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  • Burns Patho

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    chelle_asenjo
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    The document discusses the effects of decreased cardiac output including afterload, decreased tissue perfusion affecting renal blood flow, GI blood flow, anaerobic metabolism, tissue damage, cellular dysfunction, risk of acute renal failure, risk of ileus, and metabolic acidosis. It also discusses conditions that can lead to decreased cardiac output such as burns, neurological disorders, diabetes, chemical exposure, sun exposure, alcohol consumption, and inhalation of toxic fumes. The document then discusses the effects of burns, which can include shock, loss of skin barrier, hemolysis, hyperkalemia, thermo-regulation problems, hyponatremia, hypoxemia, inflammatory response, hemoglobin/myoglobin in urine, and

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    Burns patho

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    The document discusses the effects of decreased cardiac output including afterload, decreased tissue perfusion affecting renal blood flow, GI blood flow, anaerobic metabolism, tissue damage, cellular dysfunction, risk of acute renal failure, risk of ileus, and metabolic acidosis. It also discusses conditions that can lead to decreased cardiac output such as burns, neurological disorders, diabetes, chemical exposure, sun exposure, alcohol consumption, and inhalation of toxic fumes. The document then discusses the effects of burns, which can include shock, loss of skin barrier, hemolysis, hyperkalemia, thermo-regulation problems, hyponatremia, hypoxemia, inflammatory response, hemoglobin/myoglobin in urine, and

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOCX, PDF, TXT or read online from Scribd
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    42 views3 pages

    Burns Patho

    Uploaded by

    chelle_asenjo
    The document discusses the effects of decreased cardiac output including afterload, decreased tissue perfusion affecting renal blood flow, GI blood flow, anaerobic metabolism, tissue damage, cellular dysfunction, risk of acute renal failure, risk of ileus, and metabolic acidosis. It also discusses conditions that can lead to decreased cardiac output such as burns, neurological disorders, diabetes, chemical exposure, sun exposure, alcohol consumption, and inhalation of toxic fumes. The document then discusses the effects of burns, which can include shock, loss of skin barrier, hemolysis, hyperkalemia, thermo-regulation problems, hyponatremia, hypoxemia, inflammatory response, hemoglobin/myoglobin in urine, and

    Copyright:

    Attribution Non-Commercial (BY-NC)
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    of 3

    SUBMITTED BY: ASENJO, RICHELLE ANNE B.

    BSN218

    SUBMITTED TO: PROF. TARAGUA

    Afterload

    Cardiac Output

    Decreased Tissue Perfusion

    Renal blood flow

    GI blood flow

    Anaerobic metabolism

    Tissue damage

    Cellular Dysfunction

    Risk of acute renal failure

    Risk of ileus

    Metabolic acidosis

    Potential tissue necrosis

    Cell swelling

    Diabetic patients

    Neurological disorder such as stroke

    BURNS

    Working with chemicals, electricity and flames

    Excessive sun exposure

    Alcohol Consumption

    Toxic or hot fumes inhalation

    Cell lysis

    Capillary Permeability
    Sodium,Water and Protein shift from IVS to ITS Concentration of blood cells Blood Viscosity Circulating blood volume (up to 50%) Shock (Hypovolemia)

    Possible inhalation injury

    Loss of skin barrier

    Hemolysis

    Hyperkalemia

    Thermo-regulation problems Hyponatremia Hypoxemia Inflammatory response

    Hemoglobin / myoglobin in urine

    Impaired immune response Blood Pressure

    Myocardial depressant factor

    Massive stress response, Sympathetic Nervous System activation

    Adrenal corticoid hormones and catecholamine release Tachycardia

    Peripheral Vasoconstriction

    Hyperglycemia; Catabolism; Risk for Curlin s ulcer; Metabolism (after burn shock resolves)

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