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Alterations with Infectious, Inflammatory and Immunologic Responses
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Adalimumab
Abatacept
Non Steroidal Antiinflammatory Drugs (NSAID), Methotrexate,
Corticosteroids, and Biologic agents such as Etanercept, Adal-
imumab, and Abatacept are used to treat what
Juvenile Idiopathic Arthritis
Juvenile Idiopathic Arthritis
Allergic Rhinitis
Atopic Dermatitis
Exacerbation
PT directed toward specific joints, focusing on strengthening mus-
cles, mobilizing restricted joint motion, and preventing or correct-
ing deformities. OT for generalized mobility and performance of
activities of daily living; these are therapeutic mgt for what
Juvenile Idiopathic Arthritis
Juvenile Idiopathic Arthritis
Allergic Rhinitis
Atopic Dermatitis
Exacerbation
Relieve pain, Facilitate adherence, Encourage heat and exercise,
and Support child and family are nursing mgt for what
2/8
Alterations with Infectious, Inflammatory and Immunologic Responses
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Allergic Rhinitis
Atopic Dermatitis
Exacerbation
A type of pruritic eczema that usually begins during infancy and
is associated with an allergic contact dermatitis with a hereditary
tendency
Atopic eczema
Atopic eczema
Childhood eczema
Preadolescent eczema
Adolescent eczema
Atopic eczema is aka
Infantile eczema
Infantile eczema
Childhood eczema
Preadolescent eczema
Adolescent eczema
Usually begins at 2 to 6 months of age, Undergoes spontaneous
remission by 3 years of age, and generalized, especially cheeks,
scalp, trunk, and extensor surfaces of extremities
Infantile eczema
Infantile eczema
Childhood eczema
Preadolescent eczema
Adolescent eczema
Erythema, Vesicles/ Papules, Weeping/ Oozing/ Crusting/ Scal-
ing, and Often symmetric are lesion appearance of what type of
eczema
Infantile eczema
Infantile eczema
Childhood eczema
Preadolescent eczema
Adolescent eczema
Occurs at 2 3 years of age, 90% of children have manifestations by
age 5 years, and occurs in flexural areas (antecubital and popliteal
fossae, neck), wrists, ankles, and feet
Childhood eczema
Infantile eczema
Childhood eczema
Preadolescent eczema
Adolescent eczema
Symmetric involvement, Clusters of small erythematous or flesh
colored papules or minimally scaling patches, Dry and may be
hyperpigmented, Lichenification, and Keratosis pilaris (which is
common) are lesion appearance of what type of eczema
Childhood eczema
Atopic eczema
Infantile eczema
Childhood eczema
Preadolescent and Adolescent eczema
thickened skin with accentuation of creases
Seborrheic keratosis
Lichenification
Keratosis pilaris
Lichenification
Acanthosis
follicular hyperkeratosis
Keratosis pilaris
Seborrheic keratosis
3/8
Alterations with Infectious, Inflammatory and Immunologic Responses
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Keratosis pilaris
Lichenification
Acanthosis
Begins at about 12 years of age, May continue into the early adult
years or indefinitely, and affects face, sides of neck, hands, feet,
face, and antecubital and popliteal fossae (to a lesser extent)
Preadolescent and Adolescent eczema
Atopic eczema
Infantile eczema
Childhood eczema
Preadolescent and Adolescent eczema
Dry, thick lesions (lichenified plaques) are common, Intense itch-
ing in confluent papules, and dry and rough unaffected skin are
lesion appearance of what type of eczema
Preadolescent and Adolescent eczema
Atopic eczema
Infantile eczema
Childhood eczema
Preadolescent and Adolescent eczema
hydrate the skin, relieve pruritus, reduce flare ups or inflammation,
prevent and control secondary infection, avoid exposure to skin
irritants or allergens, and avoid overheating; these are therapeutic
management for what
Atopic Dermatitis
Juvenile Idiopathic Arthritis
Allergic Rhinitis
Atopic Dermatitis
Asthma
Antihistamines, topical immunomodulators, topical steroids, and
mild sedatives are pharmacologic mgt for what
4/8
Alterations with Infectious, Inflammatory and Immunologic Responses
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Allergic Rhinitis
Atopic Dermatitis
Asthma
Age, Hereditary, Gender, Mother <20 y/o, Smoking, Ethnicity,
Previous life threatening attacks, Lack of access in medical care,
and Psychological & physiologic problem are risk factors of what
Asthma
Juvenile Idiopathic Arthritis
Allergic Rhinitis
Atopic Dermatitis
Asthma
Irritants > Release of inflammatory mediator > Migration & activa-
tion of inflammatory cell > Alteration in epithelial integrity & auto-
nomic neutral control of airway > Smooth muscle responsiveness,
wheezing, dyspnea > Obstruction
Asthma
Juvenile Idiopathic Arthritis
Allergic Rhinitis
Atopic Dermatitis
Asthma
wheezing, breathlessness, dyspnea, chest tightness, night & early
morning cough, and activity induced cough/wheeze are clinical
manifestations of what
Asthma
Juvenile Idiopathic Arthritis
Allergic Rhinitis
Atopic Dermatitis
Asthma
Symptoms occurs <2x/week, Nighttime symptoms occurs
<2x/mo, and Meds are for acute exacerbation
5/8
Alterations with Infectious, Inflammatory and Immunologic Responses
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Juvenile Idiopathic Arthritis
Allergic Rhinitis
Asthma
Atopic Dermatitis
Asthma
most accurate test for asthma
ABG
Pulmonary function tests
CBC
Spirometry
Pulmonary function tests
Highly suggestive of asthma when >15% increase after inhaled
short acting B2 antagonist and when <15% decrease PEFR after
exercise
PEFR
ABG
CBC
PEFR
Pulmonary function tests
Maintain normal activity level, Normal pulmonary function, Pre-
vent symptoms, and Drug therapy are mgt goals for what
6/8
Alterations with Infectious, Inflammatory and Immunologic Responses
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B2 agonist B2 agonist
Methylxanthines
Cholinergic antagonist
Corticosteroid
Theophylline, aminophylline, and Oxtriphylline are examples of
what asthma medication
Methylxanthines B2 agonist
Methylxanthines
Cholinergic antagonist
Corticosteroid
Ipratropium (Atrovent) is an example of what asthma medication
B2 agonist
Cholinergic antagonist
Methylxanthines
Cholinergic antagonist
Corticosteroid
Prednisolone and Prednisone are examples of what asthma med-
ication
Oral Corticosteroid
Mast Cell Stabilizer
Inhaler Corticosteroid
Mast Cell Stabilizer
Monoclonal Antibodies
Helps prevent atopic asthma attacks by preventing membranes
from opening when an allergen binds to IgE
Mast Cell Stabilizer
Oral Corticosteroid
7/8
Alterations with Infectious, Inflammatory and Immunologic Responses
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Inhaler Corticosteroid
Mast Cell Stabilizer
Monoclonal Antibodies
Omalizumab is an example of what asthma medication
Oral Corticosteroid
Monoclonal Antibodies
Inhaler Corticosteroid
Mast Cell Stabilizer
Monoclonal Antibodies
Binds to IgE receptor sites on mast cells & basophils preventing
the release of chemical mediators for inflammation
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