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Chest :
Goals : Recognize normal. Decide if there is
pathology.
Recognize life threatening cases which necessitate
immediate management and intervention.
The normal chest. Anatomy, quality, proper
positioning, penetration, supine versus standing,
infants, cardiothoracic ratio etc.
What to examine: Remember : heart size and shape,
lungs, mediastinum, diaphragm, thoracic cage.
Patterns of diseases : Lungs, increased density.
Pneumonia, pleural effusions, tumors, atelactasis,
cysts.
Radiolucent lesions : Emphysema, bullae,
pneumothoax, hypogenetic lung etc.
CHEST , CV , RADIOLOGY
Anatomy:
– The heart is normal in size and shape.
– The lungs are clear.
– The mediastinum, diaphragm and thoracic
cage are intact.
Recognizing
A Technically
Adequate Chest X-ray
Factors to Evaluate
Penetration
Inspiration
Rotation
Angulation
Penetration
Anterior ribs
Posterior ribs will be visible
are those that but are
are most harder to see.
apparent on They run
more or less
the chest x- at a 45 degree
ray. They run angle
more or less downward
toward the
horizontally. feet.
Angulation
If the x-ray beam is angled toward the
head (mostly because the patient is
semi-recumbent), the film so obtained
is called an “apical lordotic” view
Anterior structures (like the clavicles)
will be projected higher on the film than
posterior structures
Pitfall Due to Angulation
Penetration
Inspiration
Rotation
Angulation
Correct
The image is apical
lordotic- look at the
high position of the
clavicles. It is also
underpenetrated.
You can’t tell if its
rotated and the
degree of
inspiration is
adequate.
What is most wrong with this image
(check any that apply)?
Penetration
Inspiration
Rotation
Angulations
Correct
Penetration
Inspiration
Rotation
Angulation
Correct
The film is
underpenetrated. You
can’t see the spine
through the heart . The
degree of inspiration is
probably adequate.
Rotation can not be
evaluated and there is a
slight amount of
angulation. Incidentally,
there is a large
bronchogenic ca in the
left lung.
What is most wrong with this image
(check any that apply)?
Penetration
Inspiration
Rotation
Angulation
Correct
<50%
The Cardiac Contours
Aortic knob
Ascending Aorta
Main pulmonary
“Double density” artery
of LA enlargement Indentation for
LA
Right atrium
Left ventricle
Coronary artery disease
Hypertension
Cardiomyopathy
Valvular lesions
AS, MS
L to R shunts
Congestive Heart Failure
Clinical
Usually from left heart failure
Shortness of breath
Paroxysmal nocturnal dyspnea
Orthopnea
Cough
Right heart failure
Edema
Congestive heart failure.
3 stages:
– congestion : upper lobe diversion “
cephalization”, arterial bronchial ratio “ blood
vessels become larger than near by bronchus”
distinctness, “peri vascular haze”.
– Interstitial edema: B and C lines of Kerley fluid
in the fissures and pleural effusions.
– Air space edema “ alveolar edema”.
Congestive Heart Failure
Secondary Four Signs of Pulmonary interstitial
edema
Thickening of the interlobular septa
Kerley B lines
Peribronchial cuffing
Wall is normally hairline thin
Thickening of the fissures
Fluid in the subpleural space in
continuity with interlobular septa
Pleural effusions
Kerley B Lines
B=distended interlobular septa
Location and appearance
Bases
1-2 cm long
Horizontal in direction
Perpendicular to pleural surface
Multiple Kerley B lines
at the left lung base
These are faint whites
lines perpendicular to
the pleural surface and 1-
2cm long
Kerley A and C Lines
A=connective tissue near bronchoarterial
bundle distends with fluid
Location and appearance
Near hilum
Run obliquely
Longer than B lines
C=reticular network of lines
C Lines probably don’t exist
Intersecting
network of lines
are Kerley A lines
in proper clinical
setting
Numerous small circular
“doughnuts” seen in lung
represent fluid in
bronchial walls when
seen in conjunction with
other signs of CHF
Fluid in the minor
fissure
Fissures may be seen
normally but are usually
no thicker than the point
of a sharpened pencil
CHF
There is diffuse
airspace
(alveolar)
disease which
has somewhat
of a “bat-wing”
appearance
CHF
There are
Kerley A
and B lines
at the right
lung base
and a small
right
effusion
CHF
There are Kerley B lines
visible at both lung bases
Important Points
The four reliable signs of CHF are:
Kerley B lines
Fluid in the fissures
Peribronchial cuffing
Pleural effusion
NOT cardiomegaly
NOT cephalization
The Solitary Pulmonary Nodule
An Approach
Uncalcified lesion with
extrapulmonary malignancy
Nodule could be
Solitary metastasis
New primary
Benign nodule
Manage with biopsy; resection may be considered
if no other lesions are demonstrated
Solitary Pulmonary Nodule
Five groups
Benign by virtue of clinical course
Benign by virtue of characteristic ca++
Benign by virtue of stability on serial films
Uncalcified and growing or no old films
Extrapulmonary malignancy
Solitary Pulmonary Nodule
Factors in determining malignancy
Calcification
Growth
Location
Size
Margin
Cavitation
Solitary Pulmonary Nodule
Calcification
Laminated granuloma
Target histoplasmoma
Popcorn hamartoma
No other calcification is benign
Solitary Pulmonary Nodule
Calcification
CT 33% more accurate in finding
calcification than plain films
Same rules for benign calcifications
apply to CT
Malignancies enhance >20 HU after IV
contrast
Solitary Pulmonary Nodule
Growth
A nodule that is stable for 2 years is almost
always benign
Doubling time-a 25% increase in diameter of
nodule = 1 double in volume
Most malignancies double in 6 weeks to 16
months
Some, like osteosarcoma, choriocarcinoma and
testicular ca can double faster
Solitary Pulmonary Nodule
Location
More cancers are located in the upper than
lower lobes
Solitary Pulmonary Nodule
Size
Small lesions tend to be benign
Over 80% of lesions > 5cm are malignant
Solitary Pulmonary Nodule
Margin
Ca tends to have ill-defined borders
Hamartomas and tuberculomas are usually
well-defined
A notch in the margin (Rigler’s notch) is
sign of benignancy
Solitary Pulmonary Nodule
Cavitation
Both benign and malignant lesions
cavitate
Ca tends to be thick-walled and nodular
Solitary Pulmonary Nodule
Truisms
SPN in a patient with known
extrapulmonary malignancy is more often 1°
lung ca than met
Malignant nodules are more symptomatic
than benign ones but most malignant
nodules are asymptomatic
William Herring, M.D. © 2002
Recognizing
A Pneumothorax
There are two layers of pleura- parietal and
visceral-the pleural space between them
Normally there is no air in the pleural
space
The visceral pleura is inseparable from the
lung parenchyma and moves with the lung
Visceral
pleura
Parietal Pleural
pleura space
When air enters the pleural space, the
parietal and visceral pleura separate
making the visceral pleura visible
The thin white line of the visceral pleura
is called the visceral pleural white line
You must see the visceral pleural white
line to make diagnosis of pneumothorax!
A pneumothorax
will be visible as a
thin white line -
the visceral pleural
white line. Absent
lung markings
superior to the
white line.
Lung Markings
Absence of lung markings is not sufficient
to make diagnosis of pneumothorax!
Simple Pneumothorax
In a simple pneumothorax, there is no
shift of the heart or mediastinal
structures (trachea)
Air in left hemithorax balances the air
in the right hemithorax
Tension Pneumothorax
Progressive loss of air into pleural space
causing a shift of the heart and mediastinal
structures away from side of pneumothorax
Opposite lung is compressed
Respiratory function severely compromised
Shift of
Complete right- heart and
sided trachea to
pneumothorax left
Lung is
compressed
against
mediastinum
Simple or Tension Pneumothorax
Air enters Right hemithorax either from tear in lung or hole in chest
wall on inspiration; does not exit on expiration
Simple pneumothrax
The visceral pleural
white line is seen and
there are no lung
marking distal to it.
This is a simple
pneumothorax since
there is no shift.
Skin fold or Pneumothorax
A fold of the patient’s skin may become
trapped between the patient and cassette
Skin folds are common
Especially in patient’s who have lost a great
deal of weight
This skin fold can mimic a
pneumothorax
This is an edge
Dense
Lucent
Skin Fold
Lucent
Dense
Lucent
Pneumothorax
Pneumonia
Pneumococcal pneumonia
Staphylococcal pneumonia
Streptococcal pneumonia
Nocardiosis
Day 1 Day 2
Rapid Clearing
© R3
Pneumococcal pneumonia
Rapidly Clearing Alveolar Infiltrate
1. Pneumococcal
pneumonia
2. Hemorrhage
3. Pulmonary edema
4. Aspiration
DDX
Staph Aureus Pneumonia
X-ray
Rapid spread through lungs
Empyema, especially in children
No air bronchogram
Pneumothorax, pyopneumothorax
Abscess formation, pneumatocoele
Bronchopleural fistula
Infiltrates with Effusion
1. Staph pneumonia
2. Strep pneumonia
3. TB
4. Pulmonary infarct
DDX
Day 1 Day 4
Staph pneumonia
Staph pneumonia
Staph pneumonia with pneumatocoele
© R3
Gram-positive, acid-fast bacterium
Occurs in immunocompromised
Multiple nodules with or without
cavitation
Empyemas occur
© R3
Pseudomonas
Klebsiella
Enterobacter
Serratia
Anaerobic
Pseudomonas Aeruginosa
General
Gram negative rod
Frequently hospital-acquired
Frequently related to inhalators or
nebulizers
Many patients on multiple antibiotics and/or
steroids
Pseudomonas Aeruginosa
Predisposed
COPD
CHF
Alcoholism
Kidney disease
Those with trachs
Pseudomonas Aeruginosa
X-ray Findings
Resembles staph pneumonia
Predilection for lower lobes
Usually affects both lungs
Contains multiple small lucencies
Lung abscess > 2 cm may also occur
Widespread nodular shadows another
manifestation
Pseudomonas
Pseudomonas lung abscess
Klebsiella, Enterobacter, Serratia
General
Encapsulated, gram negative rods
Most are hospital-acquired
Most occur in chronic alcoholics
Aspirated into lungs
Most are unilateral and right sided
Klebsiella Pneumonia
X-ray Findings
Produces excessive amounts of
inflammatory exudate
Affected lung gains volume and fissures bulge
Bulging fissure sign
Abscess and cavity formation common
Pleural effusion and empyema common
Klebsiella with bulging fissure
Anaerobic lower lobe cavitary pneumonia
Other Pneumonias
Cryptococcus
Varicella
Pneumocystis
Actinomycosis
Mycoplasma pneumonia
Coccidiomycosis
Cryptococcosis
(Torulosis)
Caused by Cryptococcus neoformans
Found in soil contaminated with pigeon
excrement
Granulomatous disease
Diabetics, immunocompromised
Frequently produces meningitis
Cryptococcosis
X-ray
Well circumscribed peripheral mass (40%)
Lobar/segmental consolidation (35%)
Cavitation (15%)
Hilar/ mediastinal adenopathy
Nodular Infectious Diseases
1. Nocardiosis
2. Coccidiomycosis
3. Cryptococcosis
4. Varicella
Take Home
SARS produces patchy airspace dz
Pneumococcal pneumonia can clear in
48 hrs
Staph produces loculated effusions and
pneumatocoeles
Nocardiosis has multiple nodules with
or without cavitation
Take Home
Pseudomonas - lower lobes; multiple
small lucencies
Klebsiella - heavy exudate; bulging
fissure
Actinomycosis – extends through
pleura
Take Home
Mycoplasma – lower lobe; dormitory
settings
Coccidiomycosis – thin-walled cavity
for patchy perihilar parenchymal infiltrates on a plain
CXR, which correspond to alveolar lesions of
advanced pulmonary sarcoidosis
Pleural Effusion-Types
Transudate
– Exudate
– Empyema
– Hemothorax
– Chylothorax
Transudate
Transudate
capillary hydrostatic pressure or
osmostic pressure
– CHF
– Hypoalbuminemia
– Cirrhosis
– Nephrotic syndrome
Exudate
Usually 2 neoplastic or inflammatory
dzs involving pleura [Fluid Protein]
[serum protein] > 0.5
[Fluid LDH] [serum LDH] >0.6
Fluid LDH > 2/3 highest normal
serum LDH
Side-specificity
Side-specificity
–Mostly left-sided
–Pancreatitis
–Dressler’s syndrome
Distal thoracic duct obstruction
–Mostly right-sided
–Heart failure
–Abdominal disease related to liver
or ovary
–Proximal thoracic duct obstruction
Appearances of Pleural Effusions
Subpulmonic effusion
Blunting of Costophrenic angle
Meniscus sign
Layering
Loculated
Laminar effusion
Opacified hemithorax
Air-fluid levels
Fluid rises
higher
along the
edge of a
pleural
effusion
producing
an upside
down “U”
or
meniscus Meniscus Sign
shape
A straight edge,
indicative of a fluid
interface, in this
case an air-fluid
interface, is seen on
the right.
In order to have an
air-fluid level in the
pleural space, there Hydropneumothora
must be a x
pneumothorax
present.