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Case Study Sample
Case Study Sample
MEMBERS:
ALLERGIES N/A
Family History
- her father’s side has family hx of hypertension and her mother died because
of vehicular accident
Social History
●Patient has history of smoking and drinking alcoholic beverages occasionally
Obstetrics and Gynecologic History
Fallopian Tubes (Uterine Tubes) The fallopian tubes serve as the pathway of the egg cells towards the uterus.
It is a smooth, hollow tunnel that is divided into four parts: the interstitial, which is 1 cm
in length; the isthmus, which is2 cm in length; the ampulla, which is 5 cm in length; and
the infundibular, which is 2 cm long and shaped like a funnel.
The funnel has small hairs called the fimbria that propel the ovum into the fallopian
tube.
The fallopian tube is lined with mucous membrane, and underneath is the connective
tissue and the muscle layer.
The muscle layer is responsible for the peristaltic movements that propel the ovum
forward.
The distal ends of the fallopian tubes are open, making a pathway for conception to
occur.
INTERNAL STRUCTURES FUNCTIONS
Uterus The uterus is described as a hollow, muscular, pear-shaped organ.
It is located at the lower pelvis, which is posterior to the bladder and anterior to the
rectum.
The uterus has an estimated length of 5 to 7 cm and width of 5 cm. it is 2.5 cm deep in
its widest part.
For non-pregnant women, it is approximately 60g in weight.
Its function is to receive the ovum from the fallopian tube and provide a place for
implantation and nourishment.
It also gives protection for the growing fetus.
It is divided into three: the body, the isthmus, and the cervix. f
The body forms the bulk of the uterus, being the uppermost part. This is also the part
that expands to accommodate the growing fetus.
The isthmus is just a short connection between the body and the cervix. This is the
portion that is cut during a cesarean section.
The cervix lies halfway above the vagina, and the other half extends into the vagina. It
has an internal and external cervical os, which is the opening into the cervical canal.
EXTERNAL STRUCTURES FUNCTIONS
Mons Veneris The mons veneris is a pad of fat tissues over the symphysis pubis.
It has a covering of coarse, curly hairs, the pubic hair.
It protects the pubic bone from trauma.
Labia Minora Lateral to the labia minora are two folds of fat tissue covered by loose connective tissue
and epithelium, the labia majora.
Its function is to protect the external genitalia and the distal urethra and vagina from
trauma.
It is covered in pubic hair that serves as additional protection against harmful bacteria
that may enter the structure.
Vestibule It is a smooth, flattened surface inside the labia wherein the openings to the urethra
and the vagina arise.
Clitoris The clitoris is a small, circular organ of erectile tissue at the front of the labia minora.
The prepuce, a fold of skin, serves as its covering.
This is the center for sexual arousal and pleasure for females because it is highly
sensitive to touch and temperature.
Skene’s Glands Also called as paraurethral glands, they are found lateral to the urethral meatus and
have ducts that open into the urethra.
The secretions from this gland lubricate the external genitalia during coitus.
EXTERNAL STRUCTURES FUNCTIONS
Bartholin’s Gland Also called bulbovaginal gland, this is another gland responsible for the lubrication of
the external genitalia during coitus.
It has ducts that open into the distal vagina.
Both of these glands secretions are alkaline to help the sperm survive in the vagina.
Fourchette This is a ridge of tissue which is formed by the posterior joining of the labia minora and
majora.
During episiotomy, this is the tissue that is cut to enlarge the vaginal opening.
Perineal Body This is a muscular area that stretches easily during childbirth.
Most pregnancy exercises such as Kegel’s and squatting are done to strengthen the
perineal body to allow easier expansion during childbirth and avoid tearing the tissue.
Hymen This covers the opening of the vagina.
It is tough, elastic, semicircle tissue torn during the first sexual intercourse.
During preeclampsia significant
changes in following body parts
are reported;
Fetoplacental unit
Heart
Blood
Coagulation system
Kidneys
Liver
Brain
Eyes
Fetoplacental unit:
In the uterus, hypertension-induced vasoconstriction decreases the
flow of uterine blood and vascular lesions occur in the placental bed,
which can lead to placental abruption.
The reduction in blood flow to the choriodecidual areas reduces the
amount of oxygen that diffuses into the fetal bloodstream inside the
placenta via the cells of the syncytiotrophoblast and cytotrophoblast.
The consequence is that the placental tissue becomes ischamic,
thrombosis of the capillaries in the chorionic villi, and defects occur
that lead to restriction of fetal development.
With decreased placental activity, hormonal production is also reduced
and this has serious consequences for the fetus’s survival.
This combination of factors frequently result in preterm labor and birth.
Heart:
Tremendous demands are made upon the
heart during preeclamsia.
It also must adjust to the demands of organs
systems with the increased workloads, such
as the kidneys and uterus.
In addition, the heart is physically pushed
upward and to the left by the enlarging
uterus, which may cause systolic murmurs.
In preeclampsia, cardiac contractility is
preserved but both steady and pulsatile
arterial load are increased inappropriately,
failing to decrease as would occur in normal
pregnancy, involving both conduit and small
vessels.
Women who have preeclampsia are at
greater risk of having high blood pressure
later in life. They are also at double the risk
of having blood clots, a heart attack or
stroke within five to fifteen years after
pregnancy.
Blood:
Hypertension and damage to endothelial cells impact capillary
permeability.
The leakage of plasma proteins from the damaged blood vessel
causes plasma colloid pressure to decrease and edema to
increase in intracellular space.
The reduced amount of intravascular plasma causes
hypovolemia and haemoconcentration, which is referred to as
elevated hematocrit,
The lungs become congested with fluid in extreme cases, and
pulmonary edema occurs, oxygenation is hindered and
cyanosis takes place.
The coagulation cascade is triggered with vasoconstriction and
disturbance of the vascular endothelium.
Coagulation system:
Thrombocytopenia is produced by increased platelet intake and
may be responsible for the production of disseminated
intravascular coagulation.
Fibrin and platelets are accumulated as the process
progresses, which can block blood flow to several organs,
especially the kidney, liver, brain and placenta.
Kidneys:
Hypertension in the kidney contributes to vasospasm of the
afferent arterioles, resulting in reduced renal blood flow that
causes hypoxia and edema of the glomerular capillary
endothelial cells.
Glomerulo-endotheliosis (glomerular endothelial damage)
enables proteins to fit into the urine, causing proteinuria,
predominantly in the form of albumin.
Reduced creatinine clearance and increased serum creatinine
and uric acid levels are expressed in renal injury.
Oliguria progresses as the condition worsens, suggesting
significant pre-eclampsia and damage to the kidneys.
Liver:
Hepatic vascular bed vasoconstriction can result in hypoxia
and edema of liver cells.
Edematous swelling of the liver in extreme cases causes
epigastic pain, which can lead to intracapsular haemorrhage
and, in extremely rare cases, liver rupture.
Changed liver function is displayed by falling albumin level rise
in liver enzyme levels.
Brain:
Hypertension combined with cerebrovascular endothelial
dysfunction increases the permeability of the blood-brain
barrier resulting in cerebral oedema and micro haemorrhaging.
Clinically this is characterized by the onset of headaches,
visual distturbances and convulsions.
If the mean arterial pressure (MAP, i.e. systolic blood pressure
plus twice the diastolic pressure separated by3) reaches 125
mmHg, cerebral flow autoregulation is impaired, resulting in
cerebral vasospasm, cerebral edema, and formation of blood
clots.
This is known as hypertensive ecephalopathy, which may lead
to cerebral haemorrhage and death if left untreated.
Eyes:
Preeclampsia is known to cause retinal vascular changes and
visual symptoms, including blurred vision, photopsia, diplopia
or even blindness; however, less is published about its effects
years later.
Pregnancy hormones cause fluid retention which alters the
cornea to make it thicker, along with an increase in the fluid
pressure within the eyeball. This results in a blurred vision.
PATHOPHYSIOLOGY
Predisposing Factor: PATHOPHYSIOLOGY OF Precipitating Factors:
Age: 43 PREECLAMPSIA Social HX: Patient has
Sex: Female history of smoking and
Family Hx: Hypertension drinking alcoholic
in her father side beverages occasionally
Unknown Etiology
Vasospasm
Kidney effect
Vascular effect
Interstitial effect Decreased glomeruli filtration
rate and increased permeability
of glomeruli membranes. Vasoconstriction
PREECLAMPSIA
PROGNOSIS
Resolution within days to weeks PP
Consider essential HTN if it persists 12 weeks
Prognostic issues include the risk of recurrent preeclampsia and related complications in
subsequent pregnancies and long-term maternal health risks.
Recurrence — The recurrence risk varies with the severity and time of onset of the acute
episode .
Women with early onset, severe preeclampsia are at greatest risk of recurrence (as high as 25
to 65 percent) .
The risk is much lower (5 to 7 percent) in women who had nonsevere preeclampsia during the
first pregnancy, versus less than 1 percent in women who had a normotensive first pregnancy
(does not apply to abortions) .
Long-term maternal risks:
hypertension
• ischemic heart disease
• stroke
venous thromboembolism
Graded relationship between severity of preeclampsia and risk of future cardiac disease
TREATMENT
Delivery remains the ultimate treatment for preeclampsia. Although maternal and fetal
risks must be weighed in determining the timing of delivery, clear indications for
delivery exist.
• When possible, vaginal delivery is preferable to avoid the added physiologic
stressors of cesarean delivery.
• If cesarean delivery must be used, regional anesthesia is preferred because it
carries less maternal risk.
• In the presence of coagulopathy, use of regional anesthesia generally is
contraindicated.
MILD PREECLAMPSIA
Maternal and Fetal
Evaluation
NO YES
PRESENT ABSENT
• Corticosteroids. If you have severe preeclampsia or HELLP syndrome, corticosteroid medications can
temporarily improve liver and platelet function to help prolong your pregnancy. Corticosteroids can also help
your baby's lungs become more mature in as little as 48 hours
• Anticonvulsant medications. If your preeclampsia is severe, your doctor may prescribe an anticonvulsant
medication, such as magnesium sulfate, to prevent a first seizure.
Bed rest
• Bed rest used to be routinely recommended for women with preeclampsia. But research hasn't shown a
benefit from this practice, and it can increase your risk of blood clots, as well as impact your economic
and social lives. For most women, bed rest is no longer recommended.
Hospitalization
• Severe preeclampsia may require that you be hospitalized. In the hospital, your doctor may perform
regular nonstress tests or biophysical profiles to monitor your baby's well-being and measure the volume
of amniotic fluid. A lack of amniotic fluid is a sign of poor blood supply to the baby.
LABORATORY /
DIAGNOSTIC
EXAMINATION
DATE OF LABORATORY OR
DIAGNOSTIC RESULTS LABORATORY
REFERENCE VALUE ACTUAL RESULTS SIGNIFICANCE /
OR DIAGNOSTIC PROCEDURE INTERPRETATION
MICROSCOPIC EXAMINATION
PUS CELLS 1-3 /hpf Normal - pus cells called pyuria 0-3 per high
power field is normal .
RED CELLS 0-1 /hpf Normal - there are red blood cells (RBCs) in the
urine. Often, the urine looks normal to the naked
eye. But when checked under a microscope, it
contains a high number of red blood cells.
April 19,2021
6:20am - CHEST XRAY PA
Radiographic Report
-Lungs Fields are clear Normal - no lungs
problem
-Heart is not enlarged Normal - no heart
problem
-Diaphragm and sulci Normal - no diaphragm
are intact and sulci problem
-The rest of the Normal - no problem
visualized chest with the structure of the
structures are intact chest
April 19,2021
6:20am - ECG
3. Encourage patient to
decrease intake of • To prevent increase
caffeine,cola and sugar intake that may
chocolate. cause high blood
pressure.
Collaborative:
• Report any
unusualities in cardiac • facilitates to other
monitor. health care provider
for better
improvements.
ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Subjective: Acute pain Short-term Indepenent Goal was met te
related to uterine Patient will Assess patient will
“Ang sakit ng contractions and maintain less maintain less
tiyan ko.” as stretching of than 2 pain on than 2 pain on
verbalized by the cervix and birth scale of 0-10. scale of 0-10.
pt. canal Dependent
Long-term Goal was met
Objectives: Patient will feel that the patient
Face grimace relaxed and Collaborative will feel relaxed
Restlessness comfortable. and comfortable.
Irritability
Pain scale: 9
ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Subjective: Excessive Fluid Short term: Independent: Rationale: Goal was met that
Volume related to After 3 hours of • Monitored vital • For baseline data after After 3 hours
“namamanas ang decreasing plasma nursing intervention signs and record. and to note of nursing
mga binti at paa ko.” colloid osmotic the patient will be progress intervention the
as verbalized by the pressure allowing able to demonstrait • position the • To promote patient will be
pt. fluid shifts out of behaviors to patient comport of patient able to
vascular improved circulation. comportably. demonstrait
compartment as • To know what behaviors to
evidenced by edema • Encourage nursing improved
formation verbalization of interventions will circulation.
feelings. be perform.
Dependent: Rationale:
• Maintain • Reduces total
fluid/sodium body
restrictions as water/prevents
indicated fluid
reccumulation.
• Apply cream or • Lotion and
ointment as ointment may be
prescribe. desired to relieve
dry craked skin.
Collaborative: Rationale:
• Consult dietician, • To promote
as needed. optimal wellness.
ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Risk for fetal
injury related to
reduced
placental
perfusion
secondary to
vasospasm.
ASSESSMENT DIAGNOSIS PLANNING INTERVENTION RATIONALE EVALUATION
Subjective: Deficient Knowledge Short term: Independent: Rationale: The goal was met
related to Lack of After 30 mins. of nursing • Provide information • May assist with further after 30 mins. of
“hindi ko po alam, kung exposure/unfamiliarity intervention the patient about additional learning/promote nursing intervention
ba itong nangyayare with information will verbalized learning process. learning at own pace. the patient had
saken.” as verbalized by resources understanding of • Motivate client by • To help client acquire verbalized as
the pt. condition/disease providing information relevant nformation evidenced by the
process and treatment. relevant to the response of patient to
situation. learning plan.
Objective: • Determine patient • which may differ and
Irritable at times Long term: most urgent need from require adjustment in The goal was met that
teary eyed After 2 days of nursing both client and nurse teaching plan. after 2 days of nursing
intervention the patient viewpoint intervention in to the
will understand the • State objectives clearly • To meet learner’s need patient had understand
learning process plan. in learner’s term. to know. the learning process
• Provide mutual goal • Clarifies expectations plan.
setting and learning of nurse and patient.
contact.
• Determines patient • To know the patient
ability to learn. level of learning ability.
• Assess the level of the • To know the patient
patient’s capabilities coping ability towards
and the possibilities of the situation.
situation.
Dependent: