROMAS, LISCH NODULES IN EYES AND FAMILY HISTORY
POINT TO DIAGNOSIS OF NEUROFIBROMATOSIS.
• PITYRIASIS ALBA IS PALE WHITE PATCHES ON THE FACE, MORE COMMON IN CHILDREN
AND NEEDS TO BE DIFFERENTIATED FROM VITILIGO WHERE THE PIGMENT IS LOST
RATHER THAN DECREASED. SINCE IMAGE LOOKS TO BE OF AN ADULT AND HAS
HYPERPIGMENTATION, PITYRIASIS ALBA IS RULED OUT.
• PITYRIASIS ROSEA REPRESENTS A HYPERSENSITIVE RASH WITH COLLARET OF SCALES
SECONDARY TO VIRAL INFECTION INVOLVING THE TRUNK AND PROXIMAL EXTREMITIES
AND HAS A CHARACTERISTIC FIR TREE APPEARANCE ON THE BACK.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
4. A PATIENT AFTER A TRIP TO
BANGKOK HAS DEVELOPED
FEVER AND PERIORAL
VESICLES, DIAGNOSIS IS:
(A) HERPES SIMPLEX
(B) IMPETIGO
(C) MOLLUSCUM
CONTAGIOSUM
(D) BULLOUS PEMPHIGOID

ANS: (A) HERPES SIMPLEX.

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
4. ANS. A. HERPES SIMPLEX
• NOTICE THE GROUPED VESICLES WITH LESIONS SHOWING UMBILICATION
WHICH IS A PRESENTATION OF PRIMARY HERPES GINGIVOSTOMATITIS.
• PRIMARY EPISODE IS QUITE DISTRESSING FOR THE PATIENT WITH FEVER
CONSTITUTIONAL SYMPTOMS AND SMALL ERODED PAINFUL LESIONS ON LIPS
ORAL MUCOSA AND PERIORAL AREA.
• IMPETIGO PRESENTS WITH HONEY COLORED CRUSTS ON THE SKIN AND IS
RULED OUT.
• MOLLUSCUM CONTAGIOSUM IS CAUSED BY POX VIRUS INFECTION AND
PRODUCES CROPS OF DOME SHAPED PAPULES WITH UMBILICATED CENTRE ON
BODY WHICH ARE ASYMPTOMATIC.
• IN THE QUESTION PATIENT HAS FEVER AND LOCATION OF VESICLES IS PERI-
ORAL AND TRIP TO A PLACE KNOWN FOR SEXTOURISM WHICH FAVORS
DIAGNOSIS OF HERPES SIMPLEX.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
5.IDENTIFY THE LESION?
A.MOLLUSCUM
CONTAGIOSUM
B.HERPES SIMPLEX
C.PITYRIASIS ROSEA
D.GIANOTTI-CROSTI
SYNDROME

ANS: A.MOLLUSCUM
CONTAGIOSUM
CAUSED BY POX VIRUS
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
6. A 25-YEAR OLD FEMALE WITH
HISTORY OF USING TOPICAL
STEROIDS. IDENTIFY THE
LESION?
A. TINEA CORPORIS
B. BLACK DOT TINEA
C. DERMOGRAPHISM
D. TINEA INCOGNITO

ANS: A. TINEA CORPORIS

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
6. ANS. A. TINEA CORPORIS
• TINEA CORPORIS PRESENTS AS AN ANNULAR
EXPANDING LESION WITH SCALY EDGE AND CENTRAL
CLEARING.
• THE PHYSICAL APPEARANCE OF CENTRAL CLEARING IS
LOST WHEN PATIENT APPLIES TOPICAL STEROIDS
CALLED AS TINEA INCOGNITO.
• FUNGAL SPORES INVOLVING INTERIOR OF HAIR SHAFT
MAKE IT WEAKER, HAIR TEND TO BREAK NEAR THE
SCALP PRODUCING THE CHARACTERISTIC CLINICAL
BLACK DOT APPEARANCE
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
7. A 16-YEAR-OLD BOY WITH
CRYPTORCHIDISM PRESENTS
WITH DRY SKIN. IDENTIFY THE
LESION?
A. X-LINKED ICHTHYOSIS
B. ICHTHYOSIS VULGARIS
C. LAMELLAR ICHTHYOSIS
D. KINDLER SYNDROME

ANS: (A) X-LINKED ICHTHYOSIS

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
7. ANS. A. X-LINKED ICHTHYOSIS
• X-LINKED RECESSIVE ICHTHYOSIS PRESENTS IN BOYS DUE TO DEFICIENCY OF
ENZYME STEROID SULFATASE. BOTH THE GENE AND ENZYME CAN BE ASSESSED
FOR CONFIRMATION OF DIAGNOSIS. IT HAS LARGE BROWN SCALES SEEN ON
THE LEGS AND TRUNK AND INVOLVES THE FLEXURES. IT IS ASSOCIATED WITH
CRYPTORCHIDISM.
• ICHTHYOSIS VULGARIS IS AN AUTOSOMAL DOMINANT CONDITION INVOLVING
DRY SKIN CAUSED BY FILAGGRIN GENE MUTATION WITH LIGHT BROWN
SCALES SPARING THE FLEXURES. PALMS AND CREASES SHOW
HYPERLINEARITY.
• LAMELLAR ICHTHYOSIS LEADS TO RETENTION OF SCALES AND CHILD BEING
BORN WITH A SHINY MEMBRANE COVERING THE BODY CALLED COLLODION
MEMBRANE
• KINDLER SYNDROME IS CHARACTERIZED BY ACRAL FRAGILITY,
PHOTOSENSITIVITY, POIKILODERMA AND CUTANEOUSATROPHY.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
8. NEONATE WITH
BLOTCHY RASH ON
THE ABDOMEN.
DIAGNOSIS IS:
A. CONGENITAL SYPHILIS
B. CUTIS MARMORATA
C. ERYTHEMA TOXICUM
NEONATORUM
D. MILIA

ANS: (C) ERYTHEMA TOXICUM

NEONATORUM
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY

8. ANS. C. ERYTHEMA TOXICUM NEONATORUM

• ERYTHEMA TOXICUM NEONATORUM PRESENTS IN NEONATES
WITH BLOTCHY MACULOPAPULAR RASH WITH PUSTULES. THESE
PUSTULES ARE STERILE AND CONTAIN EOSINOPHILS AND THE
RASH USUALLY SETTLES BY DAY 10 OF LIFE.
• CUTIS MARMORATA PRESENTS WITH MOTTLING WHICH
DISAPPEARS WHEN THE SKIN BECOMES WARMER. THE
MOTTLING IS LIKELY TO DISAPPEAR BEYOND NEONATAL PERIOD.
PERSISTENCE BEYOND NEONATAL PERIOD MAY BE ASSOCIATED
WITH HYPOTHYROIDISM, TRISOMY 21 AND CORNELIA DE LANGE
SYNDROME.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
9. A 13-YEAR-OLD BOY WITH
PATCHY DEPIGMENTED SKIN
ON THE RIGHT FLANK AND
UPPER THIGH IN SEGMENTAL
DISTRIBUTION. THE
DEPIGMENTATION STARTED 1-
YEAR BACK BUT HAS BEEN
STATIC FOR LAST FOUR
MONTHS. MOTHER REPORTS
USEOF TOPICAL STEROIDS
WHICH WAS INEFFECTIVE.
A. PIEBALDISM
B. SEGMENTAL VITILIGO
C. HYPOMELANOSIS OF ITO
D. HYPOPIGMENTED STREAKS
ANS: (B) SEGMENTAL VITILIGO
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
9. ANS. B. SEGMENTAL VITILIGO.
• VITILIGO IS AN ACQUIRED AUTOIMMUNE CONDITION TARGETING
MELANOCYTES AND PRESENT WITH LOCALIZED OR WIDESPREAD WHITE
DEPIGMENTED PATCHES. THYROID AUTOIMMUNE DYSFUNCTION IS
ASSOCIATED.
• PIEBALDISM IS AN AUTOSOMAL DOMINANT CONDITION CHARACTERIZED BY
WHITE FORELOCK AND CIRCUMSCRIBED DEPIGMENTED PATCHES AFFECTING
THE BODY. IT IS CAUSED BY A DEFECT IN PROLIFERATION AND MIGRATION OF
MELANOCYTES DURING EMBRYOGENESIS. UNLIKE VITILIGO, IT IS CONGENITAL
AND NON-PROGRESSIVE. IN THE QUESTION THE DEPIGMENTATION STARTED
AT AGE OF 12 YEARS AND PATIENT PRESENTED AT 13 YEARS.
• NEVOID HYPOMELANOSIS IS CHARACTERIZED BY HYPOPIGMENTED PATCHES
OR STREAKS WHICH FOLLOW THE LINES OF BLASCHKO. THEY ARE PRESENT AT
BIRTH AND MAY DEVELOP IN THE FIRST 2 YEARS OF LOFE.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
10. IDENTIFY THE LESION?
A. PROTEUS SYNDROME
B. CRUVEIHIER-BAUMGARTEN
DISEASE
C. DERMAL NEUROFIBROMA
D. PLEXIFORM NEUROFIBROMA

ANS: (C) DERMAL NEUROFIBROMA

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
10. ANS. C. DERMAL NEUROFIBROMA
PROTEOUS Proteus syndrome is a progressive condition wherein children are usually born
SYNDROME without any obvious deformities. Tumors of skin and bone growths appear as
. they age. The severity and locations of these various asymmetrical growths vary
greatly but typically the skull, one or more limbs, and soles of the feet will be
affected.

CRUVEILHIER- Cruveihier-Baumgarten disease is medical condition in which the umbilical or

BAUMGARTEN
DISEASE paraumbilical veins are distended, with an abdominal wall bruit.

DERMAL Dermal neurofibroma typically arise in the teenage years and are often associated
NEUROFIBROMA
with the onset of puberty. They continue to increase in number and size
throughout adulthood, although there are limits to how big they get.

PLEXIFORM Plexiform neurofibromas can grow from nerves in the skin of from more internal
NEUROFIBROMA
nerve bundles, and can be very large. Internal plexiform neurofibromas are very
difficult to remove completely because they extend through multiple layers of
tissue and the attempt would damage healthy tissue or organs.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY

11. IDENTIFY THE

LESION?
A.KOENEN TUMOR
B.ONYCHOMYCOSIS
C.ONYCHODYSTROPHY
D.ONYCHOGRYPHOSIS
ANS: (A) KOENEN TUMOR
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DERMATOLOGY

11. ANS. A. KOENEN TUMOR.

KOENEN TUMOR IS SEEN IN TUBEROUS SCLEROSIS AND
IS SUBUNGUAL FIBROMA.
TUBEROUS SCLEROSIS (TS) IS INHERITED AS AN
AUTOSOMAL DOMINANT TRAIT WITH VARIABLE
PENETRANCE AND A PREVALENCE OF 1/6,000 PEOPLE.
SPONTANEOUS GENETIC MUTATIONS OCCUR IN UP TO
75% CASES. MOLECULAR GENETIC STUDIES HAVE
IDENTIFIED TWO FOCI FOR THE TS COMPLEX. THE TSCI
GENE IS LOCATED ON CHROMOSOME 9Q34, AND THE
TSC2 GENE IS ON CHROMOSOME 16P13.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
12. A 25-YEAR-OLD GIRL PRESENTS
WITH PAPULES, ERYTHEMA AND
TALANGIECTASIAE OVER THE
FACE AS SHOWN BELOW. SHE
ALSO GIVES A HISTORY OF
FLUSHING AND BURNING
SENSATION ON EXPOSURE TO
SUN AND ON ANY EMOTIONAL
DISTURBANCE. THE MOST LIKELY
DIAGNOSIS IS:
A. ACNE VULGARIS
B. ROSACEA
C. SYSTEMIC LUPUS
ERYTHEMATOSUS
D. SCABIES
ANS: (B) ROSACEA
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY
13. AN ELDERLY PATIENT
PRESENTS WITH ITCHY TENSE
BLISTERS ON NORMAL
LOOKING SKIN AS WELL AS ON
URTICARIAL PLAQUES AS
SHOWN BELOW. THE MOST
PROBABLE DIAGNOSIS IS:

A. PEMPHIGUS VULGARIS
B. LINEAR IGA DISEASE
C. BULLOUS PEMPHIGOID
D. DERMATITIS HERPETIFORMIS

ANS: (C) BULLOUS PEMPHIGOID

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
14. A 42-YEARS MALE COMPLAINS
OF ITCHING, HIS CLINICAL
PRESENTATION IS GIVEN IN THE
IMAGE, WHICH OF THE
FOLLOWING STATEMENT IF
FALSE:
A. MOST LIKELY THIS IS TINEA
COPORIS
B. THIS INFECTS NON-HAIRY SKIN
C. CLASSICAL PRESENTATION IS RING
LIKE AND ACRIFORM LESION
D. INITIALLY PRESENTS AS PAPULE
THAT SPREADS INWARDS
ANS: (D) INITIALLY PRESENTS AS PAPULE THAT
SPREADS INWARD.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY
14. Ans. D. Initially presents as papule that spreads
inwards.
Tinea corporis –infection of the non-hairy skin of trunk and limbs.
• The typical lesions start as itchy erythematous macule or papules
that spread outward and develop into annular (ring like) and
arciform lesions with sharp, scaling or papulovesicular advancing
margin and healing centers.
• A variant of tinea corporis called tinea imbricate (imbricate is
Latin for tiled) caused by Trichophyton concentricum, is
characterized by large concentric rings, one inside another, which
manifest commonly in childhood.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
15. ALL OF THE FOLLOWING
STATEMENTS REGARDING
THIS IMAGE IS TRUE
EXCEPT?
A. KNOWN AS DHOBI ITCH
B. KNOWN AS JOCK ITCH
C. MOST COMMON IN
TROPIC
D. THIS IS CAUSED BY
CANDIDA
ANS: (D) THIS IS CAUSED BY CANDIDA
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DERMATOLOGY

15. ANS. D. THIS IS CAUSED BY CANDIDA.

TINEA CRURIS-RINGWORM OF THE GROIN (“JOCK’S ITCH “DHOBIE
ITCH”)
• MOST COMMON IN THE TROPICS.
• DIFFERENTIAL DIAGNOSIS ARE CANDIDIASIS (MORE OFTEN
INVOLVES CONCAVITIES OF THE FLEXURES AND PRODUCES
MACERATED, MOIST, GLAZED, ERYTHEMATOUS PLAQUES WITH
SATELLITE PUSTULES AND IT MORE OFTEN INVOLVES THE
SCROTUM), INVERSE PSORIASIS (TENDS TO BE ASYMPTOMATIC,
WELL-DEFINED ERYTHEMATOUS PLAQUE), AND ERYTHRASMA
(LACKS A SCALING BORDER AND INFLAMMATION,
ASYMPTOMATIC IN NATURE, FLUORESCES CORAL RED UNDER A
WOOD LIGHT).
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY
16. WHICH OF THE FOLLOWING
STATEMENT REGARDING THE
IMAGE IS FALSE?
A. THIS IS TRANSMITTED FROM
CHILD TO CHILD
B. CAUSED BY A RING WORM
C. MOSTLY THE ORGANISM
CAUSES ECTOTHRIX INFECTION
D. MOST COMMONLY CAUSED BY
T-TONSURANS

ANS: ( C) MOSTLY THE ORGANISM CAUSES

ECTOTHRIX INFECTION
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY
16. ANS. C. MOSTLY THE ORGANISM CAUSES ECTOTHRIX INFECTION.
TINEA CAPITIS – RINGWORM OF THE SCALP, TRANSMITTED FROM
CHILD TO CHILD, MOST COMMONLY CAUSED BY TRICHOPHYTON
VIOLACEUM. IN THE WESTERN LITERATURE, COMMONEST
CAUSATIVE ORGANISM RECORDED IS T. TONSURANS. BOTH OF
THEM CAUSE ENDOTHRIX INFECTION (ARTHROCONIDIA OF
DERMATOPHYTES CONTAINED WITHIN THE HAIR SHAFT) OF HAIR.
LESS FREQUENTLY, ECTOTHRIX INFECTION (ARTHROCONIDIA OF
DERMATOPHYTES SURROUNDING THE HAIR SHAFT AS A SHEATH)
OF HAIR ALSO OCCURS.
• PATCHY HAIR LOSS AND BROKEN HAIRS, INFLAMMATION AND
SCALING ARE CHARACTERISTIC OF (BACK DOT, GREY PATCH,
SEBORRHEIC DERMATITIS LIKE) TINEA CAPITIS.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY

17. WHAT IS THE MOST

LIKELY DIAGNOSIS OF
THE IMAGE PROVIDED
BELOW:
A. TINEA VERSICOLOR
B. TINEA CORPORIS
C.PITYRIASIS ALBA
D.VITILIGO
ANS: A. TINEA VERSICOLOR
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DERMATOLOGY
17. ANS. A. TINEA VERSICOLOR.
A PECULIAR ASPECT OF TINEA VERSICOLOR IS ITS PROPENSITY TO PRESENT
AS EITHER HYPOPIGMENTED OR HYPERPIGMENTED, FINELY SCALING,
ROUND OR PERIFOLLICULAR COALESCING MACULAR PATCHES FOUND
PRIMARILY OVER THE TRUNK. THE HYPOPIGMENTATION IS EXPLAINED
ON THE BASIS OF DICARBOXYLIC ACIDS PRODUCED BY MALASSEZIA
SPECIES (E.G., AZELAIC ACID) CAUSING COMPETITIVE UNHIBITION OF
TYROSINASE AND PERHAPS A DIRECT CYTOTOXIC EFFECT ON
HYPERACTIVE MELANOCYTES. THE HYPERPIGMENTATION MAY BE DUE
TO ABNORMALLY LARGE MELANOSOMES AND THICKER KERATIN LAYER.
TO ELICIT FINE BRANNY POWDERY SCALES, CANDLE GREASE SIGN OR
COUP D’ONGLE SIGN IS ELICITED.
• THE ERUPTION IS ALMOST ALWAYS ASYMPTOMATIC AND ONLY OF
COSMETIC SIGNIFICANCE.
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DERMATOLOGY
18. IDENTIFY THE LESION?
A.LICHEN PLANUS
B. DERMATOMYOSITIS
C. PSORIASIS
D.DERMATITIS
HERPETIFORMIS

ANS: C. PSORIASIS
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DERMATOLOGY
19. ALL OF THE FOLLOWING
STATEMENT REGARDING
IMAGE GIVEN BELOW ARE
TRUE EXCEPT?
A. IT IS DEEP FUNGAL INFECTION
B. IT IS CAUSED BY ANAEROBIC
ACTINOMYCETES
C. COMMONLY OCCURS IN FOOT
D. PRESENTS WITH
DISCHARGING SINUS

ANS. B. IT IS CAUSED BY ANAEROBIC

ACTINOMYCETES
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY
19. ANS. B. IT IS CAUSED BY ANAEROBIC ACTINOMYCETES.
MYCETOMA IS A DEEP FUNGAL INFECTION,
CHARACTERIZED BY A CLINICAL TRIAD OF SWELLING,
DISCHARGING SINUSES AND DISCHARGE CONTAINING
GRANULES. IT COMMONLY OCCURS ON THE FOOT,
HENCE CALLED AS MADURA FOOT.
MYCETOMA-CAUSED BY SPECIES OF FUNGAI IS KNOWN
AS EUMYCETOMA, AND THAT CAUSED BY AEROBIC
ACTINOMYCETES OR FILAMENTOUS BACTERIA AS
ACTINOMYCETOMA.
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
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DERMATOLOGY
20. IDENTIFY THE LESION?
A.PSORIASIS
B. DERMATITIS
HERPETIFORMIS
C. ERYTHEMA
MARGINATUM
D.DERMATOMYOSITIS

ANS: B. DERMATITIS HERPETIFORMIS

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY

21. IDENTIFY THE LESION?

A. PSORIASIS
B. DERMATITIS HERPETIFORMIS
C. ERYTHEMA MARGINATUM
D. ERYTHEMA MULTIFORME

ANS: D. ERYTHEMA MULTIFORME

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
22. A 26 YEAR OLD FEMALE WITH
A HISTORY OF EXTENSIVE
EXPOSURE TO SUN PRESENT
TO YOUR CLINIC WITH
PRESENTATION SIMILAR TO
THAT OF IMAGE GIVEN
BELOW. WHAT IS THE MOST
LIKELY DIAGNOSIS?
A. URTICARIA
B. DERMATITIS HERPETIFORMIS
C. ERYTHEMA MARGINATUM
D. DERMATOMYOSITIS

ANS: A. URTICARIA
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
23. IDENTIFY THE LESION?

A. VITILIGO
B. DERMATITIS
HERPETIFORMIS
C. ERYTHEMA
MARGINATUM FAMOUS example
D. DERMATOMYOSITIS

ANS: A. VITILIGO
 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
24. IDENTIFY THE LESION?

A. PSORIASIS
B. DERMATITIS HERPETIFORMIS
C. ERYTHEMA MARGINATUM
D. LICHEN PLANUS

ANS: D. LICHEN PLANUS

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
25. A 41-YEAR OLD MALE
COMPLAINS OF ITCHING FOR
OVER A DURATION OF SIX
MONTHS. HIS CLINICAL
PRESENTATION IS GIVEN IN THE
IMAGE BELOW. WHAT IS THE
MOST LIKELY DIAGNOSIS FROM
THE OPTION GIVEN BELOW?
A. PITYRIASIS ROSEA
B. DERMATITIS HERPETIFORMIS
C. ERYTHEMA MARGINATUM
D. DERMATOMYOSITIS

ANS: A. PITYRIASIS ROSEA

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
26. 32-YEAR-OLD FEMALE WITH
A COMPLAINT OF FEVER AND
ITCHING PRESENTS TO YOU
WITH THE FEATURES SIMILAR
TO THE IMAGE GIVEN BELOW.
WHAT IS THE MOST LIKELY
DIAGNOSIS?
A. PSORIASIS
B. DERMATITIS HERPETIFORMIS
C. ERYTHEMA MARGINATUM
D. DERMATOMYOSITIS

ANS: B. DERMATITIS HERPETIFORMIS

 DR: MANJUNATH, MBBS, MD (MAMC NEW DELHI) DIRECTOR
DOCTORS ACADEMY DAVANAGERE & SHIMOGA (www.doctorsacademydvg.com)

DERMATOLOGY
27. WHAT IS THE MOST
LIKELY DIAGNOSIS OF THE
IMAGE GIVEN BELOW?
A. ACNE ROSACEA
B. DERMATITIS HERPETIFORMIS
C. ERYTHEMA MARGINATUM
D. DERMATOMYOSITIS

ANS: ACNE ROSACEA

27. IDENTIFY THE CONDITION-SIVERY
SCALES
 PSOARIASIS
• DEFINITION
• CHRONIC DERMATOSIS WITH UNPREDICTABLE REMISSIONS AND
RELAPSE CHARACTERISED BY WELL DEFINED SILVERY WHITE
SCALES PAPULES AND PLAQUES ON ERYTHEMATOUS BASE

• PATHOLGY-RAPID TURN OVER OF EPIDERMIS-3 TO 4 DAYS

(NORMALLY 28 DAYS)

• HLA DW6

• PARAKERATOSIS HYPERKERATOSIS AND MUNROMICROABSCESS

 PSOARIASIS
• LEISON-ROUND PLAQUE
• WELL DEFINED AND INDURATED
• SILVERY WHITE SCLAES
• BRIGHT RED ERYTHEMATOUS BASE
• EASILY REMOVABLE
• DEVELOPES AT THE SITE OF TRAUMA (KOEBNOERS
PHENOMENA)
• BILATERALLY SYMMETRICAL
• EXTENSOR SURFACE
• COMMON SITES-SCALP LUMBOSCARAL AREA ABD
SPINE PRESSURE POINTS
 PSOARIASIS
• FACE UNCOMMON

• MUCOAS SURFACE INVOLVEMENT IS

UNCOMMON

• SCALP INVOLVED BUT NO ALOPECIA

 PSOARIASIS
• NAILS-PITTING

• JOINTS-DIP AND KNEE SPINE

• BED SIDE TESTS

• 1. GRUTTAGE TEST-SCRAPING THE LEISONS WITH
GLASS SLIDE INCREASES THE SILVERY SCALES

• 2. AUSPITZ SIGN-FURTHER SCRAPING MAY SHOW

BURKLEY MENBRANE AND PIN POINT BLEEDING
SPOTS
 TREATMENT OF PSOARIASIS
• 1. TOPICAL STEROID+SALICYLIC ACID

• 2. TOPICAL COAL TAR

• 3. EXTENSIVE-UV B RAYS
Clinical features of psoriasis
 Sites
 Bilaterally symmetrical
 Extensors areas
 Pressure points
 Scalp
 Lumbosacral area and back

Note :
 Face (uncommon)
 Mucosal surfaces (oral, genital)
are not involved.
 Scalp involvement does not
cause alopecia.
29. IDENTIFY THE FINDING IN BELOW?
29. WICKHAMS STRAIE-LICHEN
PLANUS
 LICHEN PLANUS
• 1. CHRONIC INFLAMMATORY DISORDER OF SKIN MUCOUS
MEMBRANE AND NAILS AND HAIRS

• 2. AUTOIMMUNE CONDITION

• 3. WHITE LINES –WICKHAMS STRAIE

• 4. PRURITIC POLYGONAL PURPLE AND PLANE (FLAT TOPPED)

AND PAPULE OR PLAQUE

• 5. PTERYGHUIM UNGUIS IS MOST CHARACTERISTIC

• 6. STEROIDS TREATMENT
30. IDENTIFY THE FINDING
DERMATOMYOSITIS?
• HELIOTROPHE RASH
HELIOTROPHE RASH
 DERMATOMYOSITIS
• PROXIMAL MUSLE
• CARDIOMYOPATHY
• RAYNAULDS PHENOMENA
• GOTTRONS SIGN
• MECHANIC HAND
• SHAWL SIGN
• STEROID AND IMMUNOSUPRESSANTS
32. IDENTIFY THE CONDITION?
• PITYRIASIS ROSEA
CHRISTMASS TREE APPERANCE-
PITYRIASIS ROSEA
• 1. EXACT ETILOGY IS
UNKOWN
• 2. COMMON SCALY
DISORDER IN CHIDREN
AND YOUNG ADULTS
• 3. ASSOVIATED WITH
HHV-6
• 4. USULALY NO
TREATMENT REQUIRED.
ORAL ANTIHISTAMINICS
33. IDENTIFY THE DISEAS CAUSED K
GRANULOMATIS?
• GRANULOMA
INGUINALE
34. IDENTIFY THE DISEAS CAUSED
K GRANULOMATIS?
• GRANULOMA INGUINALE

• ULCER IS PAINLESS AND

BLEEDING ON TOUCH
INDURATED

• SUBCUANEOUS
GRANULOMA RESEMBLE
LIKE BUBOES

• AZITHROMYCIN IS DOC
35. IDENTIFY THE CONDITION
• ERYTHRASMA

• CORYNEBACTERIUM
MINUTISSIUM
36. IDENTIFY THE FINDING
TUBERCULOSIS?
• LUPUS VULGARIS
APPLE JELLY NODULES ARE SEEN IN?
• LUPUS VULGARIS
38. IDENTIFY THE LEISON
38. TINEA VERSICOLOR
39. TINEA VERSICOLOR APPEARANCE?
SPAGHETTI MEAT BALL APPEARANCE
 PITYRIASIS VERSICOLOR
• TINEA VERSICOLOR

• TRUNK AND PROXIMAL EXTREMITIES

• MALASSEZIA FURFUR AND M GLOBOSA

• PETER LAMS DISEASE

• PALE LESIONS WITH FINE SCALING

• YEAST-SPAGHETTI MEATBALL APPERANCE

• LIGHT SKIN HYPERPIGMENTATION

• YEAST-AZELAIC ACID-BLEACHING EFFECT

• TOIPICAL ANTIFUNGAL-NIZORAL SELENIUM SULFIDE OINTMENT

• CICLOPEROX OLAMINE

• CLOTRIMAZOLE AND MICONAZOLE AND ITRACONAZOLE