Infectious Coryza

Presenter: Dr. Bikash Puri

 Introduction

 Infectious coryza is an acute to sub acute

respiratory disease of chickens.
 The disease was named infectious Coryza
because it was infectious and affected primarily
the nasal passages (nasal discharge).
 Also known as “fowl Coryza”.
Cont. …

 Worldwide disease of chickens having economic

importance
 This disease is mainly observed in layers, and
Marked drop in egg production in layers and
breeders (10% to more than 40%) is observed.

Multi-age farms are more susceptible.

Etiology

Haemophilus paragallinarum
( gram-negative, pleomorphic, non-motile catalase-negative,
microaerophilic rod)
 Transmission

 Direct contact, airborne droplets, and contamination of

drinking water.
 It is not egg transmitted.

 Chickens that have recovered from the disease and

chronically sick chickens are carriers of the bacterium
and are the main source of the infection.
 Pathogenesis

 Inhalation of infected air droplets

 Binds to the ciliated mucosa of upper respiratory,

 With capsule and H.A Colonization

Endotoxins
 Clinical Disease
 Infectious coryza may occur in broilers and layers.
 The most common clinical signs are:
1. Nasal discharge
2. Facial swelling
3. Lacrimation
4. Anorexia
5. Diarrhea
 Decreased feed and water consumption
1. Retards growth in young stock
2. Reduces egg production in laying flocks.
Typical facial edema and swelling on the face of a chicken
conjunctivitis, infraborbital sinusitis and lacrimation
conjunctiva, periorbital region
 Postmortem Findings

1. Exudate in infraorbital sinuses, congestion of nasal

mucosa
2. Fetoid watery discharge form nostril
3. Congestion of nasal mucosa
4. When layers are infected, peritonitis, due to deposited
eggs in the peritoneal cavity, soft shell eggs and eggs
with hematomas are often observed in the ovary.
 Microscopic Findings
1. Fibrinopurulent cellulitis,
2. Air sacs: oedema thickening, mesothelial hyperplasia,
fibrin deposition.
3. Fibrin and heterophilic infiltration is higest in subcutis.
Diagnosis
A presumptive diagnosis may be made on:
1. Clinical signs.
2. Post mortem lesions.
3. Identification of the bacteria in a Gram-stained smear from
sinus. On examination of smear, gram negative, bipolar
filametous or coccoid organish my be found
Differential Diagnosis
 Mycoplasmosis,
 respiratory viruses,
 chronic or localized pasteurellosis and
 vitamin A deficiency.