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8 
Chapter |
The kinesiopathological model and mechanical
low back pain
Linda R. Van Dillen*, Shirley A. Sahrmann† and Barbara J. Norton‡
*Program in Physical Therapy and Department of Orthopaedic Surgery, †Program in Physical Therapy, Department of Cell Biology and Physiology,
Department of Neurology and Neurophysiology and ‡Program in Physical Therapy and Department of Neurology, Washington University School
of Medicine at Washington University Medical Center, St Louis, Missouri, USA

review the findings from studies that have been conducted


CHAPTER CONTENTS
to test assumptions of the model in patients with LBP.
The kinesiopathological model 89
The kinesiopathological model and low
back pain 90 THE KINESIOPATHOLOGICAL MODEL
Low back pain subgroups 90
Studies of select aspects of the The kinesiopathological model, hereinafter referred to as
kinesiopathological model 90 the Model, describes a process that is proposed to contrib­
Movement and alignment patterns 91 ute to the development, as well as the course, of many
Repetition of movements and musculoskeletal pain conditions, including mechanical
alignments 92 LBP. The process begins with repetition of a daily activity
Contributing factors 94 associated with specific movements or specific alignments.
The repetitions are proposed to lead to adaptations in the
Summary 96
musculoskeletal and neural systems, for example, changes
References 97 in muscle strength, flexibility, stiffness, timing and level of
muscle activity, to name just a few. Although such adapta­
Characteristic patterns of movements and alignments can tions often may be beneficial, continual repetition of
be recognized in many musculoskeletal pain conditions, specific movements and alignments may be increasingly
including mechanical low back pain (LBP). In some detrimental because the associated adaptations contribute
instances the patterns displayed by patients can be linked to imbalances about the joint(s). For example, repeated
to a specific pathology, disease or injury. There are, trunk rotation to the right to reach for the phone (a trunk
however, several musculoskeletal pain conditions in which rotation activity) could cause the muscles that contribute
a specific causal link cannot be identified. A key question to right trunk rotation to become shorter and stronger
in all instances is whether the movements and alignments while the muscles that contribute to left trunk rotation to
displayed by the person with musculoskeletal pain are a become longer and weaker. With continued repetition, the
consequence of the condition or contribute to the devel­ adaptations would result in alterations in the joint motions
opment of the condition. One potential answer to this (accessory and physiological) and in joint alignments. The
question may be found in the kinesiopathological model alterations due to repeated performance of trunk rotation
(Sahrmann 2002). A basic premise of the kinesiopatho­ to the right, in turn, lead to alterations in the motions and
logical model is that continual repetition of specific move­ alignments associated with many other daily activities, i.e.
ments and alignments which are requisite to performing patterns of altered movements and alignments. The repeti­
daily activities can lead to the development of musculo­ tion of altered patterns of movements and alignments are
skeletal pain conditions (Fig. 8.1). The purposes of this proposed to result in localized regions of tissue stress,
chapter are to: (i) describe the kinesiopathological model; symptoms and, eventually, tissue injury (Adams 2004).
(ii) describe the application of the model to LBP; and (iii) According to the Model, the deleterious process can be

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