Professional Documents
Culture Documents
SUBDIVISION
o Young Old (60-74)
o Middle Old (75-84)
o Old Old (85-99)
o Centenarian (100 up)
OTHERS
o Septuagenarian (70-79)
o Octogenarian (80-89)
* ↑ Population = ↑ life expectancy
*Aging starts at the moment of
conception and continues to the AGING IN THE PHILIPPINES
remainder of life
*Old age-final stage of life (best and
worst experiences)
AGEISM
o Negative feedback towards old
age
NURSING DIAGNOSES
o Elder mistreatment may be
addressed by the following
nursing diagnoses from the North
American Nursing Diagnosis
Association:
▪ Caregiver Role Strain
▪ Coping, Ineffective Family
❖ Compromised
❖ Disabled
▪ Coping, Ineffective
Individual
▪ Protection, Ineffective
▪ Rape-trauma Syndrome
▪ Self-care deficit (poor
INSTITUTIONAL MISTREATMENT hygiene
▪ Self-esteem Situational
o There is death of information
low (unmotivated)
about mistreatment in nursing
▪ Social Isolation (alone)
homes and other residential care
facilities INTERVENTIONS
o Nurses working with older adults
ASSESSMENT must be aware of the elder
o Interdisciplinary comprehensive mistreatment reporting laws in
geriatric assessment of the their states
older adult’s cognitive and o Elder mistreatment requires an
psychosocial function is interdisciplinary team approach
essential in identifying elder
DOCUMENTATION 1. Explain the interaction between
o Excellent documentation is normal aging and responses to drug
extremely important in elder therapy in older people
mistreatment case
AGE-RELATED CHANGES THAT INCREASE
FUTURE CONSIDERATIONS RISK FOR ADVERSE DRUG AFFECTS
o Accurate and uniform data must Older persons tend to have acute and
be continuously collected at chronic conditions that may alter
state and national levels so pharmacokinetics (what the body does
that elder mistreatment trends to the drug) and pharmacodynamics
can be monitored (what the drug does to the body)
o Future research focusing on
evidence-based interventions to Age-related Process
prevent elder mistreatment is 1. Decrease function
needed 2. Slowed function
Nursing Responsibilities:
o Check for liver function tests NURSES’ NOTES
(enzymes in the liver: AST o If a patient is receiving two or
ALT/SGPT SGOT) more drugs that are highly
o Know drugs that are hepatotoxic: protein bound, the nurse should
antibiotics, NSAIDS, COX2 observe for drug interactions
inhibitors (Celecoxib, Arcoxia) and variations in responses to
each drug.
Decrease in serum albumin o Oral medications should be given
❖ Leads to altered binding with a nutritious liquid (e.g.,
capacity juice) rather than water if a
❖ May cause increased serum levels patient is anorexic or is likely
of the “free” or unbound to refuse to take adequate
proportion of protein-bound amounts of liquid. This
drugs maximizes the nutritional values
❖ May result in toxic levels of of liquids ingested. (Do not use
highly protein-bound drugs liquids that are contraindicated
because more unbound drug is due to drug–food interactions.)
available to produce its effects
Pharmacodynamic Alterations
Nursing Responsibilities: ✓ Decreased number of receptors
o Check serum (proteins) ✓ Decreased receptor binding
o Watch out for manic disorder ✓ Altered cellular response to the
medications (lithium/Zoloft) drug-
o Check creatinine (kidneys) ✓ receptor interaction
PRESSURE ULCERS
LEARNING OUTCOMES
o Discuss nursing interventions
that can be implemented to assist
the aging patient with vision and
hearing
o Describe importance of health
education and screening for eye
diseases to prevent unnecessary
vision loss in older adults
ARMD
o Age-related macular degeneration
(ARMD) is the leading cause of
blindness in adults over the age
Thickening of lens (responsible for of 65
focusing light inside the eyes) if lens o ARMD is a degenerative disorder
will thicken and harden, it will have of the macula that affects both
a harder time to focus and become central vision (scotoma) and
yellowish and opaque. Light scatters visual acuity
and there will be color discrimination o Patients with ARMD often require
problems and cause increased risk of more light for reading. They often
falling and vehicular accident experience blurry vision, central
scotomas (blind spots within the
VISION: NURSING CARE visual field), and
o Safety is a major concern with metamorphopsia, in which images
vision changes in the older adult are distorted to look smaller
o Because pupillary reaction slows (micropsia) or larger (macropsia)
with age, an older adult requires than they actually are
more time to become acclimated to o Central vision is mainly affected
changes in light intensity. by this disorder, and peripheral
Nurses should instruct patients vision remains intact (NEI,
on the importance of walking 2009). A person with macular
slowly when entering a room with degeneration will experience a
brighter or dimmer light. dark spot in the center of the
o Provide adequate lighting in field of vision and must learn to
high-traffic areas rely on and interpret peripheral
o Recommend motion sensors to turn vision in order to function
on light when an older person o 25% reduction in the development
walks into a room of age-related macular
o Look for areas where lighting is degeneration by consuming high
inconsistent. Dark or shadowy doses of antioxidants (vitamins C
areas can obscure objects and E and beta-carotene) and zinc.
o Use proper lampshades to prevent o Lutein and zeaxanthin are
glare antioxidant beta-carotenoid
pigments that concentrate in the CATARACTS: NURSING CARE
eye, is associated with a lower o Post-surgical education includes
risk of ARMD reinforcement not to lift any
o These nutrients are found in eggs, heavy objects, strain at stool,
spinach, romaine lettuce, or bend at the waist. (cause
broccoli, corn, and brussels intraocular pressure and delay
sprouts. healing)
o As a nurse, position yourself on o Patients with cognitive
the area/side where there is no impairments such as Alzheimer’s
ARMD disease must be carefully
supervised for at least 24 hours
RISK FACTORS after surgery to ensure that they
o Age do not remove the protective eye
o Smoking patch and do not rub their eye.
o Family history of ARMD o When surgery is needed in both
o Exposure to UV light eyes, one eye is done first and
o Comorbidities the second procedure is scheduled
a month or so later to allow
CATARACTS healing and recovery.
o Cataracts cloud the lens,
decrease the amount of light able GLAUCOMA
to reach the retina, and inhibit o Glaucoma is associated with optic
vision. nerve damage due to an increase
o Development is slow and painless, in IOP (intraocular pressure),
and may be unilateral or which can ultimately lead to
bilateral. vision loss.
o Cataracts are the leading cause o Problem with the optic nerve
of blindness in the world. o Aqueous humor (produced in
o Patients with cataracts may anterior chamber of eye). The one
experience blurry vision, glare, maintaining intraocular pressure
halos around objects, double (10-20; average 15)
vision, difficulty sensing o If outflow of AH is obstructed,
contrasting colors because colors it will accumulate and increase
appear faded or discolored, and pressure in the eyes damaging the
poor night vision. optic nerve and gradual visual
o Patients with cataracts should be loss
recommended for surgery o Greater than 21 the optic nerve
(treatment of choice) will atrophy and will experience
▪ outpatient basis blindness
▪ 15-30-minute procedure o Manifestation: pain
▪ local anesthesia (topical)
▪ lens will be removed and RISK FACTORS
replaced with an artificial o Age
lens (phacoemulsification) o Increased ICP
▪ 1-3 weeks of rest o Diabetes
▪ 20k for procedure; lens 60k o Hypertension
but depends on where it’s
made GLAUCOMA: NURSING CARE
o When administering eyedrops
RISKFACTORS (lowers ICP), it is important for
oAge the nurse to first wash his or her
oSmoking and alcohol hands, ask the patient to tip the
oObesity head backward and look upward,
oComorbidities like diabetes, then pull the lower lid down
hyperlipidemia slightly to make a small pouch.
o UV rays o The nurse should try not to drop
the medication directly onto the
eye but rather into the eyelid o The nurse should educate patients
pouch to prevent a violent blink on how to check serum glucose
reflex and excessive tearing. levels, when and how to administer
medications (insulin or oral
DIABETIC RETINOPATHY hypoglycemic medications), and
o Diabetic retinopathy is a signs and symptoms of
microvascular disease of the eye hypoglycemia and hyperglycemia.
occurring in both type 1 and type
2 diabetes. AGE-RELATED CHANGES IN HEARING
o Damage to the ocular o Hearing loss can interfere with
microvascular system impairs the communication, enjoyment of
transportation of oxygen and certain forms of entertainment
nutrients to the eye (Huether & such as music and television,
McCance, 2012). safety, and ultimately,
o Develop micro aneurysms and independence.
create new blood vessels o Hearing impairments make
(neovascularization) that can communication difficult and are
cause drainage of the aqueous often frustrating for both the
humor may be impaired and cause patient and family.
another glaucoma (neovascular o In the older adult, cerumen tends
glaucoma) to be drier and harder, and tends
o Prevention of diabetic to accumulate in the ear canal due
retinopathy is dependent on tight to decreased activity of the
glycemic control in addition to apocrine glands. Hearing may
managing hypertension and become impaired if cerumen
hyperlipidemia. accumulates to impact the canal.
o Goals of treatment for patients o Cerumen impaction is one of the
with diabetes include maintaining most common and reversible causes
an average pre-prandial blood of conductive hearing loss in
glucose of 80 to 120 mg/dL, an older adults.
average bedtime capillary blood o Auto toxic medications, exposure
glucose of 100 to 140 mg/dL, and to excessive noise, smoking and
a hemoglobin (HbA1c) of less than head injury are also factors
7.
o Gradual vision loss with HEARING: NURSING CARE
generalized blurring and areas of o Recommended aural hygiene
focal vision loss. involves gentle cleansing of the
auricles (out-side of the ears)
DIABETIC RETINOPATHY: NURSING CARE while bathing or showering. The
o Nurses educate patients about use of cotton-tipped applicators
diabetes mellitus and the to cleanse the ear canal is not
importance of glycemic control to recommended because the
prevent retinopathy. applicator may push the cerumen
o Proper nutrition, including a deeper into the canal and thus
low-carbohydrate and low- increase the risk of impaction,
cholesterol diet, is imperative as well as traumatize the canal
to keep blood glucose levels down wall and tympanic membrane
and decrease the risk not only of (McPhee & Papadakis, 2011).
cardiovascular disease and o Curette. A small instrument with
hypertension but also to decrease a scoop on the end is inserted
the risk of microvascular damage into the ear canal while the helix
to the eyes. is lifted posteriorly and
o Exercise helps to lower glucose laterally.
levels, burns extra calories for ▪ Increased risk of injury to
weight management, and reduces the tympanic membrane/ear
insulin resistance in people with canal
type 2 diabetes.
o Lavage or irrigation. Irrigation o Pause at the end of each phrase
is the simpler and more or sentence.
straightforward approach to o If the patient has a hearing aid,
cerumen removal. provide assistance with the
▪ Saline solution/warm water device, plus glasses if needed.
▪ Risk for infection o Assess the illumination in the
room and make sure that the
o Contraindications patient can see you. Face the
▪ Perforated tympanic patient at all times during the
membrane conversation.
▪ Ear traumas o The patient may read lips, so it
▪ Tumor is important not to cover your
▪ Diabetic patient because of mouth or chew gum. Do not speak
high risk for infection into the chart or converse with
someone over your shoulder. The
o Examination of the ear may reveal patient will misinterpret your
an external infection or message.
impaction that can be treated o Speak slowly and clearly in a
appropriately to resolve the normal tone of voice—do not shout.
hearing loss. If the problem is o If the patient does not understand
not that obvious, a few basic your message, rephrase it rather
screening tests can be performed: than repeating the same words.
the whisper, Weber, and Rinne o Gestures, if appropriate, may
tests. help.
o The working condition of the o Use written communication if the
hearing aid is then assessed. patient is able to see and read.
Assessment parameters include: o Ask the patient for an oral or
▪ Integrity of the ear mold. written response to determine if
the communication was successful.
▪ Battery.
▪ Dials.
▪ Switches.
▪ Tubing for behind the ear
aids.
Hearing Aids
▪ Check batteries
▪ Checked by audiologist for
tuning in 2-3 months
▪ Clean with water or saline
with a cotton pad. Don’t use
alcohol because it would
degrade the plastic
▪ Batteries should be removed
in a dry place
Hypothalamus
o Controls body temperature,
hunger, fatigue, and sleep
Basal ganglia
o Control of movements, hearing,
habit, cognition, and sensation
Thalamus
o Regulation of sleep,
consciousness, and alertness
Hippocampus
o Motivation, emotion, learning,
and memory
▪ APP (amyloid precursor
protein) if this breaks down
becomes beta amyloid
- This bridges impulses
(chemical and electrical)
from one neuron to the
other
- If this metabolizes
becomes beta amyloid
- Half of Beta broken down
and metabolized and
eventually thrown away
- Half remains and forms
plaques in the
hippocampus
o Tau protein
▪ A normal protein existing in
every cell body (neurons)
but for some reasons these
proteins undergo a process
where phosphate molecules
target tau proteins > loses
its shape and then becomes
tangled > connects to the
microtubules of the neuron
and then it blocks the
signal of the chemical and
electrical signals from one
neuron to the other, thus,
o Two problems: formation of degrading neurons leading to
amyloid plaques and death
neurofibrillary proteins or tau o No cure for Alzheimer’s currently
proteins ▪ But can be prevented by
▪ Amyloid plaques combine then balanced diet (fasting
stops transmission of diets) and exercising
neurotransmitters > neuron ▪ Memory exercising
dies > (x) transmission of o Increasing sulcus spaces and
impulses > (x) memory, brain increasing ventricles is evident
control, function in Alzheimer’s
▪ Tau protein combine > cause
tangles > also hinder STAGES
impulse transmission > death
of neuron
▪ Increase and accumulation >
damage cortical area which
controls motor, memory, and
executive
▪ Number of synapses decreases
> neuron deprived on
nutrients > death
o As people age amyloid and tau
proteins increase in number
o Function of beta amyloid and tau o Not easily diagnosed because the
proteins only manifestation is memory loss
▪ There is already a lot of
beta amyloid in the synapse
MILD o Prompt treatment of all
o Begins with forgetfulness reversible and irreversible
o Progresses to disorientation and conditions
confusion o Coordination between care
o Personality changes providers and family members
o Symptoms of depression/manic
behaviors Goal
o Preserve self-esteem
MODERATE o Retain self-care abilities
o Need assistance with ADLs o Prevent complications
o Unable to remember names
o Loss of short-term recall Additional notes
o May display anxious, agitated, o Parkinson’s is considered as
delusional, or obsessive behavior dementia because it also causes
o May be physically or verbally shrinkage of the brain
aggressive o Travels from the hippocampus to
o Poor personal hygiene the right area where it reaches
o Disturbed sleep the thalamic area and the basal
o Inability to carry on a ganglia where dopamine is created
conversation and stored
o May use “word salad” (sentence o Dopamine is responsible for
fragments) accuracy of movement
o Posture may be altered
o Disoriented to time and place
o May ask questions repeatedly
SEVERE
o Loss of verbal articulation
o Loss of ambulation
o Bowel and bladder incontinence
o Extended sleep patterns
o Unresponsive to stimuli
MANAGEMENT
o Cure is NOT POSSIBLE
o Pharmacologic – START LOW, GO SLOW
to decrease cognitive decline
o Non-Pharmacologic
▪ CBT: Cognitive Behavior
Therapy ASSESSMENT TOOLS
▪ Communication, Consistency o CAM: Confusion Assessment Method
▪ SAFETY AND COMMUNICATION (Delirium)
o MMSE: Mini Mental Status
Pharmacologic Examination or Mini-Cog
Cholinesterase Inhibitors (Dementia)
o Block the breakdown of ACH o CDT: Clock Drawing Test
o Acetylcholine, a neurotransmitter (Dementia) supplement to MMSE
for thinking and memory o GDS: Geriatric Depression Scale
o Side effects: nausea and diarrhea (Depression)
o Donepezil, Rivastigmine,
Galantamine (mild to moderate)
o Memantine (moderate)
Nurse’s Role
o Appropriate use of available
pharmacologic and
nonpharmacologic interventions
PARKINSON’S DISEASE (PD)
o A chronic, progressive neurologic
disorder
o Symptoms are caused by the loss
of nerve cells in the pigmented
substantia nigra pars compacta
and the locus coeruleus in the
midbrain
▪ Substantia nigra pars
compacta- has dopamine
o Lewy bodies are present in the
basal ganglia, brain stem, spinal
cord, and sympathetic ganglia
▪ Lewy body will be tangled in
the synapse of a neuron
▪ This will result to the
motor deficits
o Considered an extrapyramidal
syndrome - chorea (involuntary
twitching of the limbs or facial
muscles), and dystonia
(involuntary muscle contractions
forcing unusual or painful
positions)
▪ EPS- because of its
anatomical structure
- Tremors
▪ Chorea and dystonia- both
are involuntary due to the
imbalance of acetylcholine
(excitatory
neurotransmitter) and
dopamine (inhibitory
neurotransmitter)
▪ In Parkinson’s there is
decrease of the dopamine
resulting to involuntary
movements
o Increases with AGE; more common
in men than women
▪ Parkinson’s disease is
prevalent in 1-2 persons/
1,000 people increases 2%
above 65 years old
o Unknown cause
▪ Idiopathic symptoms will
appear even without other
symptoms
o Loss of the dopaminergic cells
situated deep in the midbrain in
the substantia nigra (the black
substance so named because of the
melanin seen in those neurons).
With the depletion of dopamine,
which inhibits neurotransmitters,
an abnormal movement syndrome
characterized by rigidity and o Message passed to end of axon >
tremor can occur sacs containing dopamine is
o Aside from the dopaminergic stimulated to be released in the
activity, exposure to synapse > crosses to bind to
environmental toxins that results dopamine receptors > opening >
to genetic predisposition can transmitting the message to the
lead to this disease receptor cells > dopamine goes
▪ Toxins > becomes neurotoxins back to the synapse > reabsorbed
> inflammation of the brain in the axon
> especially in the blood o There are enzymes that destroys
vessels > decrease in dopamine if it is not reabsorbed,
nutrients especially in the MAO-B (Monoamine Oxidase Type B)
substantia nigra > enzymes, cleans the synapse so it
decreasing dopamine release is ready for the next message
or production o Destruction of the dopamine
o Will worsen without treatment neuronal cell in the substantia
o Also, a neurodegenerative nigra of the basal ganglia >
disorder decrease/ depletion of dopamine
o Patients with this disease may stores > imbalance of the
also present cognitive impairment excitatory and inhibitory
o May happen to first degree neurotransmitters
relatives of older people who had o Excitation occurs (because
Parkinson’s acetylcholine is higher) > no
o Currently, no exact diagnostic restriction in the movement >
test, but is assessed and tested occurs in the corpus striatum,
through PET scans, MRIs, dopamine where EPS tract is located
tests controlling the complex body
o For confirmatory test, autopsy is movements
done o With severity of PD depletion of
dopamine leads to other changes >
PATHOLOGY OF PARKINSON’S DISEASE affect other neurotransmitters
like glutamate, GABA, and
serotonin
o GABA and glutamate are excitatory
neurotransmitters
o Serotonin – mood stabilizer
o That is why you see PD a mask-like
expression
Akinesia
o No movement
o Sudden arrest of movement
Bradykinesia
o Slowness of active movements
o Peristalsis will still be normal
because this is involuntary, but
the problem will be in the
chewing, eating, swallowing
because this are voluntary > high
risk for aspiration, may present
dysphagia (inability to swallow)
in progressive stages
Mid stage
o Urinary and bladder problems are
presented here
▪ Do not give food high in
vitamin B or pyridoxine or
Advanced vitamin B complex because
o Can no longer walk or stand vitamin B facilitates
without assistance breakdown of levodopa,
o Not able to do activities of daily increases action of dopa
living decarboxylase
o There are present cognitive ▪ Older person may develop
manifestations already drug induced tolerance
▪ Drug becomes ineffective at
PHARMACOLOGICAL TREATMENT FOR PD this time hyperactive
movements will show, take
note of the time when
levodopa is administered
o Amantadine
▪ Combined with levodopa and
carbidopa or cholinergic
▪ Potentiated release of
dopamine (remaining
dopamine in the substantia
nigra)
▪ This is transitory, does not
happen all the time this is
why It is combined
o In the synaptic junction there is ▪ Also, a prophylaxis in viral
dopa decarboxylase > destroys infections
dopamine o Dopamine agonists
o There are two factors that
▪ Directly stimulates
complicate the mechanism of
dopamine receptors >
action of levodopa
increase dopamine in the
▪ Med must past through BBB substantia nigra > restores
dopa decarboxylase is also the balance between
in the intestinal mucosa inhibitory and excitatory
▪ Only 30-40% of the neurotransmitters
medication crosses the BBB ▪ Initiated in the early
▪ Most of it is lost before it stages of PD before starting
enters the gen circulation with levodopa
and BBB ▪ Used in combination with
o New drug- levodopa + DDC inhibitor levodopa throughout the
> destroys dopa decarboxulase progression of PD
▪ Carbidopa + levodopa ▪ Bromocriptine
(sinemet) can be divided
▪ Pramipexole
into 4
▪ Ropinorole (requip)
▪ Added to maximize absorption
▪ Patients should be monitored
and facilitate crossing of
for sleepiness and
the BBB
drowsiness
▪ Should be taken in an empty
o MAO-B inhibitors
stomach
▪ Normally we have this in the
▪ At least 1 hour before meals
synapses, it destroys
or 2 hours after meals
dopamine that is not
▪ Potential side effect nausea reabsorb signal
and vomiting, postural transmission
hypotension
▪ Inhibits breakdown of
▪ Teach strategies to avoid dopamine
falling o COMT inhibitors (peripheral)
▪ How to sit and how to stand,
hold on unto something
▪ Catechol-O- NURSING CARE
methyltransferase The goal of treatment is to control the
▪ Reduces motor fluctuations symptoms and maintain functional
in PD and increases duration independence
of levodopa and carbidopa by o Do you have leg or arm stiffness?
decreasing its o Have you experienced any
biotransformation in the irregular jerking of your arms and
peripheral tissues legs?
▪ Synergistic drugs o Do you noticed yourself grimacing
▪ May cause dizziness and turn or making faces or chewing
urine orange or brown in movements?
color o Does your mouth water
▪ Make sure avoids alcohol, excessively?
this will potentiate risk of
liver failure o Improve functional mobility
o COMT inhibitor (central) o Maintain independence in
▪ Anticholinergic activities of daily living
▪ Used for symptomatic o Achieve adequate bowel movement
treatment o Attain and maintain acceptable
nutritional status
▪ Relieves tremors and
o Achieve effective communication
rigidity
o Develop positive coping
▪ Blocks action of
mechanisms
Acetylcholine and corrects
imbalance
FUBCTIONAL MOBILITY
▪ WOF: dry mouth, o Encourage exercises for joint
constipation, blurred mobility and joint flexibility
vision (not given to client (e.g., stationary bike, walking,
w glaucoma) urinary and range-of-motion exercises)
retention o Teach patient to walk erect, watch
▪ Do not give if the client the horizon, use a wide-based
has urinary retention gait, swing arms with walking,
because it could complicate walk heel-toe, and practice
an enlarged prostate marching to music
o Encourage breathing exercises
NURSING DIAGNOSIS while walking and frequent rest
o Impaired physical mobility periods to prevent fatigue or
related to muscle rigidity and frustration
motor weakness
o Self-care deficit related to FALL
tremor and motor disturbances o Falls are a significant public
o Constipation related to health problem. Worldwide, falls
medication and reduced activity are the second leading cause of
o Imbalanced nutrition: less than accidental or unintentional
body requirements related to injury deaths
tremors, slowness in eating, o The presence of dementia
difficulty in eating and increases risk for falls,
swallowing twofold, and individuals with
o Impaired verbal communication dementia are also at increased
related to decreased speech risk of major injuries (fracture)
volume, slowness of speech and related to falls
inability to move facial muscles
o Ineffective coping related to
depression and dysfunction due to
disease progression
o Risk for falls/risk for injury
related to rigidity and motor
weakness
assistance in pulling up without
help
o Enlist assistance of an
occupational therapist as
indicated
BOWEL ELIMINATION
o Increase oral fluid intake and
encourage to eat food with
moderate fiber content
o Establish a regular bowel routine
o A raised toilet seat is useful,
because the patient has
difficulty in moving from a
standing to a sitting position
Question
Question The nursing diagnosis for a patient is
A patient with Parkinson’s disease has imbalanced nutrition less than body
a diagnosis of impaired physical requirements related to the progress of
mobility related to bradykinesia. Which Parkinson’s disease and adverse effects
action will the nurse include in the of pharmacotherapy. What is the best
plan of care? nursing intervention to include in the
a. Instruct the patient in plan of care?
activities that can be done while a. Cheese-and-cracker snacks
lying or sitting b. Whole grains and vegetables
b. Suggest that the patient rock form c. Cereals fortified with vitamin B6
side to side to initiate led
movement COMMUNICATION
c. Have the patient take small steps o Remind patient to face the
in a straight line directly in listener, speak slowly and
front of the feet deliberately, and exaggerate
pronunciation of words; a small
SELF-CARE electronic amplifier is helpful
o Environmental modifications are if the patient has difficulty
necessary to compensate for being heard
functional disabilities o Instruct patient to speak in short
o A hospital bed at home with sentences and take a few breaths
bedside rails, an overbed frame before speaking
with a trapeze, or a rope tied to o Enlist speech therapist to assist
the foot of the bed can provide patient