This document discusses diabetes mellitus (DM), a chronic disorder caused by deficient insulin or defective insulin action that results in hyperglycemia. There are two main types of DM - type 1, which typically occurs before age 30 and requires insulin injections, and type 2, which usually develops after age 30 and may be managed with diet, exercise, or oral medications. The document outlines the differences between the two types and lists signs and symptoms of DM like polyuria, polydipsia, and blurred vision. Complications if DM is not properly managed include atherosclerosis, infections, and neuropathy.
This document discusses diabetes mellitus (DM), a chronic disorder caused by deficient insulin or defective insulin action that results in hyperglycemia. There are two main types of DM - type 1, which typically occurs before age 30 and requires insulin injections, and type 2, which usually develops after age 30 and may be managed with diet, exercise, or oral medications. The document outlines the differences between the two types and lists signs and symptoms of DM like polyuria, polydipsia, and blurred vision. Complications if DM is not properly managed include atherosclerosis, infections, and neuropathy.
This document discusses diabetes mellitus (DM), a chronic disorder caused by deficient insulin or defective insulin action that results in hyperglycemia. There are two main types of DM - type 1, which typically occurs before age 30 and requires insulin injections, and type 2, which usually develops after age 30 and may be managed with diet, exercise, or oral medications. The document outlines the differences between the two types and lists signs and symptoms of DM like polyuria, polydipsia, and blurred vision. Complications if DM is not properly managed include atherosclerosis, infections, and neuropathy.
EXOCRINE FUNCTION. DIABETES MELLITUS Chronic disorder caused by deficient insulin or defective insulin action in the body. It is characterized by hyperglycemia and disruption of carbohydrates, fats and proteins. It has two types DM type 1 DM type 2 PATHOLOGY SPECIAL PHYSIOLOGIC CHANGES WHEN INSULIN IS LACKING OR INEFFECTIVE
GLUCOSE ACCUMULATION IN BLOOD
IMPAIRED PROTEIN SYNTHESIS
ALTERED PROTEIN AND FAT METABOLISM
DIFFERENCE BETWEEN TYPE 1 AND TYPE 2 DIABETES FACTORS TYPE 1 TYPE 2 AGE OF ONSET < 30 >30
TYPE OF ONSET ABRUPT GRADUAL
EDOGENOUS LITTLE OR NONE BELOW OR ABOVE NORMAL INCIDENCE 10 % 90% KETOACIDOSIS MAY OCCUR UNLIKELY INSULIN INJECTIONS REQUIRED 20-30% 0F PATIENTS BODY WEIGHT NORMAL OR THIN OBESE MANAGEMENT DIET, EXERCISE, DIET;EXERCISE, ORAL INSULIN HYPOGLYCEMICS OR INSULIN ETIOLOGY VIRAL /AUTOIMMUNE INSULIN RESISTANCE HEREDITARY YES YES CLINICAL S/S POLYURIA POLYDIPSIA POLYPHAGIA WEIGHT LOSS HYPERGLYCEMIA GLUCOSURIA KETONURIA FATIGUE AND WEAKNESS BLURRED VISION IRRITABILITY RECURRENT INFECTIONS NUMBNESS BRUISES COMPLICATIONS OF DM ATHEROSCLEROSIS INFECTIONS NEUROPATHY PERIARTHRITIS HAND STIFFNESS