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Burn Injury: General Surgery Department, FKUI/RSCM, Jakarta, Indonesia, May 2011
Burn Injury: General Surgery Department, FKUI/RSCM, Jakarta, Indonesia, May 2011
Budhi Arifin Noor, Dion Ade Putra, Oktaviati, Ridho Ardhi Syaiful, Rizky Amaliah, Mursid
Translator: Adrian Salim, Andrio Wishnu Prabowo, Arnetta Naomi L. Lalisang, Julistian,
Muliyadi, Sony Sanjaya, Stefanny, Zamzania Anggia Shalih.
General Surgery Department, FKUI/RSCM, Jakarta, Indonesia, May 2011.
CASE ILLUSTRATION
A man, 43 years old came with complaint of burn injury 8 hours before hospital admission. The
patient was exposed to flame sparks on the face, body and both upper trunk while working as a
construction worker. Patient was holding steel when the steel was exposed to electrical wires and
caused fire. The patient was unconscious for 5 minutes and taken to a private hospital, there
patient treated with MEBO, RL 1 kolf infusion, urinary catheters, anti-tetanus and analgesics.
The patient was then referred to the RSCM due to limited facility.
The primary survey during physical examination was clear airway, spontaneous breathing, vital
signs was within normal limits, with GCS 15 (E4M6V5), and on secondary survey there was
found burn wounds on the face, neck and chest (see local status). Other physical examinations
were within normal limits with height 165 cm, and weight 62 kg. Laboratory tests result showed
hemoglobin level of 12.2 g / dL, hematocrit 35%, leukocytes 10,480 / ul, 82.2 fl platelets,
albumin 2.2, random blood glucose 152 mg / dl, procalsitonin 16.06 and other laboratory results
within normal result. The diagnosis for this patient was second degree burn injury 37.5% wide.
Patient was treated with fluid resuscitation (37.5 x 4 x 62) 9300 ml, 4650 ml within the first 8
hours, and continued with 4650 ml in the next 16 hours and then titrated until the urine output
reached 0.5 to 1 ml / kg / hour. Patients were also given co-amoksiklav injection of 3 x 1 gram,
ketorolac injection of 3 x 300 mg, ranitidine injection of 3 x 150 mg, and vitamin E injection of 1
x 400 mg. Patients were then consulted to anesthesiologist for CVP installation. After 1 day of
treatment in the ER patients were then moved to the RSCM Burn Unit.
LITERATURE REVIEW
Burn injuries can be caused by fire, exposure to high temperature such as the sun, electrocution,
chemicals and radiation. Most of burn injuries admitted to RSCM are caused by fire with 56% of
the total case, 40% of boiling water, 3% of electrocution and 1% of chemicals.5
I. PATHOPHYSIOLOGY
Areas of burn wounds are divided into three zones, which are coagulation zone, stasis zone and
hyperemic zone.1,2
a.
Coagulation zone
The tissue in this zone is irreversibly damaged during traumatic burn.
b.
Stasis zone
There are moderate perfusion disturbances in the area surrounding the necrotic zone. In the
stasis zone, there is vascular damage thus causes vascular leakage.
c.
Hyperemic zone
The character of the hyperemic zone is vasodilatation due to inflammation process.
Acute Phase / shock phase. The patients may experience disturbance in the airway,
breathing and circulation.
Sub-acute phase, which takes place after the shock phase is resolved. Lost or damaged
tissue resulting from contact with the heat source will cause inflammatory process with
exudation of plasma protein and infection that can cause sepsis.
Late Phase occurred after wound closure until maturation. The problem that arises during
this phase are scarring, contractures and deformities due to the fragility of tissue or
structured organ.
II.
DIAGNOSIS
a.
ii.
c.
d.
Circumferential Grade II and III Burn Injury cause blood flow restriction at
extremities, disturb respiration process if located at chest, therefore
escharotomy is needed.
Burn injury wound care could be divided into 3 major steps, which are
emergency/resuscitation phase, acute phase, and rehabilitation phase.
1. Acute/shock phase : to protect patient from the source of burn injury, ABC
evaluation, evaluation of any other trauma, fluid resuscitation, urine catheter,
nasogastric tube, vital sign and laboratory, pain management, tetanus prophylaxis,
administration of antibiotics and wound care.
2. Sub acute phase started when patient is hemodinamically stable. Management for
acute phases: to prevent infection, wound care, and nutrition.
3. Phase rehabilitation : to increase self-sufficiency through the achievement of
improved full
functionality.
Baxter formula
IV. DISCUSSION
In this patient, the diagnosis of Burn injury Grade II A-B was upheld on the grounds that the
injuries occurred on dermis; there were blisters, and reddish white colored injury that were very
painful. Total burn surface area was 37,5%, it was determined by Lund and Browder charts.
The patients treatment was consist of fluid resuscitation with baxter formula (TBSA x body
weight (kg) x 4 cc (RL) in 24 hours), in this case, patient was given 9300 cc for 24 hours,
divided into 4650 cc or 50% in the first 8 hours, then 4650 cc or 50% in the next 16 hours. Urine
production is needed to be monitored in the resuscitation fluid because it describes the
circulation of the fluid and the adequacy of fluid given. Nomally, urine production is 0.5 cc / kg /
hour. Installation of CVP is indicated to monitor systemic circulation of fluid resuscitation and to
access other solution.
First Day
Wound dressing