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Critical Thinking M.H. Marigowda and Krishnaswamy. Plant wealth of Lal-Bagh, Bangalore,
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A. Razi. Flora of Bangalore district. Mysore, Prasaranga, University of Mysore, 1973, pp: 739.
Jochen W L Cals Maastricht University 253 PUBLICATIONS 3,786 CITATIONS SEE PROFILE
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Pawilai Chinwangso, Stanko Brankovic, Richard C. Willson, and Dmitri Litvinov. “Development of
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I want to place an order 45. Krongelb, S., C. Bonhǒte, S. R. Brankovic, H. H. Gatzen, P. Hesketh, Y.
Kitamoto, T. Osaka, W. Schwarzacher, and G. Zangari. “Preface (ECS Transactions 50: 10).” ECS
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density of TiO2, the voltage for the former was found to be only about half of that of the latter. 42.
Krongelb, S., C. Bonhôte, S. R. Brankovic, H. H. Gatzen, Y. Kitamoto, T. Osaka, W. Schwarzacher,
and G. Zangari. “ECS Transactions (33: 34): Preface.” ECS Transactions 33, no. 34 (2011). Seek
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Assumptions are typically made in research to aid in creating excellent theories and research
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Kahn found that with an overproduction of GLUT4 in adipose tissue and muscle, glucose tolerance
and insulin sensitivity are significantly increased. Both aerobic and resistance exercise increase the
amount of GLUT4 concentrations and stimulate blood glucose uptake (Colberg et al., 2010).
Research has shown that an individual who exercise regularly will have a better glucose tolerance
and lower insulin resistance level than an individual who is constantly sedentary (Ivy, 1997). Using
training, insulin action can be increased, and this can cause a significant improvement in glycemic
control. Glycemic Control After a meal, blood glucose will increase, and skeletal muscle is
responsible for most of the glucose uptake (Adams, 2013). Glycemic control is dependent on insulin
levels and the translocation of glucose via GLUT4. When a healthy individual exercises, the glucose
uptake by muscle cells is balanced out with an equal amount of hepatic glucose production. During
moderate-intensity exercise of short duration, a patient with type 2 diabetes will have a higher
glucose uptake by muscles than hepatic glucose production; this will lower individual’s blood
glucose and improve glycemic control (Adams, 2013). To maintain glucose homeostasis, glucagon
and catecholamines play a huge role. Glucagon sends signals to the liver and increases glucose
production when insulin level decreases while catecholamines signal increases glucose production
during moderate-intensity exercise greater than 2 hours duration (Adams, 2013). Resistance training
is found to be effective for glycemic control: muscles utilize more blood glucose and become more
sensitive to insulin (Ishiguro et al., 2016). During resistance exercise, skeletal muscle contractions
increase blood glucose uptake in order to supplement intramuscular glycogenolysis. The effects of
this contraction-mediated pathway continue for a couple hours after exercise (Colberg et al., 2010).
Contractions trigger glucose transporter (GLUT4), which compensates for the impaired insulin-
stimulated GLUT4 in diabetic individuals (Colberg et al., 2010). CONCLUSION Current research
indicates that aerobic and resistance trainings leads to a decrease Forget about the size of font and
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1. How Does Exercise Help Prevent the Progression of Chronic Hyperglycemia in Individuals with
Type 2 Diabetes Mellitus? INTRODUCTION Definition, Criteria, and Prevalence Diabetes mellitus
is a metabolic disorder where the individual cannot regulate blood glucose normally due to insulin
abnormalities (Karoline, 2015). Diabetes type 1 is characterized by the autoimmune and non-
autoimmune destruction of β-cell, which is responsible for the production of insulin, resulting in the
inability to regulate blood glucose (Alberti & Zimmet, 1998). Type 2 diabetes mellitus, commonly
known as adult onset diabetes, is defined as a decline in insulin sensitivity from the onset of the
disease (Weyer, Bogardus, Mott, & Pratley, 1999). A long-term exposure to abnormally high levels
of glucose and free fatty acids (FFA) can cause β-cell dysfunction and cell death; individuals will
have varying degrees of insulin resistance and insulin hyposecretion (Alberti & Zimmet, 1998; Cnop
et al., 2005). Type 2 diabetes mellitus has become one of the fastest-growing epidemics, affecting
more than 170 million people worldwide. Almost 60 million residents in the US have prediabetes,
increasing their risk for type 2 diabetes due to their abnormally high blood glucose level (Stumvoll,
Goldstein, & van Haeften, 2005; Colberg et al.,2010). 90% of the population suffering from diabetes
mellitus are non-insulin dependent (type 2 diabetes), while the other 10% are mostly insulin
dependent diabetes, also known as type 1 diabetes mellitus (Stumvoll et al., 2005). Type 2 diabetes
mellitus is often diagnosed by identifying the initial stages of insulin resistance (Karoline et al.,
2015). According to the American Diabetes Association (ADA), there are four criteria that are used
to determine whether an individual suffers from type 2 diabetes: 1) glycated hemoglobin (A1C)
value of 6.5% or higher (mg/dl), 2) fasting plasma glucose ≥126 mg/dl, 3) 2-hour plasma glucose
≥200 mg/dl, and/or 4) classic symptoms of hyperglycemia (Colberg et al., 2010). Any of the above
conditions can be used to diagnose this disease. Long term diabetes-related problems include
blindness, amputation, dialysis, cardiovascular disease, and chronic