Professional Documents
Culture Documents
Vitamin D Deficiency
Secondary Increased Bone
Aging Decline Absorption of Ca+
Hyperparathyroidism Resorption
Decline Renal 1.25(OH)
Decreased Physical Loss of Mechanical Decreased Bone Low Bone Mass and loss
Activity Stimulation Formation of bone architecture
Increased RANKL
Menopausal Osteoblast function Increased Bone
Decreased Increased Osteoclast
Estrogen declined declined Resorption
Osteoprogenitor
Kamis, 27 Oktober 2022
4. Perbedaan fraktur pada anak dan dewasa
• Fracture more common
• Stronger and more active periosteum
• More rapid fracture healing
• Special problem of diagnosis
• Spontaneous correction of certain residual deformities
• Differences in complications
• Different emphasys in methods of treatment
• Torn ligament and dislocations less common
• Less tolerance of major blood loss
Jumat, 28 Oktober 2022
5. Indikasi absolut dan relatif operasi pada
fraktur clavicula
Absolute
• open fractures
• displaced fracture with skin tenting
• subclavian artery or vein injury
• floating shoulder (clavicle and scapular neck fracture)
Relative
• displaced with > 2cm shortening
• bilateral displaced clavicle fractures
• brachial plexus injury (questionable because 66% have spontaneous return)
• closed head injury
• seizure disorder
• polytrauma patient
Minggu, 30 Oktober 2022
6. Stabilizer pada knee
Medial:
• Static: superficial and deep medial collateral ligaments (MCL), posterior
oblique ligament (POL).
• Dynamic: semimembranosus, vastus medialis, medial gastrocnemius, PES
tendons
Lateral:
• Static: lateral collateral ligament (LCL), iliotibial band (ITB), arcuate ligament.
• Dynamic: popliteus, biceps femoris, lateral gastrocnemius
7. Definisi, anamnesis, pemeriksaan fisik,
diagnosis, klasifikasi dan treatment dari skoliosis
Scoliosis is a coronal plane spinal deformity, that consisting of lateral Classification based on age
Conginetal: abnormal vertebrae due to failure of formation and or failure of
curvature and rotation of the vertebrae
segmentation
History Taking Infantile: < 3 years old
- Age at onset Juvenile: 3-10 years old
- Maturation Adolescent: >10 years old
- Back pain Treatment
1. Congenital
- Neurological symptoms
Bracing not indicated as primary treatment
- Cosmestic complaint about asymetrical back Progression & need for surgery depend on severity/type
- Family histroy 2. Infantile:
Physical Examination: observation if cobb angle less than 30 degrees or RVAD less than 20 degrees
1. Asymetrical shoulder height TLSO if > 30 degree or RVAD >20 degree
Operative if cobb angle>50-60 degrees
2. Scapular prominence
3. Juvenile
3. Chest cage asymetry Observation cobb angle <20 degrees
4. Asymetrical distance between lateral trunk and medial arm Bracing cobb angle 20-50 degrees
5. Asymetrical paraspinal muscle Operative cobb angle >50 degrees
6. Asymetrical Waist Line 4. Adolescent
Observation if cobb angle <25 degree
Diagnosis:
Bracing 25-45 degree in patient with risser sign 0,1,2
>10 degrees abnormal curvature on AP and Lateral standing x-ray Operative >45 degree
8. Mention the composition of bone
Inorganic phase : 60%
-Osteocalcium Phospate
-Calcium hydroxyapetite
Organic phase : 35%
-Type I Collagen
-Proteoglican
-Noncollagen Proteins
-Cells : osteoblast, osteoclast, osteocytes
Water : 5%
Senin, 31 Oktober 2022
9. Definisi dan klasifikasi cerebral palsy
Cerebral Palsy is a common congenital condition caused by Anatomic Classification :
injury to the immature brain that leads to upper motor Quadriplegic : Total body involvement and nonambulatory.
neuron disease and presents with cognitive and
musculoskeletal manifestations of varying severity. Diplegic : Legs more than arms but usually still ambulatory.
IQ may be normal (injury in brain is midline)
Classification :
Hemiplegic : Arms and legs on one side of the body, usually
Physiologic Classification : with spasticity. Will eventually be able to walk, regardless of
Spastic : Most common. Velocity-dependent increased treatment
muscle tone and hyperreflexia with slow, restricted Gross Motor Function Classification Scale (GMFCS)
movement due to simultaneous contraction of agonist and Type I : Near normal gross motor function, independent
antagonist muscles. ambulator
Athetoid : Characterized by a constant succession of slow, Type II : Walks independently, but difficulty with uneven
writhing, involuntary movements. surfaces, minimal ability to jump
Ataxic : Characterized by inability to coordinate muscle Type III : Walks with assistive devices
movements. Results in unbalanced, wide based gait.
Type IV : Severely limited walking ability, primary mobility is
Mixed : Usually mixed spastic and athetoid features and wheelchair
involves the entire body.
Type V : Nonambulator with global involvment, dependent in
Hypotonic : Usually precedes spastic or ataxic for 2-3 years all aspects of care
10. Explain about POSI of the hand
The POSI position for safe hand splinting
Type of Ossification:
1. Intramebranous Ossficiation: Bone develops directly from mesenchymal
cells without a cartilage anlage
2. Endochondral Ossification: Bone replaces a cartilage anlage (template).
Osteoclasts remove the cartilage, and osteoblasts make the new bone
matrix, which is then mineralized
3. Appositional: Osteoblasts make new matrix/bone on top of existing bone.
15. Mention type of bone and mention
examples of bones from each type?
• Long bone: humerus, radius, ulna, femur, tibia fibula
• Short bone: carpal and tarsal bones
• Flat bone: cranium, clavicle, sternum, scapula, pelvis
• Sesamoid bone: patella, pisiform, interphalangeal joint sesamoid,
hallucal sesamoid
• Irregular: vertebrae
16. Please draw brachial plexus and label it!
17. Please draw cross section dorsal
compartment of wrist!
18. Please describe about bone healing
phase
Secondary bone healing phase
1. Haematoma formation: bleeding occurs from the
bone and tissue at the time of injury
2. Inflammation: 1-7 days after fracture. starts when
the fracture haematoma forms and cytokine are
released until fibrous tissue, cartolage, or bone
formation begins. Osteoclasts are formed to
remove the necrotic ends of bony fragments
3. Soft callus formation: 2-3 weeks, the cells form a
cuff of woven bone periosteally
4. Hard callus formation: 3-4 months, the fracture is
solidly united
5. Remodelling: months to years. The woven bone
slowly replaced by lamellar bone
19. Describe about perkins time table
with requirement of bilateral grade 2-4 or unilateral grade 3-4 sacroiliitis AND any
clinical criteria (see X-Ray Grading of SI joints)
22. Accepted criteria buat humeral fracture
• < 20° anterior angulation
• < 30° varus/valgus angulation
• < 3 cm shortening
23. Please mention forearm compartement +
origin, insertion, nerve!
Muscles Origin Insertion Nerve
Superficial Flexors
Flexor carpi radialis Medial epicondyle Base of 2nd (and 3rd) Median
(FCR) metacarpal
Flexor carpi ulnaris 1. Medial epicondyle Pisiform, hook of hamate, 5th Ulnar
(FCU) 2. Posterior ulna metacarpal
Flexor digito- 1. Medial epicondyle proximal ulna Middle phalanges of digits (not Median
rum superficialis (FDS) 2. Anteroproximal radius thumb)
23. Please mention forearm compartement +
origin, insertion, nerve!
Deep Flexors
Flexor digitorum Anterior ulna & interosseous Distal phalanx of digits Median/AIN
profundus (FDP) membrane Ulnar
Flexor pollicis longus Anterior radius & proximal ulna Distal phalanx of thumb Median/AIN
(FPL)
Pronator quadratus (PQ) Medial distal ulna Anterior distal radius Median/AIN
23. Please mention forearm compartement +
origin, insertion, nerve!
Muscles Origin Insertion Nerve
Superficial Extensors
Extensor digiti minimi (EDM) Lateral epicondyle MCP, P2, P3 of 5th digit Radial-PIN
Extensor carpi ulnaris (ECU) Lateral epicondyle Base of 5th metacarpal Radial-PIN
Mobile Wad
Deep Extensors
Secondary Stabilizers:
- Radiocapitellar Articulation (radial
head)
- Anterior and posterior joint capsule
- Common flexor and extensor origins
25. Explain about Vit D Metabolism
26. Mention the differences between
lamellar and woven bone?
lamellar bone woven bone
- stress oriented - non stress oriented
- mature bone - Immature bone
- highly organized - poorly organize
- pathologic process
- occur in callus forming
- less mineralized.
27. Function of synovial fluid
1. Lubrication of Joint
2. Nutrition to articular
cartilage
28. Pathophysiology of OA & 4 cardinal sign
of OA
Articular Cartilage: Synovial Membrane and Fibrous Capsule:
1. loss of proteoglycan from the matrix -> 1. dead cartilage might float in the fluid which will
susceptible to friction of joint function -> increase 5he giscosity
fissuring 2. fibrous capsule become thicker and limiting
2. in central area loss of articular surface shown joint motion.
by narrowing of cartilage space Muscles
3. in peripheral area, cartilage respods by muscle controlling joint develop spasm in
hypertrophy which outgrowth cartilage to response to pain and might becoame contracted.
become osteophyte. the late resule might become a fibrous ankylosis.
Subchondral Bone:
1.in centeal area, the stress and friction cause cardinal signs of OA
hypetrophy and become radiographically dense
(sclerotic) - joint space narrowing
2. in peripheral, the low stress cause less dense. - subcondral bone sclerosis
3. the excessive pressure lead to development of - osteophyte
cystic lesions. - subcondral cyst
Kamis, 3 November 2022
29. Function of Meniscus
1. load transmission
2. joint congruity abd stability
3. joint lubrication
4. joint nutrition
5. proprioception
30. Draw the anatomy and the vascular zone
of meniscus
31. Mention zone of physis
• Resting Zone
• Proliferative Zone
• Hypertrophic Zone:
• Maturation Zone
• Degeneration Zone
• Zone of Provisional
Calcification
32. Anatomy and histology of Nerve &
mention seddon dan sunderland classification
1. Slow-twitch muscle fibers are fatigue resistant muscle. They contain more
mitochondria and myoglobin, and are aerobic in nature compared to fast-twitch
fibers. Slow-twitch fibers are also sometimes called type I or red fibers because
of their blood supply.
2. Fast-twitch muscle fibers provide bigger and more powerful forces, but for
shorter durations and fatigue quickly. They are more anaerobic with less blood
supply, hence they are sometimes referred to as white fibers or type II.
Jumat, 4 November 2022
38. Explained about DCO, ETC and EAC
Early total care used until 1980, no longer used because lead to Early appropriate care
exacerbation of the second hit in patient with hemodynamic 1. Identifies major trauma patient
instability.
2. Definitively treats the most
time-critical orthopedic injuries
Damage control orthopedics is defined as definitive treatment while;
delayed until physiology has improved, staging definitive 3. Minimizing the secondary
management avoid adding trauma to patient during vulberable inflammatory response (lactate
period. Treated only the potentially life threatening. <4.0mmol/L, pH>7,25, base
The parameters are: ISS score >40 without thoracic trauma, ISS excess >= 5,5mmol/L)
score >20 with thoracic trauma, GCS <9, multiple injuries with The goal is to definitively treat
severe pelvic/abdominal and hemorrhagic, bilateral femora (spine, pelvis, femur, acetabulum)
fractures, pulmonary contusion noted on radiographs, within 36 hours of injury
hypothermia, head injury with AIS >2, Interleukin 6 >500pg
39. Definition and criteria of multiple trauma
Polytrauma (multitrauma) is a short verbal equivalent used for severely injured patients
usually with associated injury (itwo or more severe injuries in at least two areas of the
body), less often with a multiple injury (two or more severe injuries in one body area).
Used to know the flexibility and make decision for treatment in scoliosis, bracing
just effective in skeletally immature patient (risser 0,1,2)
47. Please explain about the Young’s modulus
of elasticity and its function
Young’s modulus describes the relationship between
stress (force per unit area) and strain (proportional
deformation in an object). A solid object deforms when
a particular load is applied to it. The body regains its
original shape when the pressure is removed if the
object is elastic. Many materials are not linear and
elastic beyond a small amount of deformation. The
constant Young’s modulus applies only to linear elastic
substances.
Parameters :
- Midfoot : curved lateral border, medial foot crease, talar head coverage
- Hindfood : posterior crease, rigid equinus, empty heel
Sabtu, 5 November 2022
48. Pathophysiologi of Osteomyelitis
Port de entry of bacteria on the skin secondary to infected scratches,
abrasions, pimples, or boils; mucous membranes of the upper
respiratory tract as a complication of a nose or throat infection.
Initially small focus of bacterial inflammation process & forming of pus
increasing intraosseous pressure and compromising local circulation
and leads to vascular thrombosis & consequent necrosis of bone.
After few days, the infection penetrates periosteum and produce
cellulitis and soft tissue abscess. In joints it becomes septic arthritis
If uncontrolled the infection may lead to metastatic or chronic
osteomyelitis
49. Kenapa hematogenous osteomyelitis
seringnya di daerah metafisis pada anak?
Karena aliran darah di metafisis dipenuhi banyak vaskularisasi
dengan aliran cenderung lambat yang disebabkan terminal dari
arteri di metafisis berbentuk hairpin loop sebelum menjadi vena
sinusoidal sehingga bakteri gampng menempel dan
berkolonisasi.
50. What is definition of Osteosarcoma
Osteosarcoma is high grade
primary skeletal malignancy
characterized by spindle cells
of mesenchymal origin
depositing immature osteoid
matrix.
Minggu, 6 November 2022
51. What is the diagnosis?
Brust fracture
high-energy axial loading spinal trauma that results in disruption of a
vertebral body endplate and the posterior vertebral body cortex that
leads to fracture of the anterior and posterior vertebral body (two-
column injury)
Chance fracture
commonly referred as seatbelt fractures, are flexion-distraction type
injuries of the spine that extend to involve all spinal columns (three-
column injury)
58. Explain about jumper’s knee
Jumper's knee also known as patellar tendinitis Special test:
defined as tendinopathy of the patellar tendon. Basset sign: tenderness to palparion at distal
pole of patella in full extension, but no
Classified into 3 phase tenderness in full flexion
Phase 1: pain after activity Imaging:
Phase 2: Pain during and after activity Xray: enthesophyte +/-
USG: thickening of tendon
Phase 3: Persistent pain
MRI in chronic cases may be thickening of
tendon, loss of the posterior border of fat pad
Diagnosis:
History taking: Pain in the anterior knee at inferior Treatment:
border of patella usually related to activity.
Nonoperative: rest, ice, activity modification,
Physical Exam: physical therapy
- Inspection: swelling +/- over tendon Operative: in stage III disease, chronic pain,
- Palpation: tenderness pastial tears
59. What is the differential diagnose of
jumper’s knee
• Patellar injury
• Patellar dislocation
• Osgood Schaltter disease
• Knee Osteochondritis dissecans
• Meniscus injuries
60. What is osgood schlatter
disease?
Definition
Osgood-Schlatter disease is
osteochondrosis or traction
apophysitis of the tibial tubercle, Additional Exams
commonly presenting as anterior knee
- Radiographs : irregularity and fragmentation of the tibial
pain in the pediatric population. tubercle
How to diagnose - MRI : soft tissue swelling; thickening and edema of inferior
- Symptoms : pain on anterior aspect patellar tendon; fragmentation and irregularity of
ossification center;
of knee; exacerbated by kneeling;
Treatment
- Physical exam :
- Nonoperative : NSAIDS, rest, ice, activity modification,
* inspection : enlarged tibial tubercle; strapping/sleeves to decrease tension on the apophysitis
tenderness over tibial tubercle; and quadriceps stretching; cast immobilization x 6 weeks;
- Operative : ossicle excision
61. What is terrible triad of elbow?.
A traumatic injury pattern of the elbow that is
characterized by:
-elbow dislocation
-radial head or neck fracture
-coronoid feacture
Senin, 14 November 2022
62. Function of annular and cruciate pulleys?
• The function of the Annular pulleys
is to enhance effective flexor
tendon functions by holding the
tendons close to the phalanges,
thereby preventing the “bow-
stringing” effect across the small
joints when the digits are in flexion.
• The function of Cruciate pulleys is
to prevent sheath collapse and
expansion during digital motion and
facilitates approximation of annular
pulleys during flexion
Rabu, 16 November 2022
63. Borders and contents of carpal tunnel?
Borders:
• Roof: transverse carpal ligament
• Floor: central proximal carpal bones
• Medial wall: pisiform and hook of hamate
• Lateral wall: trapezium and scaphoid
tubercle
Contents:
• 4 flexor digitorum superficialis (FDS)
tendons
• 4 flexor digitorum profundus (FDP)
tendons
• flexor pollicis longus (FPL)
• median nerve
64. Borders and contents of ulnar tunnel?
Borders:
• Roof : volar carpal ligamet
• Floor : transverse carpal ligament
• Medial wall : pisiform
• Lateral wall : hook of hamate
Contents:
• ulnar nerve
• ulnar artery