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Definition
The ‘Unterkreuz syndrome’ is also known as pelvic crossed syndrome, lower crossed syndrome or distal crossed syndrome. The
lower crossed syndrome (LCS) is the result of muscle strength imbalances in the lower segment. These imbalances can occur
when muscles are constantly shortened or lengthened in relation to each other. The lower crossed syndrome is characterized by
specific patterns of muscle weakness and tightness that cross between the dorsal and the ventral sides of the body. In LCS
there is overactivity and hence tightness of hip flexors and lumbar extensors. Along with this there is underactivity and weakness
of the deep abdominal muscles on the ventral side and of the gluteus maximus and medius on the dorsal side.[1] The hamstrings
are frequently found to be tight in this syndrome as well. This imbalance results in an anterior tilt of the pelvis, increased flexion
of the hips, and a compensatory hyperlordosis in the lumbar spine.
Figure 1: lower
crossed syndrome [2]
There are two known subtypes, A and B, of lower crossed syndrome. The two types are similar and involve the same main
muscle imbalance characteristics. For type A the imbalance manifests mainly in the hip, while for type B the imbalance mainly
manifests in the lower back. The two subgroups can be distinguished based upon the altered postural alignment and also
changed regional myofascial activation patterns. An observation of the lower pole of the thorax and the anterolateral abdominal
wall shows whether there are problems with the activity level and balance between the diaphragm and transversus abdominis.
Mostly, there is an underactivity of the deep transversus associated with either increased or decreased superficial activity in the
obliques and rectus.[3]
Type A: The first subgroup is the posterior pelvic crossed syndrome. In this subgroup there is a domination of the axial extensor.
[3] Because the hip flexors are shortened, the pelvis is tilted anteriorly and the hip and knee are in slight flexion. Associated with
this is an anterior translation of the thorax because of an increased thoraco-lumbar extensor activity.This gives an expression for
the compensatory hyperlordosis of the lumbar spine and hyperkyphosis in the transition from thoracic to lumbar spine. This
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leads to a decrease in the quality of breathing and of the postural control. Above that the entire thorax will move up, due to the
minimal inferior stabilization created by the abdominals. The infra-sternal angle will go up to more than 90° and the postero-
inferior thorax will be hyper-stabilized through which it will cause a limited postero-lateral costo-vertebral movement.[3]
The more anterior and elevated position of the thorax will disturb the stabilization synergies of the Lower Pelvic Unit. The patient
will lift the thorax during inspiration which causes an upper chest breathing pattern. This means that the active exhalation will be
difficult, because the abdominal activation fails to bring the thorax down and back into the more expiratory caudal (or neutral)
position. The abdominal activation is also not sufficient to create the essential intra abdominal pressure. We will notice that the
expiratory phase is shortened. This problem arises when the coordination and co-activation between the transverses and the
diaphragm is missing. The patient is forced to use the Central Posterior Clinch behavior, which results in an overactivity of the
psoas.[3]
Type B: It is also called ‘The Anterior Pelvic Crossed syndrome. In this type the abdominal muscles are too weak and too short.
This is associated with a predominant tendency of the axial flexor activity.[3] The compensation is reflected by a minimal
hypolordosis of the lumbar spine, a hyperkyphosis of the thoracic spine and protraction of the head. The pelvis is postured more
anteriorly and the knees are in hyperextension.[4]
Examination
Examination for Lower crossed syndrome should follow the same patterns as for examining a patient for Low Back Pain.
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Some specific examination points for LCS include the following:
- Position of the pelvis. There is usually an increase of anterior tilt of the pelvis. This can be associated with increased lumbar
lordosis.
- Next the shape, size and tone of the tightened/inhibited muscles. (see Definition/Description)
Hip extension - is examined to analyze the hyperextension phase of the hip in gait. Use straight leg lifting.
Hip abduction –the patient with LCS, will combine the abduction with an lateral rotation and a flexion of the hip.
Trunk curl up – is tested to estimate the interplay between usually strong iliopsoas and the abdominal muscles.
PASSIVE EXAMINATION : [5]
-Hip flexors are tested with the patient in a modified Thomas position. This test can be influenced by the stretch of the
joint capsule and thus more specific test should be performed to confirm the tightness of the adductors. Confirmation of
tightness is clear when excessive soft tissue resistance and decreased range of motion are encountered on application
of pressure.
-Thigh adductors are tested with the patient lying supine at the edge of the plinth. Tight hamstrings may contribute to the
range limitation. If this situation occurs, bending the knee should increase the range of movement.
-The piriformis muscle is tested with the patient in a supine position. If the muscle is tight, the end feel is hard and may
be associated with pain deep in the buttocks.
-Quadratus lumborum is difficult to examine. In principle, passive trunk side bending is tested while the patient assumes
a side-lying position. The reference point is the level of inferior angle of the scapula. A simpler screening test entails
observation of the spinal curve during active lateral flexion of the trunk.
-Spinal erectors are also difficult to examine. As a screening test, forward bending in a short sit allows observation of the
gradual curvature of the spine.
-Triceps surae are tested by performing passive dorsiflexion of the foot. Normally, the therapist should be able to
achieve passive dorsiflexion to 90 degrees.
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Physical Therapy Management
The treatment of tightness is not in strengthening as it would further increase tightness and possibly result in more pronounced
weakness. A tight muscle should be stretched efficiently. Stretching of tight muscles results in improved strength of inhibited
antagonistic muscles, probably mediated via the Sherrington’s law of reciprocal innervation (level of evidence: 2C). [4] [6]
This may involve purely soft tissue approaches. Stretch the specific muscle for a duration of 15 seconds. A five week active
stretching program significantly increases active and passive ROM in the lower extremity. [7] (level of evidence: 1B)
The patient lies supine in the fetal position, their knees to their
chest with their arms wrapped around their knees. Exhale and
stretch. Hold this position for 15 seconds. Iliopsoas stretch in thomas position
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Retraining patients with Posterior Pelvic Crossed
Syndrome
to improve the active exhalation, which will bring the thorax caudally on a stable pelvis. It is important to
It is important
assist the patient, while maintaining the neutral position.. While executing this exercise, it is crucial that the patient
breaths down and not up. The patient has to be able to create sufficient intra-abdominal pressure , while maintaining a
regular breathing pattern.
The patient has to lie down on his back, in supine supported hip flexion to eliminate gravity. The therapist asks the
patient to ‘breathe down to the lower placed hand’. It is then important to encourage an active and long exhalation. This
gives the patient the sense of the required action. When the correct pattern is mastered it can be progressed into
unsupported hip flexion. It is important to push the ribs wide and back, without lifting the thorax. To realize this, the client
is asked to push out sideways into the hands of the therapist.[[11], [3]](grades of recommendation: C)
References
1. ↑ Key J. The Pelvic Crossed Syndromes: A reflection of imbalanced function in the myofascial envelope; a further
exploration of Janda's work. Journal of bodywork and movement therapies. 2010 July;14:299-301
2. ↑ 2.0 2.1 2.2 Janda V. Muscles and motor control in low back pain: Assessment and management. In: Twomey Lt. Physical
therapy of the low back. New York, Edinburgh, London: Churchill Livingston, 1987;253-87
3. ↑ 3.0 3.1 3.2 3.3 3.4 3.5 3.6 Ishida, H., Hirose, R., Watanabe, S., 2012. Comparison of changes in the contraction of the
lateral abdominal muscles between the abdominal drawing-in maneuver and breathe held at the maximum expiratory
level. Man. Ther. 17 (5), 427- 431. Level of Evidence: 2C
4. ↑ Roberts, J., & Wilson, K. (1999). Effect of stretching duration on active and passive range of motion in the lower
extremity. Br J Sports Med , 259-263.fckLRLevel of evidence: 1B
5. ↑ Roberts, J., Wilson, K. (1999). Effect of stretching duration on active and passive range of motion in the lower
extremity. Br J Sports Med , 259-263.fckLRLevel of evidence: 1B
6. ↑ Liebenson, C. (2007). Evaluation of Muscular Imbalance. In Rehabilitation of the Spine: A Practitioner's Manual (p.
209). Philadelphia: Lippincott Williams & Wilkins.fckLRLevel of evidence: 2C
7. ↑ Liebenson, C. (2007). Evaluation of Muscular Imbalance. In Rehabilitation of the Spine: A Practitioner's Manual (p.
209). Philadelphia: Lippincott Williams & Wilkins.fckLRLevel of evidence: 2C
8. ↑ Simons D.G., Understanding Effective Treatments of Myofascial Trigger Points: Journal of Bodywork and Movement
Therapies, 2002, Volume 6, issue 2. fckLRLevel of evidence: 1A
9. ↑ Key J. (2013), ‘The core’: Understanding it, and retraining its dysfunction, Journal of Bodywork; Movement Therapies
17, p. 541- 559fckLRLevel of evidence: 1A
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