Pathophysiology

Modifiable Factors Non Modifiable Factors

1) Diabetes Meliitus 1) Age

2) Atrial Fibrillation 2) Heredity
3) Other heart disease 3) Sex
4) Poor diet 4) Prior Stroke, TIA or heart attack
5) Physical inactivity

Destruction of alpha
and beta cells of the
pancreas

Failure to produce Production of excess

insulin glucagon

Increase Increase serum glucose

Polyuria osmolarity due level Production of
to glucose glucose from
protein and fat
Glycoprotein stores
cell wall
deposits
Wasting of lean
Impaired immune body mass
function (decrease Small vessel
level of disease
morphonuclear
leukocytes) Diabetic DX: CT Scan
Nephropathy Accelerated Arteriosclerotic cardiovascular
atherosclerosis disease with prominent left
ventricle and signs of bilateral
Infection Delayed Diabetic pulmonary congestion.
wound Retinopathy
healing

Blurred Vision

Pus Cell Renal

Affectation

Urinalysis
Albumin: Trace
Sugar: (+++) Glucosuria Decreased
Pus cells: 5- Production of
Erythropoeitin
10 /hpf Proteinuria
RBC: 0-1 /hpf
WBC: 11.6
(06/12/10)
Decreased RBC
production in the
bone marrow
Lab results:
Hemoglobin: 3.99
(4.0-6.0)
Anemia Hematocrit: .367
(.370-.540)
(06/12/10)

Paleness of the skin,

conjunctiva, lips, buccal
mucosa and gums was
observed
 Thrombus CEREBROVASCULAR
ACCIDENT
Emboli
Decreased Tissue
perfusion (brain)

Cerebral Hypoxia

Cerebral ischemia

Short term
Eschemia
(<10-
15mins)

Temporary
Deficit

No
permanent
damage

VERTEBROBASILA
MID CEREBRAL ANTERIOR POSTERIOR R ARTERY
ARTERY CEREBRAL A. CEREBRAL A.

Dysarthia

Apraxia

Ataxia Hemisensory loss Left

upper extremities
Hemiparesis (left upper
extremities) Hemiplegia
(left lower extremities)