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Case study On
Gastritis
CASE STUDY
ON GASTRITIS
PATIENT ASSESSMENT
FAMILY HISTORY
Head of the Family : Mr. Dindayal Sharma
Family Status : Nuclear
No. of Children : 2
House : Own
Locality : Rural
Type of House : Semi Pucca
Ventilation : Proper
Kitchen : Separate
Bathroom : Separate
Drainage : proper
Water Supply : Well/hand pump
Electricity : Adequate
SOCIO-ECONOMIC STATUS
NUTRITIONAL STATUS
OTHER HABITS
VITAL SIGNS
INVESTIGATIONS
PHYSICAL EXAMINATION
1) General appearance
Consciousness :conscious
Look :worried and depressed
3) Posture
Body curves :no abnormal curves found
Movement :NAD
4) Height and weight :5’6”,56kgs
5) Skin conditions
Color :pallor
Texture :dryness
Temperature :cold and clammy
Lesion :NAD
6) Head and face
Scalp :clean
Face :pale, fatigued
7) Eyes
Eyeballs :sunken
Conjunctiva :pale
Sclera :normal
Pupils :normally react to light
Vision : normal
7) Ears
External ear :no abnormal discharges
Hearing :normal
8) Nose
External nares :no abnormal discharges
Nostrils :normal
9) Mouth and pharynx
Lips : pale but no redness, swelling etc.
Odor of the mouth : no foul smell
Teeth :clean
Gums :no inflammation and swelling
Tongue :coated
Throat and pharynx :NAD
10) Neck
Lymph nodes :non palpable
Thyroid gland :non palpable
Range of motion :normal ROM
11) Chest
Thorax :normal shape and symmetry of
expansion
Breath sounds :normal
Heart :normal heart sounds
12) Abdomen
Observation :no scar and skin rashes
Auscultation :bowel sounds heard
Palpation :no liver and spleen enlargements
Percussion :gas present but no fluid present
The stomach has four main regions: the cardia, fundus, body, and pylorus.
The cardia surrounds the superior opening of the stomach. The rounded portion
superior to and to the left of the cardia is the fundus. Inferior to the fundus is the
large central portion of the stomach, called the body. The region of the stomach
that connects to the duodenum is the pylorus; it has 2 parts, the pyloric antrum,
which connects to the body of the stomach, and the pyloric canal, which leads into
the duodenum. When the stomach is empty, the mucosa lies in large folds, called
rugae. The concave medial border of the stomach is called the lesser curvature,
and the convex lateral border is called the greater curvature.
The stomach wall is composed of the same four basic layers as the rest of
the rest of the GI tract. The surface of the mucosa is a layer of simple columnar
cells called mucous surface cells. The mucosa contains a lamina propria and
muscularis mucosae. Epithelial cells extend down into the lamina propria, where
they form columns of secretory cells called gastric glands that line many narrow
channels called gastric pits. The gastric glands contain 3 types of exocrine gland
cells namely: mucous neck cells, chief cells, parietal cells. Both mucous surface
cells and mucous neck cells secrete mucous. The chief cells (zymogenic) cells
secrete pepsinogen and gastric lipase. Parietal cells produce hydrochloric acid and
intrinsic factor (needed for the absorption of vitamin B12). The secretions of the
mucous, chief and parietal cells form gastric juice which totals 2000-3000ml per
day.
DESCRIPTION OF DISEASE
GASTRITIS
DEFINITION
It is the inflammation of the stomach mucosa.
TYPES
Chronic Gastritis appears in 3 different forms:-
1) Superficial gastritis: - Causes a reddened, edematous mucosa with
hemorrhage and small erosions.
2) Atrophic gastritis: - Occurs in all levels of the stomach, develops
frequently in association with gastric ulcer and gastric cancer and is
invariably present in pernicious anemia characterized by decreased number
of parietal and chief cells.
3) Hypertrophic gastritis: - Produces a dull and nodular mucosa with irregular
thickened or nodular rugae. Hemorrhage occur frequently
ETIOLOGY
IN BOOK IN PATIENT
1) DRUGS:-
a) Aspirin
b) Corticosteroids
c) NSAIDS
2) Diet 1) Spicy irritating food
a) Alcohol
b) Spicy irritating food
3) Micro organism
a) H pylori
b) Salmonella
c) Staphylococcus organisms
4) Environmental factors 2) smoking
a) Radiation
b) Smoking
5) Pathophysiologic conditions 3) Reflux of bile and pancreatic
a) Burns secretions
b) Large Hiatal Hernia
c) Physiologic stress
d) Reflux of bile and pancreatic
secretions
e) Renal Failure
f) Sepsis
g) Shock
6) Other Factors 4) Psycho logic stress
a) Endoscopic procedures
b) NG tube
c) Psycho logic stress
RISK FACTORS
IN BOOK IN PATIENT
1) Drug related gastritis
2) H pylori 1)Diet
3) Autoimmune
4) Diet
PATHOPHYSIOLOGY
Due to etiological factors
Tissue edema, disruption of capillary walls with loss of plasma into the gastric
lumen and possible hemorrhage
CLINICAL MANIFESTATIONS
IN BOOK IN PATIENT
1) Common Anorexia
Anorexia Feeling of fullness
Nausea and vomiting Dyspepsia
Abdominal cramping or diarrhea Belching
Epigastric pain epigastric pain
Fever Nausea, vomiting
Painless GI bleeding may occur Intolerance of hot and
and is more likely if the person spicy food
uses aspirin or NSAIDS regularly
2) Chronic
Symptoms maybe vague or absent
Anorexia
Feeling of fullness
Dyspepsia
Belching
Vague epigastric pain
Nausea, vomiting
Intolerance of spicy or fatty foods
DIAGNOSTIC EVALUATION
IN BOOK IN PATIENT
History taking History taking
Physical examination Physical examination
Complete blood count Complete blood count
Gastroscopic examination with
biopsy
Breath, urine, serum and stool
test
COMPLICATIONS:
pernicious anemia
gastrointestinal bleeding
peptic ulcer
gastric cancer
MANAGEMENT
MEDICATION CHART
S.n Medication Dose Rou Ti Action Side effects Nurses’
o (trade te me responsibility
name/generic
name)
01 Tab 500 Oral BD Antibiot INTEG: rash, Monitor I &O
Ampicillin mg ic urticaria chart
HEMA: Culture,
anemia, sensitivity before
increased drug therapy
bleeding Monitor bowel
time, bone movement before
marrow and after drug
depression, therapy
granulocytop Assess
enia respiratory status
GI: nausea, Monitor for any
vomiting, allergic reactions
diarrhea before initiation
GU: oliguria, of treatment
proteinuria, Teach patient or
hematuria, family about the
vaginitis, different aspects
moniliasis of drug therapy
CNS: The drug must
lethargy, be taken in equal
hallucinations intervals of time
, anxiety,
depression,
twitching,
coma,
convulsions
02 Inj. 50mg I/V BD H2 INTEG: rash, Administer with
Ranitidine receptor urticaria, meals for
antagon
ist fever prolonged effect
GI: Storage at room
constipation, temperature
abdominal Teach that
pain, impotence and
diarrhea, gynaecomastia
nausea, are reversible
vomiting, Teach that the
hepato patient is to
toxicity avoid driving,
GU: other hazardous
impotence, activities until
gynaecomasti stabilized on this
a medications
CNS: The drug is to be
hallucinations continued for
prescribed time
, depression, to be effective
headache,
sleeplessness,
confusion,
dizziness,
agitation
EENT:
blurred
vision,
increased
IOP
03 Tab 1 Oral OD Vit INTEG: Administer with
Biminox/Vita capsu suppl. itching, rash, fruit juice to
min B le
complex pain at the disguise taste
injection site With meals if
CV: CHF, possible for
peripheral better absorption
vascular By IM injection
thrombosis, for pernicious
pulmonary anemia
edema IV route not
GI: diarrhea recommended
CNS: but may be
admixed in TPN
flushing, solution
optic nerve
atrophy
META:
Hypokalemia
04 Tab Iron 1 Oral OD Iron INTEG: Administer in
ferrous tablet suppl. temporarily between meals
sulphate
discolored for best
tooth enamel absorption
and eyes May give with
GI: nausea, juice
constipation, Do not give with
epigastric antacids and
pain, black milk
and red tarry Teach patient
stools, that iron will
diarrhea, change color of
vomiting stool to black or
dark green
That iron
poisoning may
occur if increased
beyondrecomme
nded level
IN BOOK
A) Acute gastritis
1) Intervention involves removing the cause or treating the condition
symptomatically.
2) Vomiting frequently responds to phenothiazine group.
3) Pain responds to antacids or H2 antagonists such as aluminium –
magnesium combinations (Maalox) or ranitidine hydrochloride.
4) Foods and fluids are withheld until nausea and vomiting subside.
5) Avoid spicy food, caffeine and large heavy meals.
6) In the continued absence of nausea, vomiting and bloating, the client can
slowly return to a normal diet.
B) Chronic gastritis
1) Discomfort may lessen with a bland diet.
2) A non irritating diet consisting of 6 small feedings a day
3) Antacid after meals may help relieve symptoms.
4) Cholinergic
5) Sedatives
6) Avoidance of food that cause symptoms.
7) Administer Vitamin B12 if the client has pernicious anemia.
8) Antibiotic combinations are used for eradicating infection with H.pylori.
9) For patient with pernicious anemia, oral cobalamin, or injection of
cobalamin is needed.
10) Smoking is contraindicated in all forms of gastritis.
11) Incidence of gastric cancer is higher in the patient who has a history
of chronic gastritis, especially atrophic gastritis; close medical follow-up
should be stressed.
NURSING MANAGEMENT
Assessment –
NURSING DIAGNOSIS:
1) Pain related to increased gastric secretions and decreased mucosal
protection.
2) Altered nutritional balance, anorexia, less intake of food, related to
dyspepsia
3) Fluid and electrolyte balance, nausea and vomiting, related to disease
condition
4) Fear and anxiety related to hospitalization and disease prognosis
5) Knowledge deficit related to disease condition and drug regimen.
NURSING CARE PLAN
NURSING GOAL INTERVENTION RATIONALE EVALUATION
DIAGNOSIS
Pain related To Assess the Provide base
to increased relieve intensity, duration line data for The following
gastric pain & frequency of better interventions
secretions pain interventions helped to
and decreased Encourage patient To relieve relieve the
mucosal patient’s pain to
for bed rest pain
protection some extent.
Monitor the vital To assess the
signs of the patient nature of pain
Provide To relieve the
comfortable patient’s pain
position to the To avoid the
patient irritation of
Avoid hot, spicy the gastric
food mucosa
Administer H2 To decrease
antagonist as the gastric
prescribed by the secretions
physician
HEALTH EDUCATION
1) The patient is advised to modify his diet.
2) He is to avoid smoking and drinking alcohol
3) Promotion of rest
4) Reducing stress
5) The patient has to adhere to the pharmacotherapy.
6) The patient is advised to consult the physician if symptoms persist.
7) Close medical follow-up should be stressed.
8) Discussion about life long need for cobalamin is provided.
NURSES’ NOTES
Name of the patient : Mr. Dindayal Sharma Diagnosis:
Chr.Gastritis
Age/sex : 45yrs/M Dr. concerned: Dr. Jain
Ward/bed no : M.M.W II/17
Date : 5/10/2019