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IN CARDIOVASCULAR
DISORDERS
Georgie S. De La Cruz BSND 3A
CORONARY HEART DISEASE (CHD)
also known as ischemic heart disease (IHD), coronary artery disease
(CAD), coronary occlusion, atherosclerotic heart disease (ASHD), or
coronary thrombosis
a common form of heart disease among individuals over 40 years of
age and is a leading cause of death.
a condition in which the oxygen supply to the myocardium is
inadequate, usually due to atherosclerosis in the coronary circulation.
It can also result in hypertrophy of the cardiac muscle.
Modifiable and Unmodifiable Risk Factor
Risk factors are conditions and habits that have been demonstrated to be
associated with an increased probability of cardiovascular disease.
Major risk factors for CHD other than LDL-C are the following:
UNMODIFIABLE/IRREVERSIBLE
The principal symptom of an infarction is precordial pain, which the patient might
mistake for severe angina or indigestion. Changes in serum enzymes are
commonly used to diagnose an MI. Laboratory determination of serum glutamic
oxaloacetic transaminase (SGOT), lactic dehydrogenase (LDH), and creatinine
phosphokinase (CPK) are useful
Nutrition Therapy
• Controlled calories to reduce the heart’s work and improve weight status in
overweight and obese.
• No more than 30% of calories as fat to be taken mostly in the form of unsaturated
fats.
• Sodium restriction at 2-4 g daily
• Adequate calcium, magnesium, and potassium
• Increased intake of folic acid, B1, B6, and B12 (when serum homocysteine levels are
elevated); and
• Enough fiber intake, 25-30 g daily.
CONGESTIVE HEART FAILURE (CHF)
CHF is the condition when the heart fails to maintain a satisfactory circulation of the body’s
various metabolic needs.
CHF results in reduced heart pumping efficiency in the lower two chamber leading to
inadequate blood circulation, congestion in liver and lungs or body circulation, pedal edema,
abdominal pain, ascites, and dyspnea. Although often seen in obese patients, it can also
occur with cardiac cachexia (anorexia and fat and muscle wasting with edema). Decreased
renal flow is common and BUN may be increased.
CHF can be best treated by a combination of diet, rest, diuretics, digitalis, vasodilators and
oxygen therapy. It is best to treat the underlying cause of the condition if it can be identified.
NUTRITION THERAPY
Aims to promote rest to the heart, control edema, control body weight, correct
nutrient deficit, and prevent severe malnutrition .
Dietary concerns focus on restricting salt and fluid intake.
General Principles
Dietary instruction regarding sodium intake is recommended in all patients with
CHF.
Patients with CHF and diabetes, dyslipidemia, or obesity should be given specific
instructions regarding carbohydrate or caloric constraints.
Dietary sodium restriction (2-3 g daily) is recommended for patients with the clinical
syndrome of CHF and preserved or depressed LVEF. Further restrictions (<2 g daily)
may be considered in moderate to severe HF.
Restriction of daily fluid intake to <2 L is recommended in patients with severe
hyponatremia (serum sodium <130 mEq/L and should be considered for all patients
demonstrating fluid retention that is difficult to control despite high doses of diuretic
and sodium restriction.
Measurement of nitrogen balance, caloric intake, and prealbumin will be useful in
determining appropriate nutritional supplementation. Caloric supplementation is
recommended. Anabolic steroids are not recommended for such patients.
SODIUM
The degree of sodium restriction depends upon the severity of heart failure.
About 1.0 to 2.4 g sodium is recommended in complicated cases. However,
providing 4-6 g sodium may be satisfactory to less severe cases.
Restrict calorie intake to reduce weight if the patient is obese or overweight and
maintain DBW.
PROTEIN
Protein intake may be increased from 1.1-1.5 g/kg body weight if malnutrition is
present.
FLUIDS AND BEVERAGES
Generally 1-2 L of fluids is indicated for individuals with severe heart failure.
Reduce or eliminate alcohol intake.
Caffeine should be limited due to its potential to increases heart rate and cause
dysrhythmia.
The dietary management after cardiac surgery is designed to reduce the rate of
weight loss, maintain protein stores and support anabolism and healing.
Coronary Artery Bypass Grafts (CABG) and Percutaneous Transluminal Coronary Angioplasty (PTCA)
• Energy requirements are estimated using appropriate adjustments for activity and surgical stress
factor.
• Protein is given at 1.2 g/kg body weight during the catabolic postoperative phase and may return to
the usual requirements of 0.8-1.1 g protein/kg body weight.
• Modification of sodium, cholesterol, saturated fat, fluid and small frequent meals might also be
needed.
• The TLC diet is recommended as a maintenance diet at home and those who fail to lower lipids
after following the diet may require a more advanced regimen , limiting cholesterol further to 200
mg/day.
• Sodium limitation is often placed at 2000-3000 mg/day.
Nutrition Therapy after a Cardiac Transplant
Immediately after transplant, nutrient needs are increased. Given a high calorie,
high protein diets.
Higher protein intake (1.2 to 2.0 g/kg body weight) is needed to counteract
catabolic effects of steroids and surgical stress.
Ample protein is also needed for wound healing and tissue synthesis.
Commercial nutrition supplements, given orally or by tube, will help increase nutrient
density of oral diets immediately after operation.
Stimulant are omitted until full recovery.
Fluids are limited to 1 L daily to avoid fluid overload.
Intake of calcium, magnesium, and dietary fiber should be adequate.