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GOUT
INTRODUCTION
To better understand gout, it helps to understand the anatomy of the joints
Have cartilage A joint is a union between two bones.
Joint
Have a synovial fluid The joint allows the ends of the bones to move against one another. Have a ligaments
WHAT IS GOUT???
Do you have experience of GOUT??? Or anybody at your family have this disease???
DEFINITION
It is derived from the Latin word gutta, meaning "a drop" (of liquid).
Gout is a metabolic disorder characterized by elevated serum uric acid levels and deposits of urate crystals in synovial fluids and surrounding tissues.
Gout also is a kind of arthritis that occurs when uric acid builds up in blood and causes joint inflammation.
Acute Chronic
Lifestyle
Medical conditions
RISK FACTOR
Age and Sex Family History
ETIOLOGY
Primary Gout (Hereditary)
Classification causes of Gout
Secondary Gout
(internal/external factors, e.g. diet, kidney problems, obesity)
Etiology c0nt.
Genetic tendency Overindulgence in alcohol
A high dietary intake of purine, alcohol intake increased purine synthesis, decreased renal excretion
PATHOPHYSIOLOGY
Deposition of uric acid into the joints
GOUT
Diagnostic Test
Medical History and Physical Examination
Extremity X-ray
Medical treatment
Colchicine (Colcrys)
Comes in a tablet to take by mouth. Side effect: Mild nausea, stomach pain and mild diarrhea
Allopurinol
Comes as a tablet to take by mouth, take it around the
same time every day Side effect: Upset stomach, diarrhea and drowsiness
Corticosteroid (Prednisone)
Comes in a tablet to take by mouth. Side effect : Fluid retention, Increase BP, Mood Swings
Surgical Treatment
If gout symptom have occurred off and on without treatment, uric acid crystals may have built up in the joints to form gritty, chalky nodules called tophi.
Complication
Recurrent gout Advanced gout Joint damage
Tophi
Kidney damage
Kidney stone
The pain should be assessed on an ongoing basis using a scale of one to 10 to describe the severity. Use cognitive therapy such as creative visualization to distract the patient from his pain. Apply cold packs to inflamed joints to ease discomfort and reduce swelling. Encourage the patient to perform techniques that promote rest and relaxation.
Nursing Intervention: Evaluate orcontinuously monitor degree of joint inflammation or pain. Assess the level of intolerance and assist the patient in activities she feels unable to perform. Assist with activity passive rangeof motion.
Nursing Intervention:
Monitor vital sign Provide tepid sponge bath (if not contraindicated) Promote client safety.
Health Education
Avoid or Limit alcohol consumption
Take the prescribed medication and follow doctor`s order Avoid or Limit alcohol consumption
CONCLUSION
As we already know, gout is a monosodium urate, monohydrate crystal deposit disease. In teenagers gout also known as Rich Mans Disease, because gout can be created by eating an abundance amount of seafood. Clinical pharmacists need to be empowered with knowledge to assist prescribing clinicians in order to maximize therapeutic outcomes when treating gout. To achieve this goal, a foundation of new insights into the pathogenesis of hyperuricemia and gout has been reviewed. Both nonpharmacologic modalities and pharmacologic therapies have been discussed so that greater patient adherence through medication counseling can be achieved.
www.google.com
www.mayoclinic.com
www.ncbi.nlm.gov
REFERENCES
www.medicinet.com
www.wikipedia.com
www.emedicine.com
ANY QUESTIONS?