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Angina Pectoris
Formation of Thrombus
Presence of Atherosclerosis
Myocardial oxygenation
Aerobic O2 demand
metabolism
HR accelerates
ATP
Lactic acid
Cardiac
Myocardium Vent. filling
Lactic contraction
becomes time
acidosis strength
ischemic
SV
Chest Depression of
Vent. perform
pain vent. function
deteriorates
Auto regulation SV
LV diastole
of blood flow or
pressure rise
compensation
Changes in
In diameter of systemic BP
Pulmonary
blood vessels congestion dyspnea
and arteries