Professional Documents
Culture Documents
Influence of Personal and Family Variables On Eati
Influence of Personal and Family Variables On Eati
See discussions, stats, and author profiles for this publication at: https://www.researchgate.net/publication/357312798
CITATIONS READS
0 76
3 authors:
SEE PROFILE
Some of the authors of this publication are also working on these related projects:
Responses to social work. Faced with social emergencies and complex social problems in Mexico and Spain View project
All content following this page was uploaded by Paula Frieiro on 18 January 2022.
Paula Frieiro (1) [ORCID: https://orcid.org/0000-0001-5888-6674], Rubén González-Rodríguez (1) [ORCID: https://orcid.org/
0000-0003-1806-1103] and José Domínguez Alonso (1) [ORCID: https://orcid.org/0000-0002-1844-000X]
(1) University of Vigo. Faculty of Education and Social Work. Ourense. Spain.
SUMMARY ABSTRACT
Background: Eating disorders (ED) normally Influence of personal and
begin during puberty and adolescence, when family variables on eating disorders
attention should be paid to the factors that influence
Background: Eating disorders (EDs) usually
the development of the disease. The objective of
begin during puberty and adolescence, at a time
this work was to evaluate attitudes towards food
when attention should be paid to the factors that
and the risk of suffering from eating disorders, taking
influence the development of the disease. The aim
into account personal and family variables in a
of this study was to assess attitudes towards eating
population of secondary school students.
and the risk of developing EDs, taking into account
personal and family variables in a population of
Methods: A total of 790 Compulsory Secondary secondary school students.
Education (ESO) students enrolled in the 2019/2020
academic year in institutes in the autonomous Methods: A total of 790 Compulsory Secondary
School students enrolled in the 2019/2020 academic
community of Galicia participated, of which 410 were
year in secondary schools in the Autonomous
men and 380 women (M=13.84; SD =1.37).
Community of Galicia participated, of whom 410
In this descriptive-cross-sectional study, EDs were
were male and 380 female (M=13.84; SD=1.37). In
assessed using Garner's Eating Attitudes Test
this descriptive-cross-sectional study, eating
(EAT-26), in the version adapted to Spanish
disorders were assessed using Garner's Eating
subjects. The statistical treatment of the data was
developed through a multivariate analysis of Attitudes Test (EAT-26), adapted to Spanish
subjects. The statistical treatment of the data was
variance (MANOVA), which was reflected through
carried out by means of a multivariate analysis of
frequency analysis and contingency tables.
variance (MANOVA), which took the form of
Results: Considering the personal variables
analyzed, no statistically significant differences frequency analysis and contingency tables.
Results: With regard to the personal variables
were found in eating disorders in the gender of the
analyzed, no significant differences in eating
adolescents (p>0.05), but there were differences in
disorders were found across adolescents' gender
age (p<0.001). school year (p<0.001) and use of
(p>0.05), but there were significant differences in
social networks (p<0.05). Similarly, the data did
age (p<0.001), school year (p<0.001) and use of
present statistically significant differences in eating
social networks (p<0.05). Similarly, the data did
disorders according to the families' level of education
show significant differences in eating disorders
(p<0.01) and their family relationship (p<0.001).
according to the level of studies of the families
Conclusions: The results obtained in the (p<0.01) and their family relationship (p<0.001).
Conclusions: The results obtained in this search
research verify an influence of personal and family
confirm an influence of personal and family variables
variables on the attitudes associated with eating in attitudes associated with EDs. Further investigation
disorders. Continuing to delve deeper into these
of these variables may facilitate better intervention,
variables can facilitate better intervention, as well as
as well as improve the design of preventive
improve the design of preventive strategies.
Keywords: Adolescents, Disorders of the strategies.
Key words: Teenagers, Eating disorders, Family,
Eating behavior, Family, Social networks. Social networks.
Correspondence:
Rubén González Rodríguez
University of Vigo
Faculty of Education and Social Work Suggested citation: Frieiro P, González-Rodríguez R, Domínguez Alonso
Campus das Lagoas, s/n J. Influence of personal and family variables on eating disorders. Rev Esp
32004 Ourense, Spain Public Health. 2021; 95: December 22 e202112200.
rubgonzalez@uvigo.es
Machine Translated by Google
Paula Frieiro et al
Prevalence of the most common EDs are, first of all, significantly influenced attitudes and risk of suffering
anorexia (<1–4%), followed by binge eating disorder from eating disorders.
(<1–4%) and bulimia nervosa (<1–2%)(25). In
another systematic review analysis between 2000 ii) That the family variables level of education and
and 2018, a clear increase in the prevalence of EDs degree of family relationship were determinants for
in recent years was revealed. The weighted the development of attitudes and risk of suffering
averages of prevalence in the period 2000-2006 from eating disorders.
were 3.5%, rising to 7.8% in the period 2013-2018,
which highlighted a challenge for Public Health and SUBJECTS AND METHODS
for healthcare providers. care resources(26).
Participants and procedure. To contrast the
formulated hypotheses, an investigation was carried
Likewise, the morbidity and severity of anorexia out with Compulsory Secondary Education (ESO)
and bulimia are characterized by medical students, enrolled during the 2019/2020 school
complications that cause a large part of the mortality year in the autonomous community of Galicia
rate and by a decrease of between ten to twenty (northwestern Spain). In relation to obtaining the
years in life expectancy, also highlighting the high sample, the students enrolled in Secondary School
rate and risk of suicide(27,28). Specifically, the during the 2018/2019 school year in Galicia were
mortality rate is almost twice as high for people with taken into account, which corresponded to 92,285
EDs than for the general population, with mortality students. After consulting the public database of
in AN being higher than in other EDs(8,28,29). educational centers of the Department of Culture,
Education and University, they were contacted by
email. The centers that expressed their willingness
Due to the aforementioned, the use of agile to participate received a telephone communication
instruments that identify possible symptoms and to further specify the research and thus agree on a
risk behaviors regarding EDs contribute to early face-to-face visit to the center. An intentional
diagnosis and the development of prevention selection was made from all of them, determining
programs that, without a doubt, report improvements that the four provinces were represented and
in psychosocial intervention(18). guaranteeing that 50% of the centers were located
Without effective early diagnosis and treatment, in urban and rural areas, respectively.
the course of these disorders is prolonged through/
leading to physical, psychological, and social
morbidity and with high mortality(2).
Data collection took place between the oral). However, later studies, such as that of
months of September and March of the Constaín et al(30) have postulated the
2019/2020 school year. For this, the existence of 4 factors (bulimia nervosa; diet;
collaboration of the participating educational preoccupation with food; oral control).
centers was requested, to which the letter of
presentation of the research was delivered. In This questionnaire is a particularly useful
it, the relevance of the study, the objectives to screening tool for assessing “eating disorder
be achieved were explained to them and their risk” in high school, college, and other special
collaboration was requested for the risk samples, such as athletes(31). The EAT-26
authorization of informed consent by the legal is not designed to diagnose or assess an eating
guardians of the minor students, informing disorder and should not be used in place of a
them of the procedures that would be carried out. carried
professional
out throughout
diagnosisthe
or entire
consultation.
process.
Items
The application of the questionnaire was are presented on a 6-point forced-choice Likert
carried out during school hours at the scale ranging from 1 (“Never”) to 6 (“always”).
educational center, with one of the researchers The total score is obtained by operationalizing
always present. In the classroom, the voluntary the scores as follows: scores 1 to 3 are recoded
nature of the questions was emphasized as 0, score 4 is coded as 1, 5 is coded as 2,
again, with the option of not answering said questionnaire.
and 6 is coded as 3. The only exception is item
All personal data included in the study were 25 whose answers are scored inverted: 1 as 3,
processed and managed in accordance with the 2 as 2, 3 as 1 and from 4 to 6 as 0. The range
Organic Law 3/2018, of December 5, on the of EAT 26 varies from 0 to 78, with the number
protection of personal data and guarantee of greater than or equal to 20 being established
digital rights. The study procedure was by Castro et al(32) as a cut-off score from
developed, at all times, in accordance with the which there is a risk of suffering from an eating
Declaration of Helsinki. disorder.
Table 1
Means and Standard Deviations of the subscales of the EAT-26 questionnaire (DI, PC,
BU, CO) taking into account gender, age, academic year and social networks.
Subscales GAVE PC BU CO
Gender Male 15.99 (5.76) 11.03 (4.87) 9.81 (3.66) 15.82 (5.54)
12-13 years 17.05 (6.32) 11.85 (5.25) 10.01 (3.94) 17.05 (5.32)
14-15 years 15.83 (6.37) 10.47 (4.58) 10.24 (4.21) 15.72 (6.08)
Age
16-17-18 years 15.06 (4.83) 10.16 (4.27) 9.29 (3.31) 14.36 (5.23)
1st ESO 16.93 (6.20) 11.92 (4.93) 10.21 (3.98) 17.29 (5.34)
2nd ESO 17.11 (6.40) 11.62 (5.42) 10.02 (4.31) 16.69 (5.81)
3rd ESO 16.53 (6.73) 10.75 (4.79) 10.59 (4.47) 15.93 (6.01)
Academic course
4th ESO 14.41 (5.10) 9.72 (4.13) 9.19 (2.92) 14.44 (5.36)
With networks 16.37 (6.26) 11.01 (4.93) 10.04 (4.04) 16.06 (5.71)
Social networks Without networks 15.45 (5.83) 11.87 (4.72) 9.71 (3.42) 17.55 (5.64)
Table 2
Differences in age, school year and social networks in
diet, preoccupation with food, bulimia and oral control.
Independent 2
EAT-26 subscales F p ÿp Power
variables
power=0.615]. Thus, individuals without social groups (Academic level: primary, secondary,
networks (M=17.55; SD=5.64) presented greater university studies; Family relationship: fair, good,
oral control than those who used social networks very good) in the scores of the different subscales
(M=16.06; SD=5.71). that make up the Eating Attitudes Test (EAT) [Diet
(DI), Concern about food (PC), Bulimia (BU), and Oral
Taking into account the family environment, the Control (CO)] are found in Table 3 .
means and standard deviations obtained by the different
Table 3
Means and Standard Deviations of the subscales of the EAT-26
questionnaire (DI, PC, BU, CO) taking into account the studies of the family and its family relationshi
Subscales GAVE PC BU CO
In general, the degree of family relationships was From a univariate perspective (Table 4), the existence
perceived as “very good” in 55.6% of the cases, “good” of significant differences was shown according to the
by 33.3% of the students and defined as “regular” in a studies and the family relationship with the different
percentage of the students. 11.1%. At the multivariate subscales of the EAT-26 questionnaire (DI, PC, BU, CO).
level (MANOVA), the results showed that there were Considering the level of education of the family, the
significant differences between the risk of eating disorders differences were not significant for concern about food
and the family's level of education [Wilks' Lambda=0.97, [F(2, 787)=1.28, p>0.05, ÿ2
F(4, 785)=3.29, p <0.01, ÿ2 =0.003,
p
power=0.280] and bulimia [F(2, 787)=2.74, p>0.05, ÿ2
p
=0.014, power=0.944] and p
=0.007, power=0.543]; without em
the family relationship [Wilks Lambda=0.96, F(4, However, there were statistically significant differences
785)=4.49, p<0.001, ÿ2 p
=0.022, power=0.997]. in the diet [F(2, 787)=6.83,
p<0.01, ÿ2 p =0.017, power=0.921] and with to the family relationship, the differences were not
oral troll [F(2, 787)=2.98, p<0.05, ÿ2 p
=0.009, significant in concern about food [F(2, 787)=1.94,
power=0.670]. Thus, individuals from families with p>0.05, ÿ2 p
=0.005,
primary (M=17.60; SD=6.03) and secondary education power=0.404]; but they did show statistically significant
(M=16.75; SD=6.05) presented greater concern about differences for diet [F(2, 787)=8.70, p<0.001, ÿ2
diet than those from families with university studies p =0.022,po
(M=15.36; SD=6.38). Similarly, individuals from tension=0.969], bulimia [F(2, 787)=14.29, p<0.001, ÿ2
families with primary education (M=17.37; SD=6.41) p =0.035, power=0.999] and the with
showed greater oral control than those from families oral troll [F(2, 787)=3.99, p<0.05, ÿ2 p
=0.010,
with secondary education (M=15.82; SD=5.41). ). In power=0.715]. Regarding diet, families with a regular
reference relationship (M=18.77; SD=7.88) had greater concern
about
Table 4
Differences in family academic level and type of family relationship in diet, preoccupation with
food, bulimia, and oral control.
Independent 2
EAT-26 subscales F p Power
variables ÿp
diet than those with good relationships (M=16.37; statistically significant with respect to the DI,
SD=5.85) and very good relationships (M=15.77; SD=5.96).PC
and CO subscales. Thus, younger
Similarly, individuals from families with a individuals (12-13 years old) have greater
regular relationship (M=12.09; SD=5.32) concern about diet, food and oral control
presented a greater risk of bulimia than those than older or intermediate-aged individuals.
from good relationships (M=9.98; SD=3.55) It is evident that it is at the beginning of
and very good (M=9.63; SD=3.82). Finally, adolescence when greater concerns about
families with a regular relationship (M=17.69; eating disorders appear. In this sense, in
SD=7.16) showed greater oral control than different studies it has been observed that
those with good relationships (M=15.71; SD=5.37). eating disorders occur earlier than in other
mental health pathologies, with puberty being
DISCUSSION a critical moment for this problem(4,16) . The
age differences are not statistically significant
As previously stated, the present study for the bulimia dimension, agreeing with
sought to identify whether certain personal studies that identify the appearance of
and family variables of ESO students bulimic behaviors before adolescence as
significantly influence risk attitudes and very rare(33).
behaviors for suffering from EDs. Regarding
the personal variables, included in the first
hypothesis, the results show that it is partially Linked to age, it is observed that in the
confirmed, given that there are statistically school year variable there are also significant
significant differences between risk attitudes results in all the subscales (DI, PC, BU and
towards EDs and the variables of age, school CO). The youngest students, who are in 1st
year and social networks. , but not with and 2nd year of ESO, show greater concern
respect to gender. about food and oral control than 4th year
students. Similarly, students in the first three
years are more concerned about diet than
Consequently, taking gender into account, students in the last year. Finally, regarding
although in this study no statistically the bulimia dimension, 3rd grade students
significant differences were obtained in eating present higher values than 4th grade
disorders, slightly higher results were students. Thus, the first ESO courses are
achieved for the female gender. Different decisive for prevention-intervention in eating
studies agree that young women constitute disorders.
the majority of people who present anus rexia
and bulimia nervosa(8,26). It must be
considered that, historically, the study of EDs Regarding the use of social networks,
has focused on women, so the nosology of among the students who had networks,
eating disorders has evolved from studies attitudes of concern were seen, without being
that lack high representativeness with statistically significant, in the DI, PC and BU
respect to research that consider the subscales. However, students without social
masculine gender. networks show statistically significant
differences regarding oral control compared
to those who did have social networks. In this
The results, regarding the age variable, regard, we must keep in mind that the use of
indicate that this variable represents differences social networks can generate concern for
body image, while contributing to the risk of of the triggers of eating disorders (16,22,23,24).
developing and maintaining eating pathologies(20,21).
4. Klump KL. Puberty as a critical risk period for eating 15. Limburg K, Watson HJ, Hagger MS, Egan SJ. The
disorders: A review of human and animal studies. Horm Relationship Between Perfectionism and Psychopathology: A
Behav. 2013;64:399-410. Meta-Analysis. J Clin Psychol. 2017;73:1301-1326.
5. Monacis L, De PaloV, Griffiths MD, Sinatra M. 16. Hay P, Girosi F, Mond J. Prevalence and sociodemographic
Exploring individual differences in online addictions: The role correlates of DSM-5 eating disorders in the Australian
of identity and attachment. Int J Ment Health Addict. population. J Eat Disord. 2015;3:1-7.
2017;15:453-468.
17. Brechan I, Kvalem IL. Relationship between body
6. WHO. Press release Adolescent mental health. dissatisfaction and disordered eating: Mediating role of self-
Available at https://www.who.int/es/news-room/fact-sheets/ esteem and depression. Eat Behav.2015;17:49-58.
detail/adolescent-mental-health. [Cited September 2020].
18. Rieger E, Van Buren DJ, Bishop M, Tanofsky-Kraff M,
7. Treasure J, Duarte TA, Schmidt U. Eating disorders. Welch R, Wilfley DE. An eating disorder-specific model of
Lancet. 2020; 395:899-911. interpersonal psychotherapy (IPT-ED): Causal pathways and
treatment implications. Clin Psychol Rev. 2010;
8. Schmidt U, Adan R, Böhm I, Campbell IC, Dingemans A, 30:400-410.
Ehrlich S et al. Eating disorders: The big issue. Lancet.
2016;3:313-315. 19. Paniagua H, García S. Warning signs of eating,
depressive, learning and violent behavior disorders among
9. Hay PJ, Mond J, Buttner P, Darby A. Eating disorder be adolescents in Cantabria. Rev Esp Public Health.
haviors are increasing: Findings from two sequential 2003;77:411-422.
community surveys in South Australia. PLoS One. 2008;3:1-5.
20. Hogue JV, Mills JS. The effects of active social media
10. Qian J, Hu Q, Wan Y, Li, T, Wu M, Ren Z et al. Prevalence engagement with peers on body image in young women.
of eating disorders in the general population: a systematic Body image. 2019;28:1-5.
review. Shanghai Arch Psychiatry. 2013;25:212-223.
21. Mabe AG, Forney KJ, Keel PK. (2014). Do you “like” my
11. Smink FRE, Van Hoeken D, Hoek HW. Epidemiology of photo? Facebook use maintains eating disorder risk. Int J Eat
eating disorders: Incidence, prevalence and mortality rates. Disord. 2014;47:516-523.
Curr Psychiatry Rep 2012;14:406-414.
22. Abbott-Chapman J, Denholm C, Wyld C. Gender
12. Cancela JM, Ayán C. Prevalence and relationship differences in adolescent risk taking: are they diminis hing?
between the level of physical activity and abnormal eating An Australian intergenerational study. Youth Soc.
attitudes in Spanish university students of health sciences 2008;40:131-154.
and education. Rev Esp Public Health. 2011;85:499-505.
23. Amianto F, Ercole R, Marzola E, Abbate Daga G, Fassino
13. Fairburn CG, Harrison PJ. Eating disorders. Lancet. S. Parents' personality clusters and eating disorder red
2003 Feb;361:407-416. daughters' personality and psychopathology. Psychiatry Res.
2015;230:19-27.
14. Herle M, Abdulkadir M, Hübel C, Ferreira DS, Bryant
Waugh R, Loos R et al. The genomics of childhood eating 24. Cerniglia L, Cimino S, Tafà M, Marzilli E, Ballarotto G,
behaviors. Nat Hum Behav. 2021In:1-6. Bracaglia F. Family profiles in eating disorders: Family
functioning and psychopathology. Psychol Res Behav Manag. 29. Garner DM, Bohr Y, Garfinkel PE. The Eating Attitudes Test:
2017;10: 305-312. Psychometric Features and Clinical Correlates.
Psychol Med.1982;12:871-878.
25. Keski-Rahkonen A, Mustelin L. Epidemiology of eating disorders
in Europe: Prevalence, incidence, comorbidity, course, consequences, 30. Constaín GA, Ramírez CR, Rodríguez-Gázquez MA, Gómez M Á,
and risk factors. Curr Opinion Psychiatry. 2016;29:340-345. Múnera CM, Acosta CA. Validity and diagnostic utility of the EAT-26
scale for evaluating the risk of eating disorders in the female population
of Medellín, Colombia. Aten Primary. 2014;46:283-289.
26. Galmiche M, Déchelotte P, Lambert G, Tavolacci MP. Prevalence
of Anorexia Nervosa and Bulimia. Am J Med. 2016;129:30-37. 32. Castro J, Toro J, Salamero M, Guimerá E. The Eating Attitudes