Curriculum Vitae

Nama : Paulus Novian Harijanto, Sp.PD-KPTI, FINASIM

Tempat /Tgl Lahir : Kediri, 1 Nopember 1948
NIP : Pensiunan PNS ( ex 140087052)
Pangkat/Golongan : Pembina Utama Muda / IV/C
Pendidikan/ Jabatan sekarang
• Ketua SMF Penyakit Dalam RSU Bethesda GMIM Tomohon 2019- skrg
• Ketua Komite Medik RSU Bethesda Tomohon 2019-skrg
• Ketua Tim PPI/ PPRA/ Geriatri/ TB-program RSU Bethesda Tomohon
2011-2015
• Penanggung Jawab Unit Pelayanan VCT, RSU Bethesda Tomohon
• Dosen/ Penguji Luar biasa Ilmu Penyakit Dalam, Fak.Kedokteran Unsrat,
Manado sejak 2000
Curriculum Vitae
• Anggota Komisi Ahli Malaria Nasional, DepKes RI 2008 –
sekarang
• Ketua PETRI cabang Manado/ Sulawesi Utara, 2011-2015
• Anggota PENGURUS BESAR ( PB ) PETRI ( Perhimpunan
Peneliti Penyakit Tropik Indonesia ), 2015-2019
• Ketua PERSADIA cabang Tomohon
• Ketua PERDALIN, Sulawesi Utara
• Anggota Tim Kendali Mutu dan Kendali Biaya, BPJS
Kesehatan cabang Tondano 2016, 2017
 DIAGNOSIS & TATALAKSANA
Corona Virus 2019 (COVID-19)

Dr. Paul Harijanto, SpPD-KPTI

TATALAKSAN COVID-19
PENGEGAHAN KOMPLIKASI
PENGEGAHAN KOMPLIKASI
PENGEGAHAN KOMPLIKASI
PAKATUAN WO PAKALAWIREN
Sampai Baku Dapa !
Dr. Paul Harijanto, Sp.PD-KPTI
Div. Penyakit Tropik & Infeksi
SMF/ Bag. Penyakit Dalam
FK UNSRAT/ RSUP Manado
RSU Bethesda -Tomohon

Telp.:
0431-351024/046 ( RSU Bethesda)
0812-430-2869 ( HP)
0431-351187 (Res)
E-mail : paulharijanto@gmail.com
 Community-Acquired Pneumonia

• Infection of the pulmonary parenchyma acquired from

exposure in the community
• Classically divided into “typical” and “atypical” syndromes:
I. “Typical” CAP:
• presents with “typical” severe, acute infection
• infectious agent (usually S. pneumo or H. flu) is culturable/ identifiable
• responsive to cell-wall active antibiotics
II. “Atypical” CAP:
• presentation is usually sub-acute
• causative pathogens are difficult to culture/identify by standard methods
• not responsive to penicillins

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 Typical CAP presentation

History
• Previously healthy with sudden onset of fever and shortness of breath
Physical signs and symptoms
• fever
• tachycardia
• tachypnea
• productive cough with purulent sputum and possible hemoptysis
• pallor and cyanosis
• localized:
− dullness to percussion
− decreased breath sounds
− crackles , ronchi , egophony (“E” -to-”A” change)
Investigations
• CXR showing lobar consolidation
• CBC showing leukocytosis w/ left shift
• Sputum sample contains neutrophils, RBCs; Gram stain may be positive
depending on organism

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 Typical CAP presentation

History
• Previously healthy with sudden onset of fever and shortness of breath
Physical signs and symptoms
• fever
• tachycardia
• tachypnea
• productive cough with purulent sputum and possible hemoptysis
• pallor and cyanosis
• localized:
− dullness to percussion
− decreased breath sounds
− crackles, ronchi, egophony (“E-to-A” change)
Investigations
• CXR showing lobar consolidation
• CBC showing leukocytosis w/ left shift
• Sputum sample contains neutrophils, RBCs; Gram stain may be positive
depending on organism

58
 Typical CAP presentation

History
• Previously healthy with sudden onset of fever and shortness of breath
Physical signs and symptoms
• fever
• tachycardia
• tachypnea
• productive cough with purulent sputum and possible hemoptysis
• pallor and cyanosis
• localized:
− dullness to percussion
− decreased breath sounds
− crackles, ronchi, egophony (“E-to-A” change)
Investigations
• CXR showing lobar consolidation
• CBC showing leukocytosis w/ left shift
• Sputum sample contains neutrophils, RBCs; Gram stain may be positive
depending on organism

59
 Atypical CAP Presentation

• 32 YO healthy patient – one week of low grade fever,

sore throat, and intractable cough
• Minimal sputum production
• Able to continue to work
• No sick contacts, recent travel, or evidence of
altered immune system
• PE reveals a mildly ill-appearing patient with diffuse
wheezes on lung exam
• Primary care physician prescribes empiric antibiotics for
CAP with complete resolution
• “Walking pneumonia” syndrome

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 Complications of pneumonia

• inflammation leads to exudation of

fluid into pleural space
• can compromise lung function

• purulent exudate in pleural space

• necrosis/breakdown of visceral
pleura and/or spread of infection into
pleura
Pleural adhesions, lung fibrosis

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Gambaran Radiologis