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Cardiac Monitoring and ECG Interpretation

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0% found this document useful (0 votes)
32 views10 pages

Cardiac Monitoring and ECG Interpretation

Uploaded by

Chameka Small
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd

Cardiac Monitoring Notes

Patient Care III


MI 211c

Cardiac Monitoring

 Cardiac Conduction Cycle


- Provides electrical stimulus to cause the heart muscle to continually
 Contract (systole)
 Relax (diastole)
- SA node
- AV node
- Bundle of His
- Bundle Branches
- Purkinje Fibers
- Electrical impulses cause each myocardial cell to:
1) Change from a resting (polarized) state
2) To a state of contraction (depolarized)
3) Back to a resting state by recharging (repolarized)

1
 Cardiac Conduction Cycle Electrophysiology
- Heart cells (myocytes) are capable of conductivity, excitability, contractility, and
automaticity
- Myocytes influenced by Na+, Ca2+, and K+
- At rest – cells polarized
 Cell interior negatively charged
 Charge builds up until threshold is reached
- Na+ ions flood into the cell, followed by Ca2+ ions – cells depolarized
 Cell interior becomes more positive than outside
 Followed by mechanical event = contraction
- K ions move out of the cell – cells repolarized
+

 Interior cell charge transitions to be more negative

Cardiac Conduction Cycle

 Electrocardiogram
- An electrical picture of the heart measured externally
 Ability to do so discovered in mid-1880’s
- ECG vs. EKG
 Dr. Willem Einthoven, 1901
- Labeled with letters
 P, Q, R, S, T, U
 Letters correspond to events in the cardiac cycle

2
Electrocardiogram

 Cardiac Cycle Waves


- P-wave
- QRS complex
- T-wave
- U-wave

Cardiac Cycle Waves Description Image


Atrial depolarization -
Contraction
Location - Precedes the QRS
complex
Amplitude - Not more than
0.25 mV (2-2.5 boxes)
Duration - 0.06-0.11 seconds
P-Wave
Appearance - Rounded and
upright

3
Ventricular depolarization -
Contraction
Location - Follows the PR
interval
Amplitude - Variable with
ECG lead
QRS Complex Duration - <0.12 seconds
Appearance - 3 waves

Ventricular repolarization -
Return to resting state
Location - Follows the S wave
Amplitude - 0.5 mV or less
Duration - Not measured
Appearance - Rounded and
smooth
T-Wave

His-Purkinje repolarization -
Return to resting state
Location - Follows the T wave
Amplitude - Not measured
Duration - Not measured
U-Wave Appearance - Upright and
rounded

4
 Cardiac Cycle Waves
- PR Interval
 Beginning of the P-wave to the beginning of the QRS complex.
• Beginning of atrial depolarization to the beginning of ventricular
depolarization
• 0.12 - 0.2 sec duration
- ST segment
 End of the S-wave to the beginning of the T-wave
 Represents the end of ventricular depolarization and the beginning of
ventricular repolarization
 Duration
- Not Measured QT Interval
 Beginning of the Q wave to the end of the T Wave
 Time from ventricular depolarization to ventricular repolarization
 Duration
• 0.36 - 0.44 seconds duration
- RR Interval
 Distance from one R-wave to the next
 Used to determine if rhythm is regular

Cardiac Cycle Waves

5
 Interpreting Rhythm Strips
a. Step 1:
 Determine the rates
• Atrial rate = P waves per 6s x 10
• Ventricular rate = QRS complex (R waves) per 6s x 10
b. Step 2:
 Assess regularity of rhythm (20 – 30 s)
• Atrial = P wave to P wave
• Ventricular = R wave to R wave
c. Step 3:
 Assess atrial activity
• Compare appearance and configuration of P waves
o Present?
o Normal shaped?
o Position?
d. Step 4:
 Determine the duration of the PR interval
• Is it WNL?
o 0.12 - 0.20 sec
• Is there one P wave for each QRS complex?

e. Step 5:
 Determine the duration of the QRS complex
• Size and shape
• Duration (0.04 - 0.11 sec)
• Distance from T-wave
• Point in same direction

f. Step 6:
 Evaluate the T-wave
• Present
• Size and Shape
• Point in same direction as QRS complex
g. Step 7:
 ST segment flat on isoelectric line

6
Part II

 What is expected of you?


- Recognize normal sinus rhythms
- Be observant for signs and symptoms
- Respond appropriately
 Calm, reassuring attitude
 Assess vital signs
 Elevate patient’s head
 Be prepared to administer oxygen
 Be prepared for IV drug administration
 Be prepared to call a CODE

 Recognize Common ECG Abnormalities


- Sinus Bradycardia -Ventricular Fibrillation
- Sinus Tachycardia - Atrial Fibrillation
- Ventricular Tachycardia - Atrial Flutter
- Premature Ventricular Contractions (PVCs) - Heart Block

 Bradycardia vs. Tachycardia


- Bradycardia
 Slow heart rate
 <60 beats/min
- Tachycardia
 Fast heart rate
 >100 beats/min

Bradycardia

Tachycardia

7
 Ventricular Tachycardia
- Hear rate between 150-250 bpm
- Low Cardiac output
 Pt. lose consciousness
 Becomes hypotensive
- Solution:
 Amiodarone via IV
 May need to use defibrillator

 Premature Ventricular Contractions


- PVCs
 A ventricular contraction preceding the normal impulse initiated by the SA
node.
 May occur singly, in pairs or in threes
- ECG Characteristics
 Rhythm: irregular
 P-wave: Absent
 QRS complex
• Bizarre configuration
• Occurs early
- Causes:
 Caffeine, tobacco, alcohol ingestion, exercise, scarring from MI’s, foreign
body ‘tickling’ the heart
- Signs/Symptoms:
 Asymptomatic
 Palpitations
 Decreased cardiac output

 Ventricular Fibrillation
- Chaotic, irregular contractions of the
ventricles
- Waves are jagged
- Life threatening
- Ventricles fail to pump blood
- Leads to asystole and death
- ECG Characteristics:
 normal parameters not measurable
- Causes:
 Acute MI
 Electric Shock
 Untreated Tachycardia
 Electrolyte Imbalance
 Hypothermia
8
 Ventricular Fibrillation CONT.
- Symptoms
 Absent Pulse and BP
 Loss of Consciousness
 Cyanosis
- Treatment
 CPR
 Defibrillation
 Epinephrine

 Atrial Fibrillation
- Both atria and ventricles contracting
rapidly
- Atrial and ventricular rhythms
irregular
- May lead to PE or stroke
- Symptoms
 Palpitations
 Nausea
 Weakness & Fatigue
- Treatment
 Slow the ventricular response.
 Increase cardiac output

 Atrial Flutter
- Rapid, but regular contractions of the
atria
- Waves are rounded
- Atrial rate 250 – 400 beats/minute
- ECG Characteristics
 P-wave: Absent
• Flutter waves are present
 QRS complex: < 0.12 seconds
- Causes:
 Valve Disorder
 Infection
 Alcoholism
 Hyperthyroidism
- Symptoms
 Chest pain
 SOB
 Hypotension

9
 Heart Block
- An interference of the
normal electrical
conduction of the heart
- Defined by the location of
the block
 A-V Heart Block
- Symptoms
 SOB
 Chest pain
 Hypotension & lightheadedness

10

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