Complications of Respiratory Acidosis shock and cardiac arrest

Weaning from ventilator assess ABG values

Nonrebeather offers most O2
RATIONALES: Respiratory alkalosis results from alveolar hyperventilation. It's marked by a decrease in PaCO2 to less
than 35 mm Hg and an increase in blood pH over 7.45. Metabolic acidosis is marked by a decrease in HCO3

to less
than 22 mEq/L, and a decrease in blood pH to less than 7.35. In respiratory acidosis, the pH is less than 7.35 and the
PaCO2 is greater than 45 mm Hg. In metabolic alkalosis, the HCO3

is greater than 26 mEq/L and the pH is greater

than 7.45.
IF PH and is acidotic, and the CO2 is acidotic, then the acid base disturbance is caused by the respiratory system. If
the PH is alkolitic and the HCO3 is alkolitic, the acid base disturbance is being caused by the metabolic (or renal)
system
ARDS increased capillary permeability leading to pulmonary edema
Earliest sign increased respiratory rate PAO2 < 60
ARF cardinal physiological abnormalies are hypoventilation, hypoxemia, and
hypercapnia.

When ventilation is impaired, body retains carbon dioxide - acidosis
Morphine (analgesic) given for MI reduces pain and anxiety and decreases preload,
which decreases the workload of the heart and therefore the pain
Lidocaine decreases myocardial irritability
ARDS high pressure alarm could be PE, tube kinking. Low pressure cuff leak or
disconnected tubing
PEEP restores functional residual capacity increases MAP and improve oxygenaton
by reducing ventilation/perfusion mismatch.
Pulmonary Embolus respiratory alkalosis tachypnea and hypotension
Cyanosis sign of hypoxia