UNKNOWN ETIOLOGY

PREDISPOSING FACTOR
PRECIPITATING FACTORS
 AGE
 FETAL SEX  VASCULAR DISEASE
 ETHNICITY  ALCOHOL CONSUMPTION
 HISTORY OF ABRUPTIO  SMOKING
PLACENTA  TRAUMA
 MULTIPLE GESTATION  CHORIOAMIONITIS
 MULTIGRAVIDA  SHORT UMBILICAL CORD
 ENVIRONMENT  COITUS
 SUBSTANCES ABUSE
 PRETERM PREMATURE
RUPTURE OF MEMBRANE
 OLIGOHYDRAMIOS

DAMAGE OR RUPTURE THE SPIRAL ARTERIES



SEPERATION OF DECUDA BASALIS AND UTERINE

WALL OF THE PLACENTA

DECIDUAL HEMATOMA FORMATION (BETWEEN

PLANCENTA ND UTERUS JOIN)

INCREASE HEMATOMA FORMATION AND PRESSURE

PLACENTAL TISSUE NECROSIS

PREMATURE SEPARATION OF THE PLACENTA

 DIAGNOSTIC TOOLS:
classified as:  BLOOD TEST
 ULTRASOUND
 BIOPHYSICAL
PROFILE
Concealed Revealed
Abruptio Abruptio


SIGNS AND SYMPTOMS

VAGINAL ABDOMINAL FETAL HEART RATE PAINFUL UTERINE

BACKACHE
BLEEDING PAIN ABNORMALITIES CONTRACTION

PROGNOSIS
MADICAL MANAGEMENT

 INTREAVENOUS THERAPY
 OXYGEN INHALATION
 FIBRINOGEN
if not treated if treated, good prognosis DETERMINATION
 MEDICATION:
o MAGNESIUM
SULFATE
BAD PROGNOSIS Fetus is delivered o HYDRALAZINE
preterm o CEFUROXIME
o FUROSEMIDE
o FERROUS
SUKFATE
MATERNAL FETAL o AMLODIPIN

SURGICAL MANAGEMENT

 CESAREAN SECTION
Decrease venous Hematoma INCREASE  HYSTERECTOMY
return formation CLOTTING
Decrease concealed typed
circulating blood abruption Fetal
volume hypoxia
CONSUMPTION INCREASE
OF PLATELETS CONSUMPTIO
hematoma
N OF
accumulates and Fetal
decrease cardiac CLOTTING
seeps into the Death
output FACTOR
myometrium

decrease blood
flow couvelaire uterus
THROMBUS
FORMATION AT
THE SITE OF
decrease supply INJURY
of oxygen

HEMORRAGIC
SYNDROME
Anoxia

DIC SYNDROME

SHOCK

Organ
dysfunction

Maternal death