It is where the cell attaches itself to the bacteria and engulfs it around while engulfing

inwards. Phagocytosis happens when a cell tries to target and destroy microbial
pathogens, virus or an infected cell.  It happens initially through by binding of opsonins
or specific molecules on the pathogen(Activation) . It then triggers activation, causes
receptor cluttering (Chemotaxis) and start of phagocytosis.  The cell membrane
eventually forms (Margination)  around the pathogen and engulfs it resulting to a
phagosome. The phagosome fuses with endosomes and lysosomes to mature and
acidify making it to a phagolysosome, which the degradation of contents that will occur.
The pathogen killing could occur on 2 ways; Oxygen Dependent Pathway(use of
superoxide radicals and hydrogen peroxide)  and the Oxygen-Independent
Pathway(use of lysosomal enzymes such as proteases, phospholipases and
lysozymes.  Bacteria and parasites can live and multiply inside phagocytic cells in
some cases. Mycobacterium tuberculosis is a famous example of a bacteria whose
survival appears to be dependent on the structure and makeup of its cell surface.
Toxoplasma gondii is a parasite that has the extraordinary ability to prevent lysosomes
from fusing with the phagocytic vacuole. As a result, the hydrolytic enzymes found in
lysosomes are unable to assist in the parasite's elimination. The mechanism(s) by which
bacteria like Legionella pneumophila, Brucella abortus, and Listeriamonocytogenes
survive in phagocytes is unknown.