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ECG
Systole Diastole
The electrical potentials produced by the heart are the sum of minute
amounts of electricity generated from each individual cardiac muscle
cell.
A vector is the visual representation of the summation of the millions of
individual depolarizations.
A vector is a graphic illustration of a physical force that has both
magnitude and direction.
Electrical current can be represented by a vector.
When atrial and ventricular myocytes depolarize and repolarize, a large
current is created.
ECG reflects the electrical activity of the entire heart – the sum of all the
action potentials of all the cardiac cells.
Cardiac Vectors
Quadrants
Normal – positive lead I and aVF
Left – positive lead I and negative aVF
Right – negative lead I and positive aVF
Extreme right – negative lead I and aVF
AXIS
ECG Complexes
ECG Paper
Small boxes = 1 mm / 0.04 seconds
Large boxes = 5 mm / 0.20 seconds
(usually upright) (0.06 - 0.11 sec) (delays in the bundle branches will
widen the QRS)
T Wave – ventricular repolarization “recharging” (usually upright)
P wave
atrial
depolarization
≤ 2.5 mm in
amplitude
< 0.12 sec in
width
PR interval (0.12 -
0.20 sec.)
time of stimulus
through atria and AV
node
prolonged interval
= first-degree
heart block
ECG Waves
QRS
Ventricle depolarization
Q wave: when initial deflection is negative
R wave: first positive deflection
S wave: negative deflection after the R wave
ECG Waves
QRS
May contain R wave only
May contain QS wave
only
Small waves indicated
with small letters (q, r, s)
Repeated waves are
indicated as ‘prime’
Q waves
Pathologic Q waves are a sign of previous myocardial
infarction.
They are the result of absence of electrical activity. A myocardial
infarction can be thought of as an electrical 'hole' as scar tissue is
electrically dead and therefore results in pathologic Q waves.
QRS
width usually 0.12 second or less
ECG Waves
RR interval
interval between two consecutive QRS complexes
ECG Waves
J point:
end of QRS wave
beginning of ST segment
ST segment
beginning of ventricular repolarization
T wave
part of ventricular repolarization
asymmetrical shape
usually not measured
normally upright in lead II
ECG Waves
QT interval
RR HR QT
(sec) (bpm) (sec)
1.00 60 0.43
Long QT interval
certain drugs
electrolyte disturbances
hypothermia
ischemia
infarction
subarachnoid hemorrhage
Short QT interval
drugs or hypercalcemia
ECG Waves
U Wave
last phase of repolarization
small wave after the T wave
not always seen
significance is not known
Heart Rate Calculation
most accurate
1500 divided by the
take time to calculate
number of small boxes
only use with regular
between two R waves
rhythms