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  • Pathophysiology Activity 4 23

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    Angelokeizer Gavino
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    The document discusses the pathophysiology of meningitis caused by bacteria. It describes the process as: (1) bacteria colonizing and penetrating the nasopharyngeal mucosal membranes, (2) surviving in the blood and traveling through circulation, (3) invading the central nervous system, (4) multiplying in the subarachnoid space, and (5) increasing the permeability of the blood-brain barrier. This leads to cerebral edema, increased intracranial pressure, and impaired circulation.

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    The document discusses the pathophysiology of meningitis caused by bacteria. It describes the process as: (1) bacteria colonizing and penetrating the nasopharyngeal mucosal membranes, (2) surviving in the blood and traveling through circulation, (3) invading the central nervous system, (4) multiplying in the subarachnoid space, and (5) increasing the permeability of the blood-brain barrier. This leads to cerebral edema, increased intracranial pressure, and impaired circulation.

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    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
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    11 views4 pages

    Pathophysiology Activity 4 23

    Uploaded by

    Angelokeizer Gavino
    The document discusses the pathophysiology of meningitis caused by bacteria. It describes the process as: (1) bacteria colonizing and penetrating the nasopharyngeal mucosal membranes, (2) surviving in the blood and traveling through circulation, (3) invading the central nervous system, (4) multiplying in the subarachnoid space, and (5) increasing the permeability of the blood-brain barrier. This leads to cerebral edema, increased intracranial pressure, and impaired circulation.

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    © All Rights Reserved
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    of 4

    PATHOPHYSIOLOGY

    Activity 4 part 2 & 3

    Abes, Mark Venjohn A.

    Gavino, Keizer Angelo L.

    Gawat, Christian Ivan DG.

    Lamsen, Jhuela Marie M.

    Lising, Rosh Jezreel V.


    PATHOPHYSIOLOGY (Meningitis)

    Bacteria

    Colonization and penetration of nasopharyngeal


    mucosal membranes by bacteria

    survival of bacteria in blood


    and transportation via circulation

    invasion of central nervous system

    multiplication in subarachnoidal space

    increase permeability of blood-brain barrier

    transendothelial migration of granulocytes


    and monocytes

    release of cytokines and prostaglandins

    leakage of plasma proteins

    cerebral edema

    increased intracranial pressure

    impaired circulation
    NURSING CARE PLAN (Meningitis)
    DRUG STUDY (Meningitis)

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